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Lymphoid Hyperplasia

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821. Castleman's disease of the urachus. (Abstract)

Castleman's disease of the urachus. Castleman's disease (angiofollicular lymphoid hyperplasia) is a rare heterogeneous group of lymphoproliferative disorders of uncertain cause. Most cases occur as mediastinal masses, although extrathoracic involvement has been reported. Castleman's disease involving the genitourinary tract, one of the most infrequent organ systems affected, has so far only been described in pararenal and retroperitoneal locations. We describe a patient with an unusual case (...) of unicentric Castleman's disease that was located in a urachal remnant and bore a malignant appearance. The patient initially presented with abdominal pain and underwent laparoscopic radical excision of the urachal mass. Pathologic examination revealed Castleman's disease, hyaline-vascular type, with atypical lymphoid proliferation and follicular dendritic cell dysplasia. No evidence of concomitant lymphoma or sarcoma was found. Two years after surgical removal of the tumor, no recurrence was evident

2004 Urology

822. The effects of inoculation of Mannheimia haemolytica into the teat of lactating ewes. Full Text available with Trip Pro

and A4/B4 subgroups. In some ewes of the A2/B2 subgroups, mild leucocytic infiltration in the teat was evident; in the ewes of the A3/B3 subgroups, leucocytic infiltration (neutrophils, lymphocytes, plasma cells) was seen, as well as a lymphoid hyperplasia at the border between the teat duct and teat cistern; in ewes of the A4/B4 subgroups, intense subepithelial leucocytic infiltration was the salient feature. No differences were found in the severity of lesions between the two strains used (...) or the three treatments carried out. Although strain VSM08L had been isolated from the teat duct of a healthy ewe, it caused mastitis when inoculated intramammarily; although strain ES26L is of known pathogenicity for the mammary gland, it did not cause clinical mastitis when deposited 2 mm-deep into the teat. These findings point to a protective role of the teat of ewes, which appear to limit bacterial penetration from the teat duct or cistern to the mammary gland. The lymphoid tissue, at the border

2004 Veterinary research Controlled trial quality: uncertain

823. Nodal Inflammatory Pseudotumor Caused by Luetic Infection. (Abstract)

for luetic infection, nor specific serologic tests were available. IPT-LN areas extensively involved the nodal parenchyma in 4 cases, whereas they were focal in the remaining 5 cases. Capsular thickening and inflammation (6/9), venulitis (3/9), small granulomas (3/9), and follicular hyperplasia (7/9) were observed in the associated lymphoid parenchyma. Tp were detected in 4/9 cases of LN-IPT and in none of the extranodal IPT. Tp were extremely abundant within the IPT areas and in the perivascular tissues (...) in the surrounding parenchyma, whereas they were scattered within the capsule. In Tp+ cases, marked follicular hyperplasia was the single distinctively associated feature. Double immunostains revealed that Tp were predominantly contained in the cytoplasm of CD11c+ CD163+ macrophages, some of which co-expressed HLA-DR. In addition, scattered S100+ interdigitating dendritic cells also showed intracytoplasmic Tp. This study shows that a significant number of IPT-LN is associated with Tp infection. A spirochetal

2008 American Journal of Surgical Pathology

824. Adenotonsillar pathology in post-transplant patients. (Abstract)

for adenotonsillectomy at a tertiary care center between May 2000 and May 2003. Thirteen patients were identified. All tonsils were evaluated for follicular lymphoid hyperplasia and atypical lymphoid cells. Seven of thirteen samples were stained for EBV RNA, and twelve of thirteen samples were tested using flow cytometry. Positive flow cytometry diagnosis was based on the presence of atypical lymphocytes, gene arrangement, or both.Seven of thirteen (7/13) examined patients were found to have adenotonsillar

2008 International Journal of Pediatric Otorhinolaryngology

825. Orbital Lymphoproliferative Tumors Analysis of Clinical Features and Systemic Involvement in 160 Cases. (Abstract)

univariate and multivariate regression models.Occurrence of SL diagnosed based on the 6 monthly systemic evaluation.Of 106 patients with OLT alone, SL subsequently developed in 16% of patients and 84% patients remained free of SL. Of 17 patients in whom SL developed subsequently, 29% had marginal zone, B-cell lymphoma of the mucosa-associated lymphoid tissue (MALT), 24% had small lymphocytic lymphoma (SLL), 24% had atypical lymphoid hyperplasia (ALH), 6% each had mantle cell, follicular, and diffuse

2008 Ophthalmology

826. Evolution from primary biliary cirrhosis to primary biliary cirrhosis/autoimmune hepatitis overlap syndrome. (Abstract)

was reported to show "evolving cirrhosis with marked lymphoid hyperplasia." Although the indication was nowhere stated, she was prescribed ursodeoxycholic acid 500 mg b.i.d, on which her biochemical tests initially improved. One year later she developed itching and jaundice. Imaging studies revealed multiple gallstones. An MRCP was suggestive of cirrhosis with a questionable common bile duct stricture, and she underwent ERCP with removal of gallbladder and common bile duct stones and placement of a biliary

2008 Seminars in Liver Disease

827. Diffuse Placoid Choroidopathy in a Patient with Common Variable Immunodeficiency. (Abstract)

, the most common type of significant hypogammaglobulinemia, may be associated with a placoid choroidopathy. The cause of this CVID choroidopathy is unclear at this time, but may be secondary to lymphoid hyperplasia or granulomatous disease.Copyright 2008 S. Karger AG, Basel.

2008 International Archives of Allergy and Immunology

828. t(8;13)-positive Bilineal Lymphomas: Report of 6 Cases. (Abstract)

t(8;13)-positive Bilineal Lymphomas: Report of 6 Cases. The 8p11 myeloproliferative syndrome (EMS) is a rare hematologic malignancy characterized by myeloid hyperplasia, eosinophilia, and precursor lymphoblastic lymphoma, associated with balanced translocations involving chromosome 8p11, most commonly t(8;13)(p11;q12). Approximately 75% of EMS patients present with or develop precursor T-cell lymphoblastic lymphoma, and most subsequently develop acute myeloid leukemia. Here we describe (...) of which expressed myeloid antigens. In all cases, the latter component tended to surround residual lymphoid follicles and/or blood vessels. Numerous eosinophils and prominent high endothelial venules were present in all of the lymph node specimens. Interestingly, cells of both components expressed CD3 on immunohistochemical stains. In conclusion, EMS associated with t(8;13) should be suspected in patients with a bilineal tumor that involves lymph nodes or other extramedullary sites. We believe

2008 American Journal of Surgical Pathology

829. Developmental anatomy of the tonsil and its implications for intracapsular tonsillectomy. (Abstract)

is visible in the 4th post-conceptional week and demonstrates canalization and branching in the 8th week. Lymphoid infiltration of the lamina propria occurs in the seventh month of intrauterine life. Primary follicles form late in gestation, but germinal center stimulation does not occur until shortly after birth. During the first year of life, there is rapid proliferation of lymphoid elements and formation of active germinal centers. During the phase of maximum tonsillar hyperplasia, the lymphoid (...) elements proliferate rapidly, increasing tonsillar bulk. In senescence, there is involution of the lymphoid elements and proliferation of fibrous tissue in the capsule and trabeculae. The overall bulk of the tonsil is much decreased.There is an intimate relationship between the epithelial and lymphoid components of the tonsil which continues through life. To adequately control upper airway obstruction of tonsillar origin and recurrent tonsillar infection in childhood, total or near total intracapsular

2007 International Journal of Pediatric Otorhinolaryngology

830. Canine visceral leishmaniasis: a histopathological study of lymph nodes. (Abstract)

, but it was mainly observed in cervical and popliteal nodes. Histologically, the increased number and size of lymphoid follicles, and the marked hypertrophy and hyperplasia of medullary macrophages (cords and sinus) explained the lymphadenopathy. In addition, the clinical status or the tissue parasitism load might not be directly related to the intensity of the lesions.

2004 Acta Tropica

831. Enhancement of splenic glucose metabolism during acute malarial infection: correlation of findings of FDG-PET imaging with pathological changes in a primate model of severe human malaria. (Abstract)

indicating marked enhancement of glucose metabolism. The standardized uptake values (SUVs) of the spleen in the infected monkeys were significantly higher than those in the uninfected monkey. At autopsy, splenomegaly was clearly present in all infected monkeys, and histopathologic findings included hyperplasia of lymphoid follicles in white pulp, a large number of activated macrophage, and congestion of parasitized red blood cells (PRBCs) and malaria pigments in red pulp. We suggest that increase

2006 American Journal of Tropical Medicine & Hygiene

832. Frequency and severity of ileal adenomas in familial adenomatous polyposis after colectomy. (Abstract)

was 4.7 +/- 3.3 years. In the IRA group, 16/21 patients (77 %) presented visible polyps in the ileal mucosa [adenoma with low-grade dysplasia in 8 patients (38 %), with high-grade dysplasia in 2 (10 %), and lymphoid nodular hyperplasia in 6]. The mean interval between colectomy and adenoma diagnosis was significantly shorter in the IPAA than in the IRA group (4.76 +/- 3.3 vs. 16.4 +/- 8.5 years, P< 0.0001).Our results show a high frequency of adenomas in the ileal mucosa of patients with IPAA and IRA

2007 Endoscopy

833. Lymphoproliferative diseases of the ocular adnexa in Korea. Full Text available with Trip Pro

was 46.3 years (range, 1-87 years). The major histopathologic subtypes were mucosa-associated lymphoid tissue (MALT) lymphoma in 96 patients (75.0%), lymphoid hyperplasia in 11 (8.6%), diffuse large B-cell lymphoma in 6 (4.7%), and mantle cell lymphoma in 4 (3.1%). Ocular lymphoproliferative lesions were located in the conjunctiva (53 patients), the eyelid (33 patients), and the orbit (42 patients). Twenty patients had tumor relapses (15.6%), and 9 died of lymphoma during follow-up (7.0%). Regarding (...) Lymphoproliferative diseases of the ocular adnexa in Korea. To evaluate the clinical features, treatments, and outcomes of patients with ocular lymphoproliferative disease classified according to the World Health Organization classification and to determine prognostic factors of this disease in South Korea.Between March 1, 1995, and December 31, 2005, 128 cases of patients with ocular lymphoid tumors treated at the Samsung Medical Center were retrospectively reviewed.The mean patient age

2007 Archives of Ophthalmology

834. Proteomic analysis of apoptotic pathways reveals prognostic factors in follicular lymphoma. Full Text available with Trip Pro

overcome these obstacles, we procured lymphoid follicles from 20 cases of FL and 15 cases of benign follicular hyperplasia (FH) using laser capture microdissection. Lysates were spotted on reverse-phase protein microarrays and probed with 21 antibodies to proteins in the intrinsic apoptotic pathway, including those specific for posttranslational modifications such as phosphorylation. A panel of three antibodies [phospho-Akt(Ser473), Bcl-2, and cleaved poly(ADP-ribose) polymerase] segregated most cases

2005 Clinical Cancer Research

835. "In situ-like" mantle cell lymphoma: a report of two cases. Full Text available with Trip Pro

"In situ-like" mantle cell lymphoma: a report of two cases. Mantle cell lymphoma (MCL) is a B cell neoplasm that most often shows a diffuse growth pattern. Two cases of MCL are reported here, both with a previous diagnosis of lymphoid hyperplasia. Morphologically, germinal centres are hyperplasic with a normal or discretely enlarged mantle zone, where foci of irregularly shaped small lymphocytes are seen. These are positive for CD20, CD5 and cyclin D1, confirming a diagnosis of in situ-like MCL (...) . This type differs from the mantle zone pattern in that the neoplastic mantle zone is very thin and there is very little or no spread of tumour cells into interfollicular areas. To the best of our knowledge, this is the first report on such a pattern of MCL, which is important to recognise, as it can be confused with lymphoid hyperplasia.

2006 Journal of Clinical Pathology

836. Apoptosis is a major cause of so-called 'Caseous Necrosis' in mycobacterial granulomas in HIV-infected patients. Full Text available with Trip Pro

macrophage response, pseudo-inflammatory tumour response or non-specific lymphoid hyperplasia were examined histologically and for apoptosis by immunostaining for caspase 3 and TUNEL. Classic tuberculoid caseating granulomas in cervical lymph node and lungs from non-HIV-infected patients were also stained with caspase 3.All areas of caseous necrosis frequently displayed extensive apoptosis that readily accounted for the so-called "necrosis". Small foci of apoptosis were present in the other reaction

2007 Journal of Clinical Pathology

837. CD56: a useful tool for the diagnosis of small cell lung carcinomas on biopsies with extensive crush artefact. Full Text available with Trip Pro

hyperplasia, and five chronic non-specific inflammations) with extensive crush artefact were stained using antibodies to CD56, MNF116, thyroid transcription factor 1 (TTF-1), and CD45. All SCLCs showed strong positive staining for CD56 in 75-100% of recognisable tumour cells, even in areas where there was extensive crush artefact. Eighteen of 20 cases were positive for TTF-1 and 16 of 20 were positive for MNF116 in the tumour cells, but both of these antibodies showed little or no staining in areas (...) of crush artefact. Control cases comprising lymphoid cells were positive for CD45 in areas of crush artefact, but all cases of SCLC were negative.CD56, along with markers for cytokeratins-TTF-1, and CD45-are useful in the diagnosis of SCLC in biopsies with extensive crush artefact and can help confirm the diagnosis in cases where features are equivocal.

2005 Journal of Clinical Pathology

838. Dendritic cells accumulate in human fibrotic interstitial lung disease. (Abstract)

reaction analysis.Results were similar in idiopathic pulmonary fibrosis and nonspecific interstitial pneumonia lungs, which were heavily infiltrated by immature DCs in established fibrosis and in areas of epithelial hyperplasia. Altered epithelial cells and fibroblasts, particularly in fibroblastic foci, frankly expressed all chemokines (CCL19, CCL20, CCL22, and CXCL12) susceptible to favor the recruitment of immune cells. Lymphoid follicles were infiltrated by maturing DCs, which could originate from (...) Dendritic cells accumulate in human fibrotic interstitial lung disease. There is growing evidence that resident cells, such as fibroblasts and epithelial cells, can drive the persistent accumulation of dendritic cells (DCs) in chronically inflamed tissue, leading to the organization and the maintenance of ectopic lymphoid aggregates. This phenomenon, occurring through a chemokine-mediated retention mechanism, has been documented in various disorders, but not in fibrotic interstitial lung

2007 American Journal of Respiratory and Critical Care Medicine

839. Transmembrane activator and calcium-modulating cyclophilin ligand interactor mutations in common variable immunodeficiency: clinical and immunologic outcomes in heterozygotes. Full Text available with Trip Pro

families had the same mutations but were not immune-deficient, and their B cells produced normal amounts of IgG and IgA after APRIL stimulation.Mutations in TACI significantly predispose to autoimmunity and lymphoid hyperplasia in CVID, but additional genetic or environmental factors are required to induce immune deficiency.Additional causes of this common immune deficiency syndrome remain to be determined.

2007 Journal of Allergy and Clinical Immunology

840. Review article: capsule endoscopy - are all small intestinal lesions Crohn's disease? (Abstract)

and ulcers of the small intestine. Such erosions and ulcers may in fact be the result of non-steroidal anti-inflammatory drug intake and other pathological conditions such as lymphoid hyperplasia, lymphoma and radiation enteritis. Non-steroidal anti-inflammatory drug intake should, therefore, be excluded as a cause prior to relating the presence of lesions to CD. Misdiagnosis of CD may be harmful to the patient because if unresponsive, intensification of therapy may occur and occasionally an unnecessary

2006 Alimentary Pharmacology & Therapeutics

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