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Hyperpigmentation

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181. Hyperpigmentation and atrophy. (Abstract)

Hyperpigmentation and atrophy. We suspected a malignancy, but a conversation with the patient provided a telling clue.

2012 Journal of Family Practice

182. Postinflammatory Hyperpigmentation After Skin Prick Testing Full Text available with Trip Pro

Postinflammatory Hyperpigmentation After Skin Prick Testing 23268476 2012 12 27 2019 02 11 1939-4551 5 5 2012 May The World Allergy Organization journal World Allergy Organ J Postinflammatory hyperpigmentation after skin prick testing. 57-8 10.1097/WOX.0b013e3182488069 Ludman Sian S Department of MRC & Asthma UK Centre in Allergic Mechanisms of Asthma, Division of Asthma, Allergy and Lung Biology, King's College London, Guy's and St Thomas' NHS Foundation Trust, London, United Kingdom. Penagos

2012 The World Allergy Organization journal

183. Cyclophosphamide-induced nail hyperpigmentation in a child Full Text available with Trip Pro

Cyclophosphamide-induced nail hyperpigmentation in a child 22787324 2012 10 02 2018 11 13 1998-3662 22 2 2012 Mar Indian journal of nephrology Indian J Nephrol Cyclophosphamide-induced nail hyperpigmentation in a child. 149 10.4103/0971-4065.97144 Saha A A Department of Pediatrics, Division of Pediatric Nephrology, PGIMER and Associated Dr. RML Hospital, New Delhi, India. Arora N N Mehra N N Dubey N K NK eng Journal Article India Indian J Nephrol 8914356 0971-4065 2012 7 13 6 0 2012 7 13 6 0

2012 Indian Journal of Nephrology

184. Triple Combination and Glycolic Peels in Post-Acne Hyperpigmentation Full Text available with Trip Pro

Triple Combination and Glycolic Peels in Post-Acne Hyperpigmentation 22557866 2012 05 31 2018 11 13 0974-5157 5 1 2012 Jan Journal of cutaneous and aesthetic surgery J Cutan Aesthet Surg Triple combination and glycolic peels in post-acne hyperpigmentation. 60-1 10.4103/0974-2077.94335 Godse Kiran K Sakhia Jagdish J eng Letter India J Cutan Aesthet Surg 101525405 0974-2077 2012 5 5 6 0 2012 5 5 6 0 2012 5 5 6 1 ppublish 22557866 10.4103/0974-2077.94335 JCAS-5-60 PMC3339139 J Eur Acad Dermatol

2012 Journal of cutaneous and aesthetic surgery

185. Minocycline Hyperpigmentation Full Text available with Trip Pro

Minocycline Hyperpigmentation 22560532 2012 07 03 2013 12 13 1942-5546 87 5 2012 May Mayo Clinic proceedings Mayo Clin. Proc. Minocycline hyperpigmentation. e33 10.1016/j.mayocp.2012.02.013 Wetter David A DA Department of Dermatology, Mayo Clinic, Rochester, MN, USA. eng Case Reports Journal Article England Mayo Clin Proc 0405543 0025-6196 0 Anti-Bacterial Agents FYY3R43WGO Minocycline AIM IM Aged Anti-Bacterial Agents administration & dosage adverse effects Arthritis, Rheumatoid drug therapy (...) Humans Hyperpigmentation chemically induced diagnosis Male Minocycline administration & dosage adverse effects Risk Factors Skin drug effects 2012 02 27 2012 02 27 2012 5 8 6 0 2012 5 9 6 0 2012 7 4 6 0 ppublish 22560532 S0025-6196(12)00308-4 10.1016/j.mayocp.2012.02.013 PMC3498181

2012 Mayo Clinic Proceedings

186. Synergistic Combination of an In-office Procedure and Home Regimen for the Treatment of Facial Hyperpigmentation Full Text available with Trip Pro

Synergistic Combination of an In-office Procedure and Home Regimen for the Treatment of Facial Hyperpigmentation Hyperpigmentation disorders, such as melasma and solar lentigines, pose a significant treatment challenge for most patients. Combining a series of in-office procedures, such as chemical peels and light- and laser-based treatments, with maintenance therapies have been shown to provide greater efficacy than one treatment alone. However, receiving multiple in-office procedures may (...) . Physician-graded overall improvement in hyperpigmentation, standardized photography, and patient satisfaction were evaluated at Weeks 4, 8, and 12. At Week 12, all eight patients demonstrated improvements of at least 25 percent in overall facial hyperpigmentation, with six of the patients demonstrating a 50- or 75-percent overall improvement. One hundred percent of the patients rated their experience with the novel treatment regimen as "excellent" or "good" reflecting high patient satisfaction

2012 The Journal of clinical and aesthetic dermatology

187. Familial Progressive Hyperpigmentation: A Case Report Full Text available with Trip Pro

Familial Progressive Hyperpigmentation: A Case Report Familial progressive hyperpigmentation (FPH) is a rare genodermatosis characterized by hyperpigmented patches in the skin and mucous membranes, present in early infancy, and increase in size and number with age. The genetic basis for FPH remains unknown. We report an unusual case of familial progressive hypermelanosis in a 17-year-old male patient with family history, who presented with a peculiar progressive oral pigmentation disorder

2012 Case reports in dentistry

188. Familial glucocorticoid deficiency presenting with generalized hyperpigmentation in adolescence. Report of three siblings Full Text available with Trip Pro

Familial glucocorticoid deficiency presenting with generalized hyperpigmentation in adolescence. Report of three siblings Familial glucocorticoid deficiency (FGD) is a rare autosomal recessive disorder characterized by glucocorticoid deficiency, high ACTH levels and normal mineralocorticoid levels. FGD is caused due to defects in adrenocorticotropic hormone (ACTH) signaling. The defect can be caused by mutations in genes encoding the ACTH receptor (melanocortin 2 receptor) or its accessory (...) protein.Here we report three siblings with FGD. The second in order of siblings presented at an age of 15 years with history of diffuse hyperpigmentation since childhood. Their parents were non consanguineous. The patients were hyperpigmented and taller compared with their parents. None of the siblings had ambiguous genitalia or neurological abnormalities. There was no history of tuberculosis in the family. Biochemical investigations revealed low serum cortisol (<1 μg/dl) and elevated plasma ACTH (>1250 pg

2012 Indian journal of endocrinology and metabolism

189. Tongue hyperpigmentation during hepatitis C treatment Full Text available with Trip Pro

Tongue hyperpigmentation during hepatitis C treatment 22451683 2012 11 28 2018 12 01 1488-2329 184 13 2012 Sep 18 CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne CMAJ Tongue hyperpigmentation during hepatitis C treatment. 1498 10.1503/cmaj.111985 Bachmeyer Claude C Department of Internal Medicine, Tenon Hospital, Paris, France. claude.bachmeyer@tnn.aphp.fr Pellen Jean-Charles JC eng Case Reports Journal Article 2012 03 26 Canada CMAJ 9711805 0820-3946 (...) 0 Antiviral Agents 0 Interferon-alpha 0 Recombinant Proteins 3WJQ0SDW1A Polyethylene Glycols 49717AWG6K Ribavirin Q46947FE7K peginterferon alfa-2a AIM IM Antiviral Agents administration & dosage adverse effects therapeutic use Drug Therapy, Combination Female Hepatitis C drug therapy Humans Hyperpigmentation chemically induced pathology Interferon-alpha administration & dosage adverse effects therapeutic use Middle Aged Polyethylene Glycols administration & dosage adverse effects therapeutic use

2012 CMAJ : Canadian Medical Association Journal

190. Clinical Trial of 4% Niacinamide Versus 0.05% Desonide for the Treatment of Axillar Hyperpigmentation

Clinical Trial of 4% Niacinamide Versus 0.05% Desonide for the Treatment of Axillar Hyperpigmentation Clinical Trial of 4% Niacinamide Versus 0.05% Desonide for the Treatment of Axillar Hyperpigmentation - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please (...) remove one or more studies before adding more. Clinical Trial of 4% Niacinamide Versus 0.05% Desonide for the Treatment of Axillar Hyperpigmentation The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT01542138 Recruitment Status : Completed First Posted : March 2, 2012 Last Update Posted : November 29

2012 Clinical Trials

191. Efficacy and Safety of Clindamycin Phosphate 1.2% and Tretinoin 0.025% Gel for the Treatment of Acne and Acne-induced Post-inflammatory Hyperpigmentation in Patients with Skin of Color. Full Text available with Trip Pro

Efficacy and Safety of Clindamycin Phosphate 1.2% and Tretinoin 0.025% Gel for the Treatment of Acne and Acne-induced Post-inflammatory Hyperpigmentation in Patients with Skin of Color. To assess the efficacy and safety of a topical gel containing clindamycin 1.2% and tretinoin 0.025% for the treatment of acne and acne-induced postinflammatory hyperpigmentation (PIH) in darker skinned patients.Randomized, double-blind, placebo-controlled study.Two United States clinical sites.Thirty-three (...) patients 12 years of age or older with skin types IV to VI, mild-to-moderate facial acne, and PIH were enrolled.Patients applied clindamycin phosphate/tretinoin gel or a nonmedicated vehicle each evening and a sun protection factor 30 sunscreen daily. Changes in skin erythema and hyperpigmentation were measured using a chromameter and photographic images. Efficacy was assessed using the Evaluators Global Acne Severity Scale, lesion counts, Post-inflammatory Hyperpigmentation Severity Scales

2012 The Journal of clinical and aesthetic dermatology Controlled trial quality: uncertain

192. Hyperpigmentation of Skin Full Text available with Trip Pro

Hyperpigmentation of Skin 13969176 1998 11 01 2018 12 01 0007-1447 1 5347 1963 Jun 29 British medical journal Br Med J Hyperpigmentation of skin. A sign of vitamin-B12 deficiency. 1713-5 BAKER S J SJ IGNATIUS M M JOHNSON S S VAISH S K SK eng Journal Article England Br Med J 0372673 0007-1447 0 Vitamins OM Humans Hyperpigmentation Pigmentation Disorders Skin Vitamin B 12 Deficiency Vitamins PIGMENTATION DISORDERS VITAMIN B 12 DEFICIENCY 1963 6 29 1963 6 29 0 1 1963 6 29 0 0 ppublish 13969176

1963 British medical journal

193. Leukodystrophy, skin hyperpigmentation, and adrenal atrophy: Siemerling-Creutzfeldt disease. Transmission through several generations in two families. Full Text available with Trip Pro

Leukodystrophy, skin hyperpigmentation, and adrenal atrophy: Siemerling-Creutzfeldt disease. Transmission through several generations in two families. Two apparently unrelated families with a history of leukodystrophy associated with adrenal insufficiency are presented. Only about 20 cases of this syndrome have been reported until now. It was first described by Siemerling and Creutzfeldt; therefore we propose the designation Siemerling-Creutzfeldt disease. Our pedigrees include 15 additional

1975 American Journal of Human Genetics

194. Hyperpigmentation associated with oxprenolol administration. Full Text available with Trip Pro

Hyperpigmentation associated with oxprenolol administration. 871864 1977 09 02 2018 11 13 0007-1447 2 6082 1977 Jul 30 British medical journal Br Med J Hyperpigmentation associated with oxprenolol administration. 296 Harrower A D AD Strong J A JA eng Case Reports Journal Article England Br Med J 0372673 0007-1447 519MXN9YZR Oxprenolol AIM IM Female Humans Middle Aged Oxprenolol adverse effects Pigmentation Disorders chemically induced 1977 7 30 1977 7 30 0 1 1977 7 30 0 0 ppublish 871864

1977 British medical journal

195. An unusual case of non-malignant skin hyperpigmentation. Full Text available with Trip Pro

An unusual case of non-malignant skin hyperpigmentation. 534918 1980 05 14 2017 11 17 0007-1447 2 6205 1979 Dec 22-29 British medical journal Br Med J An unusual case of non-malignant skin hyperpigmentation. 1628-9 Potter A R AR eng Case Reports Journal Article England Br Med J 0372673 0007-1447 AIM IM Adult Animals Diagnosis, Differential Humans Insecta Male Melanoma diagnosis Pigmentation Disorders diagnosis etiology Skin Neoplasms diagnosis 1979 12 22 1979 12 22 0 1 1979 12 22 0 0 ppublish

1979 British medical journal

196. Hyperpigmentation

Hyperpigmentation Hyperpigmentation - Dermatologic Disorders - MSD Manual Professional Edition Brought to you by The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases / / / / IN THIS TOPIC OTHER TOPICS IN THIS CHAPTER Test your knowledge Diagnostic Tests for Skin (...) Education NOTE: This is the Professional Version. CONSUMERS: Topic Resources Hyperpigmentation has multiple causes and may be focal or diffuse. Most cases are due to an increase in melanin production and deposition. (See also .) Focal hyperpigmentation is most often postinflammatory in nature, occurring after injury (eg, and ) or other causes of inflammation (eg, , ). Focal linear hyperpigmentation is commonly due to phytophotodermatitis, which is a phototoxic reaction that results from ultraviolet

2013 Merck Manual (19th Edition)

197. Long-Term Follow-Up of a Case of Cheek Hyperpigmentation Associated with McCune-Albright Syndrome Treated with Q-Switched Ruby Laser. (Abstract)

Long-Term Follow-Up of a Case of Cheek Hyperpigmentation Associated with McCune-Albright Syndrome Treated with Q-Switched Ruby Laser. 21272121 2011 04 19 2015 11 19 1524-4725 37 2 2011 Feb Dermatologic surgery : official publication for American Society for Dermatologic Surgery [et al.] Dermatol Surg Long-term follow-up of a case of cheek hyperpigmentation associated with McCune-Albright syndrome treated with Q-switched ruby laser. 263-6 10.1111/j.1524-4725.2010.01864.x Ozawa Toshiyuki T (...) Department of Plastic and Reconstructive Surgery, Osaka City University Graduate School of Medicine, Osaka, Japan. Tateishi Chiharu C Shirakawa Makiko M Murakami Emi E Ishii Masamitsu M Harada Teruichi T eng Case Reports Journal Article 2011 01 27 United States Dermatol Surg 9504371 1076-0512 IM Cheek Child Female Fibrous Dysplasia, Polyostotic complications pathology Follow-Up Studies Humans Hyperpigmentation etiology pathology radiotherapy Lasers, Solid-State therapeutic use Low-Level Light Therapy

2011 Dermatologic Surgery

198. Commentary: use of oral therapy for the prevention of postinflammatory hyperpigmentation. (Abstract)

Commentary: use of oral therapy for the prevention of postinflammatory hyperpigmentation. 21518099 2011 07 26 2013 11 21 1524-4725 37 5 2011 May Dermatologic surgery : official publication for American Society for Dermatologic Surgery [et al.] Dermatol Surg Use of oral therapy for the prevention of postinflammatory hyperpigmentation. 611 10.1111/j.1524-4725.2011.01958.x McDaniel David H DH Institute of Anti-Aging Research, Eastern Virginia Medical School, Virginia Beach, Virginia, USA. mail (...) @lsvcv.com eng Comment Journal Article United States Dermatol Surg 9504371 1076-0512 0 Antifibrinolytic Agents 6T84R30KC1 Tranexamic Acid IM Dermatol Surg. 2011 May;37(5):605-10 21457392 Administration, Oral Antifibrinolytic Agents administration & dosage therapeutic use Humans Hyperpigmentation etiology prevention & control Inflammation etiology prevention & control Japan Lasers adverse effects Tranexamic Acid administration & dosage therapeutic use 2011 4 27 6 0 2011 4 27 6 0 2011 7 27 6 0 ppublish

2011 Dermatologic Surgery

199. Non-ablative 1550 nm fractional laser therapy not effective for erythema dyschromicum perstans and postinflammatory hyperpigmentation: a pilot study. (Abstract)

Non-ablative 1550 nm fractional laser therapy not effective for erythema dyschromicum perstans and postinflammatory hyperpigmentation: a pilot study. Erythema dyschromicum perstans and postinflammatory hyperpigmentation (PIH) are characterized by papillary dermal pigmentation or pigment incontinence. To date, no standard treatment is available. Fractional laser therapy (FLT) was recently reported to improve different pigment disorders.To assess the efficacy and safety of non-ablative FLT (...) in the treatment of erythema dyschromicum perstans and PIH.Eight patients with erythema dyschromicum perstans and six patients with PIH were included. In each patient, two similar test regions were randomized to receive either five fractional laser treatments in combination with intermittent daily topical bleaching or the same intermittent regimen of topical bleaching alone. Three months after the last treatment, improvement of hyperpigmentation was assessed by melanin index, reflectance spectroscopy

2011 Journal of Dermatological Treatment Controlled trial quality: uncertain

200. Efficacy and safety of azelaic acid (AzA) gel 15% in the treatment of post-inflammatory hyperpigmentation and acne: a 16-week, baseline-controlled study. (Abstract)

Efficacy and safety of azelaic acid (AzA) gel 15% in the treatment of post-inflammatory hyperpigmentation and acne: a 16-week, baseline-controlled study. Although there are few differences in the incidence and pathophysiology of acne across various races and ethnicities, there is some evidence that black patients may have larger sebaceous glands and increased sebum production. Of greater clinical relevance, patients with darker skin types are at increased risk for the development of post (...) -inflammatory hyperpigmentation (PIH), which some find as or more troubling than acne itself. This common and bothersome sequelum of acne can be difficult to manage in this population. Topical azelaic acid gel is recognized to have anti-tyrosinase activity, suggesting it may be a suitable treatment option for mild-to-moderate acne with associated moderate-to-severe PIH. This pilot study demonstrates the efficacy of topical AzA gel 15% when applied twice daily for the reduction of both acne and PIH. J

2011 Journal of drugs in dermatology : JDD

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