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Cocaine-Induced Coronary Vasospasm

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1. Cocaine-Induced Coronary-Artery Vasospasm. (PubMed)

Cocaine-Induced Coronary-Artery Vasospasm. 26840152 2016 02 12 2016 11 26 1533-4406 374 5 2016 Feb 04 The New England journal of medicine N. Engl. J. Med. IMAGES IN CLINICAL MEDICINE. Cocaine-Induced Coronary-Artery Vasospasm. e5 10.1056/NEJMicm1503339 Almaddah Nureddin N North Shore Medical Center, Salem, MA nalmaddah@partners.org. Ajayi Tokunbo O TO eng Case Reports Journal Article United States N Engl J Med 0255562 0028-4793 I5Y540LHVR Cocaine AIM IM Cocaine adverse effects Coronary (...) Angiography Coronary Vasospasm chemically induced drug therapy Electrocardiography Fatal Outcome Female Heart Arrest chemically induced Hospitalization Humans Middle Aged Pericardial Effusion diagnostic imaging Ultrasonography 2016 2 4 6 0 2016 2 4 6 0 2016 2 13 6 0 ppublish 26840152 10.1056/NEJMicm1503339

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2016 NEJM

2. Cocaine-Induced Coronary Vasospasm

Cocaine-Induced Coronary Vasospasm Cocaine-Induced Coronary Vasospasm Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Cocaine-Induced (...) Coronary Vasospasm Cocaine-Induced Coronary Vasospasm Aka: Cocaine-Induced Coronary Vasospasm , Cocaine and Chest Pain , Cocaine Abuse Cardiovascular Effects , Acute Coronary Syndrome due to Cocaine From Related Chapters II. Epidemiology contributes to nonfatal MI in 25% of patients <45 years old III. Pathophysiology increased risk MI increases to 24 fold over baseline Risk increases within first hour of use and persists for 4-7 hours Acute: Strong effects Tachyarrhythmias Coronary vasospasm

2018 FP Notebook

3. Cocaine-Induced Coronary Vasospasm

Cocaine-Induced Coronary Vasospasm Cocaine-Induced Coronary Vasospasm Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Cocaine-Induced (...) Coronary Vasospasm Cocaine-Induced Coronary Vasospasm Aka: Cocaine-Induced Coronary Vasospasm , Cocaine and Chest Pain , Cocaine Abuse Cardiovascular Effects , Acute Coronary Syndrome due to Cocaine From Related Chapters II. Epidemiology contributes to nonfatal MI in 25% of patients <45 years old III. Pathophysiology increased risk MI increases to 24 fold over baseline Risk increases within first hour of use and persists for 4-7 hours Acute: Strong effects Tachyarrhythmias Coronary vasospasm

2018 FP Notebook

4. Coronary artery vasospasm induced by cocaine (maybe)

Coronary artery vasospasm induced by cocaine (maybe) Coronary artery vasospasm induced by cocaine (maybe) » The Poison Review Coronary artery vasospasm induced by cocaine (maybe) February 4, 2016, 12:52 am Cocaine-Induced Coronary Artery Vasospasm. Almaddah N, Ajayi TO. N Engl J Med 2016 Feb 4;374:e5 In a series of amazing studies that are now about three decades old, Richard Lange and his colleagues at Parkland Hospital in Dallas investigated the cardiovascular effects of administering (...) report. The authors state that the patient’s urine drug screen (UDS) was positive for cocaine, so: “It appears that this patient used cocaine during hospitalization, which led to active coronary spasm.” This conclusion is not at all clear from the evidence presented. As any toxicologist could have told them, the UDS can be positive for 3 days (or more) after last exposure to cocaine, long after the acute effects of the drug have passed. It’s certainly possible that the coronary vasospasm could have

2016 The Poison Review blog

5. Prevalence of Coronary Vasospasm Using Coronary Reactivity Testing in Patients With Spontaneous Coronary Artery Dissection. (PubMed)

by reporting the experience of coronary vasospasm testing in patients with a history of previous SCAD in a large, tertiary referral center. We conducted a single-center retrospective review of patients with history of SCAD confirmed by angiography who received provocative testing using ergonovine in the Cleveland Clinic cardiac catheterization lab from January 1990 to December 2016. Positive vasospasm was defined as: (1) total or subtotal occlusion of at least 1 major coronary artery induced (...) Prevalence of Coronary Vasospasm Using Coronary Reactivity Testing in Patients With Spontaneous Coronary Artery Dissection. Spontaneous coronary artery dissection (SCAD) is an important cause of myocardial infarction and sudden cardiac death, particularly in young to middle-aged women. Coronary vasospasm is another condition believed to be associated with SCAD; however, this has only been shown in isolated case reports to date. We sought to examine the association of SCAD and coronary vasospasm

2019 American Journal of Cardiology

6. A Combination of Tachycardia-Mediated Heart Failure and Coronary Artery Vasospasm-Induced Silent Myocardial Infarction in a Patient with Severe Thyrotoxicosis (PubMed)

A Combination of Tachycardia-Mediated Heart Failure and Coronary Artery Vasospasm-Induced Silent Myocardial Infarction in a Patient with Severe Thyrotoxicosis Severe thyrotoxicosis can present with a myriad of cardiovascular complications. It may be mild features such as palpitations, tachycardia, and exertional dyspnea or may progress to life-threatening consequences such as atrial fibrillation, tachyarrhythmias, heart failure, myocardial infarction, and shock. In rare cases, they may present (...) with myocardial ischemia secondary to coronary artery vasospasm. We report a case of a 59-year-old Malay gentleman who presented with fast atrial fibrillation and tachycardia-mediated heart failure that evolved to a silent myocardial infarction secondary to severe coronary artery vasospasm with undiagnosed severe thyrotoxicosis. He had complete resolution of heart failure and no further recurrence of coronary artery vasospasm once treatment for thyrotoxicosis was initiated and euthyroidism achieved. This life

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2018 Case reports in cardiology

7. Follow-up Exercise Electrocardiography Can Confirm the Appropriateness of Treatment for Exercise-induced Myocardial Ischemia and Life-threatening Cardiac Arrhythmias due to Coronary Vasospasm (PubMed)

Follow-up Exercise Electrocardiography Can Confirm the Appropriateness of Treatment for Exercise-induced Myocardial Ischemia and Life-threatening Cardiac Arrhythmias due to Coronary Vasospasm 28776558 2018 10 09 2018 11 13 2542-5641 130 16 2017 08 20 Chinese medical journal Chin. Med. J. Follow-up Exercise Electrocardiography Can Confirm the Appropriateness of Treatment for Exercise-induced Myocardial Ischemia and Life-threatening Cardiac Arrhythmias due to Coronary Vasospasm. 2003-2004 10.4103 (...) diagnosis Coronary Vasospasm diagnosis Electrocardiography methods Exercise Test methods Follow-Up Studies Humans Male Myocardial Ischemia diagnosis 2017 8 5 6 0 2017 8 5 6 0 2018 10 10 6 0 ppublish 28776558 ChinMedJ_2017_130_16_2003_211891 10.4103/0366-6999.211891 PMC5555140 Heart. 2009 Nov;95(21):1792-7 19570758 Int J Med Sci. 2014 Aug 28;11(11):1161-71 25249785 Circ J. 2014;78(11):2779-801 25273915 J Clin Invest. 1989 Jun;83(6):1946-52 2723067

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2017 Chinese medical journal

8. Panbesy-induced coronary vasospasm (PubMed)

Panbesy-induced coronary vasospasm 28261741 2018 10 23 2018 11 13 0037-5675 58 9 2017 09 Singapore medical journal Singapore Med J Panbesy-induced coronary vasospasm. 569-570 10.11622/smedj.2017014 Amanullah Mohammed Rizwan MR Department of Cardiology, National Heart Centre Singapore, Singapore. Tong Khim Leng KL Department of Cardiology, Changi General Hospital, Singapore. eng Case Reports Letter 2017 03 06 Singapore Singapore Med J 0404516 0037-5675 0 Central Nervous System Stimulants (...) C045TQL4WP Phentermine N9YNS0M02X Acetylcholine IM Acetylcholine adverse effects Adult Angina, Unstable therapy Central Nervous System Stimulants adverse effects Coronary Angiography Coronary Vasospasm chemically induced Female Humans Patient Safety Phentermine adverse effects Risk Factors 2017 3 7 6 0 2018 10 24 6 0 2017 3 7 6 0 ppublish 28261741 j58/9/569 10.11622/smedj.2017014 PMC5605830 J Postgrad Med. 2006 Jan-Mar;52(1):51-6 16534169 Circulation. 2011 Oct 18;124(16):1774-82 22007100 BMJ Case Rep

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2017 Singapore medical journal

9. Acute coronary syndrome secondary to allergic coronary vasospasm (Kounis Syndrome): a case series, follow-up and literature review. (PubMed)

Acute coronary syndrome secondary to allergic coronary vasospasm (Kounis Syndrome): a case series, follow-up and literature review. Kounis syndrome (KS) is the concurrence of acute coronary syndrome associated with mast-cell and platelet activation in the setting of hypersensitivity and allergic or anaphylactic insults. Many drugs and environmental exposures had been reported as inducers, but various inducers and the mechanism of KS remained unknown till now. The widely used traditional Chinese (...) medicine (TCM) as a potential sensitizer were scarcely reported to induce allergic vasospasm due to the ignorance of the linkage between traditional medicine allergy and vasospasm.We described 5 rare cases of KS including unreported triggers of TCM and abortion, reported the treatment strategy and 1~4 years' follow-up results, and tried to probe into the etiology of KS. Case 1 and case 2 for the first time reported acute ST-segment elevation myocardial infarction (STEMI) caused by Chinese herbs related

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2018 BMC Cardiovascular Disorders

10. The potential hazard of drug-eluting stent-induced coronary vasospasm causing subacute stent thrombosis: a case report. (PubMed)

The potential hazard of drug-eluting stent-induced coronary vasospasm causing subacute stent thrombosis: a case report. Drug-eluting stent (DES) -induced coronary vasospasm is a well known phenomenon after stent implantation; however, the extent of this risk is still unknown. We report a case in which DES-induced severe coronary vasospasm was clinically suspected as a cause of subacute stent thrombosis (ST).A 67-year-old man came to our hospital due to chest pain with mild exercise. He (...) to evaluate the coronary vasomotor response; it revealed severe stent-edge spasm in the left main trunk to the LAD, except for the stented lesion, and total occlusion of the left circumflex artery.To our knowledge, the present case is the first report describing in-stent thrombosis secondary to stent-edge spasm. This case describes the potential hazard of DES-induced coronary vasospasm. Although there are several overlapping risk factors for ST development, we consider that stent-edge spasm also plays

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2016 BMC Cardiovascular Disorders

11. Angiotensin II, as well as 5-hydroxytriptamine, is a potent vasospasm inducer of saphenous vein graft for coronary artery bypass grafting in patients with diabetes mellitus (PubMed)

Angiotensin II, as well as 5-hydroxytriptamine, is a potent vasospasm inducer of saphenous vein graft for coronary artery bypass grafting in patients with diabetes mellitus Diabetes mellitus (DM) is an important risk factor for adverse outcomes of coronary artery bypass grafting. The bypass grafts harvested from patients with DM tend to go into spasm after their implantation into the coronary circulation. To clarify the contribution of 5-hydroxytriptamine (5-HT) and angiotensin II (AngII (...) ) in the bypass graft spasm, we examined the contractile reactivity to 5-HT or AngII of isolated human endothelium-denuded saphenous vein (SV) harvested from DM and non-DM patients. The 5-HT-induced constriction of the SV was significantly augmented in the DM group than in the non-DM group, which is similar to our previous report. AngII-induced constriction of the SV was also significantly augmented in the DM group than the non-DM group. Especially in the non-DM group, the AngII-induced maximal

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2016 Biochemistry and Biophysics Reports

12. Capecitabine-Induced Coronary Vasospasm (PubMed)

Capecitabine-Induced Coronary Vasospasm Capecitabine, an oral prodrug of 5-fluorouracil (5-FU), is approved for early-stage and advanced colorectal cancer and metastatic breast cancer. Cardiotoxicity of 5-FU is well described in the literature. However, cardiac adverse effects of capecitabine are poorly described. We report a case of coronary vasospasm induced by capecitabine. A 41-year-old female with metastatic breast cancer presented with chest pain 3 days after starting capecitabine

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2016 Case reports in oncology

13. Coronary Artery Vasospasm (PubMed)

Coronary Artery Vasospasm Coronary artery vasospasm (CVS) is an important mechanism of myocardial ischemia and produces any of the manifestations of coronary artery disease from silent myocardial ischemia, to effort-induced angina and variant angina, to acute coronary syndrome including myocardial infarction or sudden cardiac death. The pathogenesis, characteristic clinical features, diagnosis, and treatment of CVS are summarized. Emphasis is placed on correct diagnosis of CVS using (...) pharmacological spasm provocation test, either during coronary angiography or with echocardiographic monitoring of ventricular wall motion. Current underutilization of pharmacologic provocative test at the time of coronary angiography cannot be justified, as there is no evidence supporting that the incidence of CVS is declining. Physicians' vigilance for objective documentation of CVS is necessary for appropriate management of patients with various clinical presentations of ischemic heart disease.Copyright ©

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2018 Korean circulation journal

14. In Vivo optical coherence tomography visualization of intraplaque neovascularization at the site of coronary vasospasm: a case report. (PubMed)

In Vivo optical coherence tomography visualization of intraplaque neovascularization at the site of coronary vasospasm: a case report. Coronary plaques in patients with coronary vasospastic angina have been characterized by diffuse intima-media thickening with homogeneous fibrous tissue, without confluent necrotic tissue. However, coronary vasospasm can trigger coronary thrombosis, and may play an important role in the pathogenesis of acute coronary syndromes, though the precise morphological (...) coronary spasm-induced anterior myocardial infarction. Optical coherence tomography imaging revealed the coronary plaque with homogeneous high-intensity signal, and a clearly visualized intraplaque neovascular microchannel (NVMC) network.Neovascularization within a coronary atheroma is known to accelerate coronary atherosclerosis. The current case with coronary vasospastic angina highlights the role of NVMC formation in this process.

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2016 BMC Cardiovascular Disorders

15. Cocaine and coronary artery diseases: a systematic review of the literature. (PubMed)

Cocaine and coronary artery diseases: a systematic review of the literature. Cocaine is associated with important cardiac complications such as sudden death, acute myocarditis, dilated cardiomyopathy, life-threatening arrhythmias, and myocardial ischemia as well as infarction. It is well known that cocaine may induce vasospasm through adrenergic stimulation of the coronary arteries. Moreover, cocaine may promote intracoronary thrombosis, triggered by alterations in the plasma constituents (...) , and platelet aggregation, leading to subsequent myocardial infarction. The long-term use of cocaine may stimulate atherosclerosis, probably through endothelial cell dysfunction. Significant and severe coronary atherosclerosis is common in young chronic cocaine users and there is probably a relationship between the duration and frequency of cocaine use and the extent of coronary disease.

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2017 Journal of cardiovascular medicine (Hagerstown, Md.)

16. Cocaine-Induced Electrocardiographic Phenomenon (PubMed)

effects Cocaine-Related Disorders complications Coronary Vasospasm chemically induced diagnosis physiopathology Diagnosis, Differential Dopamine Uptake Inhibitors adverse effects Electrocardiography drug effects Humans Male 2018 10 31 6 0 2018 10 31 6 0 2018 12 12 6 0 epublish 30374245 10.14503/THIJ-18-6722 PMC6183626 Am J Emerg Med. 2006 Jan;24(1):122-3 16338519 Am Heart J. 1982 Apr;103(4 Pt 2):730-6 6121481 Ann Emerg Med. 1999 Mar;33(3):347-51 10036351 Can J Cardiol. 2008 May;24(5):404 18464948 Int (...) Cocaine-Induced Electrocardiographic Phenomenon 30374245 2018 12 11 2018 12 11 1526-6702 45 4 2018 08 Texas Heart Institute journal Tex Heart Inst J Cocaine-Induced Electrocardiographic Phenomenon. 273-274 10.14503/THIJ-18-6722 Kumar Sundeep S Sanchez Luis L Srinivasamurthy Ruthvik R Mathias Patrick F PF eng Case Reports Journal Article 2018 08 01 United States Tex Heart Inst J 8214622 0730-2347 0 Dopamine Uptake Inhibitors I5Y540LHVR Cocaine IM Adult Blood Pressure drug effects Cocaine adverse

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2018 Texas Heart Institute Journal

17. CRACKCast E154 – Cocaine and Other Sympathomimetics

for chest pain in a similar fashion to that for patients without cocaine use. Decisions regarding further investigation are based on the characteristics and course of the chest pain and results of serial troponin measurements and ECGs.” [8] List 3 pathophysiologic effects of cocaineinduced MI Accelerated atherosclerosis Enhanced platelet aggregation Vasospasm Increased demand (increased HR and afterload) LVH with time [9] What is the approach to ACS management in this patient? DAPT Consider Cath lab (...) -induced psychosis and agitation resulting from sympathomimetic agents. NOTE: Although lorazepam also can be given intramuscularly, it may take 15 minutes to reach peak sedation; repeated doses given at more frequent intervals may accumulate, causing oversedation and respiratory depression. NOTE: Safe antihypertensives to use in the patient with severe cocaine toxicity: Phentolamine Nitroglycerin Hydralazine Treatment of hyperthermia: NOTE: Patients who sustain elevated core temperatures above 106°F

2018 CandiEM

18. Immunosuppressive therapy induced coronary vasospasm and acute myocardial infarction in a patient undergoing new renal transplantation (PubMed)

Immunosuppressive therapy induced coronary vasospasm and acute myocardial infarction in a patient undergoing new renal transplantation Immunosuppressant agents such as calcineurin inhibitors (CNI) used after solid organ transplantation may cause endothelial dysfunction, and coronary and renal arterial vasospasm. We report a patient presenting acute ST segment elevation myocardial infarction (STEMI) at the second week of renal transplantation. In the case of STEMI in patients with solid organ (...) transplants under immunosuppressive therapy with CNI, coronary vasospasm associated with these drugs should be kept in mind before starting any interventional procedure. High dose nitroglycerine may immediately resolve tacrolimus or cyclosporine A induced coronary vasospasm. Calcium channel blockers should immediately be added to treatment because of the short half-life of nitroglycerine.

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2015 Postępy w kardiologii interwencyjnej = Advances in interventional cardiology

19. Aneurysmal subarachnoid hemorrhage and severe, catheter-induced vasospasm associated with excessive consumption of a caffeinated energy drink (PubMed)

Aneurysmal subarachnoid hemorrhage and severe, catheter-induced vasospasm associated with excessive consumption of a caffeinated energy drink Excessive consumption of over-the-counter stimulants is associated with coronary vasospasm, thrombotic complications, and sudden cardiac death. Their effects on cerebrovascular physiology are not yet described in the neurointerventional literature. Patients are increasingly exposed to high levels of these vasoactive substances in the form of caffeinated (...) energy drinks and specialty coffees. We report a case of aneurysmal subarachnoid hemorrhage (SAH) and severe, catheter-induced vasospasm during attempted endovascular repair of a ruptured anterior communicating artery (AComA) aneurysm in the setting of excessive energy drink consumption. We review the literature and alert clinicians to this potentially serious complication.© The Author(s) 2016.

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2016 Interventional Neuroradiology

20. Evaluation and medical therapy for coronary endothelial dysfunction induced by sirolimus-eluting stent in patient with an atherosclerotic lesion of the left main coronary artery: Case report (PubMed)

Evaluation and medical therapy for coronary endothelial dysfunction induced by sirolimus-eluting stent in patient with an atherosclerotic lesion of the left main coronary artery: Case report Sirolimus-eluting stents (SES), especially those deployed at distal sites, cause more coronary vasospasm and endothelial dysfunction in the chronic phase compared to bare-metal stents (BMS). In comparison, endothelial dysfunction is less frequently induced by the Biolimus-A9 eluting stent (BES). A 75-year (...) ). Adequate medication for CSA and CED and intervention for the atherosclerotic lesion contributed to improvement of vascular function and disappearance of his symptoms. induced by Biolimus-A9 eluting stents (BES) is less than that induced by Sirolimus-eluting stents (SES), although all drug-eluting stents are more likely to cause coronary vasospasm and endothelial dysfunction in the chronic phase, especially at distal deployment sites

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2017 Journal of cardiology cases

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