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Cushing Response

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101. Pseudo-Cushing Syndrome (Treatment)

. . Bernini GP, Argenio GF, Cerri F, Franchi F. Comparison between the suppressive effects of dexamethasone and loperamide on cortisol and ACTH secretion in some pathological conditions. J Endocrinol Invest . 1994 Nov. 17 (10):799-804. . Malerbi DA, Fragoso MC, Vieira Filho AH, Brenlha EM, Mendonça BB. Cortisol and adrenocorticotropin response to desmopressin in women with Cushing's disease compared with depressive illness. J Clin Endocrinol Metab . 1996 Jun. 81 (6):2233-7. . Tirabassi G, Papa R, Faloia E (...) Pseudo-Cushing Syndrome (Treatment) Pseudo-Cushing Syndrome: Overview, Causes and Complications, History and Physical Examination Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTI0NzE4LW92ZXJ2aWV3 processing

2014 eMedicine.com

102. Pseudo-Cushing Syndrome (Overview)

. . Bernini GP, Argenio GF, Cerri F, Franchi F. Comparison between the suppressive effects of dexamethasone and loperamide on cortisol and ACTH secretion in some pathological conditions. J Endocrinol Invest . 1994 Nov. 17 (10):799-804. . Malerbi DA, Fragoso MC, Vieira Filho AH, Brenlha EM, Mendonça BB. Cortisol and adrenocorticotropin response to desmopressin in women with Cushing's disease compared with depressive illness. J Clin Endocrinol Metab . 1996 Jun. 81 (6):2233-7. . Tirabassi G, Papa R, Faloia E (...) Pseudo-Cushing Syndrome (Overview) Pseudo-Cushing Syndrome: Overview, Causes and Complications, History and Physical Examination Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTI0NzE4LW92ZXJ2aWV3 processing

2014 eMedicine.com

103. Cushing Syndrome (Overview)

activation, [ ] changes in the enzyme adenosine monophosphate-activated protein kinase activity, [ ] and modulation of activator protein 1 (Fos/Jun) [ ] are some of the important pathways that have been described. More research still needs to be conducted to fully understand the underlying signaling pathways and glucocorticoid tissue-specific responses. A study by Serfling et al suggested that weight gain in iatrogenic Cushing syndrome may be related to a glucocorticoid-stimulated rise in the amygdala (...) Cushing Syndrome (Overview) Iatrogenic Cushing Syndrome: Background, Frequency, Mortality/Morbidity Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTE3MzY1LW92ZXJ2aWV3 processing > Iatrogenic Cushing Syndrome

2014 eMedicine.com

104. Pseudo-Cushing Syndrome (Follow-up)

. . Bernini GP, Argenio GF, Cerri F, Franchi F. Comparison between the suppressive effects of dexamethasone and loperamide on cortisol and ACTH secretion in some pathological conditions. J Endocrinol Invest . 1994 Nov. 17 (10):799-804. . Malerbi DA, Fragoso MC, Vieira Filho AH, Brenlha EM, Mendonça BB. Cortisol and adrenocorticotropin response to desmopressin in women with Cushing's disease compared with depressive illness. J Clin Endocrinol Metab . 1996 Jun. 81 (6):2233-7. . Tirabassi G, Papa R, Faloia E (...) Pseudo-Cushing Syndrome (Follow-up) Pseudo-Cushing Syndrome: Overview, Causes and Complications, History and Physical Examination Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTI0NzE4LW92ZXJ2aWV3 processing

2014 eMedicine.com

105. Glucocorticoid Therapy and Cushing Syndrome (Treatment)

Glucocorticoid Therapy and Cushing Syndrome (Treatment) Glucocorticoid Therapy and Cushing Syndrome Treatment & Management: Medical Care, Surgical Care, Consultations Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache (...) =aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvOTIxMDg2LXRyZWF0bWVudA== processing > Glucocorticoid Therapy and Cushing Syndrome Treatment & Management Updated: Dec 11, 2015 Author: George P Chrousos, MD, FAAP, MACP, MACE, FRCP(London); Chief Editor: Sasigarn A Bowden, MD Share Email Print Feedback Close Sections Sections Glucocorticoid Therapy and Cushing Syndrome Treatment Medical Care Treatment of Cushing syndrome involves identifying the underlying cause, whereas management of exogenous hypercortisolism involves optimization of glucocorticoid dose and route

2014 eMedicine Pediatrics

106. Glucocorticoid Therapy and Cushing Syndrome (Overview)

Glucocorticoid Therapy and Cushing Syndrome (Overview) Glucocorticoid Therapy and Cushing Syndrome: Background, Pathophysiology, Endogenous Cushing syndrome Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache (...) =aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvOTIxMDg2LW92ZXJ2aWV3 processing > Glucocorticoid Therapy and Cushing Syndrome Updated: Dec 11, 2015 Author: George P Chrousos, MD, FAAP, MACP, MACE, FRCP(London); Chief Editor: Sasigarn A Bowden, MD Share Email Print Feedback Close Sections Sections Glucocorticoid Therapy and Cushing Syndrome Overview Background Cushing syndrome (CS) takes its name from Harvey Cushing, who, in 1912, was one of the first physicians to report a patient affected with excessive glucocorticoid. [ ] More than 99% of cases of Cushing

2014 eMedicine Pediatrics

107. Pseudo-Cushing Syndrome (Diagnosis)

. . Bernini GP, Argenio GF, Cerri F, Franchi F. Comparison between the suppressive effects of dexamethasone and loperamide on cortisol and ACTH secretion in some pathological conditions. J Endocrinol Invest . 1994 Nov. 17 (10):799-804. . Malerbi DA, Fragoso MC, Vieira Filho AH, Brenlha EM, Mendonça BB. Cortisol and adrenocorticotropin response to desmopressin in women with Cushing's disease compared with depressive illness. J Clin Endocrinol Metab . 1996 Jun. 81 (6):2233-7. . Tirabassi G, Papa R, Faloia E (...) Pseudo-Cushing Syndrome (Diagnosis) Pseudo-Cushing Syndrome: Overview, Causes and Complications, History and Physical Examination Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTI0NzE4LW92ZXJ2aWV3 processing

2014 eMedicine.com

108. Glucocorticoid Therapy and Cushing Syndrome (Follow-up)

Glucocorticoid Therapy and Cushing Syndrome (Follow-up) Glucocorticoid Therapy and Cushing Syndrome Follow-up: Further Outpatient Care, Inpatient & Outpatient Medications, Transfer Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache (...) =aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvOTIxMDg2LWZvbGxvd3Vw processing > Glucocorticoid Therapy and Cushing Syndrome Follow-up Updated: Dec 11, 2015 Author: George P Chrousos, MD, FAAP, MACP, MACE, FRCP(London); Chief Editor: Sasigarn A Bowden, MD Share Email Print Feedback Close Sections Sections Glucocorticoid Therapy and Cushing Syndrome Follow-up Further Outpatient Care Regular follow-up care is required for patients with Cushing syndrome (CS) who are receiving adrenal steroid replacement. Obtain a history of the number of illnesses, frequency

2014 eMedicine Pediatrics

109. Variation in response to dexamethasone of a patient with Cushing's syndrome. (PubMed)

Variation in response to dexamethasone of a patient with Cushing's syndrome. A patient with Cushing's syndrome due to a nonresectable chromophobe adenoma underwent external irradiation of the hypothalamic-pituitary area. The signs of Cushing's syndrome ameliorated subsequently but recurred eventually, necessitating bilateral adrenalectomy 30 months after initial presentation. Response to dexamethasone suppression testing on four occasions varied greatly. These results may reflect alterations

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1976 Canadian Medical Association Journal

110. CORRELATION OF PLASMA ACTH CONCENTRATION WITH ADRENOCORTICAL RESPONSE IN NORMAL HUMAN SUBJECTS, SURGICAL PATIENTS, AND PATIENTS WITH CUSHING'S DISEASE (PubMed)

CORRELATION OF PLASMA ACTH CONCENTRATION WITH ADRENOCORTICAL RESPONSE IN NORMAL HUMAN SUBJECTS, SURGICAL PATIENTS, AND PATIENTS WITH CUSHING'S DISEASE 14083159 1996 12 01 2018 12 01 0021-9738 42 1963 Nov The Journal of clinical investigation J. Clin. Invest. CORRELATION OF PLASMA ACTH CONCENTRATION WITH ADRENOCORTICAL RESPONSE IN NORMAL HUMAN SUBJECTS, SURGICAL PATIENTS, AND PATIENTS WITH CUSHING'S DISEASE. 1669-77 NEY R L RL SHIMIZU N N NICHOLSON W E WE ISLAND D P DP LIDDLE G W GW eng Journal (...) Article United States J Clin Invest 7802877 0021-9738 9002-60-2 Adrenocorticotropic Hormone OM Adrenocorticotropic Hormone Blood Chemical Analysis Cushing Syndrome Endocrinology Humans Pituitary ACTH Hypersecretion Pituitary-Adrenal Function Tests Surgical Procedures, Operative BLOOD CHEMICAL ANALYSIS CORTICOTROPIN CUSHING'S SYNDROME ENDOCRINOLOGY PITUITARY-ADRENAL FUNCTION TESTS SURGERY, OPERATIVE 1963 11 1 1963 11 1 0 1 1963 11 1 0 0 ppublish 14083159 10.1172/JCI104853 PMC289451 J Clin Invest. 1962

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1963 Journal of Clinical Investigation

111. Anomalous responses to stimulation and suppression tests in Cushing's syndrome due to a calcified adrenal adenoma (PubMed)

Anomalous responses to stimulation and suppression tests in Cushing's syndrome due to a calcified adrenal adenoma 4377169 1975 11 08 2018 11 13 0032-5473 49 578 1973 Dec Postgraduate medical journal Postgrad Med J Anomalous responses to stimulation and suppression tests in Cushing's syndrome due to a calcified adrenal adenoma. 923-6 Epstein S S Goldin A R AR McLaren E H EH eng Journal Article England Postgrad Med J 0234135 0032-5473 7S5I7G3JQL Dexamethasone 9002-60-2 Adrenocorticotropic Hormone (...) IM Adenoma complications Adrenal Gland Neoplasms complications Adrenal Glands drug effects Adrenocorticotropic Hormone pharmacology Adult Calcinosis complications Cushing Syndrome etiology Dexamethasone pharmacology Female Humans 1973 12 1 1973 12 1 0 1 1973 12 1 0 0 ppublish 4377169 PMC2495454 Am J Med. 1969 Oct;47(4):619-24 4309844 J Endocrinol. 1965 Nov;33(3):515-24 5845673 Br J Radiol. 1972 Aug;45(536):621-3 5045980 Metabolism. 1958 Sep;7(5):608-23 13577415 Acta Endocrinol (Copenh). 1964 Jun

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1973 Postgraduate medical journal

112. Cardiovascular outcomes in subclinical Cushing's syndrome versus non-functioning adrenal adenoma in patients with adrenal adenomas: a systematic review

Cardiovascular outcomes in subclinical Cushing's syndrome versus non-functioning adrenal adenoma in patients with adrenal adenomas: a systematic review Print | PDF PROSPERO This information has been provided by the named contact for this review. CRD has accepted this information in good faith and registered the review in PROSPERO. CRD bears no responsibility or liability for the content of this registration record, any associated files or external websites. Email salutation (e.g. "Dr Smith

2017 PROSPERO

113. Accuracy of diagnostic tests for Cushing's syndrome: a systematic review and metaanalyses

and neither language nor publication restrictions were applied. Study selection Diagnostic cohort studies were eligible for inclusion if they evaluated the accuracy of urinary free cortisol, serum and salivary midnight or bedtime cortisol, 1mg overnight dexamethasone suppression test (DST), or the two-day 2mg DST, compared with a reference standard for Cushing's syndrome, in individuals in whom there was true diagnostic uncertainty. The eligible reference standards were pathological diagnosis, response (...) Accuracy of diagnostic tests for Cushing's syndrome: a systematic review and metaanalyses Accuracy of diagnostic tests for Cushing's syndrome: a systematic review and metaanalyses Accuracy of diagnostic tests for Cushing's syndrome: a systematic review and metaanalyses Elamin MB, Murad MH, Mullan R, Erickson D, Harris K, Nadeem S, Ennis R, Erwin PJ, Montori VM CRD summary This well-conducted review concluded that commonly used tests to diagnose Cushing's syndrome appeared to be highly accurate

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2008 DARE.

114. Maintenance of Response After rTMS for Depression Using tDCS

Maintenance of Response After rTMS for Depression Using tDCS Maintenance of Response After rTMS for Depression Using tDCS - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Maintenance of Response After rTMS (...) for Depression Using tDCS (START) The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. of clinical studies and talk to your health care provider before participating. Read our for details. ClinicalTrials.gov Identifier: NCT03708159 Recruitment Status : Recruiting First Posted : October 17, 2018 Last Update Posted : October 17, 2018 See Sponsor: Centre for Addiction

2018 Clinical Trials

115. Androgen Responses to hCG and Ovarian Morphology in PCOS

Androgen Responses to hCG and Ovarian Morphology in PCOS Androgen Responses to hCG and Ovarian Morphology in PCOS - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Androgen Responses to hCG and Ovarian (...) Morphology in PCOS The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. of clinical studies and talk to your health care provider before participating. Read our for details. ClinicalTrials.gov Identifier: NCT03489668 Recruitment Status : Recruiting First Posted : April 5, 2018 Last Update Posted : August 9, 2018 See Sponsor: Poznan University of Medical Sciences

2018 Clinical Trials

116. Efficacy and Neural Mediators of Response to Trauma Management Therapy for PTSD

Efficacy and Neural Mediators of Response to Trauma Management Therapy for PTSD Efficacy and Neural Mediators of Response to Trauma Management Therapy for PTSD - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more (...) . Efficacy and Neural Mediators of Response to Trauma Management Therapy for PTSD The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier: NCT03449576 Recruitment Status : Enrolling by invitation First Posted : February 28, 2018 Last Update Posted : January 16, 2019 Sponsor: VA Office of Research and Development

2018 Clinical Trials

117. Diagnostic accuracy and comparison of BIPSS in response to lysine vasopressin and hCRH (PubMed)

Diagnostic accuracy and comparison of BIPSS in response to lysine vasopressin and hCRH Bilateral inferior petrosal sinus sampling (BIPSS) using hCRH is currently considered the 'gold standard' test for the differential diagnosis of ACTH-dependent Cushing's syndrome (CS). Vasopressin is more potent than CRH to stimulate ACTH secretion as shown in animal studies; however, no comparative data of its use are available during BIPSS.To study the diagnostic accuracy and comparison of hCRH and lysine (...) vasopressin (LVP) stimulation during BIPSS.29 patients (27-Cushing's disease, 2-ectopic CS; confirmed on histopathology) underwent BIPSS and were included for the study. Patients were randomized to receive hCRH, 5 U LVP or 10 U LVP during BIPSS for ACTH stimulation. BIPSS and contrast-enhanced magnetic resonance imaging (CEMRI) were compared with intra-operative findings of trans-sphenoidal surgery (TSS) for localization and lateralization of the ACTH source.BIPSS correctly localized the source of ACTH

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2018 Endocrine connections

118. Differences in adiposity in Cushing syndrome caused by PRKAR1A mutations: clues for the role of cyclic AMP signaling in obesity and diagnostic implications. (PubMed)

Differences in adiposity in Cushing syndrome caused by PRKAR1A mutations: clues for the role of cyclic AMP signaling in obesity and diagnostic implications. The cAMP signaling pathway is implicated in bilateral adrenocortical hyperplasias. Bilateral adrenocortical hyperplasia is often associated with ACTH-independent Cushing syndrome (CS) and may be caused by mutations in genes such as PRKAR1A, which is responsible for primary pigmented nodular adrenocortical disease (PPNAD). PRKAR1A regulates (...) ) and without (n = 32) PRKAR1A mutations and a comparison group with aldosterone-producing adenomas (APAs) (n = 6). In addition, clinical data from a larger group of patients with Cushing disease (n = 89) and hyperaldosteronism (n = 26) were used for comparison.Body mass index (BMI), abdominal computed tomography scans, and cortisol data were collected preoperatively. PAT was assayed for PKA activity, cAMP levels, and PKA subunit expression.BMI was lower in adult patients with CS with PRKAR1A mutations

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2013 Journal of Clinical Endocrinology and Metabolism

119. Pasireotide monotherapy in Cushing's disease: a single-centre experience with 5-year extension of phase III Trial. (PubMed)

Pasireotide monotherapy in Cushing's disease: a single-centre experience with 5-year extension of phase III Trial. A recent phase III randomized controlled trial (NCT00434148) showed efficacy of pasireotide in the treatment of patients with Cushing's disease (CD). Patients were invited to participate in an extension phase of the protocol and a subgroup had a sustained response. We report the experience with 4 patients in our center of which 2 full responders have completed 5.5 and 4.25 years (...) limit of normal had a rapid sustained response to pasireotide and entered the extension phase after 12 months. They remain in clinical and biochemical disease remission and 1 patient now only requires 300 μg daily of pasireotide. All 4 patients developed glucose intolerance; however, the two patients in the extension phase were eventually able to discontinue all diabetes pharmacotherapy. Adverse events included second degree atrioventicular block type 1 without QT prolongation in a patient with pre

2013 Pituitary

120. A cellular and molecular basis for the selective desmopressin-induced ACTH release in Cushing's disease patients: key role of AVPR1b receptor and potential therapeutic implications. (PubMed)

A cellular and molecular basis for the selective desmopressin-induced ACTH release in Cushing's disease patients: key role of AVPR1b receptor and potential therapeutic implications. Desmopressin is a synthetic agonist of vasopressin receptors (AVPRs). The desmopressin stimulation test is used in the diagnosis and postsurgery prognosis of Cushing disease (CD). However, the cellular and molecular mechanisms underlying the desmopressin-induced ACTH increase in patients with CD are poorly (...) -PCR. Primary cultures derived from corticotropinomas, nonfunctioning pituitary adenomas, somatotropinomas, prolactinomas, and NPs were treated with desmopressin, and ACTH secretion/expression, [Ca(2+)]i kinetics, and AVPR expression and/or proliferative response were evaluated. The relationship between AVPR expression and plasma adrenocorticotropin/cortisol levels obtained from desmopressin tests was assessed.Desmopressin affects all functional parameters evaluated in corticotropinoma cells

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2013 Journal of Clinical Endocrinology and Metabolism

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