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Lymphoid Hyperplasia

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61. Nodular lymphoid hyperplasia of the bowel in primary hypogammaglobulinaemia: study of in vivo and in vitro lymphocyte function. Full Text available with Trip Pro

Nodular lymphoid hyperplasia of the bowel in primary hypogammaglobulinaemia: study of in vivo and in vitro lymphocyte function. In vitro and in vivo lymphocyte function was studied in six patients with primary hypogammaglobulinaemia and nodular lymphoid hyperplasia (NLH) of the bowel. Lymphocyte transformation, numbers of circulating T and B lymphocytes, and delayed hypersensitivity skin tests did not significantly differ when compared with hypogammaglobulinaemic patients without NLH. However

1977 Gut

62. Benign lymphoid hyperplasia of the colon. Full Text available with Trip Pro

Benign lymphoid hyperplasia of the colon. 919544 1977 12 29 2018 11 13 0093-0415 127 5 1977 Nov The Western journal of medicine West. J. Med. Benign lymphoid hyperplasia of the colon. 416-8 Liebman W M WM Rosental E E eng Case Reports Journal Article United States West J Med 0410504 0093-0415 IM Colon pathology Female Humans Hyperplasia Infant Lymphatic System pathology 1977 11 1 1977 11 1 0 1 1977 11 1 0 0 ppublish 919544 PMC1237891 Fed Proc. 1967 Nov-Dec;26(6):1606-11 4169458 Radiology. 1970

1977 Western Journal of Medicine

63. IRRADIATION TREATMENT OF LYMPHOID HYPERPLASIA OF THE NASOPHARYNX Full Text available with Trip Pro

IRRADIATION TREATMENT OF LYMPHOID HYPERPLASIA OF THE NASOPHARYNX Irradiation is useful in the destruction of hypertrophied lymphoid tissue which cannot be surgically removed.Beta, gamma and roentgen rays have been used for this purpose. With beta rays, surface tissue must be subjected to relatively high dosages if effective radiation is to be delivered to deeper-lying tissues. This disparity is less when gamma rays are used, less still in the case of x-rays. With the possibilities of permanent (...) damage considered, choice of method depends upon the location and extent of the excess lymphoid tissue to be destroyed and the mechanical difficulties of reaching it with effective irradiation without hazard to intervening or surrounding tissue.

1951 California Medicine

64. Epithelial control of gut-associated lymphoid tissue formation through p38α-dependent restraint of NF-κB signaling Full Text available with Trip Pro

these roles involve cell-autonomous control of signaling and gene expression by p38α. In this study, we show that p38α regulates gut-associated lymphoid tissue (GALT) formation in a noncell-autonomous manner. From an investigation of mice with intestinal epithelial cell-specific deletion of the p38α gene, we find that p38α serves to limit NF-κB signaling and thereby attenuate GALT-promoting chemokine expression in the intestinal epithelium. Loss of this regulation results in GALT hyperplasia and, in some (...) Epithelial control of gut-associated lymphoid tissue formation through p38α-dependent restraint of NF-κB signaling The protein kinase p38α mediates cellular responses to environmental and endogenous cues that direct tissue homeostasis and immune responses. Studies of mice lacking p38α in several different cell types have demonstrated that p38α signaling is essential to maintaining the proliferation-differentiation balance in developing and steady-state tissues. The mechanisms underlying

2016 Journal of immunology (Baltimore, Md. : 1950)

65. Involvement of Chemokines and a CD4-Positive T Cell Subset in the Development of Conjunctival Secondary Lymphoid Follicles in an Atopic Keratoconjunctivitis Mouse Model. Full Text available with Trip Pro

Involvement of Chemokines and a CD4-Positive T Cell Subset in the Development of Conjunctival Secondary Lymphoid Follicles in an Atopic Keratoconjunctivitis Mouse Model. Massive B cell lymphoid hyperplasia and its associated factors may play a role in exacerbating inflammation in allergic disorders. We here investigated the chemokines and CD4-positive T cell subset involved in the development of secondary lymphoid follicles (iCALT) in conjunctival tissues in an atopic keratoconjunctivitis mouse

2015 International Archives of Allergy and Immunology

66. Intrinsic functional defects of type 2 innate lymphoid cells impair innate allergic inflammation in promyelocytic leukemia zinc finger (PLZF)-deficient mice. Full Text available with Trip Pro

of eosinophils and goblet cell hyperplasia. In contrast, the adaptive allergic inflammatory response to ovalbumin and alum was unimpaired.PLZF expression at the innate lymphoid cell precursor stage has a long-range effect on the functional properties of mature ILC2s and highlights the importance of these cells for innate allergic responses in otherwise immunocompetent mice.Copyright © 2015 American Academy of Allergy, Asthma & Immunology. All rights reserved. (...) Intrinsic functional defects of type 2 innate lymphoid cells impair innate allergic inflammation in promyelocytic leukemia zinc finger (PLZF)-deficient mice. The transcription factor promyelocytic leukemia zinc finger (PLZF) is transiently expressed during development of type 2 innate lymphoid cells (ILC2s) but is not present at the mature stage. We hypothesized that PLZF-deficient ILC2s have functional defects in the innate allergic response and represent a tool for studying innate immunity

2015 Journal of Allergy and Clinical Immunology

67. Effect of Cell Surface Markers and Lymphoid Cell Distribution on the Arterial Tissue Repair (ECLAR)

Effect of Cell Surface Markers and Lymphoid Cell Distribution on the Arterial Tissue Repair (ECLAR) Effect of Cell Surface Markers and Lymphoid Cell Distribution on the Arterial Tissue Repair (ECLAR) - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please (...) remove one or more studies before adding more. Effect of Cell Surface Markers and Lymphoid Cell Distribution on the Arterial Tissue Repair (ECLAR) (ECLAR) The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT02421081 Recruitment Status : Unknown Verified October 2016 by ali eray günay, TC Erciyes

2015 Clinical Trials

68. Early lymphoid responses and germinal center formation correlates with lower viral load set points and better prognosis of SIV infection Full Text available with Trip Pro

Early lymphoid responses and germinal center formation correlates with lower viral load set points and better prognosis of SIV infection We have investigated the dynamics of germinal center (GC) formation in lymphoid tissues following acute SIV infection. SIV induces a marked follicular hyperplasia, associated with an aberrant accumulation of nonproliferating T follicular helper cells within GCs, but with an abundance of cells producing IL-21, demonstrating that the mechanisms involved (...) for these two events appear independent. IL-21-stimulated T follicular helper cells are considered a critical element for GC formation, a physiological process that seems dysregulated and excessive during HIV/SIV infection, contributing to lymphoid pathogenesis. However, the data suggest that the kinetics by which such GCs are formed may be an important predictor of the host-pathogen equilibrium, as early GC hyperplasia was associated with better control of viral replication. In contrast, monkeys undergoing

2014 Journal of immunology (Baltimore, Md. : 1950)

69. Pilot study: Association between Helicobacter pylori in adenoid hyperplasia and reflux episodes detected by multiple intraluminal impedance in children. (Abstract)

Pilot study: Association between Helicobacter pylori in adenoid hyperplasia and reflux episodes detected by multiple intraluminal impedance in children. The aim of this pilot study was to investigate an association between laryngopharyngeal reflux detected by combined multiple intraluminal impedance and pH monitoring and Helicobacter pylori in adenoid hyperplasia detected with real time polymerase chain reaction (PCR).The study group consisted of 30 children (median age 5.34 years (...) ) with extraesophageal symptoms of gastroesophageal reflux disease with adenoid hyperplasia. All children underwent adenoidectomy with subsequent PCR detection of H. pylori DNA in the tissue and multiple intraluminal impedance and pH monitoring. The most proximal impedance sensor was located 1cm caudal to the entrance of the oesophagus.We found significant differences in the number of reflux episodes among patients with PCR positivity (median 35) and negativity (median 0) of H. pylori (p-value of Mann-Whitney U-test

2014 International Journal of Pediatric Otorhinolaryngology

70. Isolated Lymphoid Follicles are Dynamic Reservoirs for the Induction of Intestinal IgA Full Text available with Trip Pro

Isolated Lymphoid Follicles are Dynamic Reservoirs for the Induction of Intestinal IgA IgA is one of the most important molecules in the regulation of intestinal homeostasis. Peyer's patches have been traditionally recognized as sites for the induction of intestinal IgA responses, however more recent studies demonstrate that isolated lymphoid follicles (ILFs) can perform this function as well. ILF development is dynamic, changing in response to the luminal microbial burden, suggesting that ILFs (...) play an important role providing an expandable reservoir of compensatory IgA inductive sites. However, in situations of immune dysfunction, ILFs can over-develop in response to uncontrollable enteric flora, resulting in ILF hyperplasia. The ability of ILFs to expand and respond to help control the enteric flora makes this dynamic reservoir an important arm of IgA inductive sites in intestinal immunity.

2012 Frontiers in immunology

71. Angiolymphoid hyperplasia with eosinophilia occurring in bilateral eyelids. Full Text available with Trip Pro

Angiolymphoid hyperplasia with eosinophilia occurring in bilateral eyelids. Angiolymphoid hyperplasia with eosinophilia (ALHE) is an uncommon benign lesion, primarily occurring in the head and neck. ALHE arising from the ocular adnexa is rare, and the bilateral presentation is especially rare in the eyelids.A 64-year-old Japanese man presented with tearing. Multiple nodules, approximately 5 mm in size, were observed in bilateral upper and lower eyelids. Surgical excisions of the both eyelids (...) masses were performed. Histopathological examination of the excised masses demonstrated proliferated blood vessels lined by plump endothelial cells together with a lymphoid and eosinopilic infiltrate, compatible with a diagnosis of ALHE. Flow cytometry studies showed that the mass consisted of mostly CD3-positive cells. During two-year follow-up, no recurrence of the mass was observed and the patient had no subjective symptom of tearingALHE may occur in the bilateral eyelids. The cause of ALHE

2013 BMC Ophthalmology

72. MR Imaging of Reactive Lymphoid Hyperplasia of the Liver. (Abstract)

MR Imaging of Reactive Lymphoid Hyperplasia of the Liver. Reactive lymphoid hyperplasia, also known as pseudolymphoma or nodular lymphoid lesion of the liver, is a rare benign lesion. It is mainly detected in the lung, stomach, small intestine, orbit, pancreas, skin, and breast. It remains difficult to distinguish reactive lymphoid hyperplasia from malignant disease clinically when it develops in the liver.We have recently encountered a patient with liver reactive lymphoid hyperplasia who had (...) undergone colon cancer surgery.Preoperative MR imaging showed some useful findings indicating reactive lymphoid hyperplasia.

2011 Journal of Gastrointestinal Surgery

73. A study of histologic and immunophenotypical staining patterns in cutaneous lymphoid hyperplasia. (Abstract)

A study of histologic and immunophenotypical staining patterns in cutaneous lymphoid hyperplasia. Cutaneous lymphoid hyperplasia (CLH) is generally classified according to clinicopathologic entities or put into broad spectrums of B-cell or T-cell predominance or co-dominance.We sought to discern histologic features and immunohistochemical staining patterns in CLH that may form a basis for a histologic classification system.We studied the clinical, histologic, immunophenotypical, and molecular (...) characteristics of 24 consecutive patients with CLH.The 24 cases were classified according to characteristic histologic features and immunophenotypical staining patterns as follows: presence of germinal center (GC) cell clusters forming well-defined lymphoid follicles (n = 10); presence of clusters of GC cell clusters not forming well-defined lymphoid follicles (n = 6); persistent arthropod assault type CLH (n = 1); CLH with a prominent histiocytic component (n = 4); and CLH without specific histologic

2011 Journal of American Academy of Dermatology

74. Benign Lymphoid Hyperplasia of the Tongue Base Causing Upper Airway Obstruction Full Text available with Trip Pro

Benign Lymphoid Hyperplasia of the Tongue Base Causing Upper Airway Obstruction Severe benign lymphoid hyperplasia (LH) is unusual in the head and neck region, but the diagnosis of LH is of clinical importance as it may be confused with malignant lymphoma, both on clinical examination and pathologically. While the etiology is poorly understood, a number of previous theories exist, which are included here in the context of a literature review. In this paper we present a case of severe pharyngeal (...) lymphoid hyperplasia causing airway obstruction and requiring tracheotomy and subsequent surgical debulking.

2011 Case reports in otolaryngology

75. Imaging Findings of Localized Lymphoid Hyperplasia of the Pancreas: a Case Report Full Text available with Trip Pro

Imaging Findings of Localized Lymphoid Hyperplasia of the Pancreas: a Case Report We report here on a case of localized lymphoid hyperplasia of the pancreas in a 70-year-old man which manifested as double lesions (uncinate process and tail) in the organ. The lesions were incidentally detected as hypoechoic lesions on ultrasonography and they appeared as delayed enhancing lesions on the contrast-enhanced dynamic CT and MRI. Total pancreatectomy was performed, because malignant tumor could (...) not be excluded according to the preoperative imaging studies and the endoscopic ultrasound-guided biopsy failed. Pathology revealed localized lymphoid hyperplasia. The patient had an uneventful postoperative course. He has been alive for 18 months after surgery.

2011 Korean Journal of Radiology

76. Etiology of the compensatory renal response: observations on the role of the lymphoid system. Full Text available with Trip Pro

Etiology of the compensatory renal response: observations on the role of the lymphoid system. 5810323 1969 10 25 2018 11 13 0035-9157 62 8 1969 Aug Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Etiology of the compensatory renal response: observations on the role of the lymphoid system. 861-4 Fox M M eng Journal Article England Proc R Soc Med 7505890 0035-9157 IM Animals Hyperplasia etiology Hypertrophy etiology Kidney physiopathology Lymphatic System physiopathology

1969 Proceedings of the Royal Society of Medicine

77. THE LYMPHOCYTE IN NATURAL AND INDUCED RESISTANCE TO TRANSPLANTED CANCER : VI. HISTOLOGICAL COMPARISON OF THE LYMPHOID TISSUE OF NATURALLY IMMUNE AND SUSCEPTIBLE MICE. Full Text available with Trip Pro

THE LYMPHOCYTE IN NATURAL AND INDUCED RESISTANCE TO TRANSPLANTED CANCER : VI. HISTOLOGICAL COMPARISON OF THE LYMPHOID TISSUE OF NATURALLY IMMUNE AND SUSCEPTIBLE MICE. The lymphoid organs of mice show definite changes after cancer inoculation. In immune mice there is a tendency towards a lymphoid hyperplasia, while in susceptible mice more or less marked depletion of the lymphoid tissue takes place. These changes are evident at the end of the 3rd week after cancer inoculation.

1921 The Journal of experimental medicine

78. The Influence of Thymic and Bursal Lymphoid Systems in Avian Tuberculosis Full Text available with Trip Pro

not significantly alter the disease in terms of survival and lesion structure. Cortisone induced earlier development of lesions.In spleen, bacilli were first seen 5 days after inoculation in macrophages in the lymphoid sheath-particularly in capsules of germinal centers. Subsequent lesions were tubercles in the lymphoid sheath, hyperplasia of penicillar arteriolar sheaths, lymphoid atrophy and amyloid deposition. Tubercles were composed of infected macrophages located centrally and surrounded by lighter (...) The Influence of Thymic and Bursal Lymphoid Systems in Avian Tuberculosis Intact and immunologically deficient chickens inoculated with M avium were examined for structural changes in the spleen, the tuberculin reactions and antibody formation. Intravenous inoculation caused miliary tuberculosis of spleen and liver but not brain; intracerebral inoculation caused massive lymphocytic tuberculosis meningitis with later spread to spleen and liver. Neonatal bursectomy and thymectomy did

1971 The American journal of pathology

79. Intrathoracic Lymphoid Hamartoma Full Text available with Trip Pro

Intrathoracic Lymphoid Hamartoma 14216979 1996 12 01 2018 12 01 0040-6376 19 1964 Sep Thorax Thorax INTRATHORACIC LYMPHOID HAMARTOMA. 469-76 HIRST E E WILLIAMS H B HB eng Journal Article England Thorax 0417353 0040-6376 OM Adolescent Hamartoma Humans Hyperplasia Lymph Nodes Mediastinal Neoplasms Pathology Surgical Procedures, Operative ADOLESCENCE HAMARTOMA HYPERPLASIA LYMPH NODES MEDIASTINAL NEOPLASMS PATHOLOGY SURGERY, OPERATIVE 1964 9 1 1964 9 1 0 1 1964 9 1 0 0 ppublish 14216979 PMC1018863

1964 Thorax

80. Studies on homotransplantable lymphomas in hamsters. I. Histologic responses in lymphoid tissues and their relationship to metastasis. Full Text available with Trip Pro

Histiocytes Hyperplasia Lymphatic Metastasis Lymphoid Tissue pathology Lymphoma Macrophages Neoplasm Transplantation Neoplasms, Experimental Transplantation, Homologous 1966 10 1 1966 10 1 0 1 1966 10 1 0 0 ppublish 5924022 PMC1907246 J Anat. 1958 Jan;92(1):74-83 13513499 Cancer. 1966 Jan;19(1):75-82 5901409 Am J Pathol. 1953 May-Jun;29(3):505-21 13040490 Cancer Res. 1960 Aug;20:1094-100 13851590 Cancer. 1960 May-Jun;13:545-9 14423891 Fed Proc. 1965 Sep-Oct;24(5):1024-9 5320102 Br J Cancer. 1959 Sep;13 (...) Studies on homotransplantable lymphomas in hamsters. I. Histologic responses in lymphoid tissues and their relationship to metastasis. 5924022 1967 01 29 2018 11 13 0002-9440 49 4 1966 Oct The American journal of pathology Am. J. Pathol. Studies on homotransplantable lymphomas in hamsters. I. Histologic responses in lymphoid tissues and their relationship to metastasis. 637-55 Carter R L RL Gershon R K RK eng Journal Article United States Am J Pathol 0370502 0002-9440 AIM IM Animals Cricetinae

1966 The American journal of pathology

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