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Polycythemia Rubra Vera

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1. Polycythemia Rubra Vera

Polycythemia Rubra Vera Polycythemia Rubra Vera Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Polycythemia Rubra Vera Polycythemia (...) Rubra Vera Aka: Polycythemia Rubra Vera , Polycythemia Vera From Related Chapters II. Definition Excessive production due to chronic myeloproliferative neoplasm III. Epidemiology Men affected more than women Age Median age of onset: 60 years old Under age 40 years old represent 20-25% of cases : 2.3 per 100,000 persons per year Prevelance: 44-57 per 100,000 persons (U.S.) IV. Pathophysiology Chronic myeloproliferative neoplasm (primary Polycythemia Vera) Associated with Janus Kinase 2 gene (JAK2

2018 FP Notebook

2. Hypertension in polycythemia rubra vera

Hypertension in polycythemia rubra vera Hypertension in polycythemia rubra vera – All About Cardiovascular System and Disorders Now Trending: | December 9, 2015 | | Hypertension in polycythemia rubra vera Multiple mechanisms have been proposed as the cause of systemic hypertension in polycythemia rubra vera: Increased number of circulating erythrocytes increase the blood viscosity and clog the renal circulation causing renal ischemia, which triggers the secretion of renin. Renin in turn

2015 Cardiophile MD blog

3. The Etiology and Treatment of Polycythemia Rubra Vera : Observations Based upon Studies of Body Fluid Changes in Dogs Subjected to Proprioceptor Depressor Neurotomy and Extensive Sympathectomy, Including the Case Report of a Man with Polycythemia Rubra Ve (PubMed)

The Etiology and Treatment of Polycythemia Rubra Vera : Observations Based upon Studies of Body Fluid Changes in Dogs Subjected to Proprioceptor Depressor Neurotomy and Extensive Sympathectomy, Including the Case Report of a Man with Polycythemia Rubra Ve 17858704 2007 09 17 2018 11 13 0003-4932 122 6 1945 Dec Annals of surgery Ann. Surg. The Etiology and Treatment of Polycythemia Rubra Vera : Observations Based upon Studies of Body Fluid Changes in Dogs Subjected to Proprioceptor Depressor (...) Neurotomy and Extensive Sympathectomy, Including the Case Report of a Man with Polycythemia Rubra Vera Treated by Extensive Para-Vertebral Sympathectomy. 1098-124 Schafer P W PW eng Journal Article United States Ann Surg 0372354 0003-4932 1945 12 1 0 0 1945 12 1 0 1 1945 12 1 0 0 ppublish 17858704 PMC1618335 Ann Surg. 1941 Oct;114(4):753-75 17857907

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1945 Annals of Surgery

4. Polycythemia Rubra Vera

Polycythemia Rubra Vera Polycythemia Rubra Vera Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Polycythemia Rubra Vera Polycythemia (...) Rubra Vera Aka: Polycythemia Rubra Vera , Polycythemia Vera From Related Chapters II. Definition Excessive production due to chronic myeloproliferative neoplasm III. Epidemiology Men affected more than women Age Median age of onset: 60 years old Under age 40 years old represent 20-25% of cases : 2.3 per 100,000 persons per year Prevelance: 44-57 per 100,000 persons (U.S.) IV. Pathophysiology Chronic myeloproliferative neoplasm (primary Polycythemia Vera) Associated with Janus Kinase 2 gene (JAK2

2015 FP Notebook

5. Chorea complicating polycythemia rubra vera (PubMed)

Chorea complicating polycythemia rubra vera A patient with polycythemia rubra vera and chorea is described and the literature on this rare combination is reviewed. This is the first report of the association of chorea and polycythemia in a male patient.The contrasting features of other forms of diffuse cerebral pathology which may be accompanied by chorea, namely disseminated lupus erythematosus, Henoch-Schönlein purpura and rheumatic chorea, are discussed.

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1972 Canadian Medical Association Journal

6. Disseminated intravascular coagulation associated with polycythaemia rubra vera. (PubMed)

Disseminated intravascular coagulation associated with polycythaemia rubra vera. 959233 1976 10 20 2018 11 13 0035-9157 69 7 1976 Jul Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Disseminated intravascular coagulation associated with polycythaemia rubra vera. 521-2 Lee C A CA Moss S S eng Case Reports Journal Article England Proc R Soc Med 7505890 0035-9157 IM Bloodletting Disseminated Intravascular Coagulation complications Female Humans Middle Aged Polycythemia Vera

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1976 Proceedings of the Royal Society of Medicine

7. Eosinophilic spongiosis with polycythaemia rubra vera. (PubMed)

Eosinophilic spongiosis with polycythaemia rubra vera. 323863 1977 06 30 2018 11 13 0035-9157 70 2 1977 Feb Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Eosinophilic spongiosis with polycythaemia rubra vera. 139-40 Black M M MM eng Case Reports Journal Article England Proc R Soc Med 7505890 0035-9157 IM Eosinophils Humans Polycythemia Vera complications Skin Diseases, Vesiculobullous complications drug therapy 1977 2 1 1977 2 1 0 1 1977 2 1 0 0 ppublish 323863 PMC1542934 Proc

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1977 Proceedings of the Royal Society of Medicine

8. Treatment of Polycythæmia Rubra Vera with Radioactive phosphorus (PubMed)

Treatment of Polycythæmia Rubra Vera with Radioactive phosphorus 13215521 2003 05 01 2018 12 01 0035-9157 47 10 1954 Oct Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Treatment of polycythaemia rubra vera with radioactive phosphorus. 857-9 VEREL D D eng Journal Article England Proc R Soc Med 7505890 0035-9157 0 Phosphorus Radioisotopes 0 Phosphorus, Dietary 27YLU75U4W Phosphorus OM Humans Phosphorus Phosphorus Radioisotopes Phosphorus, Dietary Polycythemia Vera therapy (...) Radioactivity 5527:27299:356:369 PHOSPHORUS/radioactive POLYCYTHEMIA VERA/therapy 1954 10 1 1954 10 1 0 1 1954 10 1 0 0 ppublish 13215521 PMC1918974

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1954 Proceedings of the Royal Society of Medicine

9. Treatment of Polycythaemia Rubra Vera (PubMed)

Treatment of Polycythaemia Rubra Vera 13042161 2003 05 01 2018 12 01 0007-1447 1 4820 1953 May 23 British medical journal Br Med J Treatment of polycythaemia rubra vera. 1128-31 SCOTT R B RB eng Journal Article England Br Med J 0372673 0007-1447 0 Phosphorus Radioisotopes 0 Phosphorus, Dietary 27YLU75U4W Phosphorus OM Humans Phosphorus Phosphorus Radioisotopes Phosphorus, Dietary Polycythemia Vera therapy Radioactivity 5324:12500:496:516 PHOSPHORUS/radioactive POLYCYTHEMIA VERA/therapy 1953 5

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1953 British medical journal

10. Pyrimethamine in the Treatment of Polycythaemia Rubra Vera (PubMed)

Pyrimethamine in the Treatment of Polycythaemia Rubra Vera 13733910 1998 11 01 2018 11 30 0007-1447 2 5252 1961 Sep 02 British medical journal Br Med J Pyrimethamine in the treatment of polycythaemia rubra vera. 617-21 PEGG D E DE FORD H T HT eng Journal Article England Br Med J 0372673 0007-1447 0 Antimalarials Z3614QOX8W Pyrimethamine OM Antimalarials therapy Humans Polycythemia Vera therapy Pyrimethamine ANTIMALARIALS/therapy POLYCYTHEMIA VERA/therapy 1961 9 2 1961 9 2 0 1 1961 9 2 0 0

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1961 British medical journal

11. Polycythæmia Rubra Vera: Skin Eruption Undiagnosed (PubMed)

Polycythæmia Rubra Vera: Skin Eruption Undiagnosed 14291267 1996 12 01 2018 12 01 0035-9157 58 1965 Jun Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. POLYCYTHAEMIA RUBRA VERA: SKIN ERUPTION UNDIAGNOSED. 429-30 WALLACE H J HJ eng Journal Article England Proc R Soc Med 7505890 0035-9157 VB0R961HZT Prednisone OM Diagnosis Exanthema Humans Pathology Polycythemia Vera Prednisone therapeutic use Skin Diseases DIAGNOSIS PATHOLOGY POLYCYTHEMIA VERA PREDNISONE, THERAPEUTIC SKIN

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1965 Proceedings of the Royal Society of Medicine

12. MOBILE RIGHT HEART THROMBUS WITH PULMONARY EMBOLISM IN A PATIENT WITH POLYCYTHEMIA RUBRA VERA AND SPLANCHNIC VEIN THROMBOSIS (PubMed)

MOBILE RIGHT HEART THROMBUS WITH PULMONARY EMBOLISM IN A PATIENT WITH POLYCYTHEMIA RUBRA VERA AND SPLANCHNIC VEIN THROMBOSIS Splanchnic vein thrombosis in patients with polycythemia rubra vera is well-known. Development of mobile right heart thrombus in these patients has not been reported previously. We describe a young patient with Polycythemia rubra vera and splanchnic vein thrombosis with ischemic bowel who underwent small bowel resection. He developed a large mobile right atrial thrombus

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2010 Heart views : the official journal of the Gulf Heart Association

13. Polycythemia (Overview)

erythropoiesis-stimulating factors. Relative polycythemia, or pseudoerythrocytosis, is caused by an apparent red blood cell mass increase due to plasma volume reduction (eg, due to severe diarrhea with subsequent dehydration), resulting in increased hemoconcentration. This article will review both primary and secondary polycythemias. A specific type of primary polycythemia, polycythemia rubra vera (often just called polycythemia vera) is an acquired myeloproliferative disorder which is discussed in detail (...) precursors caused by acquired or inherited mutations. These polycythemias include the diagnoses of polycythemia vera and primary familial and congenital polycythemia. Primary familial and congenital polycythemia (PFCP) is caused by germline mutations in the EpoR leading to constitutive activation of EpoR. The constantly activated "on switch" leads to excessive erythroid progenitor proliferation and differentiation, resulting in polycythemia. This autosomal dominant trait does not necessarily carry

2014 eMedicine Pediatrics

14. Polycythemia (Diagnosis)

erythropoiesis-stimulating factors. Relative polycythemia, or pseudoerythrocytosis, is caused by an apparent red blood cell mass increase due to plasma volume reduction (eg, due to severe diarrhea with subsequent dehydration), resulting in increased hemoconcentration. This article will review both primary and secondary polycythemias. A specific type of primary polycythemia, polycythemia rubra vera (often just called polycythemia vera) is an acquired myeloproliferative disorder which is discussed in detail (...) precursors caused by acquired or inherited mutations. These polycythemias include the diagnoses of polycythemia vera and primary familial and congenital polycythemia. Primary familial and congenital polycythemia (PFCP) is caused by germline mutations in the EpoR leading to constitutive activation of EpoR. The constantly activated "on switch" leads to excessive erythroid progenitor proliferation and differentiation, resulting in polycythemia. This autosomal dominant trait does not necessarily carry

2014 eMedicine Pediatrics

15. Bone Marrow Synoptic Reporting for Hematologic Neoplasms

concerns about the studies' findings. One RCS reported clinical significance for data on white blood cell counts in patients with Philadelphia-positive (Ph + ) ALL and polycythemia vera. Four studies, comprising 2 PCSs , and 2 RCS, , reported clinical significance for data on hemoglobin in patients with MDS and AA. Four studies, comprising 1 PCS and 3 RCSs, , , reported clinical significance for data on platelets in MDS, myeloproliferative disease, adult T-cell leukemia/lymphoma patients. Two RCSs (...) and constitute an integral component of any hematologic evaluation. There was high-level evidence demonstrating significantly different clinical outcomes for white blood cell counts in Ph + ALL and polycythemia vera , ; hemoglobin levels in myelodysplasia and AA – ; platelet counts in myelodysplasia, myeloproliferative disease, adult T-cell leukemia/lymphoma , , , ; absolute neutrophil count in myelodysplasia , ; and red cell distribution width and reticulocyte counts in AA. Other parameters, such as red

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2016 College of American Pathologists

16. Antithrombotics: indications and management

months. Treatment with aspirin (40-100 mg per day) was associated with a large but non-statistically significant reduction in the risk of fatal thrombotic events, without an increased risk of major bleeding, when compared to no treatment in those patients who had no clear alternative indication or contraindication to aspirin therapy (OR 0.20, 95% CI 0.03 to 1.14). 200 B Patients with polycythaemia rubra vera should be considered for treatment with aspirin, unless there are contraindications

2012 SIGN

17. Pruritus (PDQ®): Health Professional Version

, the incidence of pruritus in that condition is given. Table 1. Pruritus Differential With Little or No Primary Dermatitis Disease State Prevalence of Pruritus (%) Neoplastic – Hodgkin disease 30 [ ] – Non-Hodgkin lymphoma 15 [ ] – Leukemias 5 [ ] – Carcinoid syndrome – Paraproteinemias Iron deficiency anemia Polycythemia rubra vera ≤50 [ ] Hyper-IgE syndromes Parasitic helminthic infection Drug-induced eosinophilia Chronic renal insufficiency 57 [ ] Liver disease – Primary biliary cirrhosis 69 [ ] – Viral (...) , or makeshift tools). It is estimated that pruritus is a manifestation of an underlying systemic disease in approximately 10% to 25% of affected individuals. Nondermatologic conditions that can lead to generalized pruritus include the following:[ ] Hepatic, renal, or thyroid dysfunction. Lymphoma, myeloproliferative disorders (e.g., polycythemia vera, hypereosinophilic syndrome), and chronic lymphocytic leukemia. HIV or parasitic infections. Neuropsychiatric disorders. Despite the wide array of diseases

2016 PDQ - NCI's Comprehensive Cancer Database

18. Ruxolitinib Combined With Ibrutinib in Chronic Lymphocytic Leukemia Patients

properties of the CLL microenvironment and help to improve outcomes with Ibrutinib. The JAK2 inhibitor Ruxolitinib is licensed for use in myelofibrosis and polycythemia rubra vera and its toxicity and efficacy as a single agent in CLL has been studied in two previous clinical trials. As a single agent, Ruxolitinib was inferior to Ibrutinib. However, Ruxolitinib was shown to cooperate with Ibrutinib and increase killing of CLL cells in vitro. Based on these observations, it is hypothesized

2016 Clinical Trials

19. Pruritus

= immunoglobulin E. Neoplastic – Hodgkin disease 30 [ ] – Non-Hodgkin lymphoma 15 [ ] – Leukemias 5 [ ] – Carcinoid syndrome – Paraproteinemias Iron deficiency anemia Polycythemia rubra vera ≤50 [ ] Hyper-IgE syndromes Parasitic helminthic infection Drug-induced eosinophilia Chronic renal insufficiency 57 [ ] Liver disease – Primary biliary cirrhosis 69 [ ] – Viral hepatitis infection 15 [ ] – Cholestatic disease – Autoimmune hepatitis Thyroid dysfunction – Hashimoto thyroiditis – Hypothyroidism (...) to generalized pruritus include the following:[ ] Hepatic, renal, or thyroid dysfunction. Lymphoma, myeloproliferative disorders (e.g., polycythemia vera, hypereosinophilic syndrome), and chronic lymphocytic leukemia. HIV or parasitic infections. Neuropsychiatric disorders. Despite the wide array of diseases that may present with pruritus, a systematic evaluation of the differential using a good history, review of systems, and appropriate blood work will lead to a rational and finite group of etiologies

2012 PDQ - NCI's Comprehensive Cancer Database

20. Deep Venous Thrombosis (Diagnosis)

under . It can also be extended, as during hospitalization for pelvic, hip, or spinal surgery, or due to stroke or paraplegia. Individuals in these circumstances warrant surveillance, prophylaxis, and treatment if they develop DVT. [ , ] Reduced blood flow from increased blood viscosity or central venous pressure Increased blood viscosity may decrease venous blood flow. This change may be due to an increase in the cellular component of the blood in polycythemia rubra vera or thrombocytosis (...) trips (>4 hours) in previous 4 weeks Cancer Previous DVT Stroke Acute myocardial infarction (AMI) Congestive heart failure (CHF) Sepsis Nephrotic syndrome Ulcerative colitis Multiple trauma CNS/spinal cord injury Burns Lower extremity fractures Systemic lupus erythematosus (SLE) and the lupus anticoagulant Behçet syndrome Homocystinuria Polycythemia rubra vera Thrombocytosis Inherited disorders of coagulation/fibrinolysis Antithrombin III deficiency Protein C deficiency Protein S deficiency

2014 eMedicine Surgery

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