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Intrauterine Growth Retardation

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61. Sildenafil Citrate for the Management of Asymmetrical Intrauterine Growth Restriction

Sildenafil Citrate for the Management of Asymmetrical Intrauterine Growth Restriction Sildenafil Citrate for the Management of Asymmetrical Intrauterine Growth Restriction - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before (...) adding more. Sildenafil Citrate for the Management of Asymmetrical Intrauterine Growth Restriction The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT02678221 Recruitment Status : Unknown Verified February 2016 by Omar Mamdouh Shaaban, Assiut University. Recruitment status was: Not yet recruiting First

2016 Clinical Trials

62. Effectiveness and cost-effectiveness of routine third trimester ultrasound screening for intrauterine growth restriction: study protocol of a nationwide stepped wedge cluster-randomized trial in The Netherlands (The IRIS Study). Full Text available with Trip Pro

Effectiveness and cost-effectiveness of routine third trimester ultrasound screening for intrauterine growth restriction: study protocol of a nationwide stepped wedge cluster-randomized trial in The Netherlands (The IRIS Study). Intrauterine growth retardation (IUGR) is a major risk factor for perinatal mortality and morbidity. Thus, there is a compelling need to introduce sensitive measures to detect IUGR fetuses. Routine third trimester ultrasonography is increasingly used to detect IUGR (...) practices will be included at 22 weeks of gestation. In the intervention (n = 7,500) and control group (n = 7,500) fetal growth will be monitored by serial fundal height assessments. All practices will start offering the control condition (ultrasonography based on medical indication). Every three months, 20 practices will be randomized to the intervention condition, i.e. apart from ultrasonography if indicated, two routine ultrasound examinations will be performed (at 28-30 weeks and 34-36 weeks

2016 BMC Pregnancy and Childbirth Controlled trial quality: uncertain

63. Impetigo Herpetiformis Complicated with Intrauterine Growth Restriction Treated Successfully with Granulocyte and Monocyte Apheresis. Full Text available with Trip Pro

Impetigo Herpetiformis Complicated with Intrauterine Growth Restriction Treated Successfully with Granulocyte and Monocyte Apheresis. 27573023 2017 03 20 2017 05 05 1651-2057 97 3 2017 03 10 Acta dermato-venereologica Acta Derm. Venereol. Impetigo Herpetiformis Complicated with Intrauterine Growth Restriction Treated Successfully with Granulocyte and Monocyte Apheresis. 410-411 10.2340/00015555-2527 Saito-Sasaki Natsuko N Department of Dermatology, University of Occupational and Environmental (...) Health, 1-1 Iseigaoka, Yahatanishi-ku, Kitakyushu , 807-8555, Japan. natsuko-saito@med.uoeh-u.ac.jp. Izu Kunio K Sawada Yu Y Hino Ryosuke R Nakano Ryoji R Shimajiri Shohei S Nishimura Izumi I Nakamura Hiromasa H Sugiura Kazumitsu K Nakamura Motonobu M eng Case Reports Journal Article Sweden Acta Derm Venereol 0370310 0001-5555 IM Biopsy Dermatitis Herpetiformis blood diagnosis therapy Female Fetal Growth Retardation blood diagnosis immunology therapy Granulocytes immunology Humans Impetigo blood

2016 Acta Dermato-Venereologica

64. Morphology of early intrauterine deaths with full trisomy 15. (Abstract)

miscarriages, and no case of full trisomy 15 was observed in 62 fetal miscarriages. Fifty-eight embryos with full trisomy 15 showed structural defects on embryoscopic examination. The most common defects were craniofacial anomalies (n = 73), retarded development of the limbs (n = 39), and abnormally short umbilical cords closely attaching the embryo to the chorionic plate (n = 27). Seven embryos were classified as growth disorganized. Limb reduction defects with a prevalence of 5.6/10 000 births, all (...) Morphology of early intrauterine deaths with full trisomy 15. The morphologic features of embryos with full trisomy 15 are described.A total of 1195 pregnancy losses were examined embryoscopically and cytogenetically.Of 1173 successfully karyotyped specimens, full trisomy 15 was diagnosed cytogenetically in 59 cases (5%). All 59 trisomy 15 embryos were diagnosed cytogenetically in the group of 962 embryonic miscarriages (6%). Trisomy 15 was not registered in 171 anembryonic or yolk sac

2018 Prenatal diagnosis

65. Intrauterine Growth Retardation

are a random sampling from a Bing search on the term "Intrauterine Growth Retardation." Click on the image (or right click) to open the source website in a new browser window. Related Studies (from Trip Database) Ontology: Fetal Growth Retardation (C0015934) Definition (NCI_CTCAE) A disorder characterized by inhibition of fetal growth resulting in the inability of the fetus to achieve its potential weight. Definition (NCI) A fetus that does not grow beyond the 10th percentile of conventionally accepted (...) Intrauterine Growth Retardation Intrauterine Growth Retardation Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Intrauterine Growth

2015 FP Notebook

66. Maternal Sildenafil vs Placebo in Pregnant Women With Severe Early-Onset Fetal Growth Restriction: A Randomized Clinical Trial Full Text available with Trip Pro

articles References Perined Yearbooks Healthcare in the Netherlands [in Dutch]. Accessed April 22, 2020. Spencer R, Rossi C, Lees M, et al. ; EVERREST Consortium . Achieving orphan designation for placental insufficiency: annual incidence estimations in Europe. BJOG. 2019;126(9):1157-1167. doi:10.1111/1471-0528.15590 - - Severi FM, Rizzo G, Bocchi C, D’Antona D, Verzuri MS, Arduini D. Intrauterine growth retardation and fetal cardiac function. Fetal Diagn Ther. 2000;15(1):8-19. doi:10.1159/000020969 (...) - - Pels A, Beune IM, van Wassenaer-Leemhuis AG, Limpens J, Ganzevoort W. Early-onset fetal growth restriction: a systematic review on mortality and morbidity. Acta Obstet Gynecol Scand. 2020;99(2):153-166. doi:10.1111/aogs.13702 - - - Levine TA, Grunau RE, McAuliffe FM, Pinnamaneni R, Foran A, Alderdice FA. Early childhood neurodevelopment after intrauterine growth restriction: a systematic review. Pediatrics. 2015;135(1):126-141. doi:10.1542/peds.2014-1143 - - Show all 44 references Associated data

2020 EvidenceUpdates

67. An Insight into the Role of the Maxillary Labial Frenum in the Growth of the Maxilla: A Systematic Review Full Text available with Trip Pro

related to the development of face and its abnormal growth may lead to malformations. It serves as a stabilization element , and is influenced by that part of the cartilaginous nasal septum which is united with a fibrous connective tissue band called septopremaxillary ligament. , The maxillary labial frenum is a mucous membrane fold which develops as a posteruptive remnant of tectolabial band at around 8 to 10 weeks of intrauterine life that connects the tubercle of upper lip to the palatine papilla (...) labial frenum, a dynamic structure with a diverse morphology has a physiological importance by housing the septopremaxillary ligament and the pull of the ligament induces alveolar basal bone development and translative growth of the maxilla. Thus, the labial frenum, apart from providing stability to the upper lip, also contributes to the regulation of the midfacial growth. , - In many instances, an aberrant frenum is reported to be associated with syndromic and nonsyndromic conditions with a retarded

2019 Journal of Indian Orthodontic Society

68. Trial on the Impact of Technical Anesthetic in Pregnancies With Intrauterine Growth Restriction

Trial on the Impact of Technical Anesthetic in Pregnancies With Intrauterine Growth Restriction Trial on the Impact of Technical Anesthetic in Pregnancies With Intrauterine Growth Restriction - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one (...) or more studies before adding more. Trial on the Impact of Technical Anesthetic in Pregnancies With Intrauterine Growth Restriction The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. of clinical studies and talk to your health care provider before participating. Read our for details. ClinicalTrials.gov Identifier: NCT02666794 Recruitment Status : Recruiting

2015 Clinical Trials

69. Sildenafil Therapy In Dismal Prognosis Early-Onset Intrauterine Growth Restriction

Sildenafil Therapy In Dismal Prognosis Early-Onset Intrauterine Growth Restriction Sildenafil Therapy In Dismal Prognosis Early-Onset Intrauterine Growth Restriction - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before (...) adding more. Sildenafil Therapy In Dismal Prognosis Early-Onset Intrauterine Growth Restriction (STRIDER Canada) The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT02442492 Recruitment Status : Suspended (STRIDER-Canada trial suspended recruitment until further information can be obtained following

2015 Clinical Trials

70. Leukemia Inhibitory Factor Level in Intrauterine Growth Restriction Neonates

Leukemia Inhibitory Factor Level in Intrauterine Growth Restriction Neonates Leukemia Inhibitory Factor Level in Intrauterine Growth Restriction Neonates - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more (...) . Leukemia Inhibitory Factor Level in Intrauterine Growth Restriction Neonates The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT02518126 Recruitment Status : Unknown Verified August 2015 by Rambam Health Care Campus. Recruitment status was: Not yet recruiting First Posted : August 7, 2015 Last Update

2015 Clinical Trials

71. Evaluation of Infants With Intrauterine Growth Restriction

Study ID Numbers: 142 /181/ 20.05.2014 First Posted: August 4, 2015 Last Update Posted: August 6, 2015 Last Verified: August 2015 Keywords provided by Hășmășanu Monica Gabriela, Iuliu Hatieganu University of Medicine and Pharmacy: IUGR (intrauterine growth restriction) IGF2 (insulin-like growth factor 2) morbidity Additional relevant MeSH terms: Layout table for MeSH terms Fetal Growth Retardation Fetal Diseases Pregnancy Complications Growth Disorders Pathologic Processes (...) Evaluation of Infants With Intrauterine Growth Restriction Evaluation of Infants With Intrauterine Growth Restriction - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Evaluation of Infants With Intrauterine

2015 Clinical Trials

72. Omega-3 supplementation during pregnancy to prevent recurrent intrauterine growth restriction: systematic review and meta-analysis of randomized controlled trials Full Text available with Trip Pro

acid daily on recurrence of intrauterine growth retardation and pregnancy induced hypertension . BJOG 1994 ; 102 : 123 – 126 . 13 Onwude JL , Lilford RJ , Hjartardottir H , Staines A , Tuffnell D . A randomized double blind placebo controlled trial of fish oil in high risk pregnancy . BJOG 1995 ; 102 : 95 – 100 . 14 Malcolm CA , Hamilton R , McCulloch DL , Montgomery C , Weaver LT . Scotopicelectroretinogram in term infants born of mothers supplemented with docosahexaenoic acid during pregnancy (...) labyrinthine antioxidant capacity but does not protect against fetal growth restriction induced by placental ischaemia‐reperfusion injury . Reproduction 2013 ; 146 : 539 – 547 . 50 Andersen HJ , Andersen LF , Fuchs AR . Diet, pre‐eclampsia, and intrauterine growth retardation . Lancet 1989 ; 1 : 1146 . 51 Olsen SF , Olsen J , Frische G . Does fish consumption during pregnancy increase fetal growth? A study of the size of the newborn, placental weight and gestational age in relation to fish consumption

2015 Ultrasound in Obstetrics & Gynecology

73. LOSS OF METHYLATION AT GNAS EXON A/B IS ASSOCIATED WITH INCREASED INTRAUTERINE GROWTH. Full Text available with Trip Pro

of methylation (LOM) at exon A/B alone or sporadic PHP1B (sporPHP1B) associated with broad GNAS methylation changes. Similar to effects other imprinted genes have on early development, we recently observed severe intrauterine growth retardation in newborns, later diagnosed with pseudopseudohypoparathyroidism (PPHP) because of paternal GNAS loss-of-function mutations.This study aimed to determine whether GNAS methylation abnormalities affect intrauterine growth.Birth parameters were collected of patients who (...) LOSS OF METHYLATION AT GNAS EXON A/B IS ASSOCIATED WITH INCREASED INTRAUTERINE GROWTH. GNAS is one of few genetic loci that undergo allelic-specific methylation resulting in the parent-specific expression of at least four different transcripts. Due to monoallelic expression, heterozygous GNAS mutations affecting either paternally or maternally derived transcripts cause different forms of pseudohypoparathyroidism (PHP), including autosomal-dominant PHP type Ib (AD-PHP1B) associated with loss

2015 Journal of Clinical Endocrinology and Metabolism

74. Unstressed antepartum cardiotocography in the management of the fetus suspected of growth retardation. (Abstract)

Unstressed antepartum cardiotocography in the management of the fetus suspected of growth retardation. A detailed analysis of the antenatal cardiotocographs (CTGs) in 57 patients with suspected fetal growth retardation is presented. Four traces were normal and 53 were 'non-reactive. 'Non-reactive' traces can be sub-divided into three categories, which, in order of severity are, 'suspect', 'flat' and 'ominous'. When compared with the 'suspect' group, perinatal mortality was significantly (...) increased and the Apgar scores at one and five minutes significantly decreased in the 'flat' and 'ominous' groups. The presence of fetal distress in labour was significantly higher in the 'flat' group as compared with the 'suspect' group. Intrauterine deaths occurred only in the 'ominous' group. With experience, ten patients later in the series with 'ominous' patterns were delivered within 24 hours of the detection of such and nine infants survived. At follow-up between 6 and 34 months after birth

2014 BJOG

75. Gender-Specific Relationship Between Prenatal Exposure to Phthalates and Intrauterine Growth Restriction. Full Text available with Trip Pro

Gender-Specific Relationship Between Prenatal Exposure to Phthalates and Intrauterine Growth Restriction. No study has examined the association between prenatal phthalate exposure and intrauterine growth retardation (IUGR). This study aimed to investigate whether prenatal exposure to phthalates was associated with increased risk of IUGR.A total of 126 mother-newborn pairs, including 42 IUGR cases and 84 control newborns and their mothers, were enrolled in this case-control study. Spot urine (...) significantly inversely associated with fetal growth indicators (birth weight and Quetelet's index). When mothers were stratified by infant sex, MEHHP and MEOHP concentrations were still negatively associated with fetal growth indicators, while no significant association was observed in females. In addition, exposure-response relationships were observed between MEHHP/SumDEHP concentrations in maternal urine and IUGR.Prenatal exposure to phthalates was associated with increased risk of IUGR, and male

2014 Pediatric Research

76. Trans-ethnic meta-analysis of genome-wide association studies identifies maternal ITPR1 as a novel locus influencing fetal growth during sensitive periods in pregnancy Full Text available with Trip Pro

are nonlinear, and the growth patterns vary by gestation time [ , ]. Offspring birthweight alone cannot represent the pattern of intrauterine growth at different times during gestation [ ]. Previous studies have demonstrated that the genetic contribution to fetal growth, relative to in-utero environmental influences, varies over gestation [ – ]. Therefore, different genetic loci may influence fetal growth at different times of gestation, and the same genetic locus may have different effects on fetal growth (...) birthweight [ – ]. Using a method that partitions SNP heritability into maternal and fetal components [ ], 7.6% and 28.5% of the variance in birthweight was attributed to maternal and fetal SNPs on the genotyping arrays, respectively [ ]. Maternal genetic contributions to offspring birthweight can be due to maternally inherited offspring risk alleles or the influence of maternal genotypes on components of the intrauterine environment [ , ]. Identifying maternal genetic loci that influence fetal growth

2020 PLoS genetics

77. An approach to solving problems of growth retardation in the child and teenager. Full Text available with Trip Pro

An approach to solving problems of growth retardation in the child and teenager. The physician who looks after children and teenagers is often confronted with the problem of short stature or growth failure. Common causes of growth failure include genetic background, intrauterine disease, malnutrition, chronic illness and hormonal disorders; some cases are attributed to mental retardation or primary central nervous system disease. A major concern in the evaluation of these patients is when

1977 Canadian Medical Association Journal

78. Association of hereditary thrombophilia with intrauterine growth restriction Full Text available with Trip Pro

Association of hereditary thrombophilia with intrauterine growth restriction Intrauterine growth retardation (IUGR) contributes significantly to fetal morbidity and mortality, but its etiology is unknown in most cases.The aim of this study was to examine the association between inherited thrombophilia and IUGR.A case-control study was performed in a tertiary referral center (Afzalipour Hospital) over 2-years period (2010-2011). Cases (n=25) were women who had pregnancies complicated by IUDR (...) and control subjects (n=25) were women who had normal growth fetuses. All women were tested for inherited thrombophilia at least 4 weeks after delivery. Main outcome measure was prevalence of maternal thrombophlia. Genotyping for factor V Leiden, prothrombin gene (nucleotide G20210A), and MTHFR (C677T) mutation was performed by PCR technique. Protein C, S and antithrombin III activity were determined with a clotting assay (STA-Staclot, France).The prevalence of hereditary thrombophilia was 68% (n=17

2013 Iranian Journal of Reproductive Medicine

79. Influence of Intrauterine Growth Restriction on Amplitude-integrated EEG in Preterm Infants

Influence of Intrauterine Growth Restriction on Amplitude-integrated EEG in Preterm Infants Influence of Intrauterine Growth Restriction on Amplitude-integrated EEG in Preterm Infants - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more (...) studies before adding more. Influence of Intrauterine Growth Restriction on Amplitude-integrated EEG in Preterm Infants The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT01942525 Recruitment Status : Unknown Verified September 2013 by Katrin Klebermass-Schrehof, Medical University of Vienna. Recruitment

2013 Clinical Trials

80. Glucocorticoid mediates prenatal caffeine exposure-induced endochondral ossification retardation and its molecular mechanism in female fetal rats Full Text available with Trip Pro

Glucocorticoid mediates prenatal caffeine exposure-induced endochondral ossification retardation and its molecular mechanism in female fetal rats Our previous studies discovered that prenatal caffeine exposure (PCE) could induce intrauterine growth retardation (IUGR) and long-bone dysplasia in offspring rats, accompanied by maternal glucocorticoid over-exposure. This study is to explore whether intrauterine high glucocorticoid level can cause endochondral ossification retardation and clarify (...) its molecular mechanism in PCE fetal rats. Pregnant Wistar rats were intragastrically administered 30 and 120 mg/kg day of caffeine during gestational days (GDs) 9-20, then collected fetal serum and femurs at GD20. In vitro, primary chondrocytes were treated with corticosterone (0-1250 nM), caffeine (0-100 μM), mitogen-inducible gene 6 (Mig-6) siRNA and epidermal growth factor receptor (EGFR) siRNA, respectively, or together. Results showed that the hypertrophic chondrocytes zone (HZ) of PCE fetal

2017 Cell death & disease

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