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3,006 results for

Q Fever

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3001. Q fever: an emerging public health concern in Canada. (PubMed)

Q fever: an emerging public health concern in Canada. Q fever in humans and coxiellosis in livestock are on the increase in Canada. The progressive spread of Coxiella burnetii takes place in the animal reservoirs of the rickettsia, mainly domestic ruminants. Human Q fever is a reverberation of the coxiellosis situation on our farms. Increasing animal concentration resulting from the industrialization of agriculture and oversight of the infection in livestock permitted the extension

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1989 Canadian Journal of Veterinary Research

3002. Chronic Q fever with mixed cryoglobulinaemia. (PubMed)

Chronic Q fever with mixed cryoglobulinaemia. A 47 year old woman presented with a six month history of vasculitic rash, splenomegaly, and cardiac murmurs. Investigations showed the presence of mixed cryoglobulinaemia and raised titres to Coxiella burnetii consistent with chronic Q fever infection. The patient was treated with tetracycline (1 g four times a day).

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1989 Annals of the Rheumatic Diseases

3003. Q fever: person to person transmission within a family. (PubMed)

Q fever: person to person transmission within a family. 3590060 1987 07 23 2018 11 13 0040-6376 41 12 1986 Dec Thorax Thorax Q fever: person to person transmission within a family. 974-5 Mann J S JS Douglas J G JG Inglis J M JM Leitch A G AG eng Case Reports Journal Article England Thorax 0417353 0040-6376 IM Adult Aged Child Female Humans Male Q Fever genetics transmission 1986 12 1 1986 12 1 0 1 1986 12 1 0 0 ppublish 3590060 PMC460565 Lancet. 1949 Dec 3;2(6588):1028-30 15394107 J Am Med

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1986 Thorax

3004. Severe combined immunodeficiency syndrome, tissue transplant, leukaemia, and Q fever. (PubMed)

Severe combined immunodeficiency syndrome, tissue transplant, leukaemia, and Q fever. A child born with severe combined immunodeficiency, who was immunoreconstituted by a fetal liver and thymus transplant, developed acute lymphoblastic leukaemia in the donor cell line. During remission she contracted acute Q fever, which gave rise to unexpected complications. Early treatment of the Q fever might have altered the subsequent events and prevented her death.

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1988 Archives of Disease in Childhood

3005. Dysregulation of cytokines in acute Q fever: role of interleukin-10 and tumor necrosis factor in chronic evolution of Q fever. (PubMed)

Dysregulation of cytokines in acute Q fever: role of interleukin-10 and tumor necrosis factor in chronic evolution of Q fever. Q fever manifests as primary infection or acute Q fever and may become chronic in patients with underlying valvulopathy. Because Coxiella burnetii infection depends on host response, we measured tumor necrosis factor (TNF), interleukin (IL)-6, IL-12, and IL-10 in patients with different clinical presentations of acute Q fever. Compared with control subjects, patients (...) with uncomplicated acute Q fever exhibited increased release of the 4 cytokines. Their amounts were higher in patients with hepatitis than in patients with fever or pneumonia. In patients with valvulopathy, who exhibited the highest risk of chronic evolution, the amounts of TNF and IL-10 were higher than in patients without valvulopathy. TNF production was specifically enhanced in patients who developed Q fever endocarditis. These results show that acute Q fever is associated with cytokine overproduction

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2003 Journal of Infectious Diseases

3006. Increases in the Levels of Coxiella burnetii-Specific Immunoglobulin G1 and G3 Antibodies in Acute Q Fever and Chronic Q Fever (PubMed)

Increases in the Levels of Coxiella burnetii-Specific Immunoglobulin G1 and G3 Antibodies in Acute Q Fever and Chronic Q Fever A detailed analysis of the humoral response to Coxiella burnetii may provide insight into the pathogenesis of Q fever, a zoonosis caused by C. burnetii. The subclasses of C. burnetii-specific antibodies were determined by immunofluorescence in 20 patients with acute Q fever and 20 patients with chronic Q fever. Although immunoglobulin G1 (IgG1) and IgG3 antibodies were (...) found in acute and chronic Q fever, neither IgG2 nor IgG4 was detected. The detection of IgG1 and IgG3 antibodies was not due to an increase of the IgG1 and IgG3 subclasses. Moreover, IgG1 and IgG3 antibodies were not correlated, suggesting that they may play different roles in Q fever.

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1998 Clinical and Diagnostic Laboratory Immunology

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