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Parietal Lobe

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2721. Resection of parietal lobe gliomas: incidence and evolution of neurological deficits in 28 consecutive patients correlated to the location and morphological characteristics of the tumor. (PubMed)

Resection of parietal lobe gliomas: incidence and evolution of neurological deficits in 28 consecutive patients correlated to the location and morphological characteristics of the tumor. The goal of this study is to report the incidence and clinical evolution of neurological deficits in patients who underwent resection of gliomas confined to the parietal lobe.Patient demographics, findings of serial neurological examinations, tumor location and neuroimaging characteristics, extent of resection (...) , and surgical outcomes were tabulated by reviewing inpatient and office records, as well as all pre- and postoperative magnetic resonance (MR) images obtained in 28 consecutive patients who underwent resection of a glial neoplasm found on imaging studies to be confined to the parietal lobe. Neurological deficits were correlated with hemispheric dominance, location of the lesion within the superior or inferior parietal lobules, subcortical extension, and involvement of the postcentral gyrus. The tumors were

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2005 Journal of Neurosurgery

2722. Transient inaccuracy in reaching caused by a posterior parietal lobe lesion. (PubMed)

Transient inaccuracy in reaching caused by a posterior parietal lobe lesion. A transient disturbance of manual reaching in both hemispaces associated with a small contralateral posterior perietal lobe (area 7) lesion is described. This defect was increased when the patient tried to reach without viewing of the affected arm. Inaccurate reaching is a distinct syndrome from visuomotor or optic ataxia, and is interpreted as a failure in guiding the arm towards specific sites of the space.

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1984 Journal of neurology, neurosurgery, and psychiatry

2723. Summation of rapid tactile stimuli in parietal lobe disease. (PubMed)

Summation of rapid tactile stimuli in parietal lobe disease. The perception threshold for trains of rapid tactile pulses, applied to the index finger, has been measured in patients with parietal lobe lesions and in patients with median nerve lesions. The former patients had increased perception thresholds for single tactile pulses on the abnormal side. With successively prolonged pulse trains, the threshold decreased exponentially to reach a stable level after 150-400 ms. In contrast

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1982 Journal of neurology, neurosurgery, and psychiatry

2724. Deep left parietal lobe syndrome: conduction aphasia and other neurobehavioural disorders due to a small subcortical lesion. (PubMed)

Deep left parietal lobe syndrome: conduction aphasia and other neurobehavioural disorders due to a small subcortical lesion. A patient with sudden onset of conduction aphasia in the context of an ischaemic stroke is reported. Other neurological and neuropsychological findings included bilateral ideomotor apraxia, right hemisensory defect and paradoxical left ear extinction on a dichotic listening test. Lesion location, as inferred from magnetic resonance imaging, involved a restricted (...) subcortical area in the left parietal lobe, near the lateral wall of the cerebral ventricle. The anatomical correlate for each of the clinical findings is discussed in the light of classical anatomo-clinical correlations. It is concluded that this tetrad constitutes a specific syndrome which may be easily recognised and ascribed to a single lesion in the deep white matter of the left parietal lobe.

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1987 Journal of neurology, neurosurgery, and psychiatry

2725. Visually induced central pain and arm withdrawal after right parietal lobe infarction. (PubMed)

Visually induced central pain and arm withdrawal after right parietal lobe infarction. A 46 year old man with ischaemic infarction of the right parietal cortex had left hemianaesthesia when his eyes were closed. With eyes open, visual stimuli induced withdrawal of the arm and a burning pain in the numb side of the body. Visually induced central pain is a new clinical finding and may be related to damage of cells with anticipatory inhibitory function in the parietal association cortex.

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1994 Journal of neurology, neurosurgery, and psychiatry

2726. Micrographia associated with a parietal lobe lesion in multiple sclerosis. (PubMed)

Micrographia associated with a parietal lobe lesion in multiple sclerosis. The occurrence of micrographia in a 52 year old women two years after an isolated episode of painful sensory disturbance led to the diagnosis of multiple sclerosis. Her handwriting returned to normal after a course of intravenous methylprednisolone. Previous reports of movement disorders occurring in the context of multiple sclerosis are briefly reviewed. The finding on MRI studies of an enhancing lesion in the dominant (...) parietal white matter supports Kinnier Wilson's suggestion that the anatomical origin of micrographia lies in the cerebral hemisphere rather than the corpus striatum.

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1994 Journal of neurology, neurosurgery, and psychiatry

2727. Spatial orientation and the representation of space with parietal lobe lesions. (PubMed)

Spatial orientation and the representation of space with parietal lobe lesions. Damage to the human parietal cortex leads to disturbances of spatial perception and of motor behaviour. Within the parietal lobe, lesions of the superior and of the inferior lobule induce quite different, characteristic deficits. Patients with inferior (predominantly right) parietal lobe lesions fail to explore the contralesional part of space by eye or limb movements (spatial neglect). In contrast, superior (...) parietal lobe lesions lead to specific impairments of goal-directed movements (optic ataxia). The observations reported in this paper support the view of dissociated functions represented in the inferior and the superior lobule of the human parietal cortex. They suggest that a spatial reference frame for exploratory behaviour is disturbed in patients with neglect. Data from these patients' visual search argue that their failure to explore the contralesional side is due to a disturbed input

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1997 Philosophical Transactions of the Royal Society B: Biological Sciences

2728. Cortical integration in the visual system of the macaque monkey: large-scale morphological differences in the pyramidal neurons in the occipital, parietal and temporal lobes. (PubMed)

Cortical integration in the visual system of the macaque monkey: large-scale morphological differences in the pyramidal neurons in the occipital, parietal and temporal lobes. Layer III pyramidal neurons were injected with Lucifer yellow in tangential cortical slices taken from the inferior temporal cortex (area TE) and the superior temporal polysensory (STP) area of the macaque monkey. Basal dendritic field areas of layer III pyramidal neurons in area STP are significantly larger

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1999 Proceedings of the Royal Society B: Biological Sciences

2729. Conversion sensory symptoms associated with parietal lobe infarct: case report, diagnostic issues and brain mechanisms (PubMed)

Conversion sensory symptoms associated with parietal lobe infarct: case report, diagnostic issues and brain mechanisms This case report suggests that diagnostic difficulties and brain mechanisms related to conversion disorder associated with cerebral lesions differ from those related to conversion disorder without cerebral lesions. A 35-year-old divorced woman was admitted to a psychiatric inpatient unit with multiple physical complaints. The symptoms first appeared 5 years previous and 2 (...) months after a sexual assault. Three years later, she began to experience ill-defined sensory symptoms confined to the left half of her body (splitting the midline). Results of neurologic consultations were equivocal because of the subjective nature of the complaints, which were viewed as conversion symptoms. A magnetic resonance imaging scan demonstrated an old infarct in the right parietal lobe, suggesting a physical origin of the patient's symptoms. However, normal somatosensory-evoked responses

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2002 Journal of Psychiatry and Neuroscience

2730. Selective reduction of N-acetylaspartate in medial temporal and parietal lobes in AD (PubMed)

Selective reduction of N-acetylaspartate in medial temporal and parietal lobes in AD Both AD and normal aging cause brain atrophy, limiting the ability of MRI to distinguish between AD and age-related brain tissue loss. MRS imaging (MRSI) measures the neuronal marker N-acetylaspartate (NAA), which could help assess brain change in AD and aging.To determine the effects of AD on concentrations of NAA, and choline- and creatine-containing compounds in different brain regions and to assess (...) the extent NAA in combination with volume measurements by MRI improves discrimination between AD patients and cognitively normal subjects.Fifty-six patients with AD (mean age: 75.6 +/- 8.0 years) and 54 cognitively normal subjects (mean age: 74.3 +/- 8.1 years) were studied using MRSI and MRI.NAA concentration was less in patients with AD compared with healthy subjects by 21% (p < 0.0001) in the medial temporal lobe and by 13% to 18% (p < 0.003) in parietal lobe gray matter (GM), but was not changed

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2002 Neurology

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