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Nephrogenic Diabetes Insipidus

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81. Congenital nephrogenic diabetes insipidus. Full Text available with Trip Pro

Congenital nephrogenic diabetes insipidus. 507907 1980 01 19 2018 11 13 1468-2044 54 10 1979 Oct Archives of disease in childhood Arch. Dis. Child. Congenital nephrogenic diabetes insipidus. 807 Koepp P P eng Case Reports Letter England Arch Dis Child 0372434 0003-9888 AIM IM Child, Preschool Diabetes Insipidus congenital physiopathology Humans Infant Kidney Concentrating Ability Male 1979 10 1 1979 10 1 0 1 1979 10 1 0 0 ppublish 507907 PMC1545653 Klin Wochenschr. 1965 Jan 15;43:72-8 14309735

1979 Archives of Disease in Childhood

82. Persistent nephrogenic diabetes insipidus associated with long-term lithium carbonate treatment. Full Text available with Trip Pro

Persistent nephrogenic diabetes insipidus associated with long-term lithium carbonate treatment. 519597 1980 03 27 2018 11 13 0008-4409 121 2 1979 Jul 21 Canadian Medical Association journal Can Med Assoc J Persistent nephrogenic diabetes insipidus associated with long-term lithium carbonate treatment. 194-8 Rabin E Z EZ Garston R G RG Weir R V RV Posen G A GA eng Case Reports Journal Article Canada Can Med Assoc J 0414110 0008-4409 9FN79X2M3F Lithium AIM IM Adult Diabetes Insipidus chemically (...) induced physiopathology Diabetic Nephropathies chemically induced physiopathology Female Humans Kidney Concentrating Ability drug effects Lithium adverse effects pharmacology Polyuria chemically induced Time Factors 1979 7 21 1979 7 21 0 1 1979 7 21 0 0 ppublish 519597 PMC1704305 Psychopharmacologia. 1959;1:65-78 14443504 Ann Intern Med. 1978 Apr;88(4):576-7 637444 Kidney Int. 1977 Sep;12(3):205-13 926612 Ann Intern Med. 1977 Apr;86(4):446-7 848808 J Clin Pathol. 1975 Jun;28(6):472-5 1141447 Am J Med

1979 Canadian Medical Association Journal

83. Congenital nephrogenic diabetes insipidus in a baby girl. Full Text available with Trip Pro

Congenital nephrogenic diabetes insipidus in a baby girl. A 6-week-old girl with fever, hypernatraemia, dehydration, and polyuria failed to concentrate urine in response to exogenous vasopressin administration. There was no family history of nephrogenic diabetes insipidus. When she was 15 months old, the infusion of vasopressin did not produce an increase in urinary cyclic-AMP.

1978 Archives of Disease in Childhood

84. Nephrogenic diabetes insipidus and Werdnig-Hoffmann disease in a child: an unusual association. Full Text available with Trip Pro

Nephrogenic diabetes insipidus and Werdnig-Hoffmann disease in a child: an unusual association. The unusual association of Werdnig-Hoffmann disease and nephrogenic diabetes insipidus in a 5-month-old child is described for the first time. The association is casual, considering the different pathways of genetical transmission in these two diseases. The possibility of identifying the heterozygote is discussed and it appears to be limited to nephrogenic diabetes insipidus.

1978 Journal of Medical Genetics

85. Nephrogenic diabetes insipidus induced by demethylchlortetracycline (Declomycin). Full Text available with Trip Pro

Nephrogenic diabetes insipidus induced by demethylchlortetracycline (Declomycin). 4966354 1968 04 27 2018 11 13 0008-1264 107 5 1967 Nov California medicine Calif Med Nephrogenic diabetes insipidus induced by demethylchlortetracycline (Declomycin). 420-2 Torin D E DE eng Case Reports Journal Article United States Calif Med 0410260 0008-1264 5R5W9ICI6O Demeclocycline IM Adult Demeclocycline adverse effects Diabetes Insipidus chemically induced Female Humans Kidney Diseases chemically induced

1967 California Medicine

86. Studies with angiotensin in nephrogenic diabetes insipidus. Full Text available with Trip Pro

Studies with angiotensin in nephrogenic diabetes insipidus. 4293073 1967 12 15 2018 11 13 0008-4409 97 14 1967 Sep 30 Canadian Medical Association journal Can Med Assoc J Studies with angiotensin in nephrogenic diabetes insipidus. 841-5 Orr F R FR Filipich R L RL eng Case Reports Comparative Study Journal Article Canada Can Med Assoc J 0414110 0008-4409 11000-17-2 Vasopressins 11128-99-7 Angiotensin II 9NEZ333N27 Sodium AYI8EX34EU Creatinine RWP5GA015D Potassium AIM IM Adult Angiotensin II (...) therapeutic use Blood Pressure drug effects Creatinine Diabetes Insipidus drug therapy genetics Female Humans Kidney Failure, Chronic genetics Kidney Function Tests Male Potassium urine Sodium urine Vasopressins therapeutic use 1967 9 30 1967 9 30 0 1 1967 9 30 0 0 ppublish 4293073 PMC1923438 Lancet. 1965 Nov 13;2(7420):987-8 4159217 Can Med Assoc J. 1961 Feb 25;84:403-19 13704363 J Clin Invest. 1955 Sep;34(9):1410-6 13252088 J Clin Invest. 1965 Jul;44:1171-86 14328394 Proc R Soc Med. 1965 Dec;58(12):1005

1967 Canadian Medical Association Journal

87. Nephrogenic Diabetes Insipidus Full Text available with Trip Pro

Nephrogenic Diabetes Insipidus 20789865 2011 04 04 2011 04 04 0007-1447 1 5346 1963 Jun 22 British medical journal Br Med J Nephrogenic Diabetes Insipidus. 1630 eng Journal Article England Br Med J 0372673 0007-1447 2010 8 27 6 0 1963 6 22 0 0 1963 6 22 0 1 ppublish 20789865 PMC2124392

1963 British medical journal

88. Overview of diabetes

and/or target-tissue insulin resistance. Complications of diabetes mellitus include both macrovascular (cardiovascular) and microvascular (retinopathy, nephropathy, or neuropathy) sequelae. Diabetes insipidus (DI) is much less common and refers to disorders of vasopressin secretion (central DI) or action (nephrogenic DI), resulting in urinary concentrating abnormality. Related conditions Condition Description Common disorder characterised by insulin resistance and relative insulin deficiency. Most patients (...) syndrome: time for a critical appraisal: joint statement from the American Diabetes Association and the European Association for the Study of Diabetes. Diabetes Care. 2005 Sep;28(9):2289-304. http://care.diabetesjournals.org/content/28/9/2289.full http://www.ncbi.nlm.nih.gov/pubmed/16123508?tool=bestpractice.com Characterised by polydipsia, polyuria, increased thirst, and hypotonic urine. Central DI is due to defective synthesis or release of arginine vasopressin (AVP). Nephrogenic DI is due to renal

2018 BMJ Best Practice

89. Overview of diabetes

and/or target-tissue insulin resistance. Complications of diabetes mellitus include both macrovascular (cardiovascular) and microvascular (retinopathy, nephropathy, or neuropathy) sequelae. Diabetes insipidus (DI) is much less common and refers to disorders of vasopressin secretion (central DI) or action (nephrogenic DI), resulting in urinary concentrating abnormality. Related conditions Condition Description Common disorder characterised by insulin resistance and relative insulin deficiency. Most patients (...) syndrome: time for a critical appraisal: joint statement from the American Diabetes Association and the European Association for the Study of Diabetes. Diabetes Care. 2005 Sep;28(9):2289-304. http://care.diabetesjournals.org/content/28/9/2289.full http://www.ncbi.nlm.nih.gov/pubmed/16123508?tool=bestpractice.com Characterised by polydipsia, polyuria, increased thirst, and hypotonic urine. Central DI is due to defective synthesis or release of arginine vasopressin (AVP). Nephrogenic DI is due to renal

2018 BMJ Best Practice

90. The water deprivation test and a potential role for the arginine vasopressin precursor copeptin to differentiate diabetes insipidus from primary polydipsia Full Text available with Trip Pro

The water deprivation test and a potential role for the arginine vasopressin precursor copeptin to differentiate diabetes insipidus from primary polydipsia The water deprivation test is the gold standard test to differentiate central or nephrogenic diabetes insipidus (DI) from primary polydipsia (PP) in patients with polyuria and polydipsia. Few studies have addressed the diagnostic performance of this test. The aim of this retrospective cohort study was to evaluate the diagnostic performance (...) of the standard water deprivation test, including plasma arginine vasopressin (AVP) measurements, in 40 consecutive patients with polyuria. We compared initial test results with the final clinical diagnosis, i.e., no DI, central DI, or nephrogenic DI. The median length of follow-up was 8 years. In a subset of ten patients, the novel marker copeptin (CP) was measured in plasma. Using the final diagnosis as a gold standard, a threshold for urine osmolality of >800 mOsmol/kg after water deprivation yielded

2015 Endocrine connections

91. Diabetes Insipidus (Treatment)

References Earley LE, Orloff J. The mechanism of antidiuresis associated with the administration of hydrochlorothiazide to patients with vasopressin-resistant diabetes insipidus. J Clin Invest. Nov 1962;41(11):1988-97. Babey M, Kopp P, Robertson GL. Familial forms of diabetes insipidus: clinical and molecular characteristics. Nat Rev Endocrinol . 2011 Jul 5. 7(12):701-14. . Bockenhauer D, van't Hoff W, Dattani M, Lehnhardt A, Subtirelu M, Hildebrandt F, et al. Secondary nephrogenic diabetes insipidus (...) as a complication of inherited renal diseases. Nephron Physiol . 2010. 116(4):p23-9. . Los EL, Deen PM, Robben JH. Potential of nonpeptide (ant)agonists to rescue vasopressin V2 receptor mutants for the treatment of X-linked nephrogenic diabetes insipidus. J Neuroendocrinol . 2010 May. 22(5):393-9. . Rochdi MD, Vargas GA, Carpentier E, Oligny-Longpré G, et al. Functional characterization of vasopressin type 2 receptor substitutions (R137H/C/L) leading to nephrogenic diabetes insipidus and nephrogenic syndrome

2014 eMedicine.com

92. Diabetes Insipidus (Overview)

on the US market) Thiazides Nonsteroidal anti-inflammatory drugs (NSAIDs), such as indomethacin (may be used in nephrogenic DI, but only when no better options exist) See and for more detail. Next: Background Diabetes insipidus (DI) is defined as the passage of large volumes (>3 L/24 h) of dilute urine (< 300m Osm/kg). DI has 2 major forms: central and nephrogenic. Central DI is characterized by decreased secretion of antidiuretic hormone (ADH)—also known as arginine vasopressin (AVP)—which gives rise (...) mellitus do not induce thirst; rather, the increased thirst in uncontrolled diabetes mellitus is secondary to volume depletion from osmotic diuresis. Previous Next: Etiology DI is usually an acquired disorder, with central DI having different causes than does nephrogenic DI. In rare cases, central or nephrogenic DI may be an inherited disorder. Central diabetes insipidus Central DI has many possible causes. According to the literature, the principal causes of central DI and their oft-cited approximate

2014 eMedicine.com

93. Diabetes Insipidus (Follow-up)

References Earley LE, Orloff J. The mechanism of antidiuresis associated with the administration of hydrochlorothiazide to patients with vasopressin-resistant diabetes insipidus. J Clin Invest. Nov 1962;41(11):1988-97. Babey M, Kopp P, Robertson GL. Familial forms of diabetes insipidus: clinical and molecular characteristics. Nat Rev Endocrinol . 2011 Jul 5. 7(12):701-14. . Bockenhauer D, van't Hoff W, Dattani M, Lehnhardt A, Subtirelu M, Hildebrandt F, et al. Secondary nephrogenic diabetes insipidus (...) as a complication of inherited renal diseases. Nephron Physiol . 2010. 116(4):p23-9. . Los EL, Deen PM, Robben JH. Potential of nonpeptide (ant)agonists to rescue vasopressin V2 receptor mutants for the treatment of X-linked nephrogenic diabetes insipidus. J Neuroendocrinol . 2010 May. 22(5):393-9. . Rochdi MD, Vargas GA, Carpentier E, Oligny-Longpré G, et al. Functional characterization of vasopressin type 2 receptor substitutions (R137H/C/L) leading to nephrogenic diabetes insipidus and nephrogenic syndrome

2014 eMedicine.com

94. Diabetes Insipidus (Diagnosis)

on the US market) Thiazides Nonsteroidal anti-inflammatory drugs (NSAIDs), such as indomethacin (may be used in nephrogenic DI, but only when no better options exist) See and for more detail. Next: Background Diabetes insipidus (DI) is defined as the passage of large volumes (>3 L/24 h) of dilute urine (< 300m Osm/kg). DI has 2 major forms: central and nephrogenic. Central DI is characterized by decreased secretion of antidiuretic hormone (ADH)—also known as arginine vasopressin (AVP)—which gives rise (...) mellitus do not induce thirst; rather, the increased thirst in uncontrolled diabetes mellitus is secondary to volume depletion from osmotic diuresis. Previous Next: Etiology DI is usually an acquired disorder, with central DI having different causes than does nephrogenic DI. In rare cases, central or nephrogenic DI may be an inherited disorder. Central diabetes insipidus Central DI has many possible causes. According to the literature, the principal causes of central DI and their oft-cited approximate

2014 eMedicine.com

95. Diabetes Insipidus (Follow-up)

Endocrinologist - the presence of central DI should prompt an evaluation of anterior pituitary function Diagnostic radiologist Regular follow-up visits with an endocrinologist (for central DI) or a nephrologist (for nephrogenic DI) are necessary for dosage adjustment. When indomethacin is used in long-term therapy, carefully observe renal function for any signs of toxicity. Previous References Central diabetes insipidus. Orphannet. Available at . July 2012; Accessed: September 15, 2017. Di Iorgi N, Allegri AE (...) , Napoli F, Calcagno A, Calandra E, Fratangeli N, et al. Central diabetes insipidus in children and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266

2014 eMedicine Pediatrics

96. Diabetes Insipidus (Treatment)

Endocrinologist - the presence of central DI should prompt an evaluation of anterior pituitary function Diagnostic radiologist Regular follow-up visits with an endocrinologist (for central DI) or a nephrologist (for nephrogenic DI) are necessary for dosage adjustment. When indomethacin is used in long-term therapy, carefully observe renal function for any signs of toxicity. Previous References Central diabetes insipidus. Orphannet. Available at . July 2012; Accessed: September 15, 2017. Di Iorgi N, Allegri AE (...) , Napoli F, Calcagno A, Calandra E, Fratangeli N, et al. Central diabetes insipidus in children and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266

2014 eMedicine Pediatrics

97. Diabetes Insipidus (Overview)

and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266-77. . Faerch M, Christensen JH, Corydon TJ, et al. Partial nephrogenic diabetes insipidus caused (...) by a novel mutation in the AVPR2 gene. Clin Endocrinol (Oxf) . 2008 Mar. 68(3):395-403. . Mulders SM, Bichet DG, Rijss JP, et al. An aquaporin-2 water channel mutant which causes autosomal dominant nephrogenic diabetes insipidus is retained in the Golgi complex. J Clin Invest . 1998 Jul 1. 102(1):57-66. . . Chandrasekaran K, Karolina DS, Sepramaniam S, Armugam A, Wintour EM, Bertram JF, et al. Role of microRNAs in kidney homeostasis and disease. Kidney Int . 2012 Apr. 81(7):617-27. . Agre P, King LS

2014 eMedicine Pediatrics

98. Diabetes Insipidus (Diagnosis)

and young adults: etiological diagnosis and long-term outcome of idiopathic cases. J Clin Endocrinol Metab . 2014 Apr. 99 (4):1264-72. . . Knepper MK, Kwon T, Nielsen S. Molecular physiology of water balance. New Engl J Med . 2015 April 2. 372;14:1349-58. . Wildin RS, Antush MJ, Bennett RL. Heterogeneous AVPR2 gene mutations in congenital nephrogenic diabetes insipidus. Am J Hum Genet . 1994 Aug. 55(2):266-77. . Faerch M, Christensen JH, Corydon TJ, et al. Partial nephrogenic diabetes insipidus caused (...) by a novel mutation in the AVPR2 gene. Clin Endocrinol (Oxf) . 2008 Mar. 68(3):395-403. . Mulders SM, Bichet DG, Rijss JP, et al. An aquaporin-2 water channel mutant which causes autosomal dominant nephrogenic diabetes insipidus is retained in the Golgi complex. J Clin Invest . 1998 Jul 1. 102(1):57-66. . . Chandrasekaran K, Karolina DS, Sepramaniam S, Armugam A, Wintour EM, Bertram JF, et al. Role of microRNAs in kidney homeostasis and disease. Kidney Int . 2012 Apr. 81(7):617-27. . Agre P, King LS

2014 eMedicine Pediatrics

99. Use of Copeptin in Diabetes Insipidus

: Prospective evaluation of the novel biomarker copeptin in the differential diagnosis of diabetes insipidus against the standard diagnostic test methods. Condition or disease Intervention/treatment Diabetes Insipidus Primary Polydipsia Other: Water deprivation test Other: Hypertonic saline infusion Detailed Description: Purpose of this study is to compare the overall diagnostic accuracy of the three following diagnostic test procedures in diabetes insipidus (central, nephrogenic) and primary polydipsia (...) : University Hospital, Basel, Switzerland ClinicalTrials.gov Identifier: Other Study ID Numbers: CODDI2013 First Posted: September 12, 2013 Last Update Posted: April 17, 2018 Last Verified: April 2018 Keywords provided by University Hospital, Basel, Switzerland: Copeptin differential diagnosis of polyuria polydipsia syndrome diabetes insipidus compulsive water drinking water deprivation test Diabetes insipidus (central and nephrogenic) Additional relevant MeSH terms: Layout table for MeSH terms Diabetes

2013 Clinical Trials

100. Nephrogenic Diabetes Insipidus

Nephrogenic Diabetes Insipidus Nephrogenic Diabetes Insipidus - Genitourinary Disorders - MSD Manual Professional Edition Brought to you by The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases / / / / IN THIS TOPIC OTHER TOPICS IN THIS CHAPTER Test your (...) Catheterization in Males SOCIAL MEDIA Add to Any Platform Loading , MD, Texas Tech University Click here for Patient Education NOTE: This is the Professional Version. CONSUMERS: Nephrogenic diabetes insipidus (NDI) is an inability to concentrate urine due to impaired renal tubule response to vasopressin (ADH), which leads to excretion of large amounts of dilute urine. It can be inherited or occur secondary to conditions that impair renal concentrating ability. Symptoms and signs include polyuria and those

2013 Merck Manual (19th Edition)

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