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Eosinophilic Cellulitis

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21. Distinguishing cellulitis from its mimics. Full Text available with Trip Pro

Distinguishing cellulitis from its mimics. Distinguishing true cellulitis from its many imitators is challenging but critical if we are to avoid unnecessary use of antibiotics and delays in treatment. Common imitators of cellulitis are stasis dermatitis, lipodermatosclerosis, contact dermatitis, lymphedema, eosinophilic cellulitis, and papular urticaria. Specific criteria do not exist for the diagnosis of cellulitis, but the alert physician can find clues in the history and physical examination (...) that point toward cellulitis.

2012 Cleveland Clinic Journal of Medicine

22. Neutrophilic and eosinophilic dermatitis caused by contact allergic reaction to paraphenylenediamine in hair dye. Full Text available with Trip Pro

Neutrophilic and eosinophilic dermatitis caused by contact allergic reaction to paraphenylenediamine in hair dye. Paraphenylenediamine (PPD) in hair dyes can cause systemic as well as cutaneous allergic reactions such as neutrophilic and eosinophilic dermatitis. The symptoms are often severe. The acute lesion is normally histologically indistinguishable from any eczematous reaction with marked spongiosis.We report a case of allergic contact dermatitis caused by the use of hair dye containing (...) PPD that developed in a patient who had been using the same hair dye for many years. Her symptoms included scalp dermatitis and widespread skin lesions as well as lymphadenopathy and quite possibly dyspnea resembling asthma. What is most remarkable about this case is the histopathologic finding of neutrophilic cellulitis and a marked neutrophilic infiltrate with variable spongiosis. This unique finding was confirmed by histologic analysis of a patch test lesion specimen.It is always important

2012 Archives of Dermatology

23. Assessment of eosinophilia

Allergic rhinitis Clonorchiasis Fascioliasis Fasciolopsiasis Opisthorchiasis Filariasis Gnathostomiasis Toxocariasis Paragonimiasis Ancylostoma caninum infestation Coccidioidomycosis Herpes gestationis Drug hypersensitivity Allergic bronchopulmonary aspergillosis Eosinophilic granulomatosis with polyangiitis Eosinophilic oesophagitis Eosinophilic gastroenteritis Eosinophilic cellulitis (Wells syndrome) Chronic eosinophilic leukaemia Other leukaemias Non-Hodgkin's lymphoma Hodgkin's lymphoma Non (...) Assessment of eosinophilia Assessment of eosinophilia - Differential diagnosis of symptoms | BMJ Best Practice You'll need a subscription to access all of BMJ Best Practice Search  Assessment of eosinophilia Last reviewed: February 2019 Last updated: August 2018 Summary Eosinophilia is defined as an increase in the peripheral blood eosinophil count. The upper limit of normal is usually taken as about 0.6 × 10⁹/L (600/microlitre), but it is lower (about 0.4 × 10⁹/L [400/microlitre]) if people

2018 BMJ Best Practice

26. Scleroderma Morphea

symmetrically and predominantly located on the extremities. Mixed type of LS Mixed types of LS predominantly affect children, occurring in up to 15% of patients with juvenile LS. Mixed types often consist of linear LS and morphea (plaques type of LS) or a combination of linear and generalized LS. 5 Eosinophilic fasciitis Eosinophilic fasciitis (or Shulman syndrome) is considered by many experts to be a special subtype belonging to the spectrum of LS. 1 . A mechanical trauma often precedes the first (...) manifestation of the disease. Clinically, eosinophilic fasciitis predominantly affects the extremities and presents with a rapid onset of symmetrical swelling of the skin. In the later stage of disease, lesions become more indurated and fibrotic, leading to the typical “peau d’orange” like appearance. A distinctive clinical finding in later stages of eosinophilic fasciitis is that cutaneous veins might appear depressed compared with the surrounding tissue (called “negative vein sign”). Association

2018 European Dermatology Forum

30. Management of Immune-Related Adverse Events in Patients Treated With Immune Checkpoint Inhibitor Therapy Full Text available with Trip Pro

. If BP is diagnosed, it may be possible to avoid long-term use of systemic corticosteroids and treat with rituximab as an alternative approach to treating the irAE. Seek infectious disease consultation if patient might have secondary cellulitis or if patient has other infection risk factors, such as neutropenia, etc. It is recommended that clinicians manage grade 4 toxicities as follows: Should permanently discontinue ICPi. Should admit patient immediately and place under supervision (...) of a dermatologist. Should administer IV (methyl)prednisolone (or equivalent) 1 to 2 mg/kg/d. When toxicity improves to grade 2 or less, start corticosteroid taper. Taper should be at least 4 weeks. If BP is diagnosed, it may be possible to avoid long-term use of systemic corticosteroids and treat with rituximab as an alternative approach to treating the irAE. Seek infectious disease consultation if patient might have secondary cellulitis or if patient has other infection risk factors, such as neutropenia, etc

2018 American Society of Clinical Oncology Guidelines

33. CRACKCast E109 – CNS Infections

a CSF infection is suspected: remember that typical signs and symptoms may be absent! Contraindications: There are no absolute contraindications, just relative contraindications (need to weigh the risk benefits out!): The bottom line according to Rosen’s is: “we recommend against LP in patients who have rapid neurological deterioration, seizures, or signs of herniation.” Here’s the longer version: SSTI at the puncture site (cellulitis or spinal epidural abscess) Anyone with signs of increased ICP (...) by pretreatment with ABx) < 5 leukocytes / mm3 eosinophils See formula below table in question 14 for calculating true CSF WBC in cases of a traumatic tap Gram stain, and bacterial culture (affected by pretreatment with ABx) No organisms Antigen testing ● Cryptococcal antigen ( replaced India-Ink stain ) ● Stain for acid-fast bacilli ● VDRL (syphilis test) May be measured in CSF, serum

2017 CandiEM

34. Farydak - panobinostat

Sal. Gland (f), ? foll. cell. hypertrophy thyroid, ? fatty atrophy sternum, ? heamosiderin spleen, trend of ? # of females in oestrus phase of cycle BoneMarrow smears: ? # erythroid cells & ? M:E, ? proportion of eosinophilic cells ± abnormal morphology, ? # of granulocytic cells show maturation arrest. (all effects are associated with each other) =30 ? FC (m), ? Hb (m), MCV, MCHC, WBC, L, N, Eos, B (m), LUC (m, f only wk 13))), Plat (wk13 & 26) ? MCH (f) ? ALT (m), ? TP (f), ?rT3 (f) ? rel. brain

2015 European Medicines Agency - EPARs

35. Orbactiv - oritavancin

severe or life-threatening, depending on the pathogen [Corey and Stryjewski, 2011; Elston, 2005]. ABSSSIs include cellulitis/erysipelas, wound infections, major cutaneous abscesses, and burn infections. They commonly involve at least a 75 cm 2 surface area of redness, oedema and/or induration accompanied by lymph node enlargement or systemic symptoms such as fever [FDA, 2010]. The morbidity associated with ABSSSI often requires rapid intervention with antibacterial therapy to minimise tissue damage (...) and dogs was a dose related accumulation of eosinophilic granules in tissue macrophages including hepatocytes, renal cortical epithelial cells, adrenal cells and macrophages of the reticulo endothelial system. The appearance of the eosinophilic granules did not occur following single dose administration and did not significantly affect innate macrophage function in vitro at intracellular levels anticipated from a single 1,200 mg dose. Assessment report EMA/183786/2015 Page 27/92 Moderate, dose-related

2015 European Medicines Agency - EPARs

36. Retinoblastoma Treatment (PDQ®): Health Professional Version

tumors present with pain, orbital cellulitis, glaucoma, or buphthalmos. As the tumor progresses, patients may present with orbital or metastatic disease. Metastases occur in the preauricular and laterocervical lymph nodes, in the CNS, or systemically (commonly in the bones, bone marrow, and liver). In the United States, children of Hispanic origin and children living in lower socioeconomic conditions have been noted to present with more advanced disease.[ ] Diagnostic and Staging Evaluation (...) , round, densely packed cells with hypochromatic nuclei and scant cytoplasm. Several degrees of photoreceptor differentiation have been described and are characterized by distinctive arrangements of tumor cells, as follows: Flexner-Wintersteiner rosettes are specific to retinoblastoma; these structures consist of a cluster of low columnar cells arranged around a central lumen that is bounded by an eosinophilic membrane analogous to the external membrane of the normal retina. The lumen contains

2018 PDQ - NCI's Comprehensive Cancer Database

37. Investigation and management of eosinophilia

). The eosinophilia usually resolves with control of the underlying condition. Dermatological causes (non-allergic) Wells syndrome (eosinophilic cellulitis) is a recurring granulomatous dermatitis with eosinophilia (Wells 1971) characterised by (i) sudden onset annular or circinate erythematous-oedematous patches that rapidly evolve to morphoea-like slate-blue plaques, (ii) a histological appearance characterized by the presence of 'flame figures' and (iii) an inconstant blood eosinophilia. Similar histological (...) clinical responses when ciclosporin is added to prednisolone in previously steroid-resistant idiopathic HES (Akiyama et al, 1997; Fukuta et al, 2001, Zabel & Schlaak 1991) and when used as a steroid-sparing agent in idiopathic paediatric HES (Hosoki et al, 2011; Nadarajah et al, 1997) (and also in cases of eosinophilic cellulitis and fasciitis (Kim et al, 2013, Tahara et al, 2008)). A variety of effective ciclosporin doses have been reported, generally with gradual tapering following clinical response

2016 British Committee for Standards in Haematology

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