How to Trip Rapid Review

Step 1: Select articles relevant to your search (remember the system is only optimised for single intervention studies)

Step 2: press

Step 3: review the result, and maybe amend the or if you know better! If we're unsure of the overall sentiment of the trial we will display the conclusion under the article title. We then require you to tell us what the correct sentiment is.

557 results for

Dermatitis Herpetiformis

by
...
Latest & greatest
Alerts

Export results

Use check boxes to select individual results below

SmartSearch available

Trip's SmartSearch engine has discovered connected searches & results. Click to show

61. Gene Expression Profiling in Dermatitis Herpetiformis Skin Lesions (PubMed)

Gene Expression Profiling in Dermatitis Herpetiformis Skin Lesions Dermatitis herpetiformis (DH) is an autoimmune blistering skin disease associated with gluten-sensitive enteropathy (CD). In order to investigate the pathogenesis of skin lesions at molecular level, we analysed the gene expression profiles in skin biopsies from 6 CD patients with DH and 6 healthy controls using Affymetrix HG-U133A 2.0 arrays. 486 genes were differentially expressed in DH skin compared to normal skin: 225 were

Full Text available with Trip Pro

2012 Clinical and Developmental Immunology

62. Distinct Characteristics in Japanese Dermatitis Herpetiformis: A Review of All 91 Japanese Patients over the Last 35 Years (PubMed)

Distinct Characteristics in Japanese Dermatitis Herpetiformis: A Review of All 91 Japanese Patients over the Last 35 Years We reviewed all 91 Japanese dermatitis herpetiformis (DH) patients reported over the last 35 years. The male-to-female ratio was 2 : 1. The mean age at onset was 43.8, and 13 years earlier for female patients. More than half of these Japanese DH patients showed granular IgA deposition in the papillary dermis, and another one-third showed fibrillar IgA deposition. The male

Full Text available with Trip Pro

2012 Clinical and Developmental Immunology

63. Dermatitis Herpetiformis: From the Genetics to the Development of Skin Lesions (PubMed)

Dermatitis Herpetiformis: From the Genetics to the Development of Skin Lesions Dermatitis herpetiformis (DH) is a rare autoimmune disease linked to gluten sensitivity with a chronic-relapsing course. It is currently considered to be the specific cutaneous manifestation of celiac disease (CD). Both conditions are mediated by the IgA class of autoantibodies, and the diagnosis of DH is dependent on the detection of granular deposits of IgA in the skin. There is an underlying genetic predisposition

Full Text available with Trip Pro

2012 Clinical and Developmental Immunology

64. Recent Advances in Dermatitis Herpetiformis (PubMed)

Recent Advances in Dermatitis Herpetiformis Dermatitis herpetiformis is an autoimmune bullous disease that is associated with gluten sensitivity which typically presents as celiac disease. As both conditions are multifactorial disorders, it is not clear how specific pathogenetic mechanisms may lead to the dysregulation of immune responses in the skin and small bowel, respectively. Recent studies have demonstrated that IgA and antibodies against epidermal transglutaminase 3 play an important (...) role in the pathogenesis of dermatitis herpetiformis. Here, we review recent immunopathological progress in understanding the pathogenesis of dermatitis herpetiformis.

Full Text available with Trip Pro

2012 Clinical and Developmental Immunology

65. Dermatitis herpetiformis: Relevance of the physical examination to diagnosis suspicion (PubMed)

Dermatitis herpetiformis: Relevance of the physical examination to diagnosis suspicion 22893336 2012 11 12 2018 11 13 1715-5258 58 8 2012 Aug Canadian family physician Medecin de famille canadien Can Fam Physician Dermatitis herpetiformis: relevance of the physical examination to diagnosis suspicion. 843-7 Criado Paulo Ricardo PR Department of Dermatology, University of São Paulo, Brazil. prcriado@uol.com.br Criado Roberta Fachini Jardim RF Aoki Valeria V Belda Walter W Jr Halpern Ilana I (...) Landman Gilles G Vasconcellos Cidia C eng Case Reports Journal Article Canada Can Fam Physician 0120300 0008-350X IM Adult Celiac Disease complications diagnosis Dermatitis Herpetiformis diagnosis etiology Diagnosis, Differential Female Humans Male Middle Aged Physical Examination 2012 8 16 6 0 2012 8 16 6 0 2012 11 13 6 0 ppublish 22893336 58/8/843 PMC3418983 Clin Exp Dermatol. 2010 Mar;35(2):206-8 20015286 J Eur Acad Dermatol Venereol. 2009 Jun;23(6):633-8 19470076 Cutis. 2002 Oct;70(4):217-23

Full Text available with Trip Pro

2012 Canadian Family Physician

66. Dermatitis Herpetiformis

Dermatitis Herpetiformis Dermatitis Herpetiformis Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Dermatitis Herpetiformis Dermatitis (...) Herpetiformis Aka: Dermatitis Herpetiformis From Related Chapters II. Epidemiology : Up to 39 cases per 100,000 Usual onset at 20 to 40 years Gender preponderance in men by 2:1 ratio Ethnicity Most commonly affects white patients Rarely affects black or asian patients III. Pathophysiology Idiopathic condition Usually associated with IV. Symptoms: Precede lesion onset by 8 to 12 hours Intense Skin burning V. Signs Grouping of lesions may occur (herpetiform-like) Altered pigmentation at sites of healed

2015 FP Notebook

67. Novel Advances in Dermatitis Herpetiformis (PubMed)

Novel Advances in Dermatitis Herpetiformis 23316253 2013 08 12 2013 01 14 1740-2530 2012 2012 Clinical & developmental immunology Clin. Dev. Immunol. Novel advances in dermatitis herpetiformis. 450109 10.1155/2012/450109 Fabbri Paolo P Calabrò Antonino Salvatore AS Hashimoto Takashi T Fasano Alessio A Caproni Marzia M eng Editorial Research Support, Non-U.S. Gov't 2012 12 19 Egypt Clin Dev Immunol 101183692 1740-2522 0 Immunoglobulin A EC 2.3.2.13 Transglutaminases IM Celiac Disease immunology (...) metabolism pathology therapy Dermatitis Herpetiformis immunology metabolism pathology therapy Humans Immunoglobulin A immunology Transglutaminases immunology metabolism 2012 11 04 2012 11 04 2013 1 15 6 0 2013 1 15 6 0 2013 8 13 6 0 ppublish 23316253 10.1155/2012/450109 PMC3535881

Full Text available with Trip Pro

2012 Clinical and Developmental Immunology

68. Comparison of Two Different Skin Creams and Their Effect on Transepidermal Water Loss (TEWL) in Pediatric Patients With Atopic Dermatitis

) AD affecting at least 3 different skin areas (contralateral arms and one lower extremity) Exclusion Criteria: Inability or unwillingness of a parent guardian to give written informed consent, or to comply with study protocol. Participants with skin disease other than AD that might compromise the stratum corneum barriers (e.g., bullous diseases, psoriasis, cutaneous T-cell lymphoma (also called Mycosis Fungoides or Sezary syndrome], dermatitis herpetiformis, Hailey-Hailey, or Darier's disease (...) Comparison of Two Different Skin Creams and Their Effect on Transepidermal Water Loss (TEWL) in Pediatric Patients With Atopic Dermatitis Comparison of Two Different Skin Creams and Their Effect on Transepidermal Water Loss (TEWL) in Pediatric Patients With Atopic Dermatitis - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study

2018 Clinical Trials

69. Prevalence and incidence of dermatitis herpetiformis: a 40-year prospective study from Finland. (PubMed)

Prevalence and incidence of dermatitis herpetiformis: a 40-year prospective study from Finland. Dermatitis herpetiformis (DH) is an external manifestation of coeliac disease presenting with blistering rash and pathognomonic cutaneous IgA deposits. Better knowledge of subclinical forms and serological testing has resulted in a sharp increase in the incidence and prevalence of coeliac disease.To investigate the prevalence of DH and analyse whether the incidence of DH changed when the occurrence

2011 British Journal of Dermatology

70. Dermatitis herpetiformis Part II. Diagnosis, management, and prognosis. (PubMed)

Dermatitis herpetiformis Part II. Diagnosis, management, and prognosis. The prompt recognition of the clinical features of dermatitis herpetiformis (DH) is important, but securing a definitive diagnosis requires further work-up. Recent advances in understanding of the immunologic basis for DH have led to the development and wider availability of serologic testing, which is rapidly becoming an essential part of the diagnosis and management of DH. Part II of this series will detail the diagnosis

2011 Journal of American Academy of Dermatology

71. Dermatitis herpetiformis Part I. Epidemiology, pathogenesis, and clinical presentation. (PubMed)

Dermatitis herpetiformis Part I. Epidemiology, pathogenesis, and clinical presentation. Dermatitis herpetiformis (DH) is an autoimmune disease that is linked to gluten sensitivity and has a clear relationship to celiac disease. Both conditions are mediated by the IgA class of autoantibodies and the diagnosis of DH is dependent on detection of granular deposits of IgA in the skin. There is an underlying genetic predisposition to the development of DH but environmental factors are also important

2011 Journal of American Academy of Dermatology

72. Novel assay for detecting celiac disease-associated autoantibodies in dermatitis herpetiformis using deamidated gliadin-analogous fusion peptides. (PubMed)

Novel assay for detecting celiac disease-associated autoantibodies in dermatitis herpetiformis using deamidated gliadin-analogous fusion peptides. Dermatitis herpetiformis (DH) is a cutaneous manifestation of celiac disease (CD), the latter being identified by circulating autoantibodies against native gliadin (nGli), tissue transglutaminase (tTG), endomysium, or a combination of these. Recently, a novel serologic assay using deamidated gliadin-analogous fusion peptides (GAF3X) showed high

2011 Journal of American Academy of Dermatology

73. Leukocytoclastic vasculitis as the presenting feature of dermatitis herpetiformis. (PubMed)

Leukocytoclastic vasculitis as the presenting feature of dermatitis herpetiformis. Dermatitis herpetiformis is an autoimmune disease typically characterized by pruritic vesicles located on the extensor surfaces. Classic disease consists of neutrophils in the dermal papillae. Additional histopathologic findings include fibrin deposition and edema within the dermal papillae. Subepidermal vesicles also may be present. Direct immunofluorescence demonstrates granular IgA in the dermal papillae.A 58 (...) -year-old man with tender and pruritic erythematous macules and papules ranging from 2 to 6 mm in diameter had bilateral knee, elbow, forearm, scalp, and neck involvement. Petechiae also were present on the hands, thigh, knee, and ankle. A biopsy specimen initially demonstrated leukocytoclastic vasculitis. The results of workup for systemic vasculitis were negative. Subsequent biopsy specimens and direct immunofluorescence showed histologic evidence of dermatitis herpetiformis and leukocytoclastic

Full Text available with Trip Pro

2011 Archives of Dermatology

74. Topical treatment of dermatitis herpetiformis with betamethasone-17-valerate and fluocinolone acetonide. (PubMed)

Topical treatment of dermatitis herpetiformis with betamethasone-17-valerate and fluocinolone acetonide. 5333683 1967 03 07 2013 11 21 0001-5148 21 6 1966 Acta allergologica Acta Allergol Topical treatment of dermatitis herpetiformis with betamethasone-17-valerate and fluocinolone acetonide. 471-2 Björnberg A A Hellgren L L eng Clinical Trial Journal Article Randomized Controlled Trial Denmark Acta Allergol 0370567 0001-5148 0CD5FD6S2M Fluocinolone Acetonide 9842X06Q6M Betamethasone IM (...) Adolescent Adult Aged Betamethasone therapeutic use Child Clinical Trials as Topic Dermatitis Herpetiformis drug therapy Female Fluocinolone Acetonide therapeutic use Humans Male Middle Aged 1966 1 1 1966 1 1 0 1 1966 1 1 0 0 ppublish 5333683

1967 Acta Allergologica

75. The cellular infiltrate of the jejunum in adult coeliac disease and dermatitis herpetiformis following the reintroduction of dietary gluten. (PubMed)

The cellular infiltrate of the jejunum in adult coeliac disease and dermatitis herpetiformis following the reintroduction of dietary gluten. The cellular infiltrate of the jejunal mucosa has been studied in patients with both treated and untreated adult coeliac disease and dermatitis herpetiformis and serially in treated patients before and after the reintroduction of gluten to the diet. In adult coeliac disease and dermatitis herpetiformis the jejunal mucosa showed similar abnormalities (...) lymphocytes. These responses were essentially similar in both adult coeliac disease and in those dermatitis herpetiformis patients who had jejunal lesions before treatment. In dermatitis herpetiformis patients with normal jejunal morphology on a normal diet there was an upward trend in lamina propria plasma cells and intraepithelial lymphocytes within one to three weeks of taking extra dietary gluten. These results are compatible with the view that more than one immunological mechanism may be responsible

Full Text available with Trip Pro

1975 Gut

76. Gastric lesion in dermatitis herpetiformis. (PubMed)

Gastric lesion in dermatitis herpetiformis. Five of 33 patients with dermatitis herpetiformis (DH) were found to have gastric parietal cell antibody in their sera, whereas it was not found in 30 healthy controls of comparable age distribution. Fifteen of the patients with DH underwent further studies to investigate the histological and functional state of their gastric mucosa. Atrophic gastritis was found in all five patients whose sera contained gastric parietal cell antibody and in three

Full Text available with Trip Pro

1976 Gut

77. Lymphoma in dermatitis herpetiformis. (PubMed)

Lymphoma in dermatitis herpetiformis. 974595 1976 12 23 2018 11 13 0007-1447 2 6038 1976 Sep 25 British medical journal Br Med J Lymphoma in dermatitis herpetiformis. 757 Fowler J M JM Thomas D J DJ eng Case Reports Letter England Br Med J 0372673 0007-1447 AIM IM Dermatitis Herpetiformis complications Humans Lymphoma complications Male Middle Aged Skin Neoplasms complications 1976 9 25 1976 9 25 0 1 1976 9 25 0 0 ppublish 974595 PMC1688812 Am J Med. 1967 Jun;42(6):899-912 5338480 Proc R Soc

Full Text available with Trip Pro

1976 British medical journal

78. Dermatitis herpetiformis: effect of gluten-free diet on skin IgA and jejunal structure and function. (PubMed)

Dermatitis herpetiformis: effect of gluten-free diet on skin IgA and jejunal structure and function. To clarify the controversy about the effectiveness of a gluten-free diet in dermatitis herpetiformis, 10 highly motivated patients were investigated. The indices used to assess improvement included deposition of sub-epidermal IgA in unaffected skin, counts of intraepithelial lymphocytes, deposition of IgA in jejunal villi, and electrical tests of glucose absorption. In every patient subepidermal (...) gluten withdrawal, which confirmed that all nine patients were adhering to their diet. Routine screening for malabsorption proved to be unsatisfactory for showing the mild jejunal disease found in patients with dermatitis herpetiformis. The electrical test of glucose absorption showed subnormal results in all eight patients tested, however, and in six the results improved after gluten withdrawal.

Full Text available with Trip Pro

1977 British medical journal

79. Dermatitis herpetiformis: a comparative assessment of skin and bowel abnormality. (PubMed)

Dermatitis herpetiformis: a comparative assessment of skin and bowel abnormality. We reviewed 18 patients with a clinical diagnosis of dermatitis herpetiformis who were being treated with dapsone and were on an unrestricted diet. Diagnosis was confirmed by finding IgA deposits in the dermal papillae of unaffected skin. Dapsone was discontinued and biopsy of affected skin was carried out when the typical rash reappeared. The biopsy findings were graded according to the severity (...) of the histological changes. Small bowel tissue from each patient was examined and graded by stereo- and routine microscopy. Thirteen specimens (72%) were stereomicroscopically abnormal; all 18 showed villous atrophy, either partial or subtotal; and in 13 (72%) the interepithelial lymphocyte count was increased. No correlation was found between the histological severity of the skin and the small bowel lesions. Seemingly the severity of the skin rash in dermatitis herpetiformis is no guide to the degree of small

Full Text available with Trip Pro

1977 Journal of Clinical Pathology

80. Dermatitis herpetiformis and leiomyomas with HLA-B8, a marker of immune diseases. (PubMed)

Dermatitis herpetiformis and leiomyomas with HLA-B8, a marker of immune diseases. Multiple cutaneous leiomyomas and dermatitis herpetiformis (DH) were found in the same patient. This association has not been previously described, although the association of malignant tumours and DH is well known. HLA-B8 was found in this patient as well as in an older brother with polymyositis; this antigen is known to be associated with other immune diseases.

Full Text available with Trip Pro

1976 Canadian Medical Association Journal

To help you find the content you need quickly, you can filter your results via the categories on the right-hand side >>>>