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Dermatitis Herpetiformis

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2. Gluten challenge induces skin and small bowel relapse in long-term gluten-free diet-treated dermatitis herpetiformis. (PubMed)

Gluten challenge induces skin and small bowel relapse in long-term gluten-free diet-treated dermatitis herpetiformis. Dermatitis herpetiformis (DH) is an extra-intestinal manifestation of coeliac disease causing an itchy, blistering rash. Skin IgA deposits are pathognomonic for DH, and the treatment of choice is a life-long gluten-free diet (GFD). Preliminary evidence suggests that there are DH patients who redevelop gluten tolerance after adherence to a GFD treatment. To examine this, we

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2019 Journal of Investigative Dermatology

3. Dermatitis herpetiformis and celiac disease increase the risk of bullous pemphigoid. (PubMed)

Dermatitis herpetiformis and celiac disease increase the risk of bullous pemphigoid. Bullous pemphigoid (BP) and dermatitis herpetiformis (DH) are autoimmune bullous skin diseases. DH has been described to evolve into BP and the two diseases can have overlapping clinical appearances and diagnostic findings, but the association between DH and BP has not previously been studied in a large population. To evaluate DH and celiac disease as risk factors for BP, we conducted a retrospective case

2018 Journal of Investigative Dermatology

4. Granular IgA Deposits in the Skin of Patients with Coeliac Disease: Is it Always Dermatitis Herpetiformis? (PubMed)

Granular IgA Deposits in the Skin of Patients with Coeliac Disease: Is it Always Dermatitis Herpetiformis? Coeliac disease is an immune-mediated enteropathy driven by gluten, which can be associated with dermatitis herpetiformis. The presence of granular IgA deposits, detected by direct immunofluorescence, is the hallmark of dermatitis herpetiformis; nevertheless, IgA deposits have also been demonstrated in healthy skin of patients with coeliac disease. The main objective of this study (...) was to investigate whether IgA deposits could be found in the skin of patients with coeliac disease who have non-dermatitis herpetiformis inflammatory skin diseases. Direct immunofluorescence was performed on perilesional skin biopsies of 6 patients with coeliac disease with non-dermatitis herpetiformis inflammatory skin diseases and, as control, on 12 non-coeliac patients with inflammatory skin diseases. IgA deposits were found in all of the patients with coeliac disease, but were absent in the control group

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2018 Acta Dermato-Venereologica

5. Why now? Delayed drug-induced pancreatitis due to dapsone for dermatitis herpetiformis (PubMed)

Why now? Delayed drug-induced pancreatitis due to dapsone for dermatitis herpetiformis Drug-induced pancreatitis can be caused by a wide array of medications. In fact, the diagnosis is likely commonly missed due to the difficulty in diagnosing one agent as the sole cause. We present a case of dapsone-induced pancreatitis in a 75-year-old male with history of celiac disease. He presented with abdominal pain and was found to have acute pancreatitis. Interestingly, he had been on dapsone for 5

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2018 Journal of community hospital internal medicine perspectives

6. Dermatitis herpetiformis in an African woman (PubMed)

Dermatitis herpetiformis in an African woman Dermatitis herpetiformis (DH) is an autoimmune blistering disease of the skin. It is a result of hypersensitivity to dietary gluten. Diagnosis of DH can be challenging in a low prevalence, resource-limited population. We present the case of an African woman who presented with clinical features of DH.

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2018 The Pan African medical journal

7. Dermatitis Herpetiformis: A Common Extraintestinal Manifestation of Coeliac Disease (PubMed)

Dermatitis Herpetiformis: A Common Extraintestinal Manifestation of Coeliac Disease Dermatitis herpetiformis (DH) is a common extraintestinal manifestation of coeliac disease presenting with itchy papules and vesicles on the elbows, knees, and buttocks. Overt gastrointestinal symptoms are rare. Diagnosis of DH is easily confirmed by immunofluorescence biopsy showing pathognomonic granular immunoglobulin A (IgA) deposits in the papillary dermis. A valid hypothesis for the immunopathogenesis

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2018 Nutrients

8. Prognosis of Dermatitis Herpetiformis Patients with and without Villous Atrophy at Diagnosis (PubMed)

Prognosis of Dermatitis Herpetiformis Patients with and without Villous Atrophy at Diagnosis Dermatitis herpetiformis (DH) is a cutaneous manifestation of coeliac disease. At diagnosis, the majority of patients have villous atrophy in the small bowel mucosa. The objective of this study was to investigate whether the presence or absence of villous atrophy at diagnosis affects the long-term prognosis of DH. Data were gathered from the patient records of 352 DH and 248 coeliac disease patients

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2018 Nutrients

9. Self-Reported Fractures in Dermatitis Herpetiformis Compared to Coeliac Disease (PubMed)

Self-Reported Fractures in Dermatitis Herpetiformis Compared to Coeliac Disease Dermatitis herpetiformis (DH) is a cutaneous manifestation of coeliac disease. Increased bone fracture risk is known to associate with coeliac disease, but this has been only scantly studied in DH. In this study, self-reported fractures and fracture-associated factors in DH were investigated and compared to coeliac disease. Altogether, 222 DH patients and 129 coeliac disease-suffering controls were enrolled

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2018 Nutrients

10. A retrospective study of dermatitis herpetiformis from an immunobullous disease clinic in north India. (PubMed)

A retrospective study of dermatitis herpetiformis from an immunobullous disease clinic in north India. Indian data on dermatitis herpetiformis (DH) is not available. The aim of this study was to investigate the demographic and clinicopathological characteristics of patients with DH and to study its association with other autoimmune diseases.All data were collected from case record forms of patients registered in immunobullous disease clinic of our institute. The diagnosis was based (...) autoimmune comorbidities such as hypothyroidism. Forty percent of patients on strict gluten-free diet achieved remission in 2 years, while 35.4% had frequent relapses as they continued gluten intake.Dermatitis herpetiformis is not a rare disease in northern India as previously believed. The clinical, histological, and immunopathological characteristics of Indian DH patients are similar to those reported in Caucasian populations. The limitations of our study include an absence of genetic testing for HLA

2018 International Journal of Dermatology

11. Diagnostic Delay in Dermatitis Herpetiformis in a High-prevalence Area. (PubMed)

Diagnostic Delay in Dermatitis Herpetiformis in a High-prevalence Area. Dermatitis herpetiformis (DH) is an extra-intestinal manifestation of coeliac disease. The highest currently reported prevalence of DH is in Finland, but knowledge of diagnostic delay is limited. This study investigated the duration of rash prior to diagnosis in 446 patients with DH, analysing the results in 3 periods of 15 years. The diagnosis was considered delayed when the duration of rash before diagnosis was 2 years

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2017 Acta Dermato-Venereologica

12. Disappearance of epidermal transglutaminase and IgA deposits from the papillary dermis of dermatitis herpetiformis patients after a long-term gluten-free diet. (PubMed)

Disappearance of epidermal transglutaminase and IgA deposits from the papillary dermis of dermatitis herpetiformis patients after a long-term gluten-free diet. 28906552 2019 02 19 1365-2133 178 3 2018 03 The British journal of dermatology Br. J. Dermatol. Disappearance of epidermal transglutaminase and IgA deposits from the papillary dermis of patients with dermatitis herpetiformis after a long-term gluten-free diet. e198-e201 10.1111/bjd.15995 Hietikko M M Coeliac Disease Research Center

2017 British Journal of Dermatology

13. Ex vivo Culture of Duodenal Biopsies from Patients with Dermatitis Herpetiformis Indicates that Transglutaminase 3 Antibody Production Occurs in the Gut. (PubMed)

Ex vivo Culture of Duodenal Biopsies from Patients with Dermatitis Herpetiformis Indicates that Transglutaminase 3 Antibody Production Occurs in the Gut. Coeliac disease and dermatitis herpetiformis (DH) are characterized by autoantibodies targeting transglutaminase (TG)2 and TG3, respectively. Previous studies show that TG2 antibodies are produced in the gut and can be assessed in organ culture of small-intestinal biopsies from patients with coeliac disease. Thus far, no studies have

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2017 Acta Dermato-Venereologica

14. Dermatitis Herpetiformis as the Initial Presentation of Primary Biliary Cholangitis in a Male with Gluten Sensitivity (PubMed)

Dermatitis Herpetiformis as the Initial Presentation of Primary Biliary Cholangitis in a Male with Gluten Sensitivity Celiac disease is commonly associated with elevated liver enzymes that normalize on a gluten-free diet. Celiac disease is rarely described in patients with primary biliary cholangitis. Dermatitis herpetiformis is the skin manifestation of the celiac disease that is very rarely associated with primary biliary cirrhosis. We present the case of a 62-year-old man who presented (...) with severe chronic pruritus, in whom a diagnosis of primary biliary cholangitis was made initially. However, in the presence of atypical skin lesions, not confirming to chronic cholestasis, an in-depth evaluation including histopathological examination led to the diagnosis of dermatitis herpetiformis associated with gluten sensitivity. Gluten-free diet and medical treatment with dapsone led to beneficial clinical outcomes.

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2017 Cureus

15. The Role of Intereukin-31 in Pathogenesis of Itch and Its Intensity in a Course of Bullous Pemphigoid and Dermatitis Herpetiformis (PubMed)

The Role of Intereukin-31 in Pathogenesis of Itch and Its Intensity in a Course of Bullous Pemphigoid and Dermatitis Herpetiformis Itch which is one of the major, subjective symptoms in a course of bullous pemphigoid and dermatitis herpetiformis makes those two diseases totally different than other autoimmune blistering diseases. Its pathogenesis is still not fully known. The aim of this research was to assess the role of IL-31 in development of itch as well as to measure its intensity

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2017 BioMed research international

16. Dermatitis herpetiformis presenting as pseudovasculitis (PubMed)

Dermatitis herpetiformis presenting as pseudovasculitis 28971130 2019 02 26 2352-5126 3 5 2017 Sep JAAD case reports JAAD Case Rep Dermatitis herpetiformis presenting as pseudovasculitis. 444-447 10.1016/j.jdcr.2017.06.033 Kern Malan M College of Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas. Kim Kevin H KH Department of Dermatology, University of Arkansas for Medical Sciences, Little Rock, Arkansas. Johnson Gina G Department of Pathology, University of Arkansas (...) for Medical Sciences, Little Rock, Arkansas. Hawkins Spencer D SD School of Medicine, Wake Forest University, Winston-Salem, North Carolina. Wong Henry K HK Department of Dermatology, University of Arkansas for Medical Sciences, Little Rock, Arkansas. eng Case Reports 2017 09 08 United States JAAD Case Rep 101665210 2352-5126 DH, dermatitis herpetiformis DIF, direct immunofluorescence EED, erythema elevatum diutinum acral petechiae dermatitis herpetiformis leukocytoclastic vasculitis pseudovasculitis 2017

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2017 JAAD Case Reports

17. Expression of the JAK/STAT Signaling Pathway in Bullous Pemphigoid and Dermatitis Herpetiformis (PubMed)

Expression of the JAK/STAT Signaling Pathway in Bullous Pemphigoid and Dermatitis Herpetiformis A family of eleven proteins comprises the Janus kinases (JAK) and signal transducers and activators of transcription (STAT) signaling pathway, which enables transduction of signal from cytokine receptor to the nucleus and activation of transcription of target genes. Irregular functioning of the cascade may contribute to pathogenesis of autoimmune diseases; however, there are no reports concerning (...) autoimmune bullous diseases yet to be published. The aim of this study was to evaluate the expression of proteins constituting the JAK/STAT signaling pathway in skin lesions and perilesional area in dermatitis herpetiformis (DH) and bullous pemphigoid (BP), as well as in the control group. Skin biopsies were collected from 21 DH patients, from 20 BP patients, and from 10 healthy volunteers. The localization and expression of selected STAT and JAK proteins were examined by immunohistochemistry

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2017 Mediators of inflammation

18. Dermatitis herpetiformis with fibrillar IgA deposition and unusual histologic findings (PubMed)

Dermatitis herpetiformis with fibrillar IgA deposition and unusual histologic findings 28761917 2019 02 26 2352-5126 3 4 2017 Jul JAAD case reports JAAD Case Rep Dermatitis herpetiformis with fibrillar IgA deposition and unusual histologic findings. 344-347 10.1016/j.jdcr.2017.04.003 Miraflor Allen P AP Department of Pathology, Dartmouth-Hitchcock Medical Center, Lebanon, New Hampshire. Paul Joan J Section of Dermatology, Department of Surgery, Dartmouth-Hitchcock Medical Center, Lebanon, New (...) Hampshire. Yan Shaofeng S Department of Pathology, Dartmouth-Hitchcock Medical Center, Lebanon, New Hampshire. LeBlanc Robert E RE Department of Pathology, Dartmouth-Hitchcock Medical Center, Lebanon, New Hampshire. eng Case Reports 2017 07 19 United States JAAD Case Rep 101665210 2352-5126 DH, dermatitis herpetiformis DIF, direct immunofluorescence blistering disease clinical pathologic correlation dermatitis herpetiformis direct immunofluorescence immunobullous disease 2017 8 2 6 0 2017 8 2 6 0 2017 8

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2017 JAAD Case Reports

19. Increased Serum Levels of Th2-type Cytokines and Eotaxin in Fibrillar-type Dermatitis Herpetiformis. (PubMed)

Increased Serum Levels of Th2-type Cytokines and Eotaxin in Fibrillar-type Dermatitis Herpetiformis. 27983746 2018 04 17 2018 04 17 1651-2057 97 5 2017 05 08 Acta dermato-venereologica Acta Derm. Venereol. Increased Serum Levels of Th2-type Cytokines and Eotaxin in Fibrillar-type Dermatitis Herpetiformis. 642-643 10.2340/00015555-2604 Makino Teruhiko T Department of Dermatology, Graduate School of Medicine and Pharmaceutical Sciences, University of Toyama, 2630 Sugitani, Toyama-shi, Toyama (...) IM Adult Aged Aged, 80 and over Chemokines blood Cytokines blood Dermatitis Herpetiformis blood GTP-Binding Proteins immunology Humans Immunoglobulin A blood Interferon-gamma blood Interleukin-12 blood Interleukin-13 blood Interleukin-4 blood Interleukin-5 blood Interleukin-8 blood Male Transglutaminases immunology 2016 12 17 6 0 2018 4 18 6 0 2016 12 17 6 0 ppublish 27983746 10.2340/00015555-2604

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2016 Acta Dermato-Venereologica

20. Rituximab Treatment for Recalcitrant Dermatitis Herpetiformis. (PubMed)

Rituximab Treatment for Recalcitrant Dermatitis Herpetiformis. Dermatitis herpetiformis (DH) is an autoimmune blistering condition seen in the context of celiac disease. While typically managed by gluten-free diet and dapsone, treatment of DH refractory to standard treatments is not well defined.A man in his 80s with DH not controlled by gluten-free diet (with poor adherence), dapsone, and conventional immune-suppressing agents responded to treatment with rituximab according to the lymphoma

2016 JAMA dermatology (Chicago, Ill.)

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