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Hypertrichosis

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101. Hirsutism and Virilization Full Text available with Trip Pro

Hirsutism and Virilization In normal females, androstenedione from both the adrenal cortex and ovary, as a result of peripheral conversion, is the source of the majority of biologically active testosterone in the circulation. The control of the secretion of precursor steroid and androgenic hormone (testosterone) in females is not clear at this time. There are a number of possibilities to explain various types of hirsutism and virilization. The presence of true virilization indicates (...) a significant disorder and requires complete investigation. The presence of increased amounts of 17-ketosteroids in the urine implicates the adrenal cortex as a source of the pathologic manifestations. The suppressibility of elevated 17-ketosteroids with cortisol analogues aids in distinguishing between adrenal hyperplasia and autonomous neoplasm of the adrenal cortex. By far the most common entity in this area is simple hirsutism without virilization. Although our knowledge of this disorder is quite

1967 California Medicine

102. Testosterone and Androstenedione Blood Production Rates in Normal Women and Women with Idiopathic Hirsutism or Polycystic Ovaries Full Text available with Trip Pro

Testosterone and Androstenedione Blood Production Rates in Normal Women and Women with Idiopathic Hirsutism or Polycystic Ovaries The average plasma testosterone concentration of women with either hirsutism or polycystic ovaries and hirsutism was higher (p < 0.01) than that of normal women although the ranges overlapped. Testosterone blood production rates averaged 830 +/- 120 SE and 1,180 +/- 310 SE mug per day in the two groups of hirsute women and 230 +/- 33 SE mug per day in normal women (...) . The ranges did not overlap. The testosterone metabolic clearance rates of hirsute women (1,090 +/- 140 SE L per day) and of men (1,240 +/- 136 SE L per day) were significantly higher than those of normal women (590 +/- 44 SE L per day). These differences persisted when the metabolic clearance rates were corrected for surface area. We suggest that testosterone metabolic clearance rates vary directly with some function of testosterone production. The mean plasma androstenedione levels (2.8 +/- 0.35 SE

1967 Journal of Clinical Investigation

103. URINARY 17-KETOSTEROID CONJUGATES IN HIRSUTISM Full Text available with Trip Pro

URINARY 17-KETOSTEROID CONJUGATES IN HIRSUTISM 14471603 1998 11 01 2018 12 01 0021-9738 41 1962 Aug The Journal of clinical investigation J. Clin. Invest. Urinary 17-ketosteroid conjugates in hirsutism. 1690-8 MAUVAIS-JARVIS P P BAULIEU E E EE eng Journal Article United States J Clin Invest 7802877 0021-9738 0 17-Ketosteroids OM 17-Ketosteroids urine Body Fluids Hirsutism Humans Hypertrichosis urine 17-KETOSTEROIDS/urine HYPERTRICHOSIS/urine 1962 8 1 1962 8 1 0 1 1962 8 1 0 0 ppublish 14471603

1962 Journal of Clinical Investigation

104. Steroid Therapy in Hirsutism and Virilism Full Text available with Trip Pro

Steroid Therapy in Hirsutism and Virilism 14424812 1998 11 01 2018 12 01 0007-1447 2 5150 1959 Sep 19 British medical journal Br Med J Steroid therapy in hirsutism and virilism. 456-9 de MOWBRAY R R SPENCE A W AW MEDVEI V C VC ROBINSON A M AM eng Journal Article England Br Med J 0372673 0007-1447 0 Adrenal Cortex Hormones OM Adrenal Cortex Hormones therapy Female Hirsutism Humans Hypertrichosis therapy Virilism therapy ADRENAL CORTEX HORMONES/therapy HYPERTRICHOSIS/therapy VIRILISM/therapy 1959

1959 British medical journal

105. Adrenal Function in Amenorrhœa, Infertility and Hirsutism: Effect of Prednisone Therapy Full Text available with Trip Pro

Amenorrhea urine Female Hirsutism Humans Hypertrichosis urine Infertility Infertility, Female urine Prednisone therapy ADRENAL CORTEX HORMONES/urine AMENORRHEA/urine HYPERTRICHOSIS/urine PREDNISONE/therapy STERILITY, FEMALE/urine 1959 11 1 1959 11 1 0 1 1959 11 1 0 0 ppublish 13838779 PMC1831155 Metabolism. 1954 Nov;3(6):489-92 13213629 J Clin Invest. 1958 Jun;37(6):794-9 13549590 Biochem J. 1938 Aug;32(8):1312-31 16746757 (...) Adrenal Function in Amenorrhœa, Infertility and Hirsutism: Effect of Prednisone Therapy 13838779 1998 11 01 2018 12 01 0008-4409 81 1959 Nov 01 Canadian Medical Association journal Can Med Assoc J Adrenal function in amenorrhoea, infertility and hirsutism: effect of prednisone therapy. 714-6 TOMPKINS M G MG MORSE W I WI BRENNAN C F CF STEWART L L eng Journal Article Canada Can Med Assoc J 0414110 0008-4409 0 Adrenal Cortex Hormones VB0R961HZT Prednisone OM Adrenal Cortex Hormones urine

1959 Canadian Medical Association Journal

106. Symposium on Adolescent Gynecology and Endocrinology. Part II: Secondary amenorrhea, hirsutism in adolescents and the clinical consequences of stilbestrol exposure in utero. Secondary amenorrhea. Full Text available with Trip Pro

Symposium on Adolescent Gynecology and Endocrinology. Part II: Secondary amenorrhea, hirsutism in adolescents and the clinical consequences of stilbestrol exposure in utero. Secondary amenorrhea. 575686 1980 07 12 2008 11 20 0093-0415 131 6 1979 Dec The Western journal of medicine West. J. Med. Symposium on Adolescent Gynecology and Endocrinology. Part II: Secondary amenorrhea, hirsutism in adolescents and the clinical consequences of stilbestrol exposure in utero. Secondary amenorrhea. 516-22

1979 Western Journal of Medicine

107. Symposium on Adolescent Gynecology and Endocrinology. Part II: Secondary amenorrhea, hirsutism in adolescents and the clinical consequences of stilbestrol exposure in utero. The clinical consequences of stilbestrol exposure in utero. Full Text available with Trip Pro

Symposium on Adolescent Gynecology and Endocrinology. Part II: Secondary amenorrhea, hirsutism in adolescents and the clinical consequences of stilbestrol exposure in utero. The clinical consequences of stilbestrol exposure in utero. 545869 1980 07 12 2018 11 13 0093-0415 131 6 1979 Dec The Western journal of medicine West. J. Med. Symposium on Adolescent Gynecology and Endocrinology. Part II: Secondary amenorrhea, hirsutism in adolescents and the clinical consequences of stilbestrol exposure

1979 Western Journal of Medicine

108. Congenital Hypertrichosis Lanuginosa (Treatment)

. Arch Dermatol . 2001 Jul. 137(7):877-84. . Chen W, Ring J, Happle R. Congenital generalized hypertrichosis terminalis: a proposed classification and a plea to avoid the ambiguous term "Ambras syndrome". Eur J Dermatol . 2015 May-Jun. 25 (3):223-7. . Chanukvadze D, Kristesashvili J. Effectiveness of different diagnostic methods for assessment of hyperandrogenism in young women with hirsutism. Georgian Med News . 2011 Nov. 25-9. . Hizli D, Kösüs A, Kösüs N, Kamalak Z, Ak D, Turhan NO. The impact (...) Nov. 39 (11):1054-1068. . Mihailidis J, Dermesropian R, Taxel P, Luthra P, Grant-Kels JM. Endocrine evaluation of hirsutism. Int J Womens Dermatol . 2017 Mar. 3 (1 Suppl):S6-S10. . . Hafsi W, Badri T. Hirsutism. StatPearls [Internet] . 2018 January. Treasure Island, Fla: . . Bou-Assi E, Bonniaud B, Grimaldi M, Faivre L, Vabres P. Neonatal Cutis Laxa and Hypertrichosis Lanuginosa in Sotos Syndrome. Pediatr Dermatol . 2016 Nov. 33 (6):e351-e352. . Mendiratta V, Harjai B, Gupta T. Hypertrichosis

2014 eMedicine.com

109. Congenital Hypertrichosis Lanuginosa (Overview)

(Dilantin), streptomycin, hexachlorobenzene, penicillamine, heavy metals, sodium tetradecyl sulfate, acetazolamide, and interferon. The hypertrichosis seen in Ambras syndrome is believed to result either from an increase in the number of hairs in anagen or from an increased number of follicular units, although no one knows for certain. [ ] Alternatively, commonly occurs in women and presents as androgen-induced male-pattern hair growth of the terminal type. [ , , , ] Hirsutism may have a congenital (...) or exogenous origin. More common causes of hirsutism include , idiopathic hirsutism, , , and medications (eg, danazol, androgenic oral contraceptives). [ ] Less common causes of hirsutism include , ovarian tumors, Sertoli-Leydig cell tumors, granulosa–thecal cell tumors, other tumors that stimulate the ovarian stroma, adrenal tumors, , tumors of the adrenal cortex, and severe insulin-resistance syndromes. [ ] Hypertrichosis lanuginosa and transient neonatal cutis laxa have been described as the initial

2014 eMedicine.com

110. Congenital Hypertrichosis Lanuginosa (Follow-up)

. Arch Dermatol . 2001 Jul. 137(7):877-84. . Chen W, Ring J, Happle R. Congenital generalized hypertrichosis terminalis: a proposed classification and a plea to avoid the ambiguous term "Ambras syndrome". Eur J Dermatol . 2015 May-Jun. 25 (3):223-7. . Chanukvadze D, Kristesashvili J. Effectiveness of different diagnostic methods for assessment of hyperandrogenism in young women with hirsutism. Georgian Med News . 2011 Nov. 25-9. . Hizli D, Kösüs A, Kösüs N, Kamalak Z, Ak D, Turhan NO. The impact (...) Nov. 39 (11):1054-1068. . Mihailidis J, Dermesropian R, Taxel P, Luthra P, Grant-Kels JM. Endocrine evaluation of hirsutism. Int J Womens Dermatol . 2017 Mar. 3 (1 Suppl):S6-S10. . . Hafsi W, Badri T. Hirsutism. StatPearls [Internet] . 2018 January. Treasure Island, Fla: . . Bou-Assi E, Bonniaud B, Grimaldi M, Faivre L, Vabres P. Neonatal Cutis Laxa and Hypertrichosis Lanuginosa in Sotos Syndrome. Pediatr Dermatol . 2016 Nov. 33 (6):e351-e352. . Mendiratta V, Harjai B, Gupta T. Hypertrichosis

2014 eMedicine.com

111. Congenital Hypertrichosis Lanuginosa (Diagnosis)

(Dilantin), streptomycin, hexachlorobenzene, penicillamine, heavy metals, sodium tetradecyl sulfate, acetazolamide, and interferon. The hypertrichosis seen in Ambras syndrome is believed to result either from an increase in the number of hairs in anagen or from an increased number of follicular units, although no one knows for certain. [ ] Alternatively, commonly occurs in women and presents as androgen-induced male-pattern hair growth of the terminal type. [ , , , ] Hirsutism may have a congenital (...) or exogenous origin. More common causes of hirsutism include , idiopathic hirsutism, , , and medications (eg, danazol, androgenic oral contraceptives). [ ] Less common causes of hirsutism include , ovarian tumors, Sertoli-Leydig cell tumors, granulosa–thecal cell tumors, other tumors that stimulate the ovarian stroma, adrenal tumors, , tumors of the adrenal cortex, and severe insulin-resistance syndromes. [ ] Hypertrichosis lanuginosa and transient neonatal cutis laxa have been described as the initial

2014 eMedicine.com

112. Hypomelanosis of Ito and multiple naevoid hypertrichosis: Rare cutaneous mosaicism. (Abstract)

Hypomelanosis of Ito and multiple naevoid hypertrichosis: Rare cutaneous mosaicism. Hypomelanosis of Ito and naevoid hypertrichosis are both uncommon disorders and their coexistence in the same patient is rarely described. Most previously reported patients, in addition to cutaneous lesions, had multiple neurological, musculoskeletal and other organ system involvements. We present the case of a 3-year-old girl with these two naevoid disorders at sites completely exclusive of each other

2013 Australasian Journal of Dermatology

113. Hypertrichosis

at inappropriate locations, such as on the extremities, the head, and the back. It is caused by genetic or acquired factors, and is an androgen-independent process. This concept does not include HIRSUTISM which is an androgen-dependent excess hair growth in WOMEN and CHILDREN. Concepts Disease or Syndrome ( T047 ) MSH ICD10 , , SnomedCT 156409007 , 201164001 , 40090008 , 201165000 , 29966009 , 271607001 English Hypertrichoses , Hypertrichosis , HYPERTRICHOSIS , Hypertrichosis, unspecified , hypertrichosis (...) (diagnosis) , hypertrichosis , excess hair , Hypertrichosis [Disease/Finding] , excessive hairiness , polytrichia (diagnosis) , hypertrichosis polytrichia , polytrichia , Hypertrichosis NOS (disorder) , Pilosis , Excessive hair growth (disorder) , Polytrichia , Excessive hair growth , Hirsutism - hypertrichosis , Excessive hairiness , Excessive hair growth (finding) , Hypertrichiasis , Hypertrichosis (disorder) , Polytrichosis , Excessive growth of hair , Hairiness , excessive; hair growth

2015 FP Notebook

114. Sequence and structure based assessment of nonsynonymous SNPs in hypertrichosis universalis Full Text available with Trip Pro

Sequence and structure based assessment of nonsynonymous SNPs in hypertrichosis universalis Hairs are complex structures, making a protective layer and serves different biological functions. TRPS1, a transcription factor is one of the candidate genes causing congenital hypertrichosis, an excessive hair growth at inappropriate body parts. SNPs of TRPS1 were retrieved from dbSNP which were screened by SIFT and PolyPhen servers based on their functional impacts. Out of the screened SNPs (...) properties of TRPS1 protein. It can thus be concluded that the changed properties due to these single nucleotide polymorphisms effect the interactions of TRPS1 which result in congenital hypertrichosis.

2012 Bioinformation

115. Localized Acquired Hypertrichosis Associated with the Application of a Splint Full Text available with Trip Pro

Localized Acquired Hypertrichosis Associated with the Application of a Splint We describe a 16-year-old boy whose left forearm and hand were cut by a piece of glass from a broken window as a result of the fall. He had surgical repair of his left extensor pollicis brevis, abductor pollicis brevis, and dorsal branch of the left radial nerve. Following the surgery, he was put on a splint so as to immobilize the left forearm and wrist. On removal of the splint 4 weeks post surgery, he was noticed (...) to have more hair growth on his left forearm and hand than his right counterparts. The patient was reassessed 2, 4, and 8 months after the removal of the splint. The hypertrichosis got better with time. At the last visit, the hair growth in the left forearm and hand was back to normal. Our patient represents the first reported case of localized acquired hypertrichosis following the application of a splint in the pediatric literature.

2012 Case reports in pediatrics

116. High Frequency of Hypertrichosis after Cast Application. (Abstract)

High Frequency of Hypertrichosis after Cast Application. Hypertrichosis following cast application is commonly observed after removing casts, due to an unknown mechanism.To determine the frequency and associated demographic and clinical risk factors of postcast hypertrichosis in patients who underwent cast application for bone fractures.The study included 117 patients (50 females, 67 males, age range: 3-91 years). Demographic information of each patient (age, gender, fracture localization, type (...) of cast, cast duration and pruritus) was recorded. After removing the cast, the presence of hypertrichosis, contact dermatitis and lymphedema of the cast area was evaluated. Demographic and clinical features of the patients were analyzed to determine the risk factors for postcast hypertrichosis.Postcast hypertrichosis was detected in 34.2% of patients. Patients with hypertrichosis were younger than those without hypertrichosis (p = 0.015). Duration of cast application, session of cast application

2012 Dermatology

117. Recurrent Peptic Ulcers with General Hypertrichosis Including Growth of Hair on the Bald Scalp and Later Development of Signs Suggesting Adrenal Failure Full Text available with Trip Pro

Recurrent Peptic Ulcers with General Hypertrichosis Including Growth of Hair on the Bald Scalp and Later Development of Signs Suggesting Adrenal Failure 14834171 2004 02 15 2018 12 01 0035-9157 44 2 1951 Feb Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Recurrent peptic ulcers with general hypertrichosis including growth of hair on the bald scalp and later development of signs suggesting adrenal failure. 155-7 LE MARQUAND H S HS BOHN G L GL eng Journal Article England Proc R (...) Soc Med 7505890 0035-9157 OM Alopecia Hair Humans Hypertrichosis Peptic Ulcer Scalp 5120:66823:142:407 HAIR PEPTIC ULCER 1951 2 1 1951 2 1 0 1 1951 2 1 0 0 ppublish 14834171 PMC2081631

1951 Proceedings of the Royal Society of Medicine

118. Hypertrichosis Lanuginosa, Acquired Type Full Text available with Trip Pro

Hypertrichosis Lanuginosa, Acquired Type 14864552 2004 02 15 2018 12 01 0035-9157 44 7 1951 Jul Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Hypertrichosis lanuginosa, acquired type. 576-7 LYELL A A WHITTLE C H CH eng Journal Article England Proc R Soc Med 7505890 0035-9157 OM Hair Humans Hypertrichosis 5120:98153:407 HAIR 1951 7 1 1951 7 1 0 1 1951 7 1 0 0 ppublish 14864552 PMC2081833 Proc R Soc Med. 1951 Feb;44(2):155-7 14834171

1951 Proceedings of the Royal Society of Medicine

119. Hypertrichosis Full Text available with Trip Pro

Hypertrichosis 15420429 2004 09 30 2018 12 01 0007-1447 1 4663 1950 May 20 British medical journal Br Med J Hypertrichosis: a report of three cases. 1171-4 BROSTER L R LR eng Journal Article England Br Med J 0372673 0007-1447 OM Hair Hirsutism Humans Hypertrichosis Neoplasms, Hormone-Dependent therapy 5019:10386:77:103 ENDOCRINE THERAPY HAIR 1950 5 20 1950 5 20 0 1 1950 5 20 0 0 ppublish 15420429 PMC2037790 Br Med J. 1949 Jul 16;2(4619):113-7 18134844

1950 British medical journal

120. Case of Congenital Hypertrichosis Full Text available with Trip Pro

Case of Congenital Hypertrichosis 19981676 2010 06 24 2010 06 24 0035-9157 14 Dermatol Sect 1921 Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Case of Congenital Hypertrichosis. 84-5 Sequeira J H JH eng Journal Article England Proc R Soc Med 7505890 0035-9157 2009 12 9 6 0 1921 1 1 0 0 1921 1 1 0 1 ppublish 19981676 PMC2153395

1921 Proceedings of the Royal Society of Medicine

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