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Hypertension Causes

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141. ISH AHA-2 A CASE OF CHRONIC INDOLENT PHEOCHROMOCYTOMA WHICH CAUSED MEDICALLY-CONTROLLED HYPERTENSION BUT TREATMENT-RESISTANT DIABETES MELLITUS. (PubMed)

ISH AHA-2 A CASE OF CHRONIC INDOLENT PHEOCHROMOCYTOMA WHICH CAUSED MEDICALLY-CONTROLLED HYPERTENSION BUT TREATMENT-RESISTANT DIABETES MELLITUS.

2016 Journal of Hypertension

143. Selective Deletion of the Brain-Specific Isoform of Renin Causes Neurogenic Hypertension. (PubMed)

Selective Deletion of the Brain-Specific Isoform of Renin Causes Neurogenic Hypertension. The renin-angiotensin system (RAS) in the brain is a critical determinant of blood pressure, but the mechanisms regulating RAS activity in the brain remain unclear. Expression of brain renin (renin-b) occurs from an alternative promoter-first exon. The predicted translation product is a nonsecreted enzymatically active renin whose function is unknown. We generated a unique mouse model by selectively (...) , despite an ablation of renin-b expression, expression of renin-a was significantly increased in rostral ventrolateral medulla. These data support a new paradigm for the genetic control of RAS activity in the brain by a coordinated regulation of the renin isoforms, with expression of renin-b tonically inhibiting expression of renin-a under baseline conditions. Impairment of this control mechanism causes neurogenic hypertension.© 2016 American Heart Association, Inc.

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2016 Hypertension

144. Sodium Intake and All-Cause Mortality Over 20 Years in the Trials of Hypertension Prevention. (PubMed)

Sodium Intake and All-Cause Mortality Over 20 Years in the Trials of Hypertension Prevention. The relationship between lower sodium intake and total mortality remains controversial.This study examined the relationship between well-characterized measures of sodium intake estimated from urinary sodium excretion and long-term mortality.Two trials, phase I (1987 to 1990), over 18 months, and phase II (1990 to 1995), over 36 months, were undertaken in TOHP (Trials of Hypertension Prevention), which (...) implemented sodium reduction interventions. The studies included multiple 24-h urine samples collected from pre-hypertensive adults 30 to 54 years of age during the trials. Post-trial deaths were ascertained over a median 24 years, using the National Death Index. The associations between mortality and the randomized interventions as well as with average sodium intake were examined.Among 744 phase I and 2,382 phase II participants randomized to sodium reduction or control, 251 deaths occurred, representing

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2016 Journal of the American College of Cardiology

145. Conditional Deletion of Hsd11b2 in the Brain Causes Salt Appetite and Hypertension. (PubMed)

Conditional Deletion of Hsd11b2 in the Brain Causes Salt Appetite and Hypertension. The hypertensive syndrome of Apparent Mineralocorticoid Excess is caused by loss-of-function mutations in the gene encoding 11β-hydroxysteroid dehydrogenase type 2 (11βHSD2), allowing inappropriate activation of the mineralocorticoid receptor by endogenous glucocorticoid. Hypertension is attributed to sodium retention in the distal nephron, but 11βHSD2 is also expressed in the brain. However, the central (...) activity in the brain does not intrinsically cause hypertension, but it promotes a hunger for salt and a transition from salt resistance to salt sensitivity. Our data suggest that 11βHSD2-positive neurons integrate salt appetite and the blood pressure response to dietary sodium through a mineralocorticoid receptor-dependent pathway. Therefore, central mineralocorticoid receptor antagonism could increase compliance to low-sodium regimens and help blood pressure management in cardiovascular disease.©

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2016 Circulation

146. Meta-analysis on the risk of all-cause mortality and cardiovascular death in the early stage of hypertension. (PubMed)

Meta-analysis on the risk of all-cause mortality and cardiovascular death in the early stage of hypertension. To evaluate the relationship among the early stage of hypertension, cardiovascular death, the mortality of coronary heart disease and stroke. Two researchers searched online data of PubMed, Embase and Cochrane library databases and other related papers and manual retrieval conference papers. A prospective cohort study of relative risks and 95% CIs about the comparison with ideal blood (...) pressure, the pre-hypertension and the all-cause mortality or the death of cardiovascular that corrected a variety of risk factors. Compared with ideal blood pressure, the corrected risk factors, the pre-hypertension couldn't increase the RR of the all caused mortality; but it could increase remarkably the mortality of cardiovascular, coronary heart disease and stroke, and there was a significant difference between the two later (P<0.001). Compared with the ideal blood pressure, the pre-hypertension

2016 Pakistan journal of pharmaceutical sciences

147. Brief Report: Patterns of End Stage Renal Disease Caused by Diabetes, Hypertension, and Glomerulonephritis in Live Kidney Donors. (PubMed)

Brief Report: Patterns of End Stage Renal Disease Caused by Diabetes, Hypertension, and Glomerulonephritis in Live Kidney Donors. Inferences about late risk of end-stage renal disease (ESRD) in live kidney donors have been extrapolated from studies averaging <10 years of follow-up. Because early (<10 years) and late (≥10 years) postdonation ESRD may differ by causal mechanism, it is possible that extrapolations are misleading. To better understand postdonation ESRD, we studied patterns (...) of common etiologies including diabetes, hypertension and glomerulonephritis (GN; as reported by providers) using donor registry data linked to ESRD registry data. Overall, 125 427 donors were observed for a median of 11.0 years (interquartile range 5.3-15.7 years; maximum 25 years). The cumulative incidence of ESRD increased from 10 events per 10 000 at 10 years after donation to 85 events per 10 000 at 25 years after donation (late vs. early ESRD, adjusted for age, race and sex: incidence rate ratio

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2016 American Journal of Transplantation

148. The association between orthostatic hypertension and all-cause mortality in hospitalized elderly persons (PubMed)

The association between orthostatic hypertension and all-cause mortality in hospitalized elderly persons Little is known about the prevalence of orthostatic hypertension (OHT) and its effect on long-term mortality in the elderly. We evaluated the prevalence of OHT and its effect on mortality in hospitalized elderly patients.Out of 1852 patients admitted between 31/12/1999 and 31/12/2000 to an acute geriatric ward, 474 patients (48% males) with a mean age of 81.5 ± 6.8 years were enrolled

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2016 Journal of geriatric cardiology : JGC

149. Multiple Giant Splenic Artery Aneurysms Causing Sinistral (Left-Sided) Portal Hypertension (PubMed)

Multiple Giant Splenic Artery Aneurysms Causing Sinistral (Left-Sided) Portal Hypertension Background. Splenic artery aneurysm is the most common type of visceral aneurysms. They are usually asymptomatic and have a potential for rupture and therefore life-threatening hemorrhage. It is rare for them to cause sinistral portal hypertension. Case Report. A 23-year-old female patient presented to our clinic with gastric varices, splenomegaly, pancytopenia, and normal liver functions. She was thus (...) diagnosed with left-sided portal hypertension. Radiologic evaluation showed splenomegaly, splenic vein obstruction, and multiple aneurysms along the splenic artery ranging from 2.5 cm to 7 cm. Splenic artery aneurysm was thought to be the cause of portal hypertension and hypersplenism. We decided splenectomy is the best course of treatment. Pancytopenia could not be corrected preoperatively despite the transfusion treatment. Surgical exploration revealed multiple aneurysms deeply embedded in pancreas

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2016 Case reports in gastrointestinal medicine

150. Raised Cecal Veillonella (Firmicutes)/S 24-7 (Bacteriodetes) May Not Cause Salt-Sensitive Hypertension (PubMed)

Raised Cecal Veillonella (Firmicutes)/S 24-7 (Bacteriodetes) May Not Cause Salt-Sensitive Hypertension 27065886 2016 04 12 2018 11 13 1664-042X 7 2016 Frontiers in physiology Front Physiol Raised Cecal Veillonella (Firmicutes)/S 24-7 (Bacteriodetes) May Not Cause Salt-Sensitive Hypertension. 118 10.3389/fphys.2016.00118 Chaudhury Arun A GIM Foundation Little Rock, AR, USA. eng Journal Article 2016 03 31 Switzerland Front Physiol 101549006 1664-042X causal link hypertension microbiome 2015 12 03 (...) 2016 03 15 2016 4 12 6 0 2016 4 12 6 0 2016 4 12 6 1 epublish 27065886 10.3389/fphys.2016.00118 PMC4814461 Eur J Gastroenterol Hepatol. 2012 Apr;9 Suppl 1:S3-6; discussion S6-7 22498905 Hypertension. 2015 Jun;65(6):1331-40 25870193 PLoS One. 2012;7(6):e36466 22719832 Philos Trans R Soc Lond B Biol Sci. 2015 Aug 19;370(1675):null 26150664 Vet Clin North Am Exot Anim Pract. 2009 May;12(2):197-208, xiii 19341948 Hypertension. 2014 Oct;64(4):897-903 25047574 Front Endocrinol (Lausanne). 2014 Jun 13;5

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2016 Frontiers in physiology

151. Disparities in socioeconomic status and neighborhood characteristics affect all-cause mortality in patients with newly diagnosed hypertension in Korea: a nationwide cohort study, 2002–2013 (PubMed)

Disparities in socioeconomic status and neighborhood characteristics affect all-cause mortality in patients with newly diagnosed hypertension in Korea: a nationwide cohort study, 2002–2013 Previous studies have shown that contextual factors and individual socioeconomic status (SES) were associated with mortality in Western developed countries. In Korea, there are few empirical studies that have evaluated the association between SES and health outcomes.We conducted cohort study to investigate (...) the socioeconomic disparity in all-cause mortality for patients newly diagnosed with hypertension in the setting of universal health care coverage. We used stratified random sample of Korean National Health Insurance enrollees (2002-2013). We included patients newly diagnosed with hypertension (n = 28,306) from 2003-2006, who received oral medication to control their hypertension. We generated a frailty model using Cox's proportional hazard regression to assess risk factors for mortality.A total of 7,825 (27.6

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2016 International journal for equity in health

152. Focal stenosis of the sigmoid sinus causing intracranial venous hypertension: Case report, endovascular management, and review of the literature (PubMed)

Focal stenosis of the sigmoid sinus causing intracranial venous hypertension: Case report, endovascular management, and review of the literature Regardless of the underlying pathology, elevated intracranial pressure is the endpoint of any impairment in either cerebrospinal fluid (CSF) absorption (including arachnoid villi) or intracranial venous drainage. In all age groups, the predominant final common pathway for CSF drainage is the dural venous sinus system. Intracranial venous hypertension (...) (ICVH) is an important vascular cause of intracranial hypertension (and its subsequent sequelae), which has often been ignored due to excessive attention to the arterial system and, specifically, arteriovenous shunts. Various anatomical and pathological entities have been described to cause ICVH. For the second time, we present a unique case of severe focal stenosis in the distal sigmoid sinus associated with concurrent hypoplasia of the contralateral transverse sinus causing a significant pressure

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2016 Interventional Neuroradiology

153. Letter regarding article ‘Focal stenosis of the sigmoid sinus causing intracranial venous hypertension: Case report, endovascular management and review of the literature’ (PubMed)

Letter regarding article ‘Focal stenosis of the sigmoid sinus causing intracranial venous hypertension: Case report, endovascular management and review of the literature’ 26964555 2017 10 26 2019 01 12 2385-2011 22 4 2016 Aug Interventional neuroradiology : journal of peritherapeutic neuroradiology, surgical procedures and related neurosciences Interv Neuroradiol Letter regarding article 'Focal stenosis of the sigmoid sinus causing intracranial venous hypertension: Case report, endovascular (...) management and review of the literature'. 481 10.1177/1591019916637357 Onder Halil H Department of Neurology, Hacettepe University Hospital, Ankara, Turkey halilnder@yahoo.com. eng Editorial 2016 03 10 United States Interv Neuroradiol 9602695 1591-0199 IM Colon, Sigmoid Constriction, Pathologic Cranial Sinuses surgery Endovascular Procedures Humans Hypertension Intracranial Hypertension Sinus Thrombosis, Intracranial 2016 3 12 6 0 2016 3 12 6 0 2017 10 27 6 0 ppublish 26964555 1591019916637357 10.1177

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2016 Interventional Neuroradiology

154. Medication Causes of Hypertension

Medication Causes of Hypertension Medication Causes of Hypertension Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Medication Causes (...) of Hypertension Medication Causes of Hypertension Aka: Medication Causes of Hypertension , Hypertension due to Medications , Hypertension due to Herbals , Herbal Causes of Hypertension , Toxin Induced Hypertension , Elevated Blood Pressure due to Medications , Hypertension due to Chemotherapy , Drug Induced Hypertension From Related Chapters II. Causes: Medication Related Syndromes Antihypertensive Withdrawal III. Causes: Sodium retaining agents s (occurs in 5% of users) is typically with doses of 50 mcg

2018 FP Notebook

155. Pulmonary Hypertension Causes

Pulmonary Hypertension Causes Pulmonary Hypertension Causes Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Pulmonary Hypertension (...) Causes Pulmonary Hypertension Causes Aka: Pulmonary Hypertension Causes , Causes of Pulmonary Hypertension , Pulmonary Arterial Hypertension Causes , Medication Causes of Pulmonary Arterial Hypertension , Drug-Induced Pulmonary Arterial Hypertension From Related Chapters II. General ( ) is idiopathic Causes below relate to Secondary III. Causes: Pulmonary Arterial Hypertension (PAH) - WHO Group 1 Previously known as (PPH) tic predisposition BMPR2 gene (bone morphogenetic protein receptor type 2) ALK1

2018 FP Notebook

156. Hypertension Causes

Hypertension Causes Hypertension Causes Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Hypertension Causes Hypertension Causes Aka (...) : Hypertension Causes , Secondary Hypertension Causes , Hypertension Causes in Children , Hypertension Causes in Adolescents From Related Chapters II. Causes: Common Secondary Hypertension by age Age under 18 years Secondary cause in 10-15% of cases (70-85% of cases if under age 11 years old) (esp. males) Renal parenchymal disease (most common in under age <12 years) Vesicoureteral Reflux Nephropathy Conngenital abnormalities Age 19 to 39 years Secondary cause in 5% of cases due to fibromuscular dysplasia

2018 FP Notebook

157. Recurrent Recessive Mutation in DGUOK Causes Idiopathic Non-Cirrhotic Portal Hypertension. (PubMed)

Recurrent Recessive Mutation in DGUOK Causes Idiopathic Non-Cirrhotic Portal Hypertension. Despite advances in the diagnosis and management of idiopathic noncirrhotic portal hypertension, its pathogenesis remains elusive. Insight may be gained from study of early-onset familial idiopathic noncirrhotic portal hypertension, in which Mendelian mutations may account for disease. We performed exome sequencing of eight subjects from six kindreds with onset of portal hypertension of indeterminate (...) and reduces catalytic activity. Loss-of-function mutations in DGUOK have previously been implicated in cirrhosis and liver failure but not in isolated portal hypertension. Interestingly, treatment of patients with human immunodeficiency viral infection with the nucleoside analogue didanosine is known to cause portal hypertension in a subset of patients and lowers deoxyguanosine kinase levels in vitro; the current findings implicate these effects on deoxyguanosine kinase in the causal mechanism.Our

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2016 Hepatology

158. Accelerometer-determined physical activity and all-cause mortality in a national prospective cohort study of hypertensive adults. (PubMed)

Accelerometer-determined physical activity and all-cause mortality in a national prospective cohort study of hypertensive adults. Research in the general population suggests an inverse association between physical activity and all-cause mortality. Less research on this topic has been conducted among hypertensive adults, but the limited studies also suggest an inverse association between physical activity and all-cause mortality among hypertensive adults. At this point, sex-specific differences (...) are not well understood, and all of the physical activity-mortality studies among hypertensive adults have employed a self-report measure of physical activity. Therefore, the purpose of this study was to examine the sex-specific association between objectively measured physical activity and all-cause mortality among a national sample of hypertensive adults.Data from the 2003 to 2006 National Health and Nutrition Examination Survey, with follow-up through 2011, were employed. Hypertension status was defined

2016 Journal of Hypertension

159. Left Atrial Area and Right Ventricle Dimensions in Non-gated Axial Chest CT can Differentiate Pulmonary Hypertension Due to Left Heart Disease from Other Causes. (PubMed)

Left Atrial Area and Right Ventricle Dimensions in Non-gated Axial Chest CT can Differentiate Pulmonary Hypertension Due to Left Heart Disease from Other Causes. It is unknown whether axial non-gated CT can distinguish World Health Organization Group 2 pulmonary hypertension (pulmonary hypertension due to left heart disease) from non-Group 2 pulmonary hypertension.The study was performed to identity imaging parameters in non-gated chest CT that differentiate Group 2 from non-Group 2 pulmonary (...) hypertension.Among 158 patients who underwent right heart catheterization for evaluation of pulmonary hypertension, 112 had sufficient data and chest CT for review. Invasive hemodynamic data and numerous variables obtained from axial CT images (maximum diameters of main, right, left pulmonary arteries, ascending aorta, main pulmonary artery to ascending aorta diameter ratio, right atrial diameter, left atrial area and right ventricular size) were collected. CT variables were validated against hemodynamic data

2016 Journal of cardiovascular computed tomography

160. Novel homozygous BMP9 nonsense mutation causes pulmonary arterial hypertension: a case report. (PubMed)

Novel homozygous BMP9 nonsense mutation causes pulmonary arterial hypertension: a case report. Pulmonary arterial hypertension (PAH) is a rare, progressive, fatal vascular disorder. Genetic predisposition plays vital roles in the development of PAH, with most mutations being identified in genes involved in the transforming growth factor beta (TGF-β) signaling pathways. Defects in the BMP9 gene have been documented in hereditary hemorrhagic telangiectasia (HHT), the most common inherited

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2016 BMC pulmonary medicine

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