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Nephrogenic Diabetes Insipidus

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81. Congenital nephrogenic diabetes insipidus. (PubMed)

Congenital nephrogenic diabetes insipidus. 507907 1980 01 19 2018 11 13 1468-2044 54 10 1979 Oct Archives of disease in childhood Arch. Dis. Child. Congenital nephrogenic diabetes insipidus. 807 Koepp P P eng Case Reports Letter England Arch Dis Child 0372434 0003-9888 AIM IM Child, Preschool Diabetes Insipidus congenital physiopathology Humans Infant Kidney Concentrating Ability Male 1979 10 1 1979 10 1 0 1 1979 10 1 0 0 ppublish 507907 PMC1545653 Klin Wochenschr. 1965 Jan 15;43:72-8 14309735

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1979 Archives of Disease in Childhood

82. Persistent nephrogenic diabetes insipidus associated with long-term lithium carbonate treatment. (PubMed)

Persistent nephrogenic diabetes insipidus associated with long-term lithium carbonate treatment. 519597 1980 03 27 2018 11 13 0008-4409 121 2 1979 Jul 21 Canadian Medical Association journal Can Med Assoc J Persistent nephrogenic diabetes insipidus associated with long-term lithium carbonate treatment. 194-8 Rabin E Z EZ Garston R G RG Weir R V RV Posen G A GA eng Case Reports Journal Article Canada Can Med Assoc J 0414110 0008-4409 9FN79X2M3F Lithium AIM IM Adult Diabetes Insipidus chemically (...) induced physiopathology Diabetic Nephropathies chemically induced physiopathology Female Humans Kidney Concentrating Ability drug effects Lithium adverse effects pharmacology Polyuria chemically induced Time Factors 1979 7 21 1979 7 21 0 1 1979 7 21 0 0 ppublish 519597 PMC1704305 Psychopharmacologia. 1959;1:65-78 14443504 Ann Intern Med. 1978 Apr;88(4):576-7 637444 Kidney Int. 1977 Sep;12(3):205-13 926612 Ann Intern Med. 1977 Apr;86(4):446-7 848808 J Clin Pathol. 1975 Jun;28(6):472-5 1141447 Am J Med

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1979 Canadian Medical Association Journal

83. Inheritance of nephrogenic diabetes insipidus (PubMed)

Inheritance of nephrogenic diabetes insipidus 13197364 2003 05 01 2018 12 01 0002-9297 6 3 1954 Sep American journal of human genetics Am. J. Hum. Genet. Inheritance of nephrogenic diabetes insipidus. 354-8 WALKER N F NF RANCE C P CP eng Journal Article United States Am J Hum Genet 0370475 0002-9297 OM Databases, Genetic Diabetes Insipidus etiology Diabetes Insipidus, Nephrogenic Heredity Humans 5527:8998:137:216 DIABETES INSIPIDUS/etiology and pathogenesis HEREDITY 1954 9 1 1954 9 1 0 1 1954 9

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1954 American Journal of Human Genetics

84. A Case of Nephrogenic Diabetes Insipidus (PubMed)

A Case of Nephrogenic Diabetes Insipidus 13572903 2000 07 01 2018 12 01 0007-1447 2 5099 1958 Sep 27 British medical journal Br Med J A case of nephrogenic diabetes insipidus. 780-1 GLASER L H LH eng Case Reports Journal Article England Br Med J 0372673 0007-1447 OM Diabetes Insipidus Diabetes Insipidus, Nephrogenic Diabetes Insipidus, Neurogenic Humans Medical Records 5935:2233:172 DIABETES INSIPIDUS/case reports 1958 9 27 1958 9 27 0 1 1958 9 27 0 0 ppublish 13572903 PMC2026293 Lancet. 1956

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1958 British medical journal

85. Nephrogenic Diabetes Insipidus (PubMed)

Nephrogenic Diabetes Insipidus 20789865 2011 04 04 2011 04 04 0007-1447 1 5346 1963 Jun 22 British medical journal Br Med J Nephrogenic Diabetes Insipidus. 1630 eng Journal Article England Br Med J 0372673 0007-1447 2010 8 27 6 0 1963 6 22 0 0 1963 6 22 0 1 ppublish 20789865 PMC2124392

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1963 British medical journal

86. Nephrogenic diabetes insipidus: absence of close linkage with Xg. (PubMed)

Nephrogenic diabetes insipidus: absence of close linkage with Xg. 5309332 1970 05 05 2018 11 13 0002-9297 22 2 1970 Mar American journal of human genetics Am. J. Hum. Genet. Nephrogenic diabetes insipidus: absence of close linkage with Xg. 221-7 Bode H H HH Miettinen O S OS eng Journal Article United States Am J Hum Genet 0370475 0002-9297 0 Isoantigens 11000-17-2 Vasopressins AIM IM Chromosome Mapping Color Vision Defects Diabetes Insipidus genetics Genotype Humans Isoantigens Kidney Tubules

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1970 American Journal of Human Genetics

87. Failure to detect the carrier in congenital nephrogenic diabetes insipidus. (PubMed)

Failure to detect the carrier in congenital nephrogenic diabetes insipidus. 5018633 1972 06 22 2018 11 13 1468-2044 47 251 1972 Feb Archives of disease in childhood Arch. Dis. Child. Failure to detect the carrier in congenital nephrogenic diabetes insipidus. 137-9 Uttley W S WS Thistlethwaite D D eng Journal Article England Arch Dis Child 0372434 0003-9888 IM Adult Diabetes Insipidus congenital genetics urine Female Genotype Humans Infant, Newborn Male Middle Aged Osmolar Concentration Pedigree

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1972 Archives of Disease in Childhood

88. Nephrogenic Diabetes Insipidus: Effects of 3,5, Cyclic-adenosine Monophosphate (PubMed)

Nephrogenic Diabetes Insipidus: Effects of 3,5, Cyclic-adenosine Monophosphate 4343784 1973 01 18 2018 11 13 1468-2044 47 255 1972 Oct Archives of disease in childhood Arch. Dis. Child. Nephrogenic diabetes insipidus. Effects of 3,5, cyclic-adenosine monophosphate. 794-7 Jones N F NF Barraclough M A MA Barnes N N Cottom D G DG eng Journal Article England Arch Dis Child 0372434 0003-9888 11000-17-2 Vasopressins 8W8T17847W Urea 9NEZ333N27 Sodium AYI8EX34EU Creatinine E0399OZS9N Cyclic AMP IM (...) Adolescent Blood Pressure drug effects Child Creatinine urine Cyclic AMP administration & dosage therapeutic use Diabetes Insipidus drug therapy Glomerular Filtration Rate Humans Infant Injections, Intravenous Kidney Tubules abnormalities Osmolar Concentration Sodium urine Urea urine Urine Vasopressins therapeutic use 1972 10 1 1972 10 1 0 1 1972 10 1 0 0 ppublish 4343784 PMC1648252 Am J Physiol. 1966 Jul;211(1):255-9 5911047 Am J Physiol. 1967 Apr;212(4):939-44 6024462 Clin Sci. 1968 Apr;34(2):253-60

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1972 Archives of Disease in Childhood

89. Lithium-induced nephrogenic diabetes insipidus: in vivo and in vitro studies (PubMed)

Lithium-induced nephrogenic diabetes insipidus: in vivo and in vitro studies The physiological basis for the polyuria and polydipsia occurring in some manic-depressive patients treated with lithium salts was studied in vivo and in vitro. Three lithium-treated polyuric patients, in whom other causes of a concentrating defect were excluded, had abnormal urinary concentrating abilities after a standard water depreviation test. Two of these patients failed to respond to exogenous vasopressin (ADH (...) for the diabetes insipidus syndrome manifested by these three patients. The defect in water transport was further characterized in toad urinary bladders in vitro. Short-circuit current (I) and water flow (W) were studied under basal, ADH-stimulated, and cyclic adenosine 3',5'-monophosphate (c-AMP)-stimulated conditions. Increasing mucosal [Li(+)] progressively inhibited basal I, and both I and W induced by ADH. Significant inhibition of basal and ADH-induced I was observed at mucosal [Li(+)] < 1.1 mEq/liter

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1972 Journal of Clinical Investigation

90. The water deprivation test and a potential role for the arginine vasopressin precursor copeptin to differentiate diabetes insipidus from primary polydipsia (PubMed)

The water deprivation test and a potential role for the arginine vasopressin precursor copeptin to differentiate diabetes insipidus from primary polydipsia The water deprivation test is the gold standard test to differentiate central or nephrogenic diabetes insipidus (DI) from primary polydipsia (PP) in patients with polyuria and polydipsia. Few studies have addressed the diagnostic performance of this test. The aim of this retrospective cohort study was to evaluate the diagnostic performance (...) of the standard water deprivation test, including plasma arginine vasopressin (AVP) measurements, in 40 consecutive patients with polyuria. We compared initial test results with the final clinical diagnosis, i.e., no DI, central DI, or nephrogenic DI. The median length of follow-up was 8 years. In a subset of ten patients, the novel marker copeptin (CP) was measured in plasma. Using the final diagnosis as a gold standard, a threshold for urine osmolality of >800 mOsmol/kg after water deprivation yielded

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2015 Endocrine connections

91. Diabetes Insipidus (Diagnosis)

and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266-77. . Faerch M, Christensen JH, Corydon TJ, et al. Partial nephrogenic diabetes insipidus caused (...) by a novel mutation in the AVPR2 gene. Clin Endocrinol (Oxf) . 2008 Mar. 68(3):395-403. . Mulders SM, Bichet DG, Rijss JP, et al. An aquaporin-2 water channel mutant which causes autosomal dominant nephrogenic diabetes insipidus is retained in the Golgi complex. J Clin Invest . 1998 Jul 1. 102(1):57-66. . . Chandrasekaran K, Karolina DS, Sepramaniam S, Armugam A, Wintour EM, Bertram JF, et al. Role of microRNAs in kidney homeostasis and disease. Kidney Int . 2012 Apr. 81(7):617-27. . Agre P, King LS

2014 eMedicine Pediatrics

92. Diabetes Insipidus (Diagnosis)

on the US market) Thiazides Nonsteroidal anti-inflammatory drugs (NSAIDs), such as indomethacin (may be used in nephrogenic DI, but only when no better options exist) See and for more detail. Next: Background Diabetes insipidus (DI) is defined as the passage of large volumes (>3 L/24 h) of dilute urine (< 300m Osm/kg). DI has 2 major forms: central and nephrogenic. Central DI is characterized by decreased secretion of antidiuretic hormone (ADH)—also known as arginine vasopressin (AVP)—which gives rise (...) mellitus do not induce thirst; rather, the increased thirst in uncontrolled diabetes mellitus is secondary to volume depletion from osmotic diuresis. Previous Next: Etiology DI is usually an acquired disorder, with central DI having different causes than does nephrogenic DI. In rare cases, central or nephrogenic DI may be an inherited disorder. Central diabetes insipidus Central DI has many possible causes. According to the literature, the principal causes of central DI and their oft-cited approximate

2014 eMedicine.com

93. Diabetes Insipidus (Overview)

on the US market) Thiazides Nonsteroidal anti-inflammatory drugs (NSAIDs), such as indomethacin (may be used in nephrogenic DI, but only when no better options exist) See and for more detail. Next: Background Diabetes insipidus (DI) is defined as the passage of large volumes (>3 L/24 h) of dilute urine (< 300m Osm/kg). DI has 2 major forms: central and nephrogenic. Central DI is characterized by decreased secretion of antidiuretic hormone (ADH)—also known as arginine vasopressin (AVP)—which gives rise (...) mellitus do not induce thirst; rather, the increased thirst in uncontrolled diabetes mellitus is secondary to volume depletion from osmotic diuresis. Previous Next: Etiology DI is usually an acquired disorder, with central DI having different causes than does nephrogenic DI. In rare cases, central or nephrogenic DI may be an inherited disorder. Central diabetes insipidus Central DI has many possible causes. According to the literature, the principal causes of central DI and their oft-cited approximate

2014 eMedicine.com

94. Diabetes Insipidus (Treatment)

References Earley LE, Orloff J. The mechanism of antidiuresis associated with the administration of hydrochlorothiazide to patients with vasopressin-resistant diabetes insipidus. J Clin Invest. Nov 1962;41(11):1988-97. Babey M, Kopp P, Robertson GL. Familial forms of diabetes insipidus: clinical and molecular characteristics. Nat Rev Endocrinol . 2011 Jul 5. 7(12):701-14. . Bockenhauer D, van't Hoff W, Dattani M, Lehnhardt A, Subtirelu M, Hildebrandt F, et al. Secondary nephrogenic diabetes insipidus (...) as a complication of inherited renal diseases. Nephron Physiol . 2010. 116(4):p23-9. . Los EL, Deen PM, Robben JH. Potential of nonpeptide (ant)agonists to rescue vasopressin V2 receptor mutants for the treatment of X-linked nephrogenic diabetes insipidus. J Neuroendocrinol . 2010 May. 22(5):393-9. . Rochdi MD, Vargas GA, Carpentier E, Oligny-Longpré G, et al. Functional characterization of vasopressin type 2 receptor substitutions (R137H/C/L) leading to nephrogenic diabetes insipidus and nephrogenic syndrome

2014 eMedicine.com

95. Diabetes Insipidus (Follow-up)

References Earley LE, Orloff J. The mechanism of antidiuresis associated with the administration of hydrochlorothiazide to patients with vasopressin-resistant diabetes insipidus. J Clin Invest. Nov 1962;41(11):1988-97. Babey M, Kopp P, Robertson GL. Familial forms of diabetes insipidus: clinical and molecular characteristics. Nat Rev Endocrinol . 2011 Jul 5. 7(12):701-14. . Bockenhauer D, van't Hoff W, Dattani M, Lehnhardt A, Subtirelu M, Hildebrandt F, et al. Secondary nephrogenic diabetes insipidus (...) as a complication of inherited renal diseases. Nephron Physiol . 2010. 116(4):p23-9. . Los EL, Deen PM, Robben JH. Potential of nonpeptide (ant)agonists to rescue vasopressin V2 receptor mutants for the treatment of X-linked nephrogenic diabetes insipidus. J Neuroendocrinol . 2010 May. 22(5):393-9. . Rochdi MD, Vargas GA, Carpentier E, Oligny-Longpré G, et al. Functional characterization of vasopressin type 2 receptor substitutions (R137H/C/L) leading to nephrogenic diabetes insipidus and nephrogenic syndrome

2014 eMedicine.com

96. Diabetes Insipidus (Treatment)

Endocrinologist - the presence of central DI should prompt an evaluation of anterior pituitary function Diagnostic radiologist Regular follow-up visits with an endocrinologist (for central DI) or a nephrologist (for nephrogenic DI) are necessary for dosage adjustment. When indomethacin is used in long-term therapy, carefully observe renal function for any signs of toxicity. Previous References Central diabetes insipidus. Orphannet. Available at . July 2012; Accessed: September 15, 2017. Di Iorgi N, Allegri AE (...) , Napoli F, Calcagno A, Calandra E, Fratangeli N, et al. Central diabetes insipidus in children and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266

2014 eMedicine Pediatrics

97. Diabetes Insipidus (Overview)

and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266-77. . Faerch M, Christensen JH, Corydon TJ, et al. Partial nephrogenic diabetes insipidus caused (...) by a novel mutation in the AVPR2 gene. Clin Endocrinol (Oxf) . 2008 Mar. 68(3):395-403. . Mulders SM, Bichet DG, Rijss JP, et al. An aquaporin-2 water channel mutant which causes autosomal dominant nephrogenic diabetes insipidus is retained in the Golgi complex. J Clin Invest . 1998 Jul 1. 102(1):57-66. . . Chandrasekaran K, Karolina DS, Sepramaniam S, Armugam A, Wintour EM, Bertram JF, et al. Role of microRNAs in kidney homeostasis and disease. Kidney Int . 2012 Apr. 81(7):617-27. . Agre P, King LS

2014 eMedicine Pediatrics

98. Diabetes Insipidus (Follow-up)

Endocrinologist - the presence of central DI should prompt an evaluation of anterior pituitary function Diagnostic radiologist Regular follow-up visits with an endocrinologist (for central DI) or a nephrologist (for nephrogenic DI) are necessary for dosage adjustment. When indomethacin is used in long-term therapy, carefully observe renal function for any signs of toxicity. Previous References Central diabetes insipidus. Orphannet. Available at . July 2012; Accessed: September 15, 2017. Di Iorgi N, Allegri AE (...) , Napoli F, Calcagno A, Calandra E, Fratangeli N, et al. Central diabetes insipidus in children and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266

2014 eMedicine Pediatrics

99. Why Do Thiazides Decrease Polyuria in Diabetes Insipidus?

Why Do Thiazides Decrease Polyuria in Diabetes Insipidus? Renal Fellow Network: Why Do Thiazides Decrease Polyuria in Diabetes Insipidus? | | | | | Tuesday, September 4, 2012 Why Do Thiazides Decrease Polyuria in Diabetes Insipidus? I was reviewing the treatment of diabetes insipidus the other day, and was reminded of the paradoxical effect of thiazide diuretics on urine output in diabetes insipidus. How does this work? The traditional thinking is that thiazide-induced blockade of the Na-Cl (...) explain how a thiazide can serve to decrease polyuria in patients with diabetes insipidus. Posted by lisajcohen at Labels: , 4 comments: said... There are also studies that show thiazides DO decrease plasma volume on the long-run. So this is still controversial. Actually, research in mice shows that this is the mechanism for thiazide-induced hypocalciuria. said... I think, that the problem with Thiazides use in treating Diabetes Indipidus(DI or NDI) is that you loose more H2O(water) than Na+(sodium

2012 Renal Fellow Network

100. Nephrogenic Diabetes Insipidus

Nephrogenic Diabetes Insipidus Nephrogenic Diabetes Insipidus - Genitourinary Disorders - MSD Manual Professional Edition Brought to you by The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases / / / / IN THIS TOPIC OTHER TOPICS IN THIS CHAPTER Test your (...) Catheterization in Males SOCIAL MEDIA Add to Any Platform Loading , MD, Texas Tech University Click here for Patient Education NOTE: This is the Professional Version. CONSUMERS: Nephrogenic diabetes insipidus (NDI) is an inability to concentrate urine due to impaired renal tubule response to vasopressin (ADH), which leads to excretion of large amounts of dilute urine. It can be inherited or occur secondary to conditions that impair renal concentrating ability. Symptoms and signs include polyuria and those

2013 Merck Manual (19th Edition)

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