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Cranial Nerve 10

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9861. Cranial Sixth-Nerve Palsy and Eosinophilia in an Outbreak of Mycoplasma Pneumonia Full Text available with Trip Pro

Cranial Sixth-Nerve Palsy and Eosinophilia in an Outbreak of Mycoplasma Pneumonia The authors discuss a case in which three siblings presented with Mycoplasma pneumonia. All three had a typical rise in complement fixation antibody titres. However, the sibling with the highest titre also developed cranial sixth-nerve palsy; in addition, she was the only one of the three who did not have an eosinophilia. The authors review the symptomatology of Mycoplasma pneumonia and the involvement

1987 Canadian Family Physician

9862. Infectious mononucleosis with cranial nerve palsies. Full Text available with Trip Pro

Infectious mononucleosis with cranial nerve palsies. 3590393 1987 07 14 2018 11 13 0041-6193 56 1 1987 Apr The Ulster medical journal Ulster Med J Infectious mononucleosis with cranial nerve palsies. 69-71 Flanagan P P Hawkins S A SA Bryars J H JH eng Case Reports Journal Article Northern Ireland Ulster Med J 0417367 0041-6193 IM Adult Cranial Nerve Diseases etiology physiopathology Facial Paralysis etiology physiopathology Female Humans Infectious Mononucleosis complications 1987 4 1 1987 4 1

1987 The Ulster medical journal

9863. Cranial nerve palsy following retrobulbar anaesthesia. Full Text available with Trip Pro

Cranial nerve palsy following retrobulbar anaesthesia. 3342224 1988 04 01 2018 11 13 0007-1161 72 1 1988 Jan The British journal of ophthalmology Br J Ophthalmol Cranial nerve palsy following retrobulbar anaesthesia. 78 Rodgers R R Orellana J J eng Case Reports Letter England Br J Ophthalmol 0421041 0007-1161 IM Aged Anesthesia, Local adverse effects Humans Male Ophthalmoplegia etiology Optic Nerve Diseases etiology 1988 1 1 1988 1 1 0 1 1988 1 1 0 0 ppublish 3342224 PMC1041372 Arch Ophthalmol

1988 The British journal of ophthalmology

9864. Neurovascular compression in cranial nerve and systemic disease. Full Text available with Trip Pro

Neurovascular compression in cranial nerve and systemic disease. As we age, our arteries elongate and our brains "sag." As a consequence of these processes, redundant arterial loops and bridging or intrinsic hindbrain veins may cause cross-compression of cranial nerve root entry zones in the cerebellopontine angle. This pulsatile compression can be seen to produce hyperactive dysfunction of the cranial nerve. Symptoms of trigeminal or glossopharyngeal neuralgia (somatic sensory), hemifacial (...) spasm (somatic motor), tinnitus and vertigo (special sensory) and some cases of "essential" hypertension are caused by these vessels compressing cranial nerves V, IX--X, VII, VIII, and left X and medulla oblongata. Using microsurgical techniques, the symptoms may be relieved by vascular decompression, findings and results in 695 paients are briefly reviewed and correlated. A chronic primate model of "essential" hypertension is briefly described.

1980 Annals of Surgery

9865. Fluctuating oculomotor hyperfunction and hypofunction caused by aneurysmal compression of the third cranial nerve Full Text available with Trip Pro

Fluctuating oculomotor hyperfunction and hypofunction caused by aneurysmal compression of the third cranial nerve 10636655 2000 01 11 2008 11 20 0007-1161 83 10 1999 Oct The British journal of ophthalmology Br J Ophthalmol Fluctuating oculomotor hyperfunction and hypofunction caused by aneurysmal compression of the third cranial nerve. 1204 Dayan M R MR Elston J S JS eng Case Reports Letter England Br J Ophthalmol 0421041 0007-1161 IM Aged Aneurysm complications Carotid Artery Diseases (...) complications Carotid Artery, Internal Female Humans Nerve Compression Syndromes etiology Oculomotor Nerve Diseases etiology 2000 1 15 2000 1 15 0 1 2000 1 15 0 0 ppublish 10636655 PMC1722821

1999 The British journal of ophthalmology

9866. Examination of the cranial nerves Full Text available with Trip Pro

Examination of the cranial nerves 10698908 2000 03 14 1756-1833 320 7235 2000 Mar 04 BMJ (Clinical research ed.) BMJ Examination of the cranial nerves 655A Rothwell P Radcliffe Infirmary, Oxford. eng Journal Article England BMJ 8900488 0959-8138 2000 3 4 2000 3 4 2000 3 4 0 0 ppublish 10698908 PMC1117678

2000 BMJ : British Medical Journal

9867. Results following treatment of third cranial nerve palsy in children. Full Text available with Trip Pro

Results following treatment of third cranial nerve palsy in children. To investigate the etiology, sensory, motor, and cosmetic results of treatment for oculomotor (CNIII) palsy in children.We conducted a retrospective review of the clinical records of children with a diagnosis of CNIII palsy who were followed up in our practice between 1981 and 1996.During the 15-year period, 49 children with 53 affected eyes were followed for a mean of 5.5 years. CNIII palsy was congenital in one third

1998 Transactions of the American Ophthalmological Society

9868. Unruptured Aneurysms with Cranial Nerve Symptoms: Efficacy of Endosaccular Guglielmi Detachable Coil Treatment Full Text available with Trip Pro

Unruptured Aneurysms with Cranial Nerve Symptoms: Efficacy of Endosaccular Guglielmi Detachable Coil Treatment To evaluate the efficacy of endosaccular Guglielmi detachable coil (GDC) treatment of unruptured aneurysms causing cranial nerve (CN) symptoms.Among a database of 218 patients whose aneurysms were treated using GDC, seven patients met the criteria for unruptured aneurysms presenting with symptoms and signs of CN palsy. Changes in CN symptoms before and after GDC treatment were (...) reviewed.Aneurysms were located in the internal carotid-posterior communicating artery (n=3), the basilar bifurcation (n=1) and the cavernous internal carotid artery (n=3). CN symptoms included ptosis (n=6), mydriasis (n=2), and extraocular muscle (EOM) disorder (CN III: n=4; CN VI: n=3). Overall, improvement or resolution of CN symptoms after treatment was noted in five patients. CN symptoms in cases involving small (10 mm) and intradural aneurysms tended to respond better to GDC treatment. Ptosis

2003 Korean Journal of Radiology

9869. Cranial nerve clock. Part 1. A declarative memory paradigm. (Abstract)

Cranial nerve clock. Part 1. A declarative memory paradigm. The authors performed this study to compare a declarative memory paradigm developed to help teach medical students about the cranial nerves with a traditional text-based approach.The authors designed a clock-based paradigm to help medical students learn about the cranial nerves. To enhance memorization and related brain activation, the paradigm uses visual, spatial, and word associations in the context of an analog clock face. Twenty (...) -one undergraduate students were randomly divided into two groups. Group T viewed traditional text slides, and group C viewed text slides followed by the corresponding cranial clock slides. Subjects were tested before and after these sessions.Group C performed significantly better than group T in learning the names of the cranial nerves and their correct order (P < .011). Recall of name, number, and function was better for 11 of 12 cranial nerves, with statistical significance reached for nerves

2001 Academic radiology Controlled trial quality: uncertain

9870. Maternal diabetes in the rat impairs the formation of neural-crest derived cranial nerve ganglia in the offspring. Full Text available with Trip Pro

staining of embryos from normal and diabetic mothers to investigate the development of cranial nerve ganglia. Neural tube explants were cultured in 10 and 40 mmol/l glucose and cell death and caspase activity was measured with flow cytometry.The development of cranial ganglia V, VII, VIII, IX and X was impaired in day 10-11 embryos of diabetic rats. There was also a higher rate of cell death of neural crest derived cells cultured in 40 mmol/l glucose for 20 h (35% compared to 12% in 10 mmol/l). However (...) Maternal diabetes in the rat impairs the formation of neural-crest derived cranial nerve ganglia in the offspring. Maternal diabetes mellitus increases the risk for fetal malformations. Several of these malformations are found in organs and tissues derived from the neural crest. Previous studies have shown changes in fetal organs of neural crest origin in experimental diabetes and changes in migration of neural crest cells exposed to high glucose in vitro.We used whole-mount neurofilament

2003 Diabetologia

9871. Brainstem dysgenesis: report of five patients with congenital hypotonia, multiple cranial nerve involvement, and ocular motor apraxia. (Abstract)

Brainstem dysgenesis: report of five patients with congenital hypotonia, multiple cranial nerve involvement, and ocular motor apraxia. This paper reports three females and two males with a distinctive congenital syndrome characterized by severe congenital hypotonia, facial diplegia, jaw ankylosis, velo-pharyngeal incoordination, pyramidal tract signs, and ocular motor apraxia. Patients were followed up at ages ranging from 20 months to 16 years. All cases of this syndrome are sporadic, without (...) dysmorphological features, chromosomal, or MRI brain abnormalities. Electrophysiological studies indicate the brainstem as the site of the neurological dysfunction. Post-mortem CNS study of one of the patients demonstrated neuronal depletion of the IV, VII, VIII, and IX cranial nerve nuclei and intact morphology of the cerebral hemispheres. A vascular accident, early in foetal life, is the most likely cause of the clinical picture. The extent of brainstem involvement and its related clinical findings

2003 Developmental Medicine and Child Neurology

9872. Fascicle preservation surgery for facial nerve neuromas involving the posterior cranial fossa. (Abstract)

Fascicle preservation surgery for facial nerve neuromas involving the posterior cranial fossa. To assess facial nerve function after fascicle preservation surgery in cases of facial nerve neuroma involving the cerebellopontine angle.Retrospective case series and literature review.Tertiary referral center.Seven patients with facial nerve neuroma involving the posterior cranial fossa were reviewed from a single neurotologist's practice and combined with a review of 648 cases reported (...) in the literature.Translabyrinthine resection was used in all patients for complete tumor removal. Nerve reconstruction was accomplished with fascicle preservation (three cases), cable nerve interposition grafting (three cases, one of which involved using cranial nerve VIII as the graft), or direct anastomosis (one case).Facial nerve function as measured by the House-Brackmann grading system. RESULTS A postoperative facial nerve (House-Brackmann) grade of II/VI was obtained in two of our three patients who underwent fascicle

2003 Otology and Neurotology

9873. Symptomatic Arnold-Chiari malformation and cranial nerve dysfunction: a case study of applied kinesiology cranial evaluation and treatment. (Abstract)

Symptomatic Arnold-Chiari malformation and cranial nerve dysfunction: a case study of applied kinesiology cranial evaluation and treatment. To present an overview of possible effects of Arnold-Chiari malformation (ACM) and to offer chiropractic approaches and theories for treatment of a patient with severe visual dysfunction complicated by ACM.A young woman had complex optic nerve neuritis exacerbated by an ACM type I of the brain.Applied kinesiology chiropractic treatment was used (...) for treatment of loss of vision and nystagmus. After treatment, the patient's ability to see, read, and perform smooth eye tracking showed improvement.Further studies into applied kinesiology and cranial treatments for visual dysfunctions associated with ACM may be helpful to evaluate whether this single case study can be representative of a group of patients who might benefit from this care.

2005 Journal of Manipulative and Physiological Therapeutics

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