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Craniotabes

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1. Craniotabes

Craniotabes Craniotabes Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Craniotabes Craniotabes Aka: Craniotabes II. Definition (...) Thinning of skull outer table III. Signs Press firmly over occiput or s Gives-way as if pressing on ping-pong ball IV. Differential Diagnosis Osteogenesis imperfecta Normal variant May be present in newborns Resolves by 1 month of age May persist normally at lines Images: Related links to external sites (from Bing) These images are a random sampling from a Bing search on the term "Craniotabes." Click on the image (or right click) to open the source website in a new browser window. Related Studies (from

2018 FP Notebook

2. Relationship between newborn craniotabes and vitamin D status Full Text available with Trip Pro

Relationship between newborn craniotabes and vitamin D status In recent studies, vitamin D deficiency during pregnancy and early infancy has been reported to predispose children to many chronic diseases, except those of the skeletal system. The aim of this study was to investigate whether craniotabes in otherwise healthy newborns is physiological, its relationship to vitamin D deficiency and whether or not it requires treatment.A total of 150 healthy newborns with a weight of over 2000 g were (...) included. Newborns were divided into two groups during postnatal discharge (1-3.'s day): those with and without craniotabes. The 25-hydroxy (OH) vitamin D levels of the newborns' mothers were measured, and all infants were re-evaluated for craniotabes, as well as tested to determine levels of serum calcium (Ca), phosphorus (P), alkaline phosphatase (ALP), parathyroid hormone (PTH) and 25(OH) vitamin D, urine calcium and creatinine.Craniotabes was present in 45 (30%) of newborns enrolled in the study

2016 Northern clinics of Istanbul

3. Craniotabes

Craniotabes Craniotabes Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Craniotabes Craniotabes Aka: Craniotabes II. Definition (...) Thinning of skull outer table III. Signs Press firmly over occiput or s Gives-way as if pressing on ping-pong ball IV. Differential Diagnosis Osteogenesis imperfecta Normal variant May be present in newborns Resolves by 1 month of age May persist normally at lines Images: Related links to external sites (from Bing) These images are a random sampling from a Bing search on the term "Craniotabes." Click on the image (or right click) to open the source website in a new browser window. Related Studies (from

2015 FP Notebook

4. Craniotabes in Four-Months-Old Twins Full Text available with Trip Pro

Craniotabes in Four-Months-Old Twins 19989904 2010 06 24 2010 06 24 0035-9157 27 10 1934 Aug Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Craniotabes in Four-Months-Old Twins. 1324-5 Tallerman K H KH eng Journal Article England Proc R Soc Med 7505890 0035-9157 2009 12 9 6 0 1934 8 1 0 0 1934 8 1 0 1 ppublish 19989904 PMC2205456

1934 Proceedings of the Royal Society of Medicine

5. Vitamin D deficiency in children

(swelling of the costochondral junctions). Craniotabes (skull softening with frontal bossing and delayed fontanelle closure). Delayed tooth eruption and enamel hypoplasia. Other symptoms or conditions associated with vitamin D deficiency, such as: Long-standing (3 months or longer), unexplained bone pain. Muscular weakness (for example difficulty climbing stairs, waddling gait, difficulty rising from a chair, or delayed walking). Features of hypocalcaemia (tetany or seizures). Cardiomyopathy (in infants

2016 NICE Clinical Knowledge Summaries

6. Safety and Efficacy of Asfotase Alfa in Patients With Hypophosphatasia (HPP)

Ultrasonographic features of prenatal, characterized by: 1) severe short extremities (femur length <-4SD in second and third trimesters) 2) extending into the metaphysis (femur metaphysis length or femur length >0.33) 3) craniotabes 4) Hypoplastic thorax (Thoracic or abdominal circumference <0.6) (3) Computed tomographic findings of prenatal, characterized by: Generalized decreased ossification Extreme shortening of tubular bones Hypoplastic thorax (4) Radiographic evidence of HPP, characterized by: 1) Flared

2015 Clinical Trials

7. Congenital rickets due to vitamin D deficiency in the mothers. (Abstract)

of these cases. In 16 cases the diagnosis of the rickets led to the identification of symptomatic osteomalacia in the mothers. Of the 12 mothers who had assays for serum 25-hydroxyvitamin D (25OHD) 11 had values less than 10 ng/mL. Presentations in the infants included craniotabes, wide skull sutures, rachitic rosaries, enlargement of the wrists, tetany and convulsions. In two cases rickets had been suspected from antenatal X-rays. In five cases fractures were found at the time of initial presentation

2014 Clinical nutrition (Edinburgh, Scotland)

8. Disorders of Bone Mineralization (Overview)

features of and hyperphosphatemia include seizures, apnea, and tetany. In children, clinical features of rickets include the following (see the images below): Delayed motor milestones Hypotonia Enlargement of wrists Progressive bowing of long bones Rachitic rosary Harrison sulcus Violin case deformity of the chest Late closure of anterior fontanelle Parietal and frontal bossing Craniotabes Craniosynostosis Delay in teeth eruption Enamel hypoplasia Decreased bone mineral density Myopathy with normal

2014 eMedicine Pediatrics

9. Disorders of Bone Mineralization (Treatment)

features of and hyperphosphatemia include seizures, apnea, and tetany. In children, clinical features of rickets include the following (see the images below): Delayed motor milestones Hypotonia Enlargement of wrists Progressive bowing of long bones Rachitic rosary Harrison sulcus Violin case deformity of the chest Late closure of anterior fontanelle Parietal and frontal bossing Craniotabes Craniosynostosis Delay in teeth eruption Enamel hypoplasia Decreased bone mineral density Myopathy with normal

2014 eMedicine Pediatrics

10. Disorders of Bone Mineralization (Follow-up)

features of and hyperphosphatemia include seizures, apnea, and tetany. In children, clinical features of rickets include the following (see the images below): Delayed motor milestones Hypotonia Enlargement of wrists Progressive bowing of long bones Rachitic rosary Harrison sulcus Violin case deformity of the chest Late closure of anterior fontanelle Parietal and frontal bossing Craniotabes Craniosynostosis Delay in teeth eruption Enamel hypoplasia Decreased bone mineral density Myopathy with normal

2014 eMedicine Pediatrics

11. Disorders of Bone Mineralization (Diagnosis)

features of and hyperphosphatemia include seizures, apnea, and tetany. In children, clinical features of rickets include the following (see the images below): Delayed motor milestones Hypotonia Enlargement of wrists Progressive bowing of long bones Rachitic rosary Harrison sulcus Violin case deformity of the chest Late closure of anterior fontanelle Parietal and frontal bossing Craniotabes Craniosynostosis Delay in teeth eruption Enamel hypoplasia Decreased bone mineral density Myopathy with normal

2014 eMedicine Pediatrics

12. Osteomalacia and Renal Osteodystrophy

. The immature skeleton may reveal the following characteristic findings: In neonates, posterior flattening and squaring of the skull (eg, craniotabes) may be observed. In early childhood, bowing deformities of arms and legs are common (see the image below). Lateral radiograph of the leg in a child with chronic renal failure reveals anterior bowing of the distal tibia. In older children, scoliosis, vertebral endplate deformities, basilar invagination of the skull, triradiate deformity of the pelvis

2014 eMedicine Radiology

13. Rickets

-old girl with hypophosphatemia depict severe fraying of the metaphysis. Radiograph in a 4-year-old girl with rickets depicts bowing of the legs caused by loading. Craniotabes of long bones (see the images below) Radiograph in a 4-year-old girl with rickets, focused on the knees. Image depicts the development of knock-knees. Radiograph of the proximal humerus in a 2.5-year-old girl who had a peripheral neuroectodermal tumor of the right brachial plexus. After treatment with ifosfamide, the patient

2014 eMedicine Radiology

14. Increasing incidence of nutritional rickets: a population-based study in olmsted county, Minnesota. Full Text available with Trip Pro

, osteomalacia, genu varum, genu valgum, craniotabes, hypocalcemia, hypocalcemic seizure, and tetany. Record abstraction was performed to select individuals with radiographic confirmation of rickets. Age- and sex-matched controls were identified for the evaluation of risk factors. The main outcome measure was radiographic evidence of rickets without identifiable inherited, genetic, or nonnutritional causes. Incidence rates were calculated using Rochester Epidemiology Project census data.Of 768 children

2013 Mayo Clinic Proceedings

15. Impact of vitamin D supplementation on markers of bone mineral metabolism in term infants. (Abstract)

-terminal propeptide of type I procollagen (PINP), C-terminal telopeptide of type I collagen (CTX), and amino-terminal propeptide of C-type natriuretic peptide (NT-proCNP) were measured. None of the newborns had craniotabes, hypocalcemia or hyperparathyroidism. The median (Q1;Q3) 25OHD increased from a baseline of 8.45 (7;11.9) ng/ml to 54.6 (34.7;67.3) ng/ml (p<0.001). The baseline 25OHD negatively correlated with total increment of 25OHD (r=-0.54; p=0.002). There were changes in ALP (241 vs. 331 IU; p

2012 Bone

16. Vitamin Deficiency, Dependency, and Toxicity - Vitamin D

weakness, and bone pain at any age. Vitamin D deficiency in a pregnant woman causes deficiency in the fetus. Occasionally, deficiency severe enough to cause maternal osteomalacia results in rickets with metaphyseal lesions in neonates. In young infants, rickets causes softening of the entire skull (craniotabes). When palpated, the occiput and posterior parietal bones feel like a ping pong ball. In older infants with rickets, sitting and crawling are delayed, as is fontanelle closure; there is bossing (...) sunlight exposure or dietary intake Symptoms and signs of rickets, osteomalacia, or neonatal tetany Characteristic bone changes seen on x-ray X-rays of the radius and ulna plus serum levels of calcium, phosphate, alkaline phosphatase, PTH, and 25(OH)D are needed to differentiate vitamin D deficiency from other causes of bone demineralization. Assessment of vitamin D status and serologic tests for syphilis can be considered for infants with craniotabes based on the history and physical examination

2013 Merck Manual (19th Edition)

17. Hypovitaminosis D Full Text available with Trip Pro

International (Review). 99 (3): 227–36. : . . Yorifuji J, Yorifuji T, Tachibana K, Nagai S, Kawai M, Momoi T, Nagasaka H, Hatayama H, Nakahata T (May 2008). "Craniotabes in normal newborns: the earliest sign of subclinical vitamin D deficiency". The Journal of Clinical Endocrinology and Metabolism . 93 (5): 1784–8. : . . Holick MF (October 2008). "Vitamin D: a D-Lightful health perspective". Nutrition Reviews . 66 (10 Suppl 2): S182–94. : . . Wang CJ, McCauley LK (December 2016). . Current Osteoporosis

2012 Wikipedia

18. Prevention and treatment of infant and childhood vitamin D deficiency in Australia and New Zealand: a consensus statement Full Text available with Trip Pro

Drugs: rifampicin, isoniazid and anticonvulsants 25-OHD = 25-hydroxyvitamin D or calcidiol. 1,25-(OH) 2 D = 1,25-dihydroxyvitamin D or calcitriol. 2 Osseous signs of vitamin D deficiency (common to less common) Swelling of wrists and ankles Rachitic rosary (enlarged costochondral joints felt lateral to the nipple line) Genu varum, genu valgum or windswept deformities of the knee Frontal bossing Limb pain and fracture Craniotabes (softening of skull bones, usually evident on palpation of cranial

2006 MJA Clinical Guidelines

19. Hypervitaminosis

present with craniotabes, irritability, failure to thrive, decreased appetite and pruritus. Craniotabes is abnormally soft bones of the skull and is unrelated to tabes dorsalis. Complications include: . Hypercalciuria and . Benign intracranial hypertension. Vitamin A may be associated with increased bone fragility and an increased risk of fractures but current evidence is inconclusive. [ ] Investigation FBC. U&E, especially if there is vomiting. LFTs. Ca ++ . Dual-energy X-ray absorptiometry (DEXA

2008 Mentor

20. Osteomalacia and Rickets (Vitamin D Deficiency)

, and a soft, deformable skull (craniotabes). Children with vitamin D deficiency may be irritable and reluctant to weight-bear, and manifest impaired growth. An increased susceptibility to infections and respiratory symptoms may be a caused by a 'rachitic lung', with reduced lung expansion and muscle weakness. Severe vitamin D deficiency can result in cardiomyopathy and potentially fatal heart failure. Adults: Pain and proximal muscle weakness are the main features of vitamin D deficiency in adults (...) -bowing and knock knees. There may be bony deformities of the chest, pelvis and skull, delayed dentition, poor growth, and bone pain. Softening of the skull (craniotabes) and frontal bossing in the first few months of life; delayed closure of fontanelles. Tender swollen joints. Enlargement of the ends of the ribs ('rachitic rosary') due to expansion of the costochondral junction in a 3- to 6-month-old child. Deformed bones, bowing of the legs, knock knees. Delayed walking or a waddling gait. Impaired

2008 Mentor

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