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Yeoman File Generation

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1. Yeoman File Generation

Yeoman File Generation Yeoman File Generation Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Yeoman File Generation Yeoman File (...) Generation Aka: Yeoman File Generation From Related Chapters II. Technique: Setup Install Node Install Yeoman npm install -g yo tsd Create a directory for project mkdir generator-MYNAMEHERE cd generator-MYNAMEHERE\ Create json config files npm init (package.json for node) tsd init (tsd.json) Open in in root directory "code ." Edit json file Add dev-dependencies object Add dependencies (e.g. Yeoman) Create the index.js file mkdir app Create file "app/index.js" Set up the index.js to run all the node code

2018 FP Notebook

2. Yeoman File Generation

Yeoman File Generation Yeoman File Generation Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Yeoman File Generation Yeoman File (...) Generation Aka: Yeoman File Generation From Related Chapters II. Technique: Setup Install Node Install Yeoman npm install -g yo tsd Create a directory for project mkdir generator-MYNAMEHERE cd generator-MYNAMEHERE\ Create json config files npm init (package.json for node) tsd init (tsd.json) Open in in root directory "code ." Edit json file Add dev-dependencies object Add dependencies (e.g. Yeoman) Create the index.js file mkdir app Create file "app/index.js" Set up the index.js to run all the node code

2016 FP Notebook

3. Diagnosis and Treatment of Low Back Pain

and Value, VA, Washington, DC & Office of Evidence Based Practice, U.S. Army Medical Command Version 2.0 – 2017 Based on evidence reviewed through October 21, 2016 VA/DoD Clinical Practice Guideline for Diagnosis and Treatment of Low Back Pain September 2017 Page 3 of 110 Table of Contents I. Introduction 5 II. Recommendations 6 III. Background 9 A. Description of Low Back Pain 9 B. Epidemiology and Impact 10 a. General Population 10 b. Veterans Affairs Population 11 c. Department of Defense Population (...) published the Clinical Practice Guideline for diagnosis and treatment of Low Back Pain (2007 LBP CPG), which was based on evidence reviewed through November 2006. Since the release of that guideline, a growing body of research has expanded the general knowledge and understanding of LBP. Improved recognition of the complex nature of these conditions has led to the adoption of new strategies for diagnosis and treatment of LBP. Consequently, a recommendation to update the 2007 LBP CPG was initiated in 2016

2017 VA/DoD Clinical Practice Guidelines

4. Cancer Prevention Overview (PDQ®): Health Professional Version

patients were matched with an equal number of controls by age, sex, and zip code. Cancer patients were 2.6 times more likely to file for bankruptcy than the cancer-free controls ( P < .05).[ ] References American Cancer Society: Cancer Facts and Figures 2019. Atlanta, Ga: American Cancer Society, 2019. . Last accessed January 23, 2019. Faller H, Schuler M, Richard M, et al.: Effects of psycho-oncologic interventions on emotional distress and quality of life in adult patients with cancer: systematic (...) of Vogelstein and Kinzler [ ] and Hanahan and Weinberg.[ ] The model of Vogelstein and Kinzler emphasizes that cancer is ultimately a disease of damaged DNA, comprised of a series of genetic mutations that can transform normal cells to cancerous cells. The genetic mutations include inactivation of tumor suppressor genes and activation of oncogenes. Compared with cancers arising in the general population, individuals with a major inherited predisposition to cancer are born with inherited (i.e., germline

2017 PDQ - NCI's Comprehensive Cancer Database

5. Langerhans Cell Histiocytosis Treatment (PDQ®): Health Professional Version

for cancer patients. It does not provide formal guidelines or recommendations for making health care decisions. This summary is reviewed regularly and updated as necessary by the PDQ Pediatric Treatment Editorial Board, which is editorially independent of the National Cancer Institute (NCI). The summary reflects an independent review of the literature and does not represent a policy statement of NCI or the National Institutes of Health (NIH). General Information About Langerhans Cell Histiocytosis (LCH (...) of cell development at which the somatic mutation occurs is critical in defining the extent of disease in LCH. LCH is now considered a myeloid neoplasm. Clinical implications Clinical implications of the described genomic findings include the following: LCH joins a group of other pediatric entities with activating BRAF mutations, including select nonmalignant conditions (e.g., benign nevi) [ ] and low-grade malignancies (e.g., pilocytic astrocytoma).[ , ] All of these conditions have a generally

2016 PDQ - NCI's Comprehensive Cancer Database

6. Langerhans Cell Histiocytosis Treatment (PDQ®): Health Professional Version

for cancer patients. It does not provide formal guidelines or recommendations for making health care decisions. This summary is reviewed regularly and updated as necessary by the PDQ Pediatric Treatment Editorial Board, which is editorially independent of the National Cancer Institute (NCI). The summary reflects an independent review of the literature and does not represent a policy statement of NCI or the National Institutes of Health (NIH). General Information About Langerhans Cell Histiocytosis (LCH (...) of cell development at which the somatic mutation occurs is critical in defining the extent of disease in LCH. LCH is now considered a myeloid neoplasm. Clinical implications Clinical implications of the described genomic findings include the following: LCH joins a group of other pediatric entities with activating BRAF mutations, including select nonmalignant conditions (e.g., benign nevi) [ ] and low-grade malignancies (e.g., pilocytic astrocytoma).[ , ] All of these conditions have a generally

2016 PDQ - NCI's Comprehensive Cancer Database

7. Cancer Prevention Overview (PDQ®): Health Professional Version

patients were matched with an equal number of controls by age, sex, and zip code. Cancer patients were 2.6 times more likely to file for bankruptcy than the cancer-free controls ( P < .05).[ ] References American Cancer Society: Cancer Facts and Figures 2019. Atlanta, Ga: American Cancer Society, 2019. . Last accessed January 23, 2019. Faller H, Schuler M, Richard M, et al.: Effects of psycho-oncologic interventions on emotional distress and quality of life in adult patients with cancer: systematic (...) of Vogelstein and Kinzler [ ] and Hanahan and Weinberg.[ ] The model of Vogelstein and Kinzler emphasizes that cancer is ultimately a disease of damaged DNA, comprised of a series of genetic mutations that can transform normal cells to cancerous cells. The genetic mutations include inactivation of tumor suppressor genes and activation of oncogenes. Compared with cancers arising in the general population, individuals with a major inherited predisposition to cancer are born with inherited (i.e., germline

2016 PDQ - NCI's Comprehensive Cancer Database

8. Behavioral and Oral Motor Interventions for Feeding Problems in Children

, D.; Yeomans, M. R.; and Wardle, J.: Eating for pleasure or profit: the effect of incentives on children's enjoyment of vegetables. Psychol Sci, 22(2): 190-6, 2011, [2a]] http://www.ncbi.nlm.nih.gov/pubmed/21191095 ?. 11. Davies, F.: Does the ends justify the means? Asia Pacific Journal of Speech Language and Hearing, 8(2): 146- 152, 2003, [1b]] ?. 12. Gentry, J. A., and Luiselli, J. K.: Treating a child’s selective eating through parent implemented feeding intervention in the home setting (...) : An Interdisciplinary Journal of Special Care Practices, 15(1): 29-41, 2002, [5a]] ?. 48. Valdimarsdottir, H.; Halldorsdottir, L. Y.; and Sigurthardottir, Z. G.: Increasing the variety of foods consumed by a picky eater: generalization of effects across caregivers and settings. J Appl Behav Anal, 43(1): 101-5, 2010, [5a]] http://www.ncbi.nlm.nih.gov/pubmed/20808499 ?. 49. VanDalen, K. H., and Penrod, B.: A comparison of simultaneous versus sequential presentation of novel foods in the treatment of food selectivity

2013 Cincinnati Children's Hospital Medical Center

9. Langerhans Cell Histiocytosis

Langerhans Cell Histiocytosis Langerhans Cell Histiocytosis Treatment (PDQ®)—Health Professional Version - National Cancer Institute Menu Search Search Search General Information About Langerhans Cell Histiocytosis (LCH) The histiocytic diseases in children and adults are caused by an abnormal accumulation of cells of the mononuclear phagocytic system. Only Langerhans cell histiocytosis (LCH), a myeloid-derived dendritic cell disorder, is discussed in detail in this summary. The histiocytic (...) conditions (e.g., benign nevi) [ ] and low-grade malignancies (e.g., pilocytic astrocytoma).[ , ] All of these conditions have a generally indolent course, with spontaneous resolution occurring in some cases. This distinctive clinical course may be a manifestation of oncogene-induced senescence.[ , ] BRAF V600E mutations can be targeted by BRAF inhibitors (e.g., vemurafenib and dabrafenib) or by the combination of BRAF inhibitors plus MEK inhibitors (e.g., dabrafenib/trametinib and vemurafenib

2012 PDQ - NCI's Comprehensive Cancer Database

10. Cancer Prevention Overview

patients and their families. In addition to the physical morbidity caused by cancer, cancer is frequently associated with emotional distress and an overall reduction in quality of life.[ ] Cancer has also been observed to be a financial stressor. In a population-based study in western Washington, 197,840 cancer patients were matched with an equal number of controls by age, sex, and zip code. Cancer patients were 2.6 times more likely to file for bankruptcy than the cancer-free controls ( P < .05 (...) cells to cancerous cells. The genetic mutations include inactivation of tumor suppressor genes and activation of oncogenes. Compared with cancers arising in the general population, individuals with a major inherited predisposition to cancer are born with inherited (i.e., germline) mutations in genes involved in cancer causation, giving them a head start on the pathway to cancer. Similar mutations would be expected to result in cancer progression among all individuals; however, in those without

2012 PDQ - NCI's Comprehensive Cancer Database

11. Risk of serious NSAID-related gastrointestinal events during long-term exposure: a systematic review

in English, French or German. Using the same search criteria, a high-specificity search of Current Contents, EMBASE, MEDLINE and Derwent Drug File databases was conducted in July 2002 (limitations: humans, 1999–2002). In this manner, relevant observational studies and further randomised trials were identified either directly (back to 1999) or indirectly (by manual search of systematic reviews from 1999–2002, representing the NSAID literature back to the 1960s). Abstracts of 479 search results were (...) reviewed; 360 records were excluded for not fulfilling the basic criteria, and 119 records plus an additional 102 records identified from manual searches were retrieved for more detailed evaluation (total, 221) ( ). When retrospective cohort studies using the same health care databases (eg, Tennessee Medicaid, UK General Practice Research Database, Tayside, or ARAMIS) were identified, only one study per database was selected; the rest , , - were excluded to avoid potentially redundant patient

2006 EvidenceUpdates

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