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Oral Allergy Syndrome

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1. Oral Allergy Syndrome in Birch Pollen-Sensitized Patients from a Korean University Hospital (Full text)

Oral Allergy Syndrome in Birch Pollen-Sensitized Patients from a Korean University Hospital Oral allergy syndrome (OAS) is a type of allergic reaction that mainly occurs on oral contact with raw fruit, vegetables, or nuts. The most common type of OAS is birch pollen-related food allergy. Although OAS is a common food allergy in adults, only few epidemiologic studies have been reported in Korea. Here we investigate the prevalence and triggers of birch pollen-related food allergy.We conducted (...) a retrospective chart review of 1,427 patients who underwent a skin prick test for inhalant allergens at the Asthma and Allergy Clinic in Seoul National University Bundang Hospital from January 2011 to December 2016.Of 1,427 patients, 125 (8.7%) were sensitized to birch pollen. Among them, 20.0% developed OAS, which was the most common food allergy (96.2%). The prevalence of OAS was higher in females, and was 18.2% in birch pollen-sensitized allergic rhinoconjunctivitis patients. Further, 72.0% OAS patients

2018 Journal of Korean medical science PubMed

2. Oral allergy syndrome. (PubMed)

Oral allergy syndrome. To review oral allergy syndrome (OAS).We searched several medical literature data bases with the following key words: "oral allergy syndrome," "OAS," "pollen-food allergy syndrome," "PFAS," "allergy," "diagnosis," "treatment."Oral allergy syndrome (OAS), also called "pollen-food allergy syndrome," is a type of food allergy brought about by flavors, nuts, raw fruit, and vegetables. The most well-known symptoms are mouth and throat itching, which starts rapidly after a food (...) is placed in the mouth, and that, as a rule, continues for just a couple of minutes after the food has been swallowed. The frequency of OAS with pollen allergy has been reported as 5-8%; 1-2% of patients with OAS with pollen allergy show extreme responses, e.g., anaphylaxis. Birch tree pollen, ragweed pollen, and grass pollen hypersensitivity cause the symptoms. The diagnosis of OAS is confirmed by a positive history and positive skin-prick test result triggered by the food's fresh extract. Oral

2018 American journal of rhinology & allergy

3. Shellfish/crustacean oral allergy syndrome among national service pre-enlistees in Singapore (Full text)

Shellfish/crustacean oral allergy syndrome among national service pre-enlistees in Singapore All Singaporean males undergo medical screening prior to compulsory military service. A history of possible food allergy may require referral to a specialist Allergy clinic to ensure that special dietary needs can be taken into account during field training and deployment.To study the pattern of food allergy among pre-enlistees who were referred to a specialist allergy clinic to work up suspected food (...) %). The most commonly reported foods were shellfish/crustaceans (78%), peanut (15.6%), and egg (6.5%). Self-limiting oral allergy syndrome, OAS (itchy lips and throat with/without lip angioedema) was the most common manifestation (n = 33, 42.9%) followed by anaphylaxis (n = 23, 29.9%). Majority of OAS was from shellfish/crustacean (90.6%); of which shrimp (30.3%), crab (15.2%), and lobster (3.0%) were the most common. Mild childhood asthma (69.7%), allergic rhinitis (6.3%), and eczema (6.1%) were the most

2018 Asia Pacific allergy PubMed

4. Allergy to apple cultivars among patients with birch pollinosis and oral allergy syndrome. (PubMed)

Allergy to apple cultivars among patients with birch pollinosis and oral allergy syndrome. Oral allergy syndrome (OAS) is caused by cross-reacting allergens found in pollen, raw fruits, vegetables, and some tree nuts. The major apple allergen, Mal d 1, is a cause of food allergic reactions in birch pollen sensitized patients.To explore the allergenicity of the most popular and commonly consumed apple cultivars in Poland in patients with birch pollen allergy with or without OAS.Data were (...) obtained from 46 adults with clinical symptoms of birch allergy and allergic rhinitis or rhinoconjunctivitis. Patients were divided into 2 groups according to the occurrence of OAS to apple. Skin prick tests (SPTs) were performed with pulp from the 11 most popular apple cultivars in Poland. Specific IgE (sIgE) to Bet v 1 was measured by radioallergosorbent test.Patients with OAS had more positive responses to apple SPT vs patients without OAS (odds ratios, 4.8-11.96). Patients with OAS had distinctive

2016 Asthma & Immunology

5. Birch allergy and oral allergy syndrome: The practical relevance of serum immunoglobulin E to Bet v 1. (PubMed)

Birch allergy and oral allergy syndrome: The practical relevance of serum immunoglobulin E to Bet v 1. Birch allergy (BA) may frequently be associated with fruit-vegetables oral allergy syndrome (OAS). Bet v 1 is the major birch allergen. Previously, it was reported that serum-specific immunoglobulin E (IgE) level could differentiate allergy from sensitization. Thus, this study aimed to investigate the practical role of Bet v 1 IgE.A total of 245 subjects (128 women, 117 men; mean age, 41 years

2016 Allergy and Asthma Proceedings

6. Galectin-1 inhibits oral-intestinal allergy syndrome (Full text)

Galectin-1 inhibits oral-intestinal allergy syndrome The pathogenesis of oral-intestinal allergy syndrome (OIAS) has not been well understood. Published data indicate that galectin (Gal) 1 has immune regulatory functions. This study tests a hypothesis that Gal1 inhibits oral-intestinal allergy syndrome.Mice were sensitized to peanut extracts (PE) via the buccal mucosa with or without using Gal1 together.Upon re-exposure to specific antigen, the OIAS mice showed the systemic allergic response (...) , the oral allergic reactions, and intestinal allergic inflammation, including increases in serum histamine, drop of the core temperature, higher levels of PE-specific IgE and interleukin (IL)-4. Increases in mast cell and eosinophil in the oral mucosa and intestinal mucosa were also observed. The OIAS was inhibited by co-administration with Gal1 via a mechanism of suppressing micro RNA (miR)-98 and reversing the expression of IL-10 in CD14+ cells in the intestine.The OIAS can be induced by applying

2017 Oncotarget PubMed

7. Oral Allergy Syndrome

Oral Allergy Syndrome Oral Allergy Syndrome Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Oral Allergy Syndrome Oral Allergy (...) Syndrome Aka: Oral Allergy Syndrome , Pollen-Food Allergy Syndrome From Related Chapters II. Epidemiology Most common III. Risk Factors or patients with grass, weed or tree pollen triggers (10%) Birch pollen allergy (highly associated) IV. Causes Birch pollen allergy Carrot Celery Hazelnuts Parsnips Potatoes Fresh fruit (apples, cherries, peaches, pears...) Grass pollen Kiwi Tomato Ragweed pollen Bananas Melons V. Symptoms Limited to oral exposure to trigger foods (see above) Immediate onset when

2018 FP Notebook

8. Oral and Sublingual Immunotherapy for Treatment of IgE-Mediated Food Allergy. (PubMed)

Oral and Sublingual Immunotherapy for Treatment of IgE-Mediated Food Allergy. Development of active therapies for IgE-mediated food allergy is a critical action step toward alleviating the adverse medical, psychosocial, and economic burdens on affected patients and families. Significant progress has been observed specifically in the application of single-allergen oral and sublingual immunotherapy for treatment of IgE-mediated food allergy, with emphasis on milk, egg, and peanut as the primary (...) (SLIT) has been utilized for the treatment of food allergy and pollen-food allergy syndrome, demonstrating moderate efficacy, a favorable safety profile and variable tolerability, with oropharyngeal symptoms most commonly observed. Although studies directly comparing OIT and SLIT are limited, in general, the favorable safety profile associated with SLIT comes at the expense of reduced efficacy, while the more robust clinical effects observed with OIT come at the risk of potentially intolerable

2018 Clinical Reviews in Allergy & Immunology

9. Assessment of food allergies and sensitivities

. 2002 Aug 31;360(9334):701-10. http://www.ncbi.nlm.nih.gov/pubmed/12241890?tool=bestpractice.com IgE-mediated food disorders include anaphylaxis and oral allergy syndrome. Cell-mediated reactions involve T cells, and may occur several hours to days after ingesting the offending food. Sicherer SH. Food allergy. Lancet. 2002 Aug 31;360(9334):701-10. http://www.ncbi.nlm.nih.gov/pubmed/12241890?tool=bestpractice.com Food protein-induced enterocolitis, enteropathy, proctocolitis, and coeliac disease (...) been confirmed as a trigger of bronchospasm in sulphite-sensitive asthmatic patients. Simon RA. Update on sulfite sensitivity. Allergy. 1998;53(46 Suppl):78-9. http://www.ncbi.nlm.nih.gov/pubmed/9826006?tool=bestpractice.com Differentials Oral allergy syndrome Atopic dermatitis Food protein-induced proctocolitis Coeliac disease Lactose intolerance Food-induced anaphylaxis Food-dependent exercise-induced anaphylaxis Eosinophilic oesophagitis Food protein-induced enterocolitis Congenital sucrase

2018 BMJ Best Practice

10. Quality of Life in Systemic Nickel Allergy Syndrome

provided by (Responsible Party): Eleonora Nucera, Catholic University of the Sacred Heart Study Details Study Description Go to Brief Summary: This study evaluates the effects of Nickel oral hyposensitization treatment (NiOHT) on health-related quality of life (HRQoL) of patients suffered from Systemic Nickel Allergy Syndrome (SNAS). Condition or disease Intervention/treatment Systemic Nickel Allergy Syndrome Quality of Life Biological: Nickel oral hyposensitization treatment Detailed Description (...) rhinitis, we hypothesized similar positive results even after oral Ni desensitization. Study Design Go to Layout table for study information Study Type : Observational Estimated Enrollment : 40 participants Observational Model: Cohort Time Perspective: Prospective Official Title: Quality of Life Assessment Before and After Hyposensitization Treatment in Systemic Nickel Allergy Syndrome Actual Study Start Date : March 2015 Estimated Primary Completion Date : April 30, 2019 Estimated Study Completion

2018 Clinical Trials

11. Perioperative Anaphylaxis Including Kounis Syndrome due to Selective Cefazolin Allergy. (PubMed)

2012 and December 2016. All patients were investigated according to the European Network for Drug Allergy (ENDA) recommendations through skin testing (major and minor penicillin determinants, penicillin, amoxicillin, cefazolin, cefuroxime and ceftriaxone) and oral challenges tests.We included 7 patients (median age 40 years) with perioperative anaphylactic reactions immediately after cefazolin injection, 4 with hypotension and 1 with Kounis syndrome (KS) type I. The presence of a selective IgE (...) Perioperative Anaphylaxis Including Kounis Syndrome due to Selective Cefazolin Allergy. Perioperative use of cefazolin has been associated with severe allergic reactions, and patients are usually labelled as allergic to penicillin afterwards. The aim of our study was to describe a group of patients with immediate reactions to cefazolin, with proven selective hypersensitivity reactions.Systematic review of all patients followed at our drug centre with cefazolin-related reactions, between January

2018 International Archives of Allergy and Immunology

12. Safety and Efficacy Study of Oral Fosfomycin Versus Oral Levofloxacin to Treat Complicated Urinary Syndromes (FOCUS)

Safety and Efficacy Study of Oral Fosfomycin Versus Oral Levofloxacin to Treat Complicated Urinary Syndromes (FOCUS) Safety and Efficacy Study of Oral Fosfomycin Versus Oral Levofloxacin to Treat Complicated Urinary Syndromes (FOCUS) - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number (...) of saved studies (100). Please remove one or more studies before adding more. Safety and Efficacy Study of Oral Fosfomycin Versus Oral Levofloxacin to Treat Complicated Urinary Syndromes (FOCUS) The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. of clinical studies and talk to your health care provider before participating. Read our for details

2018 Clinical Trials

13. Characterization of oral immune cells in birch pollen-allergic patients: impact of the Oral Allergy Syndrome and sublingual allergen immunotherapy on antigen presenting cells. (PubMed)

Characterization of oral immune cells in birch pollen-allergic patients: impact of the Oral Allergy Syndrome and sublingual allergen immunotherapy on antigen presenting cells. A detailed characterization of human oral immune cells is needed to better understand local mechanisms associated with allergen capture following oral exposure.Oral immune cells were characterized by immunohistology and immunofluorescence in biopsies obtained from three healthy individuals and 23 birch pollen-allergic (...) patients with/without oral allergy syndrome (OAS), at baseline and after 5 months of sublingual allergen immunotherapy (AIT).Similar cell subsets (i.e., dendritic cells, mast cells, and T lymphocytes) were detected in oral tissues from healthy and birch pollen-allergic individuals. CD207+ Langerhans cells (LCs) and CD11c+ myeloid dendritic cells (DCs) were found in both the epithelium and the papillary layer of the Lamina propria (LP), whereas CD68+ macrophages, CD117+ mast cells, and CD4+ /CD8+ T

2015 Allergy

14. Anaphylactic shock with methylprednisolone sodium succinate in a child with short bowel syndrome and cow’s milk allergy (Full text)

Anaphylactic shock with methylprednisolone sodium succinate in a child with short bowel syndrome and cow’s milk allergy Medications with methyl-prednisolone sodium succinate containing lactose, which potentially contains traces of cow's milk proteins (CMP), could cause allergic reactions or compromise treatment of acute allergic reactions in sensitized patients.We describe the unusual case of a one-year-old child affected by short bowel syndrome and history of severe cow's milk allergy (CMA (...) and symptom resolution occurred.Children who are highly sensitive to milk may have severe allergic reactions also after exposure to CMP through a different administration route than the oral one. Patients who have food allergies need to pay particular attention to the prescription of drugs and their formulation.

2017 Italian journal of pediatrics PubMed

15. CRACKCast E119 – Allergy, Hypersensitivity, Angioedema, and Anaphylaxis

CRACKCast E119 – Allergy, Hypersensitivity, Angioedema, and Anaphylaxis CRACKCast E119 – Allergy, Hypersensitivity, Angioedema, and Anaphylaxis - CanadiEM CRACKCast E119 – Allergy, Hypersensitivity, Angioedema, and Anaphylaxis In , by Chris Lipp October 19, 2017 This episode of CRACKCast covers Rosen’s Chapter 119 (109 in 9th), Allergy, Hypersensitivity, Angioedema, and Anaphylaxis. Patients suffering from acute allergic reactions, anaphylaxis, and angioedema are amongst the most unstable (...) patients presenting to the ED. With proper management, however, these patients can be efficiently managed and discharged with relative ease. Shownotes – Also check out EMCases Rapid Review video on Rosen’s in Perspective This chapter covers disease states in which the immune system overreacts against typically harmless agents. These hypersensitivity reactions are manifested in clinical symptoms ranging from mildly inconvenient to fatal. For practical purposes, let’s define some terms: Allergy Used

2017 CandiEM

16. Unmet diagnostic needs in contact oral mucosal allergies (Full text)

Unmet diagnostic needs in contact oral mucosal allergies The oral mucosa including the lips is constantly exposed to several noxious stimuli, irritants and allergens. However, oral contact pathologies are not frequently seen because of the relative resistance of the oral mucosa to irritant agents and allergens due to anatomical and physiological factors. The spectrum of signs and symptoms of oral contact allergies (OCA) is broad and a large number of condition can be the clinical expression (...) of OCA such as allergic contact stomatitis, allergic contact cheilitis, geographic tongue, oral lichenoid reactions, burning mouth syndrome. The main etiological factors causing OCA are dental materials, food and oral hygiene products, as they contain flavouring agents and preservatives. The personal medical history of the patient is helpful to perform a diagnosis, as a positive history for recent dental procedures. Sometimes histology is mandatory. When it cannot identify a direct cause

2016 Clinical and molecular allergy : CMA PubMed

17. Nickel oral hyposensitization in patients with systemic nickel allergy syndrome. (Full text)

Nickel oral hyposensitization in patients with systemic nickel allergy syndrome. This is the first randomized, double-blind, placebo-controlled trial (EUDRACT No. 2009-013923-43) evaluating nickel oral hyposensitizing treatment (NiOHT) in patients with "systemic nickel allergy syndrome" (SNAS), characterized by Ni-allergic contact dermatitis and systemic reactions after eating Ni-rich food.Adults with positive Ni-patch test, who reported symptoms suggesting SNAS, which improved after Ni-poor (...) diet, and were positive to Ni-oral challenge were eligible. Patients were randomly assigned to three treatments (1.5 μg, 0.3 μg, or 30 ng Ni/week) or placebo for a year, with progressive reintroduction of Ni-rich foods form the 5(th) month. Out of 141 patients randomized, 113 completed the trial. Endpoints were efficacy and tolerability of treatment.During Ni-rich food re-introduction, the 1.5 μg Ni/week group had a mean VAS score significantly higher than placebo (p = 0.044), with significant

2014 Annals of Medicine PubMed

18. Allergen cross-reactivity in allergic rhinitis and oral-allergy syndrome: a bioinformatic protein sequence analysis. (PubMed)

Allergen cross-reactivity in allergic rhinitis and oral-allergy syndrome: a bioinformatic protein sequence analysis. Clinical allergy cross-reactivity that is seen with related inhalant allergens or between unrelated inhalant allergens and foods in oral allergy syndrome (OAS) remains poorly understood. The goal of this study is to determine whether clinical cross-reactivity can be identified from primary protein sequences in allergy epitopes and food proteins.High-throughput analysis (...) was performed by assembling all known allergy epitopes within the Immune Epitope Database (IEDB; http://www.iedb.org) for 5 common species from 5 inhalant allergen subclasses and comparing their protein sequences to each other, as well as to sequences of intact proteins from known cross-reactive foods in the European Molecular Biology Laboratory-European Bioinformatics Institute (EMBL-EBI) protein database (http://www.uniprot.org) that have been implicated in OAS. Computational methods were employed

2014 International forum of allergy & rhinology

19. IgE, IgG4 and IgA specific for Bet v 1-related food allergens do not predict oral allergy syndrome. (Full text)

IgE, IgG4 and IgA specific for Bet v 1-related food allergens do not predict oral allergy syndrome. Birch pollen-associated plant food allergy is caused by Bet v 1-specific IgE, but presence of cross-reactive IgE to related allergens does not predict food allergy. The role of other immunoglobulin isotypes in the birch pollen-plant food syndrome has not been investigated in detail.Bet v 1-sensitized birch pollen-allergic patients (n = 35) were diagnosed for food allergy by standardized (...) interviews, skin prick tests, prick-to-prick tests and ImmunoCAP. Concentrations of allergen-specific IgE, IgG1, IgG4 and IgA to seven Bet v 1-related food allergens were determined by ELISA.Bet v 1, Cor a 1, Mal d 1 and Pru p 1 bound IgE from all and IgG4 and IgA from the majority of sera. Immunoglobulins to Gly m 4, Vig r 1 and Api g 1.01 were detected in <65% of the sera. No significant correlation was observed between plant food allergy and increased or reduced levels of IgE, IgG1, IgG4 or IgA

2014 Allergy PubMed

20. Nickel oral hyposensitization in patients with systemic nickel allergy syndrome. (Full text)

Nickel oral hyposensitization in patients with systemic nickel allergy syndrome. This is the first randomized, double-blind, placebo-controlled trial (EUDRACT No. 2009-013923-43) evaluating nickel oral hyposensitizing treatment (NiOHT) in patients with "systemic nickel allergy syndrome" (SNAS), characterized by Ni-allergic contact dermatitis and systemic reactions after eating Ni-rich food.Adults with positive Ni-patch test, who reported symptoms suggesting SNAS, which improved after Ni-poor (...) diet, and were positive to Ni-oral challenge were eligible. Patients were randomly assigned to three treatments (1.5 μg, 0.3 μg, or 30 ng Ni/week) or placebo for a year, with progressive reintroduction of Ni-rich foods form the 5(th) month. Out of 141 patients randomized, 113 completed the trial. Endpoints were efficacy and tolerability of treatment.During Ni-rich food re-introduction, the 1.5 μg Ni/week group had a mean VAS score significantly higher than placebo (p = 0.044), with significant

2014 Annals of medicine PubMed

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