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Dermatitis Herpetiformis

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541. Recurrent genital ulceration: a case of dermatitis herpetiformis. (PubMed)

Recurrent genital ulceration: a case of dermatitis herpetiformis. 2391115 1990 10 04 2018 11 13 0266-4348 66 4 1990 Aug Genitourinary medicine Genitourin Med Recurrent genital ulceration: a case of dermatitis herpetiformis. 270-1 Sivakumar K K Department of Genitourinary Medicine, Royal Victoria Hospital, Bournemouth, UK. Coelho D C DC Roy R B RB eng Case Reports Journal Article England Genitourin Med 8503853 0266-4348 IM Adult Dermatitis Herpetiformis complications pathology Humans Male Penile

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1990 Genitourinary Medicine

542. Antigliadin antibodies in dermatitis herpetiformis. (PubMed)

Antigliadin antibodies in dermatitis herpetiformis. 6681090 1985 05 01 2018 11 13 0041-6193 52 2 1983 The Ulster medical journal Ulster Med J Antigliadin antibodies in dermatitis herpetiformis. 113-7 McMillan S A SA Hutchison T T Haire M M Allen G G Kelly A A eng Journal Article Northern Ireland Ulster Med J 0417367 0041-6193 0 Antibodies 0 Plant Proteins 9007-90-3 Gliadin IM Adolescent Adult Aged Antibodies analysis Child Dermatitis Herpetiformis immunology pathology Diagnosis, Differential

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1983 The Ulster medical journal

543. Foods for patients with dermatitis herpetiformis (correction) (PubMed)

Foods for patients with dermatitis herpetiformis (correction) 6839237 1983 06 17 2018 11 13 0008-4409 128 10 1983 May 15 Canadian Medical Association journal Can Med Assoc J Foods for patients with dermatitis herpetiformis (correction) 1145 eng Letter Canada Can Med Assoc J 0414110 0008-4409 8002-80-0 Glutens AIM IM Dermatitis Herpetiformis diet therapy Glutens administration & dosage Humans 1983 5 15 1983 5 15 0 1 1983 5 15 0 0 ppublish 6839237 PMC1875284 Br J Dermatol. 1982 Dec;107(6):631-40

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1983 Canadian Medical Association Journal

544. Foods for patients with dermatitis herpetiformis. (PubMed)

Foods for patients with dermatitis herpetiformis. 6825017 1983 04 07 2018 11 13 0008-4409 128 5 1983 Mar 01 Canadian Medical Association journal Can Med Assoc J Foods for patients with dermatitis herpetiformis. 512 Hogan D J DJ eng Letter Canada Can Med Assoc J 0414110 0008-4409 8002-80-0 Glutens AIM IM Dermatitis Herpetiformis diet therapy Feeding Behavior Glutens administration & dosage Humans 1983 3 1 1983 3 1 0 1 1983 3 1 0 0 ppublish 6825017 PMC1874980 Harefuah. 1961 May 1;60:289-91

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1983 Canadian Medical Association Journal

545. Dermatitis herpetiformis: diagnosis, diet and demography. (PubMed)

Dermatitis herpetiformis: diagnosis, diet and demography. We describe a long term study of 76 patients with dermatitis herpetiformis. Unlike patients with coeliac disease, where the peak incidence was during the first and fourth decades, no dermatitis herpetiformis patients presented in the first decade; also, there was a male preponderance in dermatitis herpetiformis which contrasts with the excess of females in coeliac disease. The apparent prevalence of dermatitis herpetiformis was 11 per (...) 100 000 in our population; approximately one fifth of that of coeliac disease. Jejunal villous atrophy was present in 78% of our dermatitis herpetiformis patients, and a single jejunal biopsy was as effective at detecting this as the multiple biopsy technique. A majority of patients were able to stop, or radically reduce their dapsone or sulphapyridine treatment after the institution of a gluten free diet. Spontaneous remission of the skin lesion occurred in only two patients not receiving

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1984 Gut

546. Intestinal permeability in patients with coeliac disease and dermatitis herpetiformis. (PubMed)

Intestinal permeability in patients with coeliac disease and dermatitis herpetiformis. Intestinal permeability was investigated in patients with coeliac disease and dermatitis herpetiformis by a 51Chromium-labelled ethylenediaminetetraacetate (51Cr-EDTA) absorption test and the results correlated with histomorphometric analysis and intraepithelial lymphocyte counts of jejunal biopsies. The mean (+/- SD) 24 hour urine excretion of 51Cr-EDTA in 34 healthy volunteers was 1.9 +/- 0.5% of the orally (...) administered test dose. Patients with untreated coeliac disease (19) or untreated dermatitis herpetiformis (five) excreted significantly more 51Cr-EDTA than controls (5.9 +/- 2.7% and 4.6 +/- 2.1%, respectively, p less than 0.001) and all were outside the normal range of 1.0-2.6%. Patients with coeliac disease (42) treated for 6 months-23 years (mean 5 years) and patients with dermatitis herpetiformis (11) treated for 6 months-8 years (mean 3 years) excreted significantly more 51Cr-EDTA than controls, 4.2

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1985 Gut

547. Ultrastructural binding sites of endomysium antibodies from sera of patients with dermatitis herpetiformis and coeliac disease. (PubMed)

Ultrastructural binding sites of endomysium antibodies from sera of patients with dermatitis herpetiformis and coeliac disease. The ultrastructural binding sites of endomysium antibodies, specific serological markers of gluten sensitive enteropathy, were investigated in the rabbit oesophagus using the immunogold technique. Endomysium antibodies from sera of patients with dermatitis herpetiformis and with coeliac disease bound in an identical manner in a non-fibrillar material closely associated (...) with fine collagenous-reticulin fibrils and also with similar fibrils connecting smooth muscle cells and elastic tissue in the endomysial connective tissue. These observations suggest that IgA antibodies in sera from patients with dermatitis herpetiformis and coeliac disease recognise a common antigen in an amorphous component associated with the reticular connective tissue of oesophageal lamina muscularis mucosae and thus confirm the probable identity of IgA class endomysium and jejunal antibodies.

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1992 Gut

548. HLA association with dermatitis herpetiformis is accounted for by a cis or transassociated DQ heterodimer. (PubMed)

HLA association with dermatitis herpetiformis is accounted for by a cis or transassociated DQ heterodimer. HLA-DR, DQ, and DP restriction fragment genotyping was undertaken in 23 dermatitis herpetiformis patients and 53 healthy control subjects. HLA-DQw2 was present in 100% of patients with dermatitis herpetiformis (23 of 23) versus 40% of control subjects (21 of 53). Significant secondary associations occurred with HLA-DR3 (91% of patients versus 28% of control subjects) and DPw1 (39 (...) % of patients versus 11% of control subjects). Dermatitis herpetiformis and coeliac disease thus share an identical HLA class II association. It is likely that HLA class II genes directly influence the immune responses leading to mucosal damage in both diseases. The strongest candidate for disease susceptibility to dermatitis herpetiformis is DQw2. The HLA molecule most likely to be involved in coeliac disease is a specific DQ alpha/DQ beta heterodimer, encoded in cis arrangement in DR3 haplotypes

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1991 Gut

549. Helicobacter pylori serology in patients with coeliac disease and dermatitis herpetiformis. (PubMed)

Helicobacter pylori serology in patients with coeliac disease and dermatitis herpetiformis. To investigate whether Helicobacter pylori infection or autoimmune gastritis is responsible for the reported increase in gastric pathology and abnormalities of gastric function in patients with coeliac disease and dermatitis herpetiformis (DH).Serum H pylori IgG antibodies were assayed by enzyme linked immunosorbent assay and intrinsic factor antibodies by radioimmunoassay in 99 patients with coeliac (...) disease and 58 patients with dermatitis herpetiformis from two geographic areas.H pylori positivity in patients with coeliac disease and dermatitis herpetiformis increased with age, reaching 50% and 70%, respectively, in patients over 50 years. The percentage H pylori seropositivity in coeliac disease did not differ from the percentage positivity observed in 250 similarly aged blood donors from the same geographic area (Leeds). Seropositivity in patients with dermatitis herpetiformis

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1992 Journal of Clinical Pathology

550. Celiac Disease and Dermatitis Herpetiformis: National survey indicates delays in diagnosis (PubMed)

Celiac Disease and Dermatitis Herpetiformis: National survey indicates delays in diagnosis A national survey of 1937 members of the Canadian Celiac Association was conducted by mail questionnaire in June 1989 to study problems in diagnosing and managing celiac disease (CD) and dermatitis herpetiformis. Although 82% of the 1294 respondents were biopsied, 14% were diagnosed by their dramatic response to the gluten-free diet. Fifteen percent of biopsy-proven respondents saw five or more doctors

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1992 Canadian Family Physician

551. Monozygous twins concordant for duodenojejunal villous atrophy and dermatitis herpetiformis. (PubMed)

Monozygous twins concordant for duodenojejunal villous atrophy and dermatitis herpetiformis. A pair of monozygous twins concordant for both duodenojejunal villous atrophy and dermatitis herpetiformis are described, the first example to be reported in the literature. The observation provides strong support for the concept of an underlying genetic component being involved in the pathogenesis of these two conditions.

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1986 Gut

552. Gluten challenge in children with dermatitis herpetiformis: a clinical, morphological and immunohistological study. (PubMed)

Gluten challenge in children with dermatitis herpetiformis: a clinical, morphological and immunohistological study. Twenty one children with dermatitis herpetiformis were studied in an attempt to evaluate the response in the skin, in jejunal morphology, and in jejunal immunoglobulin containing cell counts to gluten elimination and subsequent gluten challenge. In all of the 15 patients whose jejunal biopsy was studied after the eventual gluten challenge the jejunal lesion had returned in 2.4

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1986 Gut

553. Different HLA associated gene combinations contribute to susceptibility for coeliac disease and dermatitis herpetiformis. (PubMed)

Different HLA associated gene combinations contribute to susceptibility for coeliac disease and dermatitis herpetiformis. Forty two white patients of British or Irish descent with coeliac disease and 28 with dermatitis herpetiformis were typed for class I HLA-A, B, and C, and class II DR and DQ antigens. In coeliac disease there was a significant increase in the frequencies of A1, B8, DR3, DR7, and DQw2 compared with controls but no increase of DR2. In dermatitis herpetiformis there were

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1986 Gut

554. HLA class II alpha chain gene polymorphisms in patients with insulin-dependent diabetes mellitus, dermatitis herpetiformis, and celiac disease. (PubMed)

HLA class II alpha chain gene polymorphisms in patients with insulin-dependent diabetes mellitus, dermatitis herpetiformis, and celiac disease. We have investigated DNA polymorphism of the class II alpha chain genes in HLA typed patients with insulin dependent diabetes mellitus (IDDM; n = 79), celiac disease (CD; n = 46), dermatitis herpetiformis (DH; n = 53), and controls (n = 86). Preferential allelic associations of HLA genes and gene products have thus been constructed for susceptibility

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1987 Journal of Clinical Investigation

555. Local immunoglobulin production is different in gastritis associated with dermatitis herpetiformis and simple gastritis. (PubMed)

Local immunoglobulin production is different in gastritis associated with dermatitis herpetiformis and simple gastritis. The degree of inflammation and atrophy in gastric body mucosal specimens (n = 38) from 28 patients with dermatitis herpetiformis (DH) was graded histologically. Immunoglobulin (Ig) producing cells were enumerated by paired immunofluorescence staining in a 500 microns wide section area from the muscularis mucosae to the lumen (mucosal 'tissue unit'). The number of immunocytes

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1987 Gut

556. Cellobiose/mannitol sugar absorption test in patients with dermatitis herpetiformis: a preliminary report (PubMed)

Cellobiose/mannitol sugar absorption test in patients with dermatitis herpetiformis: a preliminary report An abnormal cellobiose/mannitol ratio is present in new patients presenting with dermatitis herpetiformis and in patients who have never adhered to a strict gluten-free diet. In patients with dermatitis herpetiformis in remission on a strict gluten-free diet, small bowel absorption as measured by the cellobiose/mannitol ratio is normal. It is suggested that cellobiose/mannitol ratio (...) absorption is a useful screening test for the intestinal abnormality associated with dermatitis herpetiformis and failure of the test to return to normal could suggest poor dietary compliance.

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1988 The Ulster medical journal

557. Manic depression induced by dapsone in patient with dermatitis herpetiformis. (PubMed)

Manic depression induced by dapsone in patient with dermatitis herpetiformis. 2510873 1989 12 26 2018 11 13 0959-8138 299 6703 1989 Sep 30 BMJ (Clinical research ed.) BMJ Manic depression induced by dapsone in patient with dermatitis herpetiformis. 860 Gawkrodger D D Royal Hallamshire Hospital, Sheffield. eng Case Reports Journal Article England BMJ 8900488 0959-8138 8W5C518302 Dapsone AIM IM Adult Bipolar Disorder chemically induced Dapsone adverse effects therapeutic use Dermatitis (...) Herpetiformis drug therapy Humans Male 1989 9 30 1989 9 30 0 1 1989 9 30 0 0 ppublish 2510873 PMC1837664 J Am Acad Dermatol. 1983 Aug;9(2):274-5 6886118 BMJ. 1989 Jul 29;299(6694):324 2504425 BMJ. 1989 Jun 3;298(6686):1524 2503107

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1989 BMJ : British Medical Journal

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