Latest & greatest articles for Nephrogenic Diabetes Insipidus

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Top results for Nephrogenic Diabetes Insipidus

2. AVPR2 variants and mutations in nephrogenic diabetes insipidus: review and missense mutation significance. (Abstract)

AVPR2 variants and mutations in nephrogenic diabetes insipidus: review and missense mutation significance. Almost 90% of nephrogenic diabetes insipidus (NDI) is due to mutations in the arginine-vasopressin receptor 2 gene (AVPR2). We retrospectively examined all the published mutations/variants in AVPR2. We planned to perform a comprehensive review of all the AVPR2 mutations/variants and to test whether any amino acid change causing a missense mutation is significantly more or less common than

2008 Journal of cellular physiology

3. Causes of reversible nephrogenic diabetes insipidus: a systematic review. (Abstract)

Causes of reversible nephrogenic diabetes insipidus: a systematic review. In nephrogenic diabetes insipidus (NDI), the kidney is unable to produce concentrated urine because of the insensitivity of the distal nephron to antidiuretic hormone (arginine vasopressin). In settings in which fluid intake cannot be maintained, this may result in severe dehydration and electrolyte imbalances. The risk for conversion of reversible to irreversible NDI seems to be a potential complication. This review

2005 American journal of kidney diseases : the official journal of the National Kidney Foundation

4. Desmopressin for nocturnal enuresis in nephrogenic diabetes insipidus. (Abstract)

Desmopressin for nocturnal enuresis in nephrogenic diabetes insipidus. We have investigated two unrelated families, in which two children had inherited primary nocturnal enuresis, and nephrogenic diabetes insipidus caused by new mutations in the aquaporin-2 gene (AQP2). The mutant AQP2 proteins were inactive, suggesting that administration of desmopressin could not concentrate the urine in these patients. However, treatment with desmopressin resolved primary nocturnal enuresis completely

2002 Lancet