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Glomerulonephritis Causes

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61. Identification of Ceruloplasmin as a Gene that Affects Susceptibility to Glomerulonephritis Through Macrophage Function Full Text available with Trip Pro

Identification of Ceruloplasmin as a Gene that Affects Susceptibility to Glomerulonephritis Through Macrophage Function Crescentic glomerulonephritis (Crgn) is a complex disorder where macrophage activity and infiltration are significant effector causes. In previous linkage studies using the uniquely susceptible Wistar Kyoto (WKY) rat strain, we have identified multiple crescentic glomerulonephritis QTL (Crgn) and positionally cloned genes underlying Crgn1 and Crgn2, which accounted for 40

2017 Genetics

62. Case Report of Spontaneous Remission of Biopsy-Proven Idiopathic Immune Complex-Mediated Membranoproliferative Glomerulonephritis Full Text available with Trip Pro

Case Report of Spontaneous Remission of Biopsy-Proven Idiopathic Immune Complex-Mediated Membranoproliferative Glomerulonephritis Membranoproliferative glomerulonephritis (MPGN) is a histopathologic diagnosis causing microscopic hematuria, nephrotic range proteinuria, and chronic renal failure. Current understanding divides pathogenesis into two broad categories: immune complex mediated and complement mediated (now termed C3 glomerulopathy). The term idiopathic immune complex-mediated MPGN

2017 Case Reports in Nephrology and Dialysis

63. Hepatitis C virus-associated cryoglobulinemia with membrano-proliferative glomerulonephritis treated with prednisolone and interferon: A case report Full Text available with Trip Pro

Hepatitis C virus-associated cryoglobulinemia with membrano-proliferative glomerulonephritis treated with prednisolone and interferon: A case report Hepatitis C virus (HCV) is a major cause of liver-associated morbidity and has an increasing prevalence worldwide. Hepatitis C virus infection may lead to chronic hepatitis, cirrhosis and liver failure. However, it is also associated with a wide range of extra-hepatic complications, such as cryoglobulinemia, an immune complex disease associated (...) with cryoglobulin leading to multiple organ damage and, while the major symptom is vasculitis. The present study reported on a-58-year-old woman who was diagnosed with HCV-associated cryoglobulinemia with skin, kidney and blood system damage and biopsy-proven cryoglobulinemia membrano-proliferative glomerulonephritis. HCV RNA clearance occurred within a few weeks of interferon treatment and the patient was then treated by prednisolone and sustained interferon. While the therapeutic effect was obvious at first

2017 Experimental and therapeutic medicine

64. Acute Post Streptococcal Glomerulonephritis

. Condition or disease Intervention/treatment PSGN - Post-Streptococcal Glomerulonephritis Diagnostic Test: anti streptolysin o titre and serum complement test Detailed Description: Acute post streptococcal glomerulonephritis are caused by group A beta haemolytic streptococci and follow upper airway infections such as pharyngitis or tonsillitis, by 14 to 21 days and 3-6 weeks after skin infection especially in warmer climates . In recent decades the number of patients with post streptococcal (...) glomerulonephritis has decreased considerably in the United States and Europe industrialized countries. In other parts of the world ,some developing communities. the incidence of post streptococcal glomerulonephritis has remained high. post streptococcal glomerulonephritis is one of the leading cause requiring hospital admissions in children , and it is also an important cause of acute renal failure in developing countries. Though deaths due to this disease are rare, it can cause serious complications

2017 Clinical Trials

65. Clinical Features and Outcomes of a Racially Diverse Population with Fibrillary Glomerulonephritis. Full Text available with Trip Pro

glomerulonephritis represents a secondary glomerular disease process (associated with autoimmune disease, infection or malignancy) in many cases and hence screening is essential. As the screening for comorbidities increased over time, more underlying causes were identified. We noted a high frequency of HCV among black patients, suggesting a possible causative association. Treatment of underlying disease is essential for patients for the best outcome.© 2017 S. Karger AG, Basel. (...) Clinical Features and Outcomes of a Racially Diverse Population with Fibrillary Glomerulonephritis. Fibrillary glomerulonephritis is characterized by randomly arranged fibrils, approximately 20 nm in diameter by electron microscopy. Patients present with proteinuria, hematuria and kidney insufficiency, and about half of the reported patients progress to end-stage kidney disease within 4 years. The dependence of patient characteristics and outcomes on race has not been explored. In this study

2017 American journal of nephrology

66. Increased glomerulonephritis recurrence after living related donation. Full Text available with Trip Pro

Increased glomerulonephritis recurrence after living related donation. Kidney transplantation confers superior outcomes for patients with end stage kidney disease, and live donor kidneys associate with superior outcomes compared to deceased donor kidneys. Modern immunosuppression has improved rejection rates and transplant survival and, as a result, recurrence of glomerulonephritis has emerged as a major cause of allograft loss. However, many glomerulonephritides have significant genetic risk (...) which may manifest through kidney intrinsic or systemic mechanisms. We hypothesise that heritable kidney intrinsic predisposition to glomerulonephritis will result in increased risk of glomerulonephritis recurrence in kidneys transplanted from genetically related donors.We investigated the effect of living related donation on rates of recurrence and subsequent graft outcomes in 7236 patient from 28 years of ANZDATA transplant registry data. Data were analysed in R, using Kaplan Meier Survival

2017 BMC Nephrology

67. Different types of glomerulonephritis associated with the dysregulation of the complement alternative pathway in 2 brothers: A case report. Full Text available with Trip Pro

Different types of glomerulonephritis associated with the dysregulation of the complement alternative pathway in 2 brothers: A case report. C3 glomerulonephritis (C3GN) and complement-mediated hemolytic uremic syndrome (HUS) both result from the abnormal regulation of the complement system. A significant number of patients with C3GN or complement-mediated HUS have mutations of more than 1 complement protein. This discovery has had a major impact on identifying the underlying cause of familial

2017 Medicine

68. C3 glomerulonephritis with a severe crescentic phenotype. (Abstract)

treatment. It is important to recognize this rare cause of crescentic glomerulonephritis so that appropriate evaluation and treatment can be carried out. (...) C3 glomerulonephritis with a severe crescentic phenotype. C3 glomerulopathy (C3G) is rare type of glomerulonephritis resulting from the glomerular deposition of C3 due to dysregulation of the alternative pathway of complement. It is further subdivided into C3 glomerulonephritis (C3GN) and dense deposit disease (DDD), depending on the ultrastructural features. C3GN usually presents with a membranoproliferative pattern of injury. Crescents may or may not be present. However, we have noted

2017 Pediatric Nephrology

69. Recurrent glomerulonephritis after kidney transplantation: risk factors and allograft outcomes. Full Text available with Trip Pro

recipients were determined using adjusted Cox proportional hazard and competing risk modeling. A total of 6,597 recipients with biopsy-proven glomerulonephritis as the primary cause of end-stage kidney disease were followed for 51,871 person-years (median duration 7.7 years). The four most common types of glomerulonephritis were IgA nephropathy in 2501 patients, focal segmental glomerulosclerosis (FSGS) in 1403, membranous in 376, and membranoproliferative (MPGN) nephropathy in 357 patients. Among (...) as likely to lose their allografts compared to those without recurrence (adjusted hazard ratio 2.04 [1.81-2.31]). Thus, recurrent glomerulonephritis remains a significant cause of graft loss in transplant recipients.Crown Copyright © 2017. Published by Elsevier Inc. All rights reserved.

2017 Kidney International

70. Cardiovascular risk is similar in patients with glomerulonephritis compared to other types of chronic kidney disease: a matched cohort study. Full Text available with Trip Pro

Cardiovascular risk is similar in patients with glomerulonephritis compared to other types of chronic kidney disease: a matched cohort study. Patients with chronic kidney disease (CKD) due to glomerulonephritis (GN) are thought to be at high risk for cardiovascular disease (CVD). However, no study has examined whether GN directly contributes to CV risk beyond the effects conferred by pre-existing traditional risk factors and level of renal function.Matched cohort study using the previously (...) described prospective CanPREDDICT study cohort. 2187 patients with CKD (eGFR 15-45 ml/min/m2) from 25 Canadian centres were divided into GN vs non-GN cause of CKD. Patients on immunotherapy for GN were not included in the study. Standardized measures of CV risk factors, biomarkers and CV outcomes were recorded over 3 years of follow-up, with the primary outcome measure being time to first all-cause CV event.In the overall cohort, CV events occurred in 25 (8.7%) of the GN group and 338 (17.8%) of the non

2017 BMC Nephrology

71. IgG4-related membranous glomerulonephritis and generalized lymphadenopathy without pancreatitis: a case report. Full Text available with Trip Pro

glomerulonephritis is a well-established cause of membranous glomerulonephritis. It must be sought after in every patient with a previous diagnosis of IgG4-related disease and in every patient with this histological finding on renal biopsy. Corticoids are still the first-line treatment of IgG4-related disease. New therapeutic strategies are needed to avoid glucocorticoids long term side-effects. Interestingly, the patient was prescribed cyclophosphamide in addition to glucocorticoids for an immune (...) IgG4-related membranous glomerulonephritis and generalized lymphadenopathy without pancreatitis: a case report. IgG4-related disease is a recently described pathologic entity. This is the case of a patient with nephrotic syndrome and lymphadenopathy due to IgG4-related disease. Such a kidney involvement is quite peculiar and has only been described a few times recently. Renal biopsy showed a glomerular involvement with membranous glomerulonephritis in association with a tubulo-interstitial

2017 BMC Nephrology

72. Rapidly progressive glomerulonephritis due to coexistent anti-glomerular basement membrane disease and fibrillary glomerulonephritis Full Text available with Trip Pro

Rapidly progressive glomerulonephritis due to coexistent anti-glomerular basement membrane disease and fibrillary glomerulonephritis Anti-glomerular basement membrane (anti-GBM) disease is a major cause of rapidly progressive glomerulonephritis (RPGN). On the other hand, fibrillary glomerulonephritis (GN) typically presents as proteinuria, hematuria and renal insufficiency, but rarely as RPGN. Without electron microscopy, the diagnosis of fibrillary GN can be missed. We report a 68-year-old

2015 Clinical kidney journal

73. Rapidly Progressive Glomerulonephritis

Progressive Glomerulonephritis Rapidly Progressive Glomerulonephritis Aka: Rapidly Progressive Glomerulonephritis , RPGN From Related Chapters II. Causes III. Symptoms and Signs Similar to Rapidly Progressive IV. Diagnosis: Renal Biopsy Crescentic V. Course Progresses to within weeks to months Patients refusing within 6 months: 50% VI. Management d dose glucocorticoids Cytotoxic agents Cyclophosphamide Plasma exchange Images: Related links to external sites (from Bing) These images are a random sampling (...) Rapidly Progressive Glomerulonephritis Rapidly Progressive Glomerulonephritis Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Rapidly

2018 FP Notebook

74. Poststreptococcal Glomerulonephritis

Poststreptococcal Glomerulonephritis Poststreptococcal Glomerulonephritis Aka: Poststreptococcal Glomerulonephritis , Post-Streptococcal Glomerulonephritis , Acute post-streptococcal glomerulonephritis From Related Chapters II. Epidemiology Previously, most common cause of , but has decreased significantly in the U.S. As of 2018, most cases occur in underserved regions Previously most often in childhood (ages 2 to 6) Now more common in age >60 years old, especially with comorbidities III. Pathophysiology (...) Poststreptococcal Glomerulonephritis Poststreptococcal Glomerulonephritis Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4

2018 FP Notebook

75. Acute Glomerulonephritis

Glomerulonephritis Aka: Acute Glomerulonephritis , Glomerulonephritis From Related Chapters II. Definition with casts III. Causes See ( , ) Membranous Glomerulonephritis ( ) Nephritis with low complement Postinfectious proliferative Glomerulonephritis (classic) Membranoproliferative Glomerulonephritis Cryoglobulinemia Nephritis with normal complement IgA Nephropathy (Berger's Disease) Fibrillary Glomerulonephritis Renal Wegner's Granulomatosis IV. Symptoms (variably present) Malaise Low grade fever V. Symptoms (...) Treat specific cause Images: Related links to external sites (from Bing) These images are a random sampling from a Bing search on the term "Acute Glomerulonephritis." Click on the image (or right click) to open the source website in a new browser window. Related Studies (from Trip Database) Ontology: Glomerulonephritis (C0017658) Definition (NCI) A renal disorder characterized by damage in the glomeruli. It may be acute or chronic, focal or diffuse, and it may lead to renal failure. Causes include

2018 FP Notebook

76. Proliferative glomerulonephritis causing acute renal failure in a child with Salmonella septicemia Full Text available with Trip Pro

Proliferative glomerulonephritis causing acute renal failure in a child with Salmonella septicemia 23814433 2013 07 02 2018 11 13 0971-4065 23 3 2013 May Indian journal of nephrology Indian J Nephrol Proliferative glomerulonephritis causing acute renal failure in a child with Salmonella septicemia. 240-1 10.4103/0971-4065.111875 Dhooria G S GS Department of Pediatrics, Dayanand Medical College and Hospital, Ludhiana, India. Bains H S HS Bhat D D eng Journal Article India Indian J Nephrol

2013 Indian Journal of Nephrology

77. IgG4: a tantalizing link between causes of membranous glomerulonephritis and systemic disease. Full Text available with Trip Pro

IgG4: a tantalizing link between causes of membranous glomerulonephritis and systemic disease. Alexander et al. give a detailed description of IgG4-related membranous glomerulonephritis (MGN). Their data delineate for the first time in IgG4-related disease (IgG4-RD) a second pattern of tissue injury occurring within the same organ. The paper highlights that MGN can occur alone or simultaneously with IgG4-related tubulointerstitial nephritis. Thus, it extends our understanding of IgG4-RD

2013 Kidney International

78. Glomerulonephritis Causes

Glomerulonephritis Causes Glomerulonephritis Causes Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Glomerulonephritis Causes (...) Glomerulonephritis Causes Aka: Glomerulonephritis Causes , Infectious Causes of Glomerulonephritis From Related Chapters II. Causes: Primary Glomerular Disease IgA Nephropathy Mesangial proliferative disease Membranoproliferative disease Antiglomerular basement membrane III. Causes: Infectious l Acute bacteremia Pneumococcal bacteremia Visceral Viral A and B (EBV) Parasitic IV. Causes: Multisystem Disease Postpartum Hereditary nephritis Cryoglobulinemia V. Causes: Medications (primarily cause RPGN) See Examples

2015 FP Notebook

79. Discontinuation of antiretroviral therapy causing progression to end-stage renal disease in an HIV patient diagnosed with immune complex ‘lupus-like’ glomerulonephritis Full Text available with Trip Pro

Discontinuation of antiretroviral therapy causing progression to end-stage renal disease in an HIV patient diagnosed with immune complex ‘lupus-like’ glomerulonephritis Immune complex 'lupus-like glomerulonephritis' is a type of renal injury seen infrequently in human immunodeficiency virus (HIV) patients and very little is known about the clinical course and treatment. Treatment options are limited but antiretroviral therapy and steroids have been tried with limited success. We report (...) a case of a 21-year-old HIV-positive African American male with lupus-like glomerulonephritis who progressed to end-stage renal disease upon discontinuation of highly active antiretroviral therapy. This case illustrates the importance of antiretroviral therapy as an important treatment modality for immune complex lupus-like glomerulonephritis in HIV patients.

2012 Clinical kidney journal

80. Combination of Factor H Mutation and Properdin Deficiency Causes Severe C3 Glomerulonephritis. Full Text available with Trip Pro

Combination of Factor H Mutation and Properdin Deficiency Causes Severe C3 Glomerulonephritis. Factor H (fH) and properdin both modulate complement; however, fH inhibits activation, and properdin promotes activation of the alternative pathway of complement. Mutations in fH associate with several human kidney diseases, but whether inhibiting properdin would be beneficial in these diseases is unknown. Here, we found that either genetic or pharmacological blockade of properdin, which we expected

2012 Journal of the American Society of Nephrology

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