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Intrauterine Growth Retardation

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121. Mental Retardation (Overview)

to the amount of alcohol ingested. Toxemia of pregnancy and placental insufficiency Intrauterine growth retardation has many causes, the most important being maternal toxemia with its consequences, ending in insult to the CNS. Prematurity may be of maternal or fetal origin. When it is connected with fetal developmental deviations, the prognosis depends on the infant's general condition. Prematurity and especially intrauterine growth retardation predispose to many perinatal complications, which may result (...) deficits. Neonatal bacterial infections might result in sepsis and meningitis, which, in turn, may cause hydrocephalus. Delivery problems During delivery, asphyxia is the most important factor causing an insult to the CNS. It leads to cell death, which might be demonstrated with neuroimaging techniques as leukomalacia. Premature infants and those with intrauterine growth retardation are at special risk for damage to the cortex or thalamus, which, in addition to affecting intelligence, causes various

2014 eMedicine.com

122. Mental Retardation (Treatment)

. The severity of the symptoms is related to the amount of alcohol ingested. Toxemia of pregnancy and placental insufficiency Intrauterine growth retardation has many causes, the most important being maternal toxemia with its consequences, ending in insult to the CNS. Prematurity may be of maternal or fetal origin. When it is connected with fetal developmental deviations, the prognosis depends on the infant's general condition. Prematurity and especially intrauterine growth retardation predispose to many (...) , profound mental retardation, and neurological deficits. Neonatal bacterial infections might result in sepsis and meningitis, which, in turn, may cause hydrocephalus. Delivery problems During delivery, asphyxia is the most important factor causing an insult to the CNS. It leads to cell death, which might be demonstrated with neuroimaging techniques as leukomalacia. Premature infants and those with intrauterine growth retardation are at special risk for damage to the cortex or thalamus, which

2014 eMedicine.com

123. Mental Retardation (Follow-up)

. The severity of the symptoms is related to the amount of alcohol ingested. Toxemia of pregnancy and placental insufficiency Intrauterine growth retardation has many causes, the most important being maternal toxemia with its consequences, ending in insult to the CNS. Prematurity may be of maternal or fetal origin. When it is connected with fetal developmental deviations, the prognosis depends on the infant's general condition. Prematurity and especially intrauterine growth retardation predispose to many (...) , profound mental retardation, and neurological deficits. Neonatal bacterial infections might result in sepsis and meningitis, which, in turn, may cause hydrocephalus. Delivery problems During delivery, asphyxia is the most important factor causing an insult to the CNS. It leads to cell death, which might be demonstrated with neuroimaging techniques as leukomalacia. Premature infants and those with intrauterine growth retardation are at special risk for damage to the cortex or thalamus, which

2014 eMedicine.com

124. Premature newborns with fatal intrauterine herpes simplex virus-1 infection: first report of twins and review of the literature. (PubMed)

twins (27th week of gestation) whose intrauterine growth retardation, fetal anaemia and cardiotocography abnormalities led to a caesarean emergency delivery.Accurate medical history revealed a maternal febrile gingivostomatitis at the 23rd week of gestation, which was neglected by the treating gynaecologist. Respiratory distress was present at delivery and intubation was necessary in both children. The whole skin showed extensive erosions and ulcerations and the mucosa of the eyes and genitals (...) Premature newborns with fatal intrauterine herpes simplex virus-1 infection: first report of twins and review of the literature. Neonates with blistering skin diseases are dermatologic emergencies. The pathologies involved can pose diagnostic difficulties and there exists a variety of potential life-threatening differential diagnoses.description of the first case of intrauterine acquired herpes simplex virus (HSV) 1 infection in twins.We present the case of two premature bicordial biamniotic

2014 Journal of the European Academy of Dermatology and Venereology

125. The effect of intrauterine growth retardation on the expression of developmental factors in porcine placenta subsequent to the initiation of placentation. (PubMed)

The effect of intrauterine growth retardation on the expression of developmental factors in porcine placenta subsequent to the initiation of placentation. Intrauterine growth retardation (IUGR) hinders fetal growth and postnatal development in swine; however the etiology of IUGR is essentially unknown. Expression of fourteen candidate genes associated with placental development or IUGR was examined in gestational day 50 (gd50) control and IUGR fetus whole placental tissue or areolae by real

2010 Placenta

126. Randomized Clinical Trial: Expectant Management vs Laser Treatment of Monochorionic Twins With Severe Selective Intrauterine Growth Retardation and Absent or Reverse Diastolic Flow in the Umbilical Artery

Randomized Clinical Trial: Expectant Management vs Laser Treatment of Monochorionic Twins With Severe Selective Intrauterine Growth Retardation and Absent or Reverse Diastolic Flow in the Umbilical Artery Randomized Clinical Trial: Expectant Management vs Laser Treatment of Monochorionic Twins With Severe Selective Intrauterine Growth Retardation and Absent or Reverse Diastolic Flow in the Umbilical Artery - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer (...) to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Randomized Clinical Trial: Expectant Management vs Laser Treatment of Monochorionic Twins With Severe Selective Intrauterine Growth Retardation and Absent or Reverse Diastolic Flow in the Umbilical Artery The safety and scientific validity of this study

2010 Clinical Trials

127. Pravastatin ameliorates placental vascular defects, fetal growth, and cardiac function in a model of glucocorticoid excess (PubMed)

cord velocity, normalized fetal weight, and improved the cardiac function of Hsd11b2(-/-) fetuses. This improved cardiac function occurred despite persisting indications of increased glucocorticoid exposure in the Hsd11b2(-/-) fetal heart. Thus, the pravastatin-induced enhancement of fetal capillaries within the placenta and the resultant hemodynamic changes correspond with restored fetal cardiac function. Statins may represent a useful therapeutic approach to intrauterine growth retardation due (...) Pravastatin ameliorates placental vascular defects, fetal growth, and cardiac function in a model of glucocorticoid excess Fetoplacental glucocorticoid overexposure is a significant mechanism underlying fetal growth restriction and the programming of adverse health outcomes in the adult. Placental glucocorticoid inactivation by 11β-hydroxysteroid dehydrogenase type 2 (11β-HSD2) plays a key role. We previously discovered that Hsd11b2(-/-) mice, lacking 11β-HSD2, show marked underdevelopment

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2016 Proceedings of the National Academy of Sciences of the United States of America

128. Influence of gestational salt restriction in fetal growth and in development of diseases in adulthood (PubMed)

Influence of gestational salt restriction in fetal growth and in development of diseases in adulthood Recent studies reported the critical role of the intrauterine environment of a fetus in growth or the development of disease in adulthood. In this article we discussed the implications of salt restriction in growth of a fetus and the development of growth-related disease in adulthood. Salt restriction causes retardation of fatal growth or intrauterine death thereby leading to low birth weight (...) or decreased birth rate. Such retardation of growth along with the upregulation of the renin angiotensin system due to salt restriction results in the underdevelopment of cardiovascular organs or decreases the number of the nephron in the kidney and is responsible for onset of hypertension in adulthood. In addition, gestational salt restriction is associated with salt craving after weaning. Moreover, salt restriction is associated with a decrease in insulin sensitivity. A series of alterations

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2016 Journal of biomedical science

129. Learning defects in Drosophila growth restricted chico mutants are caused by attenuated adenylyl cyclase activity (PubMed)

Learning defects in Drosophila growth restricted chico mutants are caused by attenuated adenylyl cyclase activity Reduced insulin/insulin-like growth factor signaling (IIS) is a major cause of symmetrical intrauterine growth retardation (IUGR), an impairment in cell proliferation during prenatal development that results in global growth defects and mental retardation. In Drosophila, chico encodes the only insulin receptor substrate. Similar to other animal models of IUGR, chico mutants have (...) defects in global growth and associative learning. However, the physiological and molecular bases of learning defects caused by chico mutations, and by symmetrical IUGR, are not clear.In this study, we found that chico mutations impair memory-associated synaptic plasticity in the mushroom bodies (MBs), neural centers for olfactory learning. Mutations in chico reduce expression of the rutabaga-type adenylyl cyclase (rut), leading to decreased cAMP synthesis in the MBs. Expressing a rut (+) transgene

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2016 Molecular brain

130. Growth Delay Onset Determination

Determination Growth Delay Onset Determination Aka: Growth Delay Onset Determination From Related Chapters II. Causes: Intrauterine Growth Retardation tic or chromosomal abnormality Transplacental infectious disease ( ) Toxins Nicotine Medications III. Causes: Birth Prolonged Panhypopituitarism IV. Causes: Childhood See Images: Related links to external sites (from Bing) These images are a random sampling from a Bing search on the term "Growth Delay Onset Determination." Click on the image (or right click (...) Growth Delay Onset Determination Growth Delay Onset Determination Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Growth Delay Onset

2018 FP Notebook

131. A Trial Evaluating the Role of Sildenafil in the Treatment of Fetal Growth Restriction

Information provided by (Responsible Party): Ahmed Abdel Shafy El Shahawy, Ain Shams University Study Details Study Description Go to Brief Summary: This is a Randomized Controlled Trial to evaluate the effect of sildenafil on Doppler velocity indices of the umbilical arteries in patients with placental insufficiency and fetal growth restriction, and if sildenafil can improve fetal and neonatal outcomes in those patients. Condition or disease Intervention/treatment Phase Fetal Growth Retardation Drug (...) Years to 40 Years (Adult) Sexes Eligible for Study: Female Accepts Healthy Volunteers: No Criteria Inclusion Criteria: Pregnant women, singleton pregnancy, gestational age 24-34 weeks, with Fetal growth restriction. Intact membranes. Abnormal umbilical artery Doppler waveforms. Fetal abdominal circumference at or below the tenth percentile. Normal venous fetal Doppler Exclusion Criteria: Undetermined gestational age. Intrauterine infection. High Risk for aneuploidy (e.g. maternal age ≥40 years

2015 Clinical Trials

132. Impact of inhaled nitric oxide on white matter damage in growth-restricted neonatal rats. (PubMed)

Impact of inhaled nitric oxide on white matter damage in growth-restricted neonatal rats. Fetal growth restriction is the second leading cause of perinatal morbidity and mortality, and neonates with intrauterine growth retardation (IUGR) have increased neurocognitive and neuropsychiatric morbidity. These neurocognitive impairments are mainly related to injury of the developing brain associated with IUGR. Growing evidence from preclinical models of brain injury in both adult and neonatal rodents

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2015 Pediatric Research

133. Acetylsalicylic acid for the prevention of preeclampsia and intra-uterine growth restriction in women with abnormal uterine artery Doppler: a systematic review and meta-analysis

' gestation. Authors' objectives To assess the influence of gestational age at the time of introduction of low-dose acetylsalicylic acid (ASA) on the incidence of pre-eclampsia and intrauterine growth restriction (IUGR) in women at increased risk on the basis of abnormal uterine artery Doppler. Searching MEDLINE, EMBASE and The Cochrane Library were searched without language restrictions from 1965 to 2008. Search terms were reported. Bibliographies of recent two meta-analyses were handsearched (...) Acetylsalicylic acid for the prevention of preeclampsia and intra-uterine growth restriction in women with abnormal uterine artery Doppler: a systematic review and meta-analysis Acetylsalicylic acid for the prevention of preeclampsia and intra-uterine growth restriction in women with abnormal uterine artery Doppler: a systematic review and meta-analysis Acetylsalicylic acid for the prevention of preeclampsia and intra-uterine growth restriction in women with abnormal uterine artery Doppler

2009 DARE.

134. Intrauterine infection of mice with St. Louis encephalitis virus: immunological, physiological, neurological, and behavioral effects on progeny. (PubMed)

Intrauterine infection of mice with St. Louis encephalitis virus: immunological, physiological, neurological, and behavioral effects on progeny. Intravenous injection of pregnant mice with St. Louis encephalitis (SLE) virus at 8 days of gestation resulted in infection of the fetus. Progeny developed no antibody or tolerance to SLE virus since the viral antigen was cleared by maternal antibody before antibody-forming competence developed in the young. Temporary growth retardation was observed (...) in a number of young at 3 weeks of age. After the initial setback the growth rate increased, indicating that early runting was due to an inability to adjust adequately to extrauterine life, which was subsequently overcome. In most other young there were no significant effects on growth, reproduction, or life expectancy. A few young died at or shortly after birth; in these, neurological changes ranging from gross defects such as encephaloceles and hydrocephalus to histological evidence of necrosis

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1975 Infection and immunity

135. Growth, Safety and Tolerance of a Rice Protein Hydrolysate Formula in Infants With Cow's Milk Protein Allergy

congenital malformations or neonatal diseases Severe concurrent or chronic diseases Intrauterine growth retardation Neonatal infections ( e.g. CMV, HIV) Simultaneous participation in other clinical trials Parents not signing written informed consent Unable to adhere to protocol requirements or study visits due to non compliance of parents or caregivers. Liver, kidney, haematological abnormalities as judged clinically by investigators at baseline. Contacts and Locations Go to Information from the National (...) Growth, Safety and Tolerance of a Rice Protein Hydrolysate Formula in Infants With Cow's Milk Protein Allergy Growth, Safety and Tolerance of a Rice Protein Hydrolysate Formula in Infants With Cow's Milk Protein Allergy - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies

2014 Clinical Trials

136. FACTORS ASSOCIATED WITH GROWTH PATTERNS FROM BIRTH TO EIGHTEEN MONTHS IN A BENINESE COHORT OF CHILDREN. (PubMed)

anthropometric measurements taken at delivery, every month up to 6 months and then quarterly. Infant and young child feeding (IYCF) practices and malarial morbidity were recorded. Gestational age was estimated using the Ballard method; William's sex-specific reference curve of birth weight-for-gestational-age was used to determine intrauterine growth retardation (IUGR). Analyses were performed on 520 children using a linear mixed model. Low birth weight (coef=-0.43; p=0.002), IUGR (coef=-0.49; p<0.001 (...) FACTORS ASSOCIATED WITH GROWTH PATTERNS FROM BIRTH TO EIGHTEEN MONTHS IN A BENINESE COHORT OF CHILDREN. The aim of this study was to analyze factors influencing the growth pattern of children from birth to 18 months. A longitudinal prospective study was conducted in three maternity wards in Southern Benin. Inclusion took place between June 2007 and July 2008; children were followed-up until 18 months of age. Height-for-age and weight-for-height Z-scores were computed using the newborn's

2014 Acta Tropica

137. Evaluation of growth in very low birth weight preterm babies (PubMed)

in the neonatal intensive care unit (NICU) of our hospital were examined. The target height formula was used in assessment of growht in height and the contribution of genetic properties was investigated. The points of the subjects on the growth curve were plotted according to the Percentile Curve of the Turkish Children prepared by Neyzi et al. The states of the subjects with and without intrauterine growth retardation (were compared. The study was intitiated after obtaining approval from the ethics (...) Evaluation of growth in very low birth weight preterm babies The aim of this study was to evaluate physical growth of very low birth weight (VLBW) preterm babies at a mean age of three years and to investigate the factors which affected growth.The factors including maternal problems, prenatal problems, early neonatal problems, nutrition, familial socioeconomical status and presence of chronic disease which affected catch-up growth in terms of height and weight in VLBW infants followed up

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2014 Turkish Archives of Pediatrics/Türk Pediatri Arşivi

138. Homozygous mutation of the Insulin-like Growth Factor-I Receptor gene (IGF1R) in a patient with severe pre- and postnatal growth failure, and congenital malformations. (PubMed)

Homozygous mutation of the Insulin-like Growth Factor-I Receptor gene (IGF1R) in a patient with severe pre- and postnatal growth failure, and congenital malformations. Heterozygous mutations in the IGF1 receptor (IGF1R) gene lead to partial resistance to IGF1 and contribute to intrauterine growth retardation (IUGR) with postnatal growth failure. To date, homozygous mutations of this receptor have not been described.A 13.5-year-old girl born from healthy first-cousin parents presented

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2012 European Journal of Endocrinology

139. Maternal Determinants of HIV-exposed and HIV-unexposed Fetal Growth, Birth Outcomes and Early Infant Growth

month post-partum ] weight, height, mid-upper arm circumference, triceps skinfold, fundal height Fetal growth [ Time Frame: Up to 1 month post-partum ] Pregnancy outcomes [ Time Frame: Up to 1 month post-partum ] spontaneous abortion, stillbirth, preterm delivery, perinatal mortality (death within first 7 days), neonatal mortality (death within first 28 days), small for gestational age, intrauterine growth retardation, low birth weight Early infant anthropometrics [ Time Frame: Up to 1 month post (...) Maternal Determinants of HIV-exposed and HIV-unexposed Fetal Growth, Birth Outcomes and Early Infant Growth Maternal Determinants of HIV-exposed and HIV-unexposed Fetal Growth, Birth Outcomes and Early Infant Growth - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies

2012 Clinical Trials

140. Fetal Growth Restriction (Overview)

is necessary. The ability to diagnose the disorder and understand its pathophysiology still outpaces the ability to prevent or treat its complications. The current therapeutic goals are to optimize the timing of delivery to minimize hypoxemia and maximize gestational age and maternal outcome. Further study may elucidate preventive or treatment strategies to assist the growth-restricted fetus. Previous References Severi FM, Rizzo G, Bocchi C, et al. Intrauterine growth retardation and fetal cardiac function (...) retardation and perinatal death? An overview. BJOG . 2000 Feb. 107(2):196-208. . Mandruzzato GP, Bogatti P, Fischer L, Gigli C. The clinical significance of absent or reverse end-diastolic flow in the fetal aorta and umbilical artery. Ultrasound Obstet Gynecol . 1991 May 1. 1(3):192-6. . Fong KW, Ohlsson A, Hannah ME, et al. Prediction of perinatal outcome in fetuses suspected to have intrauterine growth restriction: Doppler US study of fetal cerebral, renal, and umbilical arteries. Radiology . 1999 Dec

2014 eMedicine.com

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