How to Trip Rapid Review

Step 1: Select articles relevant to your search (remember the system is only optimised for single intervention studies)

Step 2: press

Step 3: review the result, and maybe amend the or if you know better! If we're unsure of the overall sentiment of the trial we will display the conclusion under the article title. We then require you to tell us what the correct sentiment is.

557 results for

Dermatitis Herpetiformis

by
...
Latest & greatest
Alerts

Export results

Use check boxes to select individual results below

SmartSearch available

Trip's SmartSearch engine has discovered connected searches & results. Click to show

161. Pemphigus Herpetiformis (Diagnosis)

Updated: Nov 16, 2018 Author: Blanca Anais Estupiñan; Chief Editor: William D James, MD Share Email Print Feedback Close Sections Sections Pemphigus Herpetiformis Overview Background Pemphigus herpetiformis is a rare autoimmune bullous skin disorder that is considered a clinical variant of pemphigus. Classically, it combines the clinical features of dermatitis herpetiformis with the immunopathologic features of pemphigus. Previously, pemphigus was described using various terms, including herpetiform (...) pemphigus, acantholytic herpetiform dermatitis, pemphigus controlled by sulfapyridine, and mixed bullous disease. Because pemphigus herpetiformis is a clinical variant of pemphigus, it may be more appropriately described with a term that begins with the general group term (pemphigus), followed by a term for the variant subset (herpetiformis), similar to the terms for other pemphigus variants, such as , , , and pemphigus vegetans. [ , ] Next: Pathophysiology Pemphigus herpetiformis appears to be mediated

2014 eMedicine.com

162. Dermatitis Herpetiformis

Dermatitis Herpetiformis Dermatitis Herpetiformis - Dermatologic Disorders - MSD Manual Professional Edition Brought to you by The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases The trusted provider of medical information since 1899 SEARCH SEARCH MEDICAL TOPICS Common Health Topics Resources QUIZZES & CASES Quizzes Cases / / / / IN THIS TOPIC OTHER TOPICS IN THIS CHAPTER Test your knowledge Diagnostic (...) at Dartmouth University Click here for Patient Education NOTE: This is the Professional Version. CONSUMERS: Topic Resources Dermatitis herpetiformis is an intensely pruritic, chronic, autoimmune, papulovesicular cutaneous eruption strongly associated with celiac disease. Typical findings are clusters of intensely pruritic, erythematous, urticarial lesions, as well as vesicles, papules, and bullae, usually distributed symmetrically on extensor surfaces. Diagnosis is by skin biopsy with direct

2013 Merck Manual (19th Edition)

163. Paraneoplastic pemphigus herpetiformis with IgG antibodies to desmoglein 3 and without mucosal lesions. (PubMed)

Paraneoplastic pemphigus herpetiformis with IgG antibodies to desmoglein 3 and without mucosal lesions. Pemphigus herpetiformis (PH) is a rare clinical entity that combines the clinical features of dermatitis herpetiformis and the immunopathologic features of pemphigus. The target antigen is usually desmoglein 1, with exceptional cases manifesting autoantibodies against desmoglein 3. More recently, it has been found that many patients with PH also demonstrate autoantibodies against desmocollin

Full Text available with Trip Pro

2011 Archives of Dermatology

164. The association of dermatitis herpetiformis and systemic lupus erythematosus. (PubMed)

The association of dermatitis herpetiformis and systemic lupus erythematosus. Dermatitis herpetiformis (DH) is an immune-mediated cutaneous disease occasionally associated with celiac disease, but rarely associated with systemic lupus erythematosus (SLE). The combination of DH and SLE is immunologically mediated and suggests a relationship between the two conditions. We describe a woman with DH and SLE with a novel HLA phenotype.Copyright © 2009 American Academy of Dermatology, Inc. Published

2010 Journal of American Academy of Dermatology

165. Novel Screening Assay Performance in Pediatric Celiac Disease and Adult Dermatitis Herpetiformis. (PubMed)

Novel Screening Assay Performance in Pediatric Celiac Disease and Adult Dermatitis Herpetiformis. : Several serologic assays are commercially available to aid in the diagnosis of gluten-sensitive enteropathy (GSE). Our objective in this study was to assess the performance of a novel combined antigen-screening assay for GSE.: Deidentified sera from 111 pediatric patients suspected of having celiac disease (CD), 130 adults diagnosed with dermatitis herpetiformis (DH), and 77 pediatric and 49

2010 Journal of Pediatric Gastroenterology and Nutrition

166. Use of Dapsone Gel, 5% for Treating Dermatitis Herpetiformis

Use of Dapsone Gel, 5% for Treating Dermatitis Herpetiformis Use of Dapsone Gel, 5% for Treating Dermatitis Herpetiformis - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Use of Dapsone Gel, 5% for Treating (...) Dermatitis Herpetiformis The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT01115244 Recruitment Status : Terminated (poor enrollment and lack of funding) First Posted : May 4, 2010 Results First Posted : December 14, 2017 Last Update Posted : December 14, 2017 Sponsor: Vanderbilt University Medical Center

2010 Clinical Trials

167. Atopic Dermatitis Research Network (ADRN) Influenza Vaccine Study

; Participated in another clinical trial investigating a vaccine, drug, medical device, or a medical procedure in the four weeks preceding the study vaccination or who plan to participate in another clinical trial during the present study period; Any skin disease other than AD that might compromise the stratum corneum barrier (e.g., bullous disease, psoriasis, cutaneous T cell lymphoma [also called Mycosis Fungoides or Sezary syndrome], dermatitis herpetiformis, Hailey-Hailey, or Darier's disease); Received (...) Atopic Dermatitis Research Network (ADRN) Influenza Vaccine Study Atopic Dermatitis Research Network (ADRN) Influenza Vaccine Study - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Atopic Dermatitis Research

2012 Clinical Trials

168. Registry for the Atopic Dermatitis Research Network

of complete excision are not exclusionary. Who have any skin disease other than AD that might compromise the stratum corneum barrier (e.g., bullous diseases, psoriasis, cutaneous T cell lymphoma [also called Mycosis Fungoides or Sezary syndrome], dermatitis herpetiformis, Hailey-Hailey, or Darier's disease). Who have a history of systemic immunological illness (e.g., immunodeficiency disorders such as human immunodeficiency virus [HIV] or lupus erythematosus) other than the condition being studied. Who (...) Registry for the Atopic Dermatitis Research Network Registry for the Atopic Dermatitis Research Network - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Registry for the Atopic Dermatitis Research Network

2011 Clinical Trials

169. Malignancy and dermatitis. (PubMed)

Malignancy and dermatitis. 5470450 1970 12 02 2018 11 13 0007-1447 4 5726 1970 Oct 03 British medical journal Br Med J Malignancy and dermatitis. 55 Horgan J H JH eng Case Reports Journal Article England Br Med J 0372673 0007-1447 AIM IM Adult Carcinoma complications Dermatitis Herpetiformis complications Humans Lung Neoplasms complications Male Middle Aged 1970 10 3 1970 10 3 0 1 1970 10 3 0 0 ppublish 5470450 PMC1820571 Br Med J. 1970 Mar 7;1(5696):610 5440239

Full Text available with Trip Pro

1970 British medical journal

170. Rubber boot dermatitis in Newfoundland: a survey of 30 patients. (PubMed)

Adult Aged Benzene Derivatives Carbamates Chromates Dermatitis Herpetiformis etiology Dermatitis, Contact diagnosis etiology Dermatitis, Exfoliative etiology Dermatitis, Occupational etiology Diagnosis, Differential Eczema diagnosis Eczema, Dyshidrotic etiology Fisheries Foot Dermatoses etiology Guanidines Humans Hydrocortisone therapeutic use Hypersensitivity etiology Leg Dermatoses etiology Male Middle Aged Neurodermatitis diagnosis Newfoundland and Labrador Plastics Purpura diagnosis Quinolines (...) Rubber boot dermatitis in Newfoundland: a survey of 30 patients. 4236654 1969 03 07 2018 11 13 0008-4409 100 1 1969 Jan 04 Canadian Medical Association journal Can Med Assoc J Rubber boot dermatitis in Newfoundland: a survey of 30 patients. 13-9 Ross J B JB eng Journal Article Canada Can Med Assoc J 0414110 0008-4409 0 Benzene Derivatives 0 Carbamates 0 Chromates 0 Guanidines 0 Plastics 0 Quinolines 0 Sulfides 0 Thiazoles 76H78MJJ52 Trimeprazine 9006-04-6 Rubber WI4X0X7BPJ Hydrocortisone AIM IM

Full Text available with Trip Pro

1969 Canadian Medical Association Journal

171. Prediction of Clinical and Mucosal Severity of Coeliac Disease and Dermatitis Herpetiformis by Quantification of IgA/IgG Serum Antibodies to Tissue Transglutaminase. (PubMed)

Prediction of Clinical and Mucosal Severity of Coeliac Disease and Dermatitis Herpetiformis by Quantification of IgA/IgG Serum Antibodies to Tissue Transglutaminase. We analysed whether the quantification of autoantibodies against tissue transglutaminase could be used to predict mucosal destruction and disease severity in patients with gluten sensitivity.One hundred seventy patients with coeliac disease (CD), comprising 52 children with severe malabsorption (group I), 59 children with mild (...) symptoms (group II), 59 adults (group III), 134 patients with dermatitis herpetiformis (DH), and 131 disease controls, were studied. Serial serum samples of patients in groups I and II on a gluten-free diet were also included. Serum levels of antibodies against recombinant tissue transglutaminase were determined with ELISA using standard curves for quantification of antibodies.Immunoglobulin (Ig)A antibodies against tissue transglutaminase (IgA-TGA) were detected in all of the patients with CD

2009 Journal of Pediatric Gastroenterology and Nutrition

172. Autoantibodies against epidermal transglutaminase are a sensitive diagnostic marker in patients with dermatitis herpetiformis on a normal or gluten-free diet. (PubMed)

Autoantibodies against epidermal transglutaminase are a sensitive diagnostic marker in patients with dermatitis herpetiformis on a normal or gluten-free diet. Dermatitis herpetiformis (DH) is a cutaneous manifestation of gluten-sensitive enteropathy (celiac disease). Patients with DH demonstrate circulating IgA antibodies against epidermal transglutaminase (eTG) and tissue transglutaminase (tTG). It has been suggested that eTG is the autoantigen of DH.The purpose of this study

2009 Journal of American Academy of Dermatology

173. Coeliac disease

, hypertrophy of the intestinal crypts, and increased numbers of lymphocytes in the epithelium and lamina propria. Locally these changes lead to gastrointestinal symptoms and malabsorption. Systemic manifestations are diverse, potentially affecting almost every organ system. History and exam immunoglobulin (IgA) deficiency diarrhoea bloating abdominal pain/discomfort anaemia dermatitis herpetiformis family history osteopenia/osteoporosis fatigue weight loss failure to thrive type 1 diabetes autoimmune

2018 BMJ Best Practice

174. Assessment of vesicular-bullous rash

of blisters) may be an early indicator of autoimmune blistering diseases (e.g., bullous pemphigoid and dermatitis herpetiformis). Classification Blisters occur at various depths of the epidermis or dermis and may be classified based on the depth of skin involved. A broad classification divides blisters into those arising within the epidermis (intraepidermal, which includes subcorneal) and those that develop below the epidermis (subepidermal, which includes the basement membrane zone). Differentials Herpes (...) pemphigoid) Epidermolysis bullosa acquisita Bullous lupus erythematosus Dermatitis herpetiformis Contributors Authors Assistant Clinical Professor Departments of Dermatology and Pathology University of California Davis Medical Center Sacramento CA Disclosures KLB declares that she has no competing interests. Peer reviewers Professor of Dermato-Epidemiology Center of Evidence Based Dermatology Nottingham University Hospitals NHS Trust Queen's Medical Centre Nottingham UK Disclosures HCW declares that he

2018 BMJ Best Practice

175. Assessment of pruritus

/pubmed/19663870?tool=bestpractice.com Differentials Atopic dermatitis Urticaria Insect bite Pinworm infection Scabies Lichen planus Psoriasis Skin xerosis Cholestatic pruritus Chronic renal failure (uraemic pruritus) Postherpetic itch Depression Schizophrenia Drug-induced pruritus Post-burn pruritus Ascariasis Amoebiasis Giardiasis Cutaneous larva migrans Schistosomiasis Tapeworm infection Trichinellosis Dermatitis herpetiformis Bullous pemphigoid Iron deficiency anaemia Diabetic peripheral (...) =bestpractice.com Importantly, it has been demonstrated that the brain activity in patients suffering from chronic itch upon pruritic stimuli differs significantly from that observed in healthy subjects. Ishiuji Y, Coghill RC, Patel TS, et al. Distinct patterns of brain activity evoked by histamine-induced itch reveal an association with itch intensity and disease severity in atopic dermatitis. Br J Dermatol. 2009;161:1072-1080. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784001/ http://www.ncbi.nlm.nih.gov

2018 BMJ Best Practice

176. Assessment of pruritus

/pubmed/19663870?tool=bestpractice.com Differentials Atopic dermatitis Urticaria Insect bite Pinworm infection Scabies Lichen planus Psoriasis Skin xerosis Cholestatic pruritus Chronic renal failure (uraemic pruritus) Postherpetic itch Depression Schizophrenia Drug-induced pruritus Post-burn pruritus Ascariasis Amoebiasis Giardiasis Cutaneous larva migrans Schistosomiasis Tapeworm infection Trichinellosis Dermatitis herpetiformis Bullous pemphigoid Iron deficiency anaemia Diabetic peripheral (...) =bestpractice.com Importantly, it has been demonstrated that the brain activity in patients suffering from chronic itch upon pruritic stimuli differs significantly from that observed in healthy subjects. Ishiuji Y, Coghill RC, Patel TS, et al. Distinct patterns of brain activity evoked by histamine-induced itch reveal an association with itch intensity and disease severity in atopic dermatitis. Br J Dermatol. 2009;161:1072-1080. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784001/ http://www.ncbi.nlm.nih.gov

2018 BMJ Best Practice

177. Coeliac disease

, hypertrophy of the intestinal crypts, and increased numbers of lymphocytes in the epithelium and lamina propria. Locally these changes lead to gastrointestinal symptoms and malabsorption. Systemic manifestations are diverse, potentially affecting almost every organ system. History and exam immunoglobulin (IgA) deficiency diarrhoea bloating abdominal pain/discomfort anaemia dermatitis herpetiformis family history osteopenia/osteoporosis fatigue weight loss failure to thrive type 1 diabetes autoimmune

2017 BMJ Best Practice

178. CRACKCast E120 – Dermatologic presentations

, malignancy, or treatment with corticosteroids, antibiotics, or immunosuppressive agents. [6] Describe the stepwise management of diaper dermatitis (list 4) “Contact dermatitis is an inflammatory reaction of the skin to a chemical, physical, or biologic agent, which acts as an irritant or allergic sensitizer. The primary lesions of contact dermatitis are papules, vesicles, and bullae on an erythematous base. Streaky, linear, intensely pruritic lesions are characteristic. A pattern in the region in contact (...) or vesicles should be treated with cool wet compresses of Domeboro or Burow’s solutions (aluminum acetate). Topical baths, available over the counter, may also be comforting. Systemic antihistamines, such as hydroxyzine and diphenhydramine, may help control pruritus; nonsedating antihistamines are preferred for use during the day. If present, secondary bacterial infection must also be treated. Know your ddx: Cutaneous candidiasis Contact dermatitis Atopic dermatitis Tinea cruris Intertrigo HSV

2017 CandiEM

179. Iodine thyroid blocking: Guidelines for use in planning and responding to radiological and nuclear emergencies

- trointestinal disturbances and minor rashes. There are some rare but clinically relevant reactions, e.g. in patients with dermatitis herpetiformis or hypocomplementemic vascu- litis. Risk groups for such reactions include those with pre-existing thyroid disorders and iodine hypersensitivity (38,39). In case of hypersensitivity to iodine, use of potassium perchlorate can be considered to supress iodine uptake by the thyroid gland during the time of potential exposure (40). The use of additives

2017 World Health Organisation Guidelines

180. Celiac Disease: Screening

by persons with celiac disease causes immune-mediated inflammatory damage to the small intestine mucosa, resulting in malabsorption of nutrients. Celiac disease can have several different presentations. Classic celiac disease is associated with diarrhea, abdominal pain, and weight loss. However, celiac disease is also associated with nongastrointestinal, nonspecific manifestations of disease such as anemia, osteoporosis, chronic fatigue, peripheral neuropathy or ataxia, aphthous stomatitis, dermatitis (...) herpetiformis, infertility, recurrent fetal loss, or short stature. Children may also experience pubertal delay and dental enamel defects. For patients with subclinical disease, symptoms may be mild and not recognized until after initiation of a gluten-free diet. Patients with silent, or asymptomatic, celiac disease have been diagnosed by serologic testing and intestinal biopsy but do not have the typical signs or symptoms of celiac disease. Patients with potential celiac disease have positive serology

2017 U.S. Preventive Services Task Force

To help you find the content you need quickly, you can filter your results via the categories on the right-hand side >>>>