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Macule

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1301. Treatment of acquired bilateral nevus of Ota-like macules (Hori's nevus) using a combination of scanned carbon dioxide laser followed by Q-switched ruby laser. (PubMed)

Treatment of acquired bilateral nevus of Ota-like macules (Hori's nevus) using a combination of scanned carbon dioxide laser followed by Q-switched ruby laser. Acquired bilateral nevus of Ota-like macules (Hori's nevus) is a dermal pigmented lesion commonly seen in middle-aged women of Asian descent. The Q-switched ruby laser (QSRL) has been used successfully to treat a variety of benign pigmented lesions. Multiple, sequential treatments are typically required for complete clearance

2003 Journal of the American Academy of Dermatology

1302. Effect of cold air cooling on the incidence of postinflammatory hyperpigmentation after Q-switched Nd:YAG laser treatment of acquired bilateral nevus of Ota like macules. (PubMed)

Effect of cold air cooling on the incidence of postinflammatory hyperpigmentation after Q-switched Nd:YAG laser treatment of acquired bilateral nevus of Ota like macules. To investigate the effect of cold air cooling on the incidence of postinflammatory hyperpigmentation (PIH) after laser treatment in Asian patients.Randomized, controlled, split-face study.Skin laser center of a university hospital.Twenty-three Thai women with acquired bilateral nevus of Ota-like macules.Patients were treated

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2007 Archives of Dermatology

1303. A case of agminated lentiginosis with multiple café-au-lait macules. (PubMed)

A case of agminated lentiginosis with multiple café-au-lait macules. Agminated lentiginosis is an unusual pigmentary disorder, characterized by numerous lentigines grouped within an area of normal skin. The pigmented macules are often in a segmental distribution within a sharp demarcation at the midline. We encountered a 28-year-old woman with an unusual combination of multiple café-au-lait macules and diffuse numerous lentigines involving the right cheek and ipsilateral upper thorax with sharp (...) demarcation at the midline. The multiple lentigines extended bilaterally over the back in a peppered distribution. There were 21 café-au-lait macules on both arms, and the trunk and buttocks; however, there were no Lisch nodules, neurofibromas, or any other clinical manifestations for neurofibromatosis. Histopathology of a macule revealed the features of lentigo.

2007 Clinical & Experimental Dermatology

1304. Treatment of acquired bilateral nevus of ota-like macules (Hori's nevus) with a combination of the 532 nm Q-Switched Nd:YAG laser followed by the 1,064 nm Q-switched Nd:YAG is more effective: prospective study. (PubMed)

Treatment of acquired bilateral nevus of ota-like macules (Hori's nevus) with a combination of the 532 nm Q-Switched Nd:YAG laser followed by the 1,064 nm Q-switched Nd:YAG is more effective: prospective study. Acquired bilateral nevus of Ota-like macules (Hori's nevus) is a common dyschromatosis among Asian women. Q-switched lasers have been used successfully as a treatment modality.The purpose of this study was to compare the efficacy of using the Q-switched 532 nm neodymium:yttrium-aluminum

2006 Dermatologic Surgery

1305. Recurrent pigmented macules after q-switched alexandrite laser treatment of congenital melanocytic nevus. (PubMed)

Recurrent pigmented macules after q-switched alexandrite laser treatment of congenital melanocytic nevus. Q-switch-mode laser treatment of congenital nevi does not result in complete histological clearance, and many patients have partial repigmentation within several months. In addition, the number of recurrent pigmented macules (RPMs) may increase, a major drawback to good cosmetic results. While the mechanism of recurrence is not known.To help elucidate the mechanism of RPM development, we

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2004 Dermatologic Surgery

1306. Cutaneous arteritis presenting with hyperpigmented macules: macular arteritis. (PubMed)

Cutaneous arteritis presenting with hyperpigmented macules: macular arteritis. Macular arteritis is a novel form of cutaneous arteritis in which the primary lesion is a hyperpigmented macule. Traditional stigmata of cutaneous vasculitis such as palpable purpura and erythematous nodules are not present. The disease is asymptomatic and appears to follow an indolent course. Systemic involvement has not been observed.

2003 Journal of American Academy of Dermatology

1307. Melanotic macule of nail unit and its clinicopathologic spectrum. (PubMed)

Melanotic macule of nail unit and its clinicopathologic spectrum. The clinical and histologic spectrum of melanotic macule of the nail unit is examined and the differences in the clinical appearance of longitudinal melanochychia caused by melanotic macule and by other kinds of proliferations of melanocytes are assessed. We observed that the clinical appearance of the pigmented band was of little help in establishing the underlying basic pathologic process. This underscores the importance

2006 Journal of American Academy of Dermatology

1308. Divided café-au-lait macule of the mouth. (PubMed)

Divided café-au-lait macule of the mouth. We describe a 4-year-old, otherwise healthy boy with a congenital history of a perioral and labial segmental café-au-lait macule, who was noted to have unilateral localized gingival hyperpigmentation that aligned with the café-au-lait macule. This case is highly illustrative of the embryologic timing of the genetic event locally, which leads to café-au-lait type hyperpigmentation. Because the facial features and the ectoderm overlying the facial muscles (...) develop around the third to fourth week of gestation, the distribution of this café-au-lait macule suggests development at the same time.

2007 Journal of American Academy of Dermatology

1309. Left atrial myxoma presenting with erythematous macules and loss of memory. (PubMed)

Left atrial myxoma presenting with erythematous macules and loss of memory. Left atrial myxoma was diagnosed in a 42-year-old woman who had cutaneous and neurological manifestations without cardiac symptoms or signs. A two-dimensional echocardiogram revealed an orange-sized mobile mass in the left atrium. Magnetic resonance imaging showed multiple cerebral infarctions. Cardiac surgery for removal of the left atrial myxoma was successful and histopathology confirmed myxoma. Cutaneous

2003 Clinical & Experimental Dermatology

1310. The mechanism of epidermal hyperpigmentation in café-au-lait macules of neurofibromatosis type 1 (von Recklinghausen's disease) may be associated with dermal fibroblast-derived stem cell factor and hepatocyte growth factor. (PubMed)

The mechanism of epidermal hyperpigmentation in café-au-lait macules of neurofibromatosis type 1 (von Recklinghausen's disease) may be associated with dermal fibroblast-derived stem cell factor and hepatocyte growth factor. The mechanism of the accentuated melanization in café-au-lait macules (CALMs) in patients with neurofibromatosis type 1 (NF1; von Recklinghausen's disease) has not been elucidated.To clarify the mechanism involved in the hyperpigmentation of CALMs in NF1.Using enzyme-linked

2003 British Journal of Dermatology

1311. The congenital lingual melanotic macule. (PubMed)

The congenital lingual melanotic macule. Circumscribed oral melanotic macules in adults are a well-defined entity. Congenital oral melanotic macules, however, are rare and not well characterized. We report 5 cases of congenital lingual melanotic lesions with consistent clinical and histologic features.Congenital lingual melanotic macules presented in all cases at birth, followed by proportional growth. Clinical findings were well-circumscribed, brown, single or multiple macules on the tongue, 2 (...) was noted. Two retrospective histopathology reviews mention 4 corresponding cases but report no clinical data.The congenital lingual melanotic macule represents a clinically distinct, benign, pigmented oral melanotic lesion, and may be more common than the literature suggests.

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2003 Archives of Dermatology

1312. Brown macules on the cheeks (PubMed)

Brown macules on the cheeks 15794019 2005 04 22 2018 11 13 0008-350X 51 2005 Mar Canadian family physician Medecin de famille canadien Can Fam Physician Can you identify this condition? Melasma. 353-5 Ting Patricia T PT University of Calgary, Alberta. Barankin Benjamin B eng Case Reports Journal Article Review Canada Can Fam Physician 0120300 0008-350X IM Addison Disease diagnosis Adult Diagnosis, Differential Female Humans Lentigo diagnosis Lupus Erythematosus, Discoid diagnosis Melanosis

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2005 Canadian Family Physician

1313. Dermacase. Melanotic macule of the lip. (PubMed)

Dermacase. Melanotic macule of the lip. 9512829 1998 04 22 2018 11 13 0008-350X 44 1998 Feb Canadian family physician Medecin de famille canadien Can Fam Physician Dermacase. Melanotic macule of the lip. 273, 279 Enta T T University of Calgary, Alta. eng Case Reports Journal Article Canada Can Fam Physician 0120300 0008-350X IM Adolescent Diagnosis, Differential Humans Lip Diseases pathology Lip Neoplasms diagnosis Male Melanoma diagnosis Melanosis pathology Nevus, Pigmented diagnosis Peutz

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1998 Canadian Family Physician

1314. Hyperpigmented dermal macules in children following the administration of Maloprim for malaria chemoprophylaxis. (PubMed)

Hyperpigmented dermal macules in children following the administration of Maloprim for malaria chemoprophylaxis. The occurrence of an unexpected side effect following the use of Maloprim (pyrimethamine/dapsone) for malaria chemosuppression in 3-59 months old children in Sierra Leone is reported. As part of a trial of chemoprophylaxis and insecticide-impregnated bed nets, 2000 children received either Maloprim or placebo; 4% of children who received Maloprim fortnightly for more than 3 months (...) developed hyperpigmented macules, whereas none of the children who received placebo did so. Histopathological examination of full thickness skin biopsies showed macrophages containing melanin in the dermal layer. Clustering of cases was noted among siblings, suggesting the possible involvement of genetic factors in the pathogenesis of these skin reactions. One child was accidentally re-exposed to Maloprim after the drug had been withdrawn and he developed a severe reaction. No other serious side effect

1997 Transactions of the Royal Society of Tropical Medicine and Hygiene

1315. "Cutis tricolor": congenital hyper- and hypopigmented macules associated with a sporadic multisystem birth defect: an unusual example of twin spotting? (PubMed)

"Cutis tricolor": congenital hyper- and hypopigmented macules associated with a sporadic multisystem birth defect: an unusual example of twin spotting? An uncommon coexistence of circumscribed hyperpigmentation and hypopigmentation, in close proximity to each other, is described in a 17 years old patient with various other cogenital defects, such as dysmorphic facial appearance, severe kyphoscoliosis, delayed motor development, epileptic seizures, and mental retardation. We suggest

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1997 Journal of Medical Genetics

1316. Spinal Neurofibromatosis without Café-au-Lait Macules in Two Families with Null Mutations of the NF1 Gene (PubMed)

Spinal Neurofibromatosis without Café-au-Lait Macules in Two Families with Null Mutations of the NF1 Gene Spinal neurofibromatosis (SNF) is considered to be an alternative form of neurofibromatosis, showing multiple spinal tumors and café-au-lait macules. Involvement of the neurofibromatosis type 1 (NF1) locus has been demonstrated, by linkage analysis, for three families with SNF. In one of them, a cosegregating frameshift mutation in exon 46 of the NF1 gene was identified. In the present (...) study, we report four individuals from two families who carry NF1 null mutations that would be expected to cause NF1. Three patients have multiple spinal tumors and no café-au-lait macules, and the fourth has no clinical signs of NF1. In the first family, a missense mutation (Leu2067Pro) in NF1 exon 33 was found, and, in the second, a splice-site mutation (IVS31-5A-->G) enlarging exon 32 by 4 bp at the 5' end was found. The latter mutation has also been observed in an unrelated patient

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2001 American Journal of Human Genetics

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