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Lymphoid Hyperplasia

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801. Management of Thyroid Cancer

Advisory Committee (part of Health Protection Agency) BAETS British Association of Endocrine and Thyroid Surgeons BAO-HNS British Association of Otolaryngologists, Head & Neck Surgeons CCH C-Cell hyperplasia CT Computed tomography DTC Differentiated thyroid cancer* EORTC European Organisation for Research and Treatment of Cancer FACS Fluorescent activated cell sorter FDG 18 Fluoro-deoxy-glucose FMTC Familial medullary thyroid cancer FNAC Fine-needle aspiration cytology FTC Follicular thyroid cancer* GP (...) General practitioner IJV Internal jugular vein IMRT Intensity modulated radiotherapy MACIS Metastases, Age at presentation, Completeness of surgical resection, Invasion (extrathyroidal), Size MALT Mucosa associated lymphoid tissue MDL Minimum detection level MDT Multidisciplinary team MEN Multiple endocrine neoplasia MIBG Metaiodobenzylguanidine MRI Magnetic resonance imaging MRND Modified radical neck dissection MTC Medullary thyroid carcinoma* PET Positron emission tomography PTC Papillary thyroid

2007 British Association of Endocrine and Thyroid Surgeons

802. Guidelines for 18F-FDG PET and PET-CT imaging in paediatric oncology

child’s motion during the scan; hence, the need for sedation will decrease. Interpretation It is important to keep in mind the physiologic FDG distribution to avoid image misinterpretation. FDG uptake in the thymus is a common finding in children and young adults, which can be normally identified because of its characteristic appearance of an inverted “V”.Thymus hyperplasia after chemotherapy is a common finding, and it can be difficult to differentiate it from residual/recurrent disease (...) in the anterior mediastinum. FDG uptake in naso-/ oropharyngeal lymphoid tissue such as the Waldeyer’s ring is often more prominent in children compared to the adult population. Uptake in mastication muscles may become visible in babies who suck pacifiers or are fed within the first 30 min of the tracer injection. High FDG uptake is often seen symmetrically in skeletal growth plates. Diffuse- ly elevated FDG uptake in the bone marrow has been observed in patients after the end of chemotherapy as evidence

2008 European Association of Nuclear Medicine

803. Clinical practice guidelines for the management of early breast cancer

iSource National Breast Cancer Centreix Clinical practice guidelines for the management of early breast cancer LIST OF ABBREVIATIONS ADH atypical ductal hyperplasia AH atypical hyperplasia ALH atypical lobular hyperplasia BCSS Breast Cancer Support Service CA 15.3 breast cancer tumour marker CEA non-specific tumour markers CLE complete local excision CMF cyclophosphamide, methotrexate and 5-fluorouracil CS conservative surgery CSF colony stimulating factor DCIS ductal carcinoma in situ EIC extensive (...) at any age) 28 • a substantial increase in body size in post-menopausal women 29,30 • alcohol (a daily intake of more than two drinks) 31,3213 Clinical practice guidelines for the management of early breast cancer Ductal carcinoma in situ, lobular carcinoma in situ and atypical hyperplasia Some breast diseases, including ductal carcinoma in situ (DCIS), lobular carcinoma in situ (LCIS) and atypical hyperplasia (AH), are associated with an increased risk of invasive breast cancer. 33,34 DCIS

2001 Cancer Australia

804. Lymphoma, primary pulmonary

-CD20+ cells. Differential diagnosis Radiological appearance may mimic cryptogenic organizing pneumonia, lymphoid interstitial pneumonia (LIP), diffuse lymphoid hyperplasia, follicular bronchitis and bronchioloalveolar carcinoma. LIP may be associated with PPL in case of autoimmune disease. Management and treatment The treatment options include watchful observation for indolent types of PPL, surgery in localized tumors, chemotherapy with or without rituximab if the lesions are diffuse or involve (...) of information on the Orphanet website are accepted. For all other comments, please send your remarks via . Only comments written in English can be processed. Check this box if you wish to receive a copy of your message * " for="captcha" >Please reproduce the text below: * Primary pulmonary lymphoma Disease definition Primary pulmonary lymphoma (PPL) is a rare lymphoma of the lung, defined as a clonal lymphoid proliferation affecting one or both lungs (parenchyma and/or bronchi) in a patient

2005 Orphanet

805. What might be the cause of three enlarged lymph nodes on the sternocleidomastoid (4mm across), persistent for 3 weeks, in a seemly healthy 16 month old child?

nodes that, if evaluated by adult standards, would qualify as lymphadenopathy. • Healthy children typically have palpable lymph nodes in the anterior cervical, inguinal, and often-axillary regions. Lymphoid mass in general increases steadily after birth until children are aged 8-12 years, and then, during puberty, lymphoid tissue undergoes a progressive atrophy, which continues throughout life. Bilateral anterior cervical lymph nodes up to 2 cm in diameter often are found in well children (...) lymphadenopathy: Cervical nodes drain the tongue, external ear, parotid gland, and deeper structures of the neck, including the larynx, thyroid, and trachea. Inflammation or direct infection of these areas causes subsequent engorgement and hyperplasia of their respective node groups. Adenopathy is most common in cervical nodes in children and usually is related to infectious etiologies. Lymphadenopathy posterior to the sternocleidomastoid typically is a more ominous finding, with a higher risk of serious

2006 TRIP Answers

806. AMG-479 in Treating Patients With Advanced Solid Tumors or Non-Hodgkin Lymphoma

lymphoma Primary hepatic tumors or at increased risk for hepatic tumors, including any of the following: Hepatitis of any etiology Alcohol abuse or dependency Hepatic adenoma Follicular nodular hyperplasia Autoimmune conditions associated with biliary tract cancer Alpha 1 antitrypsin deficiency Hemochromatosis History of vinyl chloride or thorotrast/thorium dioxide exposure History of histiocytic (Kupffer cell) neoplasia Presence of untreated or symptomatic CNS metastases or symptoms of brain (...) -cell lymphoma of mucosa-associated lymphoid tissue nodal marginal zone B-cell lymphoma recurrent adult Burkitt lymphoma recurrent adult diffuse large cell lymphoma recurrent adult diffuse mixed cell lymphoma recurrent adult diffuse small cleaved cell lymphoma recurrent adult grade III lymphomatoid granulomatosis recurrent adult immunoblastic large cell lymphoma recurrent adult lymphoblastic lymphoma recurrent cutaneous T-cell non-Hodgkin lymphoma recurrent grade 1 follicular lymphoma recurrent

2007 Clinical Trials

807. Endoscopic findings and pathologic characteristics of gastric eosinophilic granuloma: A report of 18 patients Full Text available with Trip Pro

pylorus and/or duodenal bulb, esophagitis, mucous hyperemia and/or mucosal erosion. Eosinophilic cell infiltration and generous hyperplasia of arterioles, venules and lymph vessels were found in the lesions of the patients. Interstitium had massive eosinophilic infiltrates and was made up of collagen fibers and fibroblasts. Lymphoid follicles were revealed in some sections of biopsies.GEG is lack of specific symptoms and physical signs. It can be misdiagnosed as gastric ulcer in most cases before

2006 World journal of gastroenterology : WJG

808. Acute Appendicitis: Are we Over Diagnosing it? Full Text available with Trip Pro

patients in whom macroscopically normal appendices had been removed, other findings were noted including fibro-obliterative changes in 10, luminal inflammation in 6, serositis in 5, lymphoid hyperplasia in 3, feacoliths in 2, and pinworm in 1, making another abdominal pathology a possibility.The number of macroscopically normal appendices removed was much larger in female patients. However in 27 of 49 normal looking appendices in females, findings such as serositis, luminal inflammation, lymphoid (...) hyperplasia, etc. were noted on histology, suggesting that another abdominal pathology may have been missed or the appendix may still have been the cause for pain. On the basis of these findings, we suggest that diagnostic laparoscopy should be performed at least in all female patients before an appendicectomy; if no other findings are noted on laparoscopy, it is likely to be worthwhile to remove the appendix.

2007 Annals of the Royal College of Surgeons of England

809. Systemic Mastocytosis Presenting with a Prominent B Lymphocyte Proliferation in the Bone Marrow and Extensive Fibrosis of the Spleen Full Text available with Trip Pro

Systemic Mastocytosis Presenting with a Prominent B Lymphocyte Proliferation in the Bone Marrow and Extensive Fibrosis of the Spleen Systemic mastocytosis is a disease characterized by multifocal mast cell proliferation in the bone marrow or other extracutaneous organs. Because of loosely scattered and hypo-/agranular mast cells, the diagnosis is sometimes very difficult. In the bone marrow, mast cell infiltration may be associated with prominent lymphoid infiltration leading to a misdiagnosis (...) . The consecutive bone marrow biopsies were similar to the first. The subsequent splenectomy specimen exhibited striking fibrosis. In the lymph node sections, there was marginal zone hyperplasia. Multifocal accumulations of mast cells were strongly positive with mast cell tryptase and CD117 on immunohistochemical staining, though no metachromasia was identified in Giemsa and Toluidine Blue stained aspirates and tissue sections, probably due to hypo-/agranulation of mast cells. The case was presented

2007 Disease markers

810. Infiltration of the synovial membrane with macrophage subsets and polymorphonuclear cells reflects global disease activity in spondyloarthropathy Full Text available with Trip Pro

, lymphoid aggregates, CD1a+ cells, intracellular citrullinated proteins, and MHC-HC gp39 complexes than RA synovitis. Unsupervised clustering of the SpA samples based on synovial features identified two separate clusters that both contained different SpA subtypes but were significantly differentiated by concentration of C-reactive protein and erythrocyte sedimentation rate. Global disease activity in SpA correlated significantly with lining-layer hyperplasia as well as with inflammatory infiltration (...) biopsies obtained from 99 SpA and 86 RA patients with active knee synovitis were analyzed for 15 histological and immunohistochemical markers. Correlations with swollen joint count, serum C-reactive protein concentrations, and erythrocyte sedimentation rate were analyzed using classical and multiparameter statistics. SpA synovitis was characterized by higher vascularity and infiltration with CD163+ macrophages and polymorphonuclear leukocytes (PMNs) and by lower values for lining-layer hyperplasia

2005 Arthritis research & therapy

811. Gastric Helicobacter Infection Induces a Th2 Phenotype but Does Not Elevate Serum Cholesterol in Mice Lacking Inducible Nitric Oxide Synthase Full Text available with Trip Pro

significantly in the iNOS-deficient mice relative to those for the wild-type mice (all P <0.01). Helicobacter-infected iNOS-/- mice had chronic lymphoid infiltration and negligible to mild glandular atrophy and mucous metaplasia in the fundic mucosa, while H. felis-infected wild-type mice had severe atrophic and metaplastic mucosal changes. The atrophic gastritis in the infected wild-type mice, particularly the female mice, was also accompanied by greater granulocytic infiltration, antral hyperplasia (...) histologically and serologically at 32 weeks postinfection. Equal numbers of uninfected controls were sham inoculated. The mice were scored for severity of gastric inflammation, hyperplasia, glandular atrophy, and mucous metaplasia in the corpus and for the level of helicobacter colonization. The immunoglobulin G1 (IgG1), IgG2a, and IgG2c antibody responses to H. felis were determined. As a secondary measure, serum cholesterol levels were assessed. iNOS-/- mice have a propensity for increased serum

2005 Infection and immunity

812. An unusual finding in a patient with iron deficiency anaemia Full Text available with Trip Pro

Female Humans Hyperplasia Lymphoid Tissue pathology 2005 9 16 9 0 2005 12 13 9 0 2005 9 16 9 0 ppublish 16162954 54/10/1491 10.1136/gut.2005.067082 PMC1774709

2005 Gut

813. CD34+ Cord Blood Cell-Transplanted Rag2−/− γc−/− Mice as a Model for Epstein-Barr Virus Infection Full Text available with Trip Pro

as newborns were recently shown to develop human B, T, and dendritic cells, constituting lymphoid organs in situ. Here we used this model to better define the strategy of EBV infection of human B cells in vivo and to compare this model system with different conditions of EBV infection in humans. Our results support the model of EBV persistence in vivo in cases that were characterized by follicular hyperplasia and a relatively normal CD4(+) and CD8(+) T-cell distribution. Intriguingly, in cases that were

2008 The American journal of pathology


and atrophic in 3. Six of the 24 sublingual glands removed were microscopically normal, while the other specimens presented acinar atrophy with hyperplasia of duct-like structures. Interstitial fibrosis was observed in 18 cases and was accompanied by adipose tissue infiltration in 15. Decreased lymphoid tissue was observed in 16 samples and oncocytosis was present in 5 cases. We suggest that MFE in edentulous or partially edentulous patients should be considered as an entity for the text books. (...) MOUTH FLOOR ENLARGEMENTS RELATED TO THE SUBLINGUAL GLANDS IN EDENTULOUS OR PARTIALLY EDENTULOUS PATIENTS. A MICROSCOPIC STUDY Mouth floor enlargements (MFE) are observed in edentulous and partially edentulous patients, impairing denture fitting, and have recently been described in the literature as hyperplasias of the sublingual glands.This study aims at describing the microscopic aspects of MFE that contribute to their final diagnosis.Twenty-four specimens were surgically removed from

2006 Journal of Applied Oral Science

815. Laparoscopic Management of Delayed Recurrent Intussusception in an Older Child Full Text available with Trip Pro

postoperative course.The specimen was remarkable for findings of multiple enlarged lymph nodes over the serosal surface of the ileum and the terminal ileum with focal edema, prominent lymphoid hyperplasia and large hemorrhagic areas.This case highlights the fact that in a child with a delayed recurrence of intussusception, the presence of a lead point should be suspected, and operative therapy should be strongly considered over hydrostatic reduction. The current management of recurrent intussusception

2007 JSLS : Journal of the Society of Laparoendoscopic Surgeons

816. Appendicitis Full Text available with Trip Pro

Appendicitis Potential causes of appendicitis include faecoliths, lymphoid hyperplasia, and caecal carcinoma, all of which can lead to obstruction of the appendix lumen. The lifetime risk is approximately 7-9% in the USA, making appendicectomy the most common abdominal surgical emergency. Mortality from acute appendicitis is less than 0.3%, but rises to 1.7% after perforation.We conducted a systematic review and aimed to answer the following clinical question: What are the effects of treatments

2007 BMJ Clinical Evidence

817. Antibiotic-induced mesenteric adenopathy in an intussusception mouse model: a randomized, controlled trial. (Abstract)

Antibiotic-induced mesenteric adenopathy in an intussusception mouse model: a randomized, controlled trial. Idiopathic intussusception is a leading cause of intestinal obstruction in young children. Although the etiology remains obscure, lymphoid hyperplasia is found in a majority of cases. Antibiotics, the most frequently prescribed medication class in the pediatric population, have been recently associated with intussusception. The authors sought to determine whether enteral antibiotic

2005 Journal of pediatric gastroenterology and nutrition

818. Mesenteric Castleman disease. (Abstract)

Mesenteric Castleman disease. Castleman disease is a rare benign lymph node hyperplasia usually presenting as an asymptomatic mediastinal mass in children. It can present at any extrathoracic site where there is lymphoid tissue. The sites include retroperitoneum, mesentery, axilla, and pelvis. Mesenteric Castleman disease is very rare in children.

2008 Journal of Pediatric Surgery

819. Nonoperative treatment of acute appendicitis in children. (Abstract)

. Antibiotic therapy probably causes regression of lymphoid hyperplasia because of suppression of bacterial infection and prevents ischemia and bacterial invasion in the early stage of appendicitis. We found that some of the patients who had a history of abdominal pain for less than 24 hours with localized abdominal tenderness and hemodynamic stability could be treated nonoperatively. (...) years; mean age, 9 years). The mean anteroposterior diameter of the appendix at the presentation was 7.11 +/- 1.01 mm (range, 6-9.5 mm). Ultrasound examination was repeated after 48 hours of treatment. The mean diameter of the appendix was 4.64 +/- 0.82 mm (range, 3.6-6.8 mm). The difference was statistically significant (t = 9.63, P < .0001). Nonoperative treatment was successful in 15 (93.7%) of the 16 patients.Hyperplasia of the appendiceal lymphoid follicle frequently causes luminal obstruction

2007 Journal of Pediatric Surgery

820. Primary abdominal lymphonodular cryptococcosis in children: 2 case reports and a literature review. (Abstract)

was mainly the persisting fever with or without abdominal pain. Imaging findings indicated dramatic lymphadenopathy and light hepatosplenomegaly. Excision biopsy of the abnormal lymph node demonstrated reactive lymphoid hyperplasia and the presence of C neoformans. The results of nested polymerase chain reaction confirmed the diagnosis of C neoformans.This is the first report on C neoformans in the primary abdominal lymph node in children. This report suggests that C neoformans infection should

2006 Journal of Pediatric Surgery

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