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Neurogenic Shock

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41. Neurogenic Bladder (Overview)

Lesions Neurogenic bladder from spinal cord lesions may take various forms, depending on the mechanism and site of injury. Spinal cord trauma When an individual sustains a spinal cord injury (eg, from a diving accident or motor vehicle injury), the initial neurologic response is spinal shock. During this spinal shock phase, the affected individual experiences flaccid paralysis below the level of injury, and the somatic reflex activity is either depressed or absent. The anal and bulbocavernosus reflex (...) Neurogenic Bladder (Overview) Neurogenic Bladder: Overview, Neuroanatomy, Physiology and Pathophysiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNDUzNTM5LW92ZXJ2aWV3 processing > Neurogenic Bladder Updated

2014 eMedicine.com

42. Neurogenic Bladder (Follow-up)

Lesions Neurogenic bladder from spinal cord lesions may take various forms, depending on the mechanism and site of injury. Spinal cord trauma When an individual sustains a spinal cord injury (eg, from a diving accident or motor vehicle injury), the initial neurologic response is spinal shock. During this spinal shock phase, the affected individual experiences flaccid paralysis below the level of injury, and the somatic reflex activity is either depressed or absent. The anal and bulbocavernosus reflex (...) Neurogenic Bladder (Follow-up) Neurogenic Bladder: Overview, Neuroanatomy, Physiology and Pathophysiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNDUzNTM5LW92ZXJ2aWV3 processing > Neurogenic Bladder Updated

2014 eMedicine.com

43. A patient with refractory shock induced by several factors, including obstruction because of a posterior mediastinal hematoma. (Abstract)

fractures, right hemopneumothorax with pulmonary contusion, and minor liver injury. After infusing 5000 mL of lactated Ringer's solution and 10 units of blood, his circulation remained unstable. On a repeat CT examination, the left atrium was found to be compressed by a posterior mediastinal hematoma induced by the fracture of the thoracic spine, and a diagnosis of shock induced by multiple factors, including hemorrhagic, neurogenic, and obstructive mechanisms, was made. After obtaining stable (...) A patient with refractory shock induced by several factors, including obstruction because of a posterior mediastinal hematoma. A 44-year-old man who drove a motorcycle experienced a collision with the side of another motorcycle. Because he had sustained a high-energy injury to the spinal cord, he was transferred to our hospital. His circulation was unstable, and received tracheal intubation in addition to thoracostomy for the hemothorax. Whole-body computed tomography (CT) revealed multiple

2015 American Journal of Emergency Medicine

44. Neurogenic stunned myocardium as a manifestation of encephalitis involving cerebellar tonsils. (Abstract)

Neurogenic stunned myocardium as a manifestation of encephalitis involving cerebellar tonsils. Neurogenic stunned myocardium is defined as a myocardial injury or dysfunction after neurological insults. It is most commonly reported in patients with subarachnoid hemorrhage, and the presenting symptoms may mimic an acute myocardial infarction or myocarditis. In severe cases, cardiogenic shock and acute pulmonary edema may occur and lead to a devastating event. Therefore, it requires prompt (...) recognition and proper intervention. We herein report the case of a 25-year-old woman who presented to our hospital with the symptoms of acute pulmonary edema, shock, and consciousness disturbance. The diagnosis of encephalitis of cerebellar tonsils complicated with acute hydrocephalus and neurogenic stunned myocardium was made. Detailed neurologic examinations, neuroimaging studies, and characteristic echocardiographic changes expedite the correct diagnosis and treatment.

2012 American Journal of Emergency Medicine

45. Dopamine-induced neurogenic vaso-dilatation in the intact hindleg of the dog. Full Text available with Trip Pro

to atropine or propranolol and (iv) it was blocked by small doses of haloperidol. When hypovolemic shock was produced, the incidence of the neurogenic decrease of vascular resistance was smaller. 5 Dopamine also increased renal blood flow. This increase was not reduced by the occurrence of the neurogenic vasodilation in the inervated femoral artery. 6 These results are consistent with the idea that the dopamine-induced neurogenic vasodilation, originally described in the isolated perfused hindleg (...) Dopamine-induced neurogenic vaso-dilatation in the intact hindleg of the dog. 1 The dopamine-induced neurogenic vasodilation, previously described in the isolated perfused hindleg of the dog, has been studied in anaesthetized dogs with intact circulation in the hindleg. Dopamine was administered intravenously and/or intra-aortically, either as a bolus injection of 4 or 1l mug/kg, or as a continuous infusion of 4, 8, 16 or 32 mug kg-1 min-1. 2 Dopamine, given as a bolus injection or by infusion

1977 British journal of pharmacology

46. Tissue and plasma catecholamines and dopamine beta-hydroxylase activity of various animal species after neurogenic sympathetic stimulation. Full Text available with Trip Pro

Tissue and plasma catecholamines and dopamine beta-hydroxylase activity of various animal species after neurogenic sympathetic stimulation. 1. The effects of reserpine and exposure to a stressing atmosphere containing 20% CO2 for 5 hr on catecholamine (CA) levels and dopamine beta-hydroxylase (DBH) activity of adrenal, spleen and plasma of cat, rabbit, rat and guinea-pig were studied. 2. Twenty-four hr after an I.P. injection of reserpine, the CA contents of the adrenals and spleens were (...) activity to CA in the spleen and adrenal were much higher in the guinea-pig than in the rat. The fraction of the total DBH activity that can be solubilized by osmotic shock of purified adrenomedullary chromaffin granules was 28% in the rat and 71% in the guinea-pig. If one makes the assumption that 'soluble' and 'releasable' DBH may be equated, the amount of 'releasable' DBH into the circulation is much greater in the guinea-pig than in the rat. 7. The results suggest that the guinea-pig is a better

1978 The Journal of physiology

47. Shock, Distributive (Diagnosis)

in intensive care units (ICUs). (See , , , and .) Other causes of distributive shock include (SIRS) due to noninfectious inflammatory conditions such as burns and pancreatitis; ( ); anaphylaxis; reactions to drugs or toxins, including insect bites, transfusion reaction, and heavy metal poisoning; addisonian crisis; hepatic insufficiency; and neurogenic shock due to brain or spinal cord injury. (See and .) Types of shock Shock is a clinical syndrome characterized by inadequate tissue perfusion that results (...) SVR is due primarily to massive histamine release from mast cells after activation by antigen-bound immunoglobulin E (IgE), as well as increased synthesis and release of prostaglandins. Neurogenic shock is due to loss of sympathetic vascular tone from severe injury to the nervous system. Previous Next: Etiology The most common etiology of distributive shock is sepsis. Other causes include the following: SIRS due to noninfectious conditions such as pancreatitis, burns, or trauma TSS Anaphylaxis

2014 eMedicine.com

48. Shock, Distributive (Overview)

in intensive care units (ICUs). (See , , , and .) Other causes of distributive shock include (SIRS) due to noninfectious inflammatory conditions such as burns and pancreatitis; ( ); anaphylaxis; reactions to drugs or toxins, including insect bites, transfusion reaction, and heavy metal poisoning; addisonian crisis; hepatic insufficiency; and neurogenic shock due to brain or spinal cord injury. (See and .) Types of shock Shock is a clinical syndrome characterized by inadequate tissue perfusion that results (...) SVR is due primarily to massive histamine release from mast cells after activation by antigen-bound immunoglobulin E (IgE), as well as increased synthesis and release of prostaglandins. Neurogenic shock is due to loss of sympathetic vascular tone from severe injury to the nervous system. Previous Next: Etiology The most common etiology of distributive shock is sepsis. Other causes include the following: SIRS due to noninfectious conditions such as pancreatitis, burns, or trauma TSS Anaphylaxis

2014 eMedicine.com

49. Shock, Hemorrhagic (Diagnosis)

difficult because not all tissues and organs will experience the same amount of oxygen imbalance for a given clinical disturbance. Clinicians struggle daily to adequately define and monitor oxygen utilization on the cellular level and to correlate this physiology to useful clinical parameters and diagnostic tests. The 4 classes of shock, as proposed by Alfred Blalock, are as follows [ ] : Hypovolemic Vasogenic (septic) Cardiogenic Neurogenic Hypovolemic shock, the most common type, results from a loss (...) Shock, Hemorrhagic (Diagnosis) Hemorrhagic Shock: Background, Pathophysiology, Epidemiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNDMyNjUwLW92ZXJ2aWV3 processing > Hemorrhagic Shock Updated: Sep 12, 2018

2014 eMedicine Surgery

50. Shock, Hemorrhagic (Overview)

difficult because not all tissues and organs will experience the same amount of oxygen imbalance for a given clinical disturbance. Clinicians struggle daily to adequately define and monitor oxygen utilization on the cellular level and to correlate this physiology to useful clinical parameters and diagnostic tests. The 4 classes of shock, as proposed by Alfred Blalock, are as follows [ ] : Hypovolemic Vasogenic (septic) Cardiogenic Neurogenic Hypovolemic shock, the most common type, results from a loss (...) Shock, Hemorrhagic (Overview) Hemorrhagic Shock: Background, Pathophysiology, Epidemiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNDMyNjUwLW92ZXJ2aWV3 processing > Hemorrhagic Shock Updated: Sep 12, 2018

2014 eMedicine Surgery

51. Reversible cardiogenic shock due to catecholamine-induced cardiomyopathy: a variant of takotsubo? (Abstract)

Reversible cardiogenic shock due to catecholamine-induced cardiomyopathy: a variant of takotsubo? Catecholamine-induced cardiomyopathy, including takotsubo, neurogenic stunned myocardium, and pheochromocytoma-related cardiomyopathy, is a reversible and generally benign condition. We are reporting a case series of young women who had cardiogenic shock and pulmonary edema due to severe left ventricular systolic dysfunction, which completely recovered in the course of 2 to 3 weeks. Both patients

2013 American Journal of Emergency Medicine

52. Heparin Anticoagulation to Improve Outcomes in Septic Shock: The HALO Pilot

) Need for invasive mechanical ventilation or a P/F ratio < 250 Platelets < 100 x109/L, or a drop of 50 x109/L in the 3 days prior to enrollment Arterial pH < 7.30 or base deficit > 5 mmol/L in association with a lactate >/= to 3.0 mmol/L Exclusion Criteria: Consent declined Clinically apparent other forms of shock including cardiogenic, obstructive (massive pulmonary embolism, cardiac tamponnade, tension pneumothorax), hemorrhagic, neurogenic, or anaphylactic Received vasopressor therapy for greater (...) Heparin Anticoagulation to Improve Outcomes in Septic Shock: The HALO Pilot Heparin Anticoagulation to Improve Outcomes in Septic Shock: The HALO Pilot - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more

2012 Clinical Trials

53. Temperature variability analysis using wavelets and multiscale entropy in patients with systemic inflammatory response syndrome, sepsis, and septic shock Full Text available with Trip Pro

2), and seven had septic shock (group 3). All temperature curves were studied during the first 24 hours of an inflammatory state. A wavelet transformation was applied, decomposing the signal in different frequency components (scales) that have been found to reflect neurogenic and metabolic inputs on temperature oscillations. Wavelet energy and entropy per different scales associated with complexity in specific frequency bands and multiscale entropy of the whole signal were calculated. Moreover (...) Temperature variability analysis using wavelets and multiscale entropy in patients with systemic inflammatory response syndrome, sepsis, and septic shock Even though temperature is a continuous quantitative variable, its measurement has been considered a snapshot of a process, indicating whether a patient is febrile or afebrile. Recently, other diagnostic techniques have been proposed for the association between different properties of the temperature curve with severity of illness

2012 Critical Care

54. Critical care - Shock

beds; this bypass plus uncoupled cellular O 2 transport cause cellular hypoperfusion (shown by decreased O 2 consumption). In other situations, blood pools in venous capacitance beds and cardiac output falls. Distributive shock may be caused by (anaphylactic shock); bacterial infection with endotoxin release ( ); severe injury to the spinal cord, usually above T4 (neurogenic shock); and ingestion of certain drugs or poisons, such as nitrates, opioids, and adrenergic blockers. Anaphylactic shock (...) Critical care - Shock Shock - Critical Care Medicine - MSD Manual Professional Edition Brought to you by The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases / / / / IN THIS TOPIC OTHER TOPICS IN THIS CHAPTER Test your knowledge Cardiopulmonary Resuscitation

2013 Merck Manual (19th Edition)

55. Spinal shock

: Ditunno, JF; Little, JW; Tessler, A; Burns, AS (2004). "Spinal shock revisited: a four-phase model". Spinal cord : the official journal of the International Medical Society of Paraplegia 42 (7): 383–95. URL: Note: Differentiate spinal shock from neurogenic shock, which refers to the hemodynamic triad of hypotension, bradycardia, and peripheral vasodilation resulting from severe autonomic dysfunction. Neurogenic shock is due to the interruption of sympathetic nervous system control in acute spinal cord (...) Spinal shock Emergency Medicine Blog: Spinal shock Sunday, June 16, 2013 Spinal shock Spinal shock is not really "shock" in the sense of circulatory collapse but rather a transient or temporary (physiologic rather than anatomical) complete loss of all neurologic function, including reflexes and rectal tone, below a specific level that is associated with autonomic dysfunction. This phenomenon was first described in 1750 by Whytt. Spinal shock begins within a few minutes following an acute spinal

2013 Emergency Medicine Blog

57. Optimisation of RIZIV – INAMI lump sums for incontinence

management 59 2.7 MANAGEMENT OF FAECAL INCONTINENCE 62 2.7.1 Initial clinical assessment 62 2.7.2 Specialised management (primary or secondary care) 66 2.7.3 Surgery for faecal incontinence 66 KCE Report 304 Incontinence 3 2.7.4 Care pathway for the management of faecal incontinence in frail older women and men 69 2.8 MANAGEMENT OF NEUROLOGICAL INCONTINENCE 72 2.8.1 Management of neurogenic urinary incontinence 72 2.8.2 Management of faecal incontinence in neurological patients 76 3 REIMBURSEMENT (...) Figure 13 – ICS Conservative management of faecal incontinence 65 Figure 14 – ICS Surgical management of faecal incontinence 68 Figure 15 – ICS Management of faecal incontinence in frail older women and men 70 Figure 16 – ICS Initial management of neurogenic urinary incontinence 73 Figure 17 – ICS Specialised management of neurogenic urinary continence 75 Figure 18 – ICS Initial management of neurogenic faecal incontinence 78 Figure 19 – ICS Specialised management of neurogenic faecal incontinence 80

2020 Belgian Health Care Knowledge Centre

58. EAN guideline on palliative care of people with severe, progressive multiple sclerosis

, fatigue, pain and blad- der problems) were produced. For each symptom, a full report is available in Appendix S5 and summary of ?ndings tables in Appendix S6. Spasticity The majority of the included publications (32/44, 73%; 31 trials) addressed spasticity as an outcome. Of these, 26 trials (84%) addressed drugs (baclofen, ben- zodiazepines, cannabinoids, GABAergics, tizanidine and botulinum toxin) and ?ve trials addressed non- pharmacological treatments (exercise, radial shock wave therapy (...) in treatment decision-making. 2) Radial shock wave therapy (weak recommenda- tion/low certainty evidence) or transcutaneous electrical nerve stimulation (preferably 8-h appli- cation time; weak recommendation/very low cer- tainty evidence) are suggested to reduce pain in patients with severe MS. Patient preferences and treatment availability should be considered in treatment decision-making. Additional recommendations based on indirect evidence (weak recommendations/very low certainty evidence) 1

2020 European Academy of Neurology

59. Donor heart and lung procurement: A consensus statement Full Text available with Trip Pro

the procuring teams. Table 1 Causes of Death Affecting the Thoracic Organ Donor Cause Description Trauma Cardiac and pulmonary contusions, myocardial stunning, neurogenic pulmonary edema CVA Indication of possible hypertension in the donor, left ventricular hypertrophy, coronary artery disease, peripheral arterial disease Asphyxia Primary respiratory failure, if unwitnessed unknown downtime Drug intoxication Possible drug-induced problems. That is, cocaine, methamphetamines—coronary artery disease—indicates (...) donors are at risk of intravascular volume depletion to loss of vascular tone and increased capillary permeability. Diabetes insipidus (DI) and hyperglycemia may also compound further losses, increasing the risk for hypovolemic shock. Munshi L Murugan L Intensive care of the deceased multiorgan donor: one donor, nine lives. in: Subramanian K Sakai T Anesthesia and perioperative care for organ transplantation. Springer , New York 2017 : 51-61 In donors with decreased pre-load, crystalloid solutions

2020 International Society for Heart and Lung Transplantation

60. United Kingdom BASHH national guideline for the management of epididymo-orchitis

or neurogenic bladder following anoplasty should be considered [19,20]. • All patients with a confirmed urinary tract pathogen should have further investigations of the urinary tract [21,22]. • Ureaplasma urealyticum is found in men with epididymo-orchitis, often in association with N. gonorrhoeae or C trachomatis infection. Evidence supporting it as a common cause of epididymo-orchitis is lacking [5, 12, 23] • Mycoplasma genitalium is a slow-growing micro-organism, replicating intracellularly (...) sepsis and septic shock: 2012. Crit Care Med. 2013 Feb;41(2):580-637. doi: 10.1097/CCM.0b013e31827e83af. 94. Nwokolo NC, Dragovic B, Patel S, et al 2015 UK national guideline for the management of infection with Chlamydia trachomatis. Int J STD AIDS 2016;27(4) 251 –267. 95. Wind CM, Schim van der Loeff MF, Unemo M, et al. Test of Cure for Anogenital Gonorrhoea Using Modern RNA Based and DNA-Based Nucleic Acid Amplification Tests: A Prospective Cohort Study. Clin Infect Dis 2016;62(11):1348-55. doi

2020 British Association for Sexual Health and HIV

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