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Dermatitis Herpetiformis

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161. Dermatitis herpetiformis Part I. Epidemiology, pathogenesis, and clinical presentation. (Abstract)

Dermatitis herpetiformis Part I. Epidemiology, pathogenesis, and clinical presentation. Dermatitis herpetiformis (DH) is an autoimmune disease that is linked to gluten sensitivity and has a clear relationship to celiac disease. Both conditions are mediated by the IgA class of autoantibodies and the diagnosis of DH is dependent on detection of granular deposits of IgA in the skin. There is an underlying genetic predisposition to the development of DH but environmental factors are also important

2011 Journal of American Academy of Dermatology

162. Prevalence and incidence of dermatitis herpetiformis: a 40-year prospective study from Finland. (Abstract)

Prevalence and incidence of dermatitis herpetiformis: a 40-year prospective study from Finland. Dermatitis herpetiformis (DH) is an external manifestation of coeliac disease presenting with blistering rash and pathognomonic cutaneous IgA deposits. Better knowledge of subclinical forms and serological testing has resulted in a sharp increase in the incidence and prevalence of coeliac disease.To investigate the prevalence of DH and analyse whether the incidence of DH changed when the occurrence

2011 British Journal of Dermatology

163. Novel assay for detecting celiac disease-associated autoantibodies in dermatitis herpetiformis using deamidated gliadin-analogous fusion peptides. (Abstract)

Novel assay for detecting celiac disease-associated autoantibodies in dermatitis herpetiformis using deamidated gliadin-analogous fusion peptides. Dermatitis herpetiformis (DH) is a cutaneous manifestation of celiac disease (CD), the latter being identified by circulating autoantibodies against native gliadin (nGli), tissue transglutaminase (tTG), endomysium, or a combination of these. Recently, a novel serologic assay using deamidated gliadin-analogous fusion peptides (GAF3X) showed high

2011 Journal of American Academy of Dermatology

164. Atopic Dermatitis Research Network (ADRN) Influenza Vaccine Study

; Participated in another clinical trial investigating a vaccine, drug, medical device, or a medical procedure in the four weeks preceding the study vaccination or who plan to participate in another clinical trial during the present study period; Any skin disease other than AD that might compromise the stratum corneum barrier (e.g., bullous disease, psoriasis, cutaneous T cell lymphoma [also called Mycosis Fungoides or Sezary syndrome], dermatitis herpetiformis, Hailey-Hailey, or Darier's disease); Received (...) Atopic Dermatitis Research Network (ADRN) Influenza Vaccine Study Atopic Dermatitis Research Network (ADRN) Influenza Vaccine Study - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Atopic Dermatitis Research

2012 Clinical Trials

165. Malignancy and dermatitis. Full Text available with Trip Pro

Malignancy and dermatitis. 5470450 1970 12 02 2018 11 13 0007-1447 4 5726 1970 Oct 03 British medical journal Br Med J Malignancy and dermatitis. 55 Horgan J H JH eng Case Reports Journal Article England Br Med J 0372673 0007-1447 AIM IM Adult Carcinoma complications Dermatitis Herpetiformis complications Humans Lung Neoplasms complications Male Middle Aged 1970 10 3 1970 10 3 0 1 1970 10 3 0 0 ppublish 5470450 PMC1820571 Br Med J. 1970 Mar 7;1(5696):610 5440239

1970 British medical journal

166. Rubber boot dermatitis in Newfoundland: a survey of 30 patients. Full Text available with Trip Pro

Adult Aged Benzene Derivatives Carbamates Chromates Dermatitis Herpetiformis etiology Dermatitis, Contact diagnosis etiology Dermatitis, Exfoliative etiology Dermatitis, Occupational etiology Diagnosis, Differential Eczema diagnosis Eczema, Dyshidrotic etiology Fisheries Foot Dermatoses etiology Guanidines Humans Hydrocortisone therapeutic use Hypersensitivity etiology Leg Dermatoses etiology Male Middle Aged Neurodermatitis diagnosis Newfoundland and Labrador Plastics Purpura diagnosis Quinolines (...) Rubber boot dermatitis in Newfoundland: a survey of 30 patients. 4236654 1969 03 07 2018 11 13 0008-4409 100 1 1969 Jan 04 Canadian Medical Association journal Can Med Assoc J Rubber boot dermatitis in Newfoundland: a survey of 30 patients. 13-9 Ross J B JB eng Journal Article Canada Can Med Assoc J 0414110 0008-4409 0 Benzene Derivatives 0 Carbamates 0 Chromates 0 Guanidines 0 Plastics 0 Quinolines 0 Sulfides 0 Thiazoles 76H78MJJ52 Trimeprazine 9006-04-6 Rubber WI4X0X7BPJ Hydrocortisone AIM IM

1969 Canadian Medical Association Journal

167. Paraneoplastic pemphigus herpetiformis with IgG antibodies to desmoglein 3 and without mucosal lesions. Full Text available with Trip Pro

Paraneoplastic pemphigus herpetiformis with IgG antibodies to desmoglein 3 and without mucosal lesions. Pemphigus herpetiformis (PH) is a rare clinical entity that combines the clinical features of dermatitis herpetiformis and the immunopathologic features of pemphigus. The target antigen is usually desmoglein 1, with exceptional cases manifesting autoantibodies against desmoglein 3. More recently, it has been found that many patients with PH also demonstrate autoantibodies against desmocollin

2011 Archives of Dermatology

168. Use of Dapsone Gel, 5% for Treating Dermatitis Herpetiformis

Use of Dapsone Gel, 5% for Treating Dermatitis Herpetiformis Use of Dapsone Gel, 5% for Treating Dermatitis Herpetiformis - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Use of Dapsone Gel, 5% for Treating (...) Dermatitis Herpetiformis The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT01115244 Recruitment Status : Terminated (poor enrollment and lack of funding) First Posted : May 4, 2010 Results First Posted : December 14, 2017 Last Update Posted : December 14, 2017 Sponsor: Vanderbilt University Medical Center

2010 Clinical Trials

169. The association of dermatitis herpetiformis and systemic lupus erythematosus. (Abstract)

The association of dermatitis herpetiformis and systemic lupus erythematosus. Dermatitis herpetiformis (DH) is an immune-mediated cutaneous disease occasionally associated with celiac disease, but rarely associated with systemic lupus erythematosus (SLE). The combination of DH and SLE is immunologically mediated and suggests a relationship between the two conditions. We describe a woman with DH and SLE with a novel HLA phenotype.Copyright © 2009 American Academy of Dermatology, Inc. Published

2010 Journal of American Academy of Dermatology

170. Novel Screening Assay Performance in Pediatric Celiac Disease and Adult Dermatitis Herpetiformis. (Abstract)

Novel Screening Assay Performance in Pediatric Celiac Disease and Adult Dermatitis Herpetiformis. : Several serologic assays are commercially available to aid in the diagnosis of gluten-sensitive enteropathy (GSE). Our objective in this study was to assess the performance of a novel combined antigen-screening assay for GSE.: Deidentified sera from 111 pediatric patients suspected of having celiac disease (CD), 130 adults diagnosed with dermatitis herpetiformis (DH), and 77 pediatric and 49

2010 Journal of Pediatric Gastroenterology and Nutrition

171. Registry for the Atopic Dermatitis Research Network

of complete excision are not exclusionary. Who have any skin disease other than AD that might compromise the stratum corneum barrier (e.g., bullous diseases, psoriasis, cutaneous T cell lymphoma [also called Mycosis Fungoides or Sezary syndrome], dermatitis herpetiformis, Hailey-Hailey, or Darier's disease). Who have a history of systemic immunological illness (e.g., immunodeficiency disorders such as human immunodeficiency virus [HIV] or lupus erythematosus) other than the condition being studied. Who (...) Registry for the Atopic Dermatitis Research Network Registry for the Atopic Dermatitis Research Network - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Registry for the Atopic Dermatitis Research Network

2011 Clinical Trials

172. Assessment of pruritus

/pubmed/19663870?tool=bestpractice.com Differentials Atopic dermatitis Urticaria Insect bite Pinworm infection Scabies Lichen planus Psoriasis Skin xerosis Cholestatic pruritus Chronic renal failure (uraemic pruritus) Postherpetic itch Depression Schizophrenia Drug-induced pruritus Post-burn pruritus Ascariasis Amoebiasis Giardiasis Cutaneous larva migrans Schistosomiasis Tapeworm infection Trichinellosis Dermatitis herpetiformis Bullous pemphigoid Iron deficiency anaemia Diabetic peripheral (...) =bestpractice.com Importantly, it has been demonstrated that the brain activity in patients suffering from chronic itch upon pruritic stimuli differs significantly from that observed in healthy subjects. Ishiuji Y, Coghill RC, Patel TS, et al. Distinct patterns of brain activity evoked by histamine-induced itch reveal an association with itch intensity and disease severity in atopic dermatitis. Br J Dermatol. 2009;161:1072-1080. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784001/ http://www.ncbi.nlm.nih.gov

2018 BMJ Best Practice

173. Assessment of vesicular-bullous rash

of blisters) may be an early indicator of autoimmune blistering diseases (e.g., bullous pemphigoid and dermatitis herpetiformis). Classification Blisters occur at various depths of the epidermis or dermis and may be classified based on the depth of skin involved. A broad classification divides blisters into those arising within the epidermis (intraepidermal, which includes subcorneal) and those that develop below the epidermis (subepidermal, which includes the basement membrane zone). Differentials Herpes (...) pemphigoid) Epidermolysis bullosa acquisita Bullous lupus erythematosus Dermatitis herpetiformis Contributors Authors Assistant Clinical Professor Departments of Dermatology and Pathology University of California Davis Medical Center Sacramento CA Disclosures KLB declares that she has no competing interests. Peer reviewers Professor of Dermato-Epidemiology Center of Evidence Based Dermatology Nottingham University Hospitals NHS Trust Queen's Medical Centre Nottingham UK Disclosures HCW declares that he

2018 BMJ Best Practice

174. Assessment of pruritus

/pubmed/19663870?tool=bestpractice.com Differentials Atopic dermatitis Urticaria Insect bite Pinworm infection Scabies Lichen planus Psoriasis Skin xerosis Cholestatic pruritus Chronic renal failure (uraemic pruritus) Postherpetic itch Depression Schizophrenia Drug-induced pruritus Post-burn pruritus Ascariasis Amoebiasis Giardiasis Cutaneous larva migrans Schistosomiasis Tapeworm infection Trichinellosis Dermatitis herpetiformis Bullous pemphigoid Iron deficiency anaemia Diabetic peripheral (...) =bestpractice.com Importantly, it has been demonstrated that the brain activity in patients suffering from chronic itch upon pruritic stimuli differs significantly from that observed in healthy subjects. Ishiuji Y, Coghill RC, Patel TS, et al. Distinct patterns of brain activity evoked by histamine-induced itch reveal an association with itch intensity and disease severity in atopic dermatitis. Br J Dermatol. 2009;161:1072-1080. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784001/ http://www.ncbi.nlm.nih.gov

2018 BMJ Best Practice

175. Coeliac disease

, hypertrophy of the intestinal crypts, and increased numbers of lymphocytes in the epithelium and lamina propria. Locally these changes lead to gastrointestinal symptoms and malabsorption. Systemic manifestations are diverse, potentially affecting almost every organ system. History and exam immunoglobulin (IgA) deficiency diarrhoea bloating abdominal pain/discomfort anaemia dermatitis herpetiformis family history osteopenia/osteoporosis fatigue weight loss failure to thrive type 1 diabetes autoimmune

2018 BMJ Best Practice

176. Coeliac disease

, hypertrophy of the intestinal crypts, and increased numbers of lymphocytes in the epithelium and lamina propria. Locally these changes lead to gastrointestinal symptoms and malabsorption. Systemic manifestations are diverse, potentially affecting almost every organ system. History and exam immunoglobulin (IgA) deficiency diarrhoea bloating abdominal pain/discomfort anaemia dermatitis herpetiformis family history osteopenia/osteoporosis fatigue weight loss failure to thrive type 1 diabetes autoimmune

2017 BMJ Best Practice

177. Coeliac disease: recognition, assessment and management

, activate an abnormal mucosal immune response. Clinical and histological improvements usually follow when gluten is excluded from the diet. Coeliac disease can present with a wide range of clinical features, both gastrointestinal (such as indigestion, diarrhoea, abdominal pain, bloating, distension or constipation) and non-gastrointestinal (such as fatigue, dermatitis herpetiformis, anaemia, osteoporosis, reproductive problems, neuropathy, ataxia or delayed puberty). Children may also present

2015 National Institute for Health and Clinical Excellence - Clinical Guidelines

178. Iodine thyroid blocking: Guidelines for use in planning and responding to radiological and nuclear emergencies

- trointestinal disturbances and minor rashes. There are some rare but clinically relevant reactions, e.g. in patients with dermatitis herpetiformis or hypocomplementemic vascu- litis. Risk groups for such reactions include those with pre-existing thyroid disorders and iodine hypersensitivity (38,39). In case of hypersensitivity to iodine, use of potassium perchlorate can be considered to supress iodine uptake by the thyroid gland during the time of potential exposure (40). The use of additives

2017 World Health Organisation Guidelines

179. CRACKCast E120 – Dermatologic presentations

, malignancy, or treatment with corticosteroids, antibiotics, or immunosuppressive agents. [6] Describe the stepwise management of diaper dermatitis (list 4) “Contact dermatitis is an inflammatory reaction of the skin to a chemical, physical, or biologic agent, which acts as an irritant or allergic sensitizer. The primary lesions of contact dermatitis are papules, vesicles, and bullae on an erythematous base. Streaky, linear, intensely pruritic lesions are characteristic. A pattern in the region in contact (...) or vesicles should be treated with cool wet compresses of Domeboro or Burow’s solutions (aluminum acetate). Topical baths, available over the counter, may also be comforting. Systemic antihistamines, such as hydroxyzine and diphenhydramine, may help control pruritus; nonsedating antihistamines are preferred for use during the day. If present, secondary bacterial infection must also be treated. Know your ddx: Cutaneous candidiasis Contact dermatitis Atopic dermatitis Tinea cruris Intertrigo HSV

2017 CandiEM

180. Celiac Disease: Screening

, osteoporosis, chronic fatigue, peripheral neuropathy or ataxia, aphthous stomatitis, dermatitis herpetiformis, infertility, recurrent fetal loss, or short stature. Children may also experience pubertal delay and dental enamel defects. For patients with subclinical disease, symptoms may be mild and not recognized until after initiation of a gluten-free diet. Patients with silent, or asymptomatic, celiac disease have been diagnosed by serologic testing and intestinal biopsy but do not have the typical signs

2017 U.S. Preventive Services Task Force

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