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Clostridium perfringens

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1401. Clostridium perfringens brain infection following a penetration wound of the orbit. (PubMed)

Clostridium perfringens brain infection following a penetration wound of the orbit. 2883262 1987 05 22 2018 11 13 0022-3050 50 2 1987 Feb Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Clostridium perfringens brain infection following a penetration wound of the orbit. 241 McHugh D D Moseley R P RP Uttley D D eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Clostridium perfringens Encephalitis etiology Gas Gangrene etiology

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1987 Journal of neurology, neurosurgery, and psychiatry

1402. Differences in the Carboxy-Terminal (Putative Phospholipid Binding) Domains of Clostridium perfringens and Clostridium bifermentans Phospholipases C Influence the Hemolytic and Lethal Properties of These Enzymes (PubMed)

Differences in the Carboxy-Terminal (Putative Phospholipid Binding) Domains of Clostridium perfringens and Clostridium bifermentans Phospholipases C Influence the Hemolytic and Lethal Properties of These Enzymes The phospholipases C of C. perfringens (alpha-toxin) and C. bifermentans (Cbp) show >50% amino acid homology but differ in their hemolytic and toxic properties. We report here the purification and characterisation of alpha-toxin and Cbp. The phospholipase C activity of alpha-toxin

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1999 Infection and immunity

1403. Chloramphenicol Resistance in Clostridium difficile Is Encoded on Tn4453 Transposons That Are Closely Related to Tn4451 from Clostridium perfringens (PubMed)

Chloramphenicol Resistance in Clostridium difficile Is Encoded on Tn4453 Transposons That Are Closely Related to Tn4451 from Clostridium perfringens The chloramphenicol resistance gene catD from Clostridium difficile was shown to be encoded on the transposons Tn4453a and Tn4453b, which were structurally and functionally related to Tn4451 from Clostridium perfringens. Tn4453a and Tn4453b excised precisely from recombinant plasmids, generating a circular form, as is the case for Tn4451. Evidence (...) been shown that Tn4453a and Tn4453b can be transferred to suitable recipient cells by RP4 and therefore are mobilizable transposons. It is concluded that, like Tn4451, they must encode a functional tnpZ gene and a target oriT or RSA site. The finding that related transposable elements are present in C. difficile and C. perfringens has implications for the evolution and dissemination of antibiotic resistance genes and the mobile elements on which they are found within the clostridia.

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1998 Antimicrobial Agents and Chemotherapy

1404. Clostridium difficile and Clostridium perfringens in upper gut of infants with protracted diarrhoea. (PubMed)

Clostridium difficile and Clostridium perfringens in upper gut of infants with protracted diarrhoea. 4056073 1985 12 03 2018 11 13 0021-9746 38 10 1985 Oct Journal of clinical pathology J. Clin. Pathol. Clostridium difficile and Clostridium perfringens in upper gut of infants with protracted diarrhoea. 1196 Gianerilli P P Luzzi I I Occhionero M M Capano G G Guarino A A Guandalini S S eng Letter England J Clin Pathol 0376601 0021-9746 AIM IM Child, Preschool Clostridium isolation & purification

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1985 Journal of Clinical Pathology

1405. Hybridization analysis of three chloramphenicol resistance determinants from Clostridium perfringens and Clostridium difficile. (PubMed)

Hybridization analysis of three chloramphenicol resistance determinants from Clostridium perfringens and Clostridium difficile. The chloramphenicol resistance determinant from a nonconjugative strain of Clostridium perfringens was cloned and shown to be expressed in Escherichia coli. Subcloning and deletion analysis localized the resistance gene, catQ, to within a 1.25-kilobase (kb) partial Sau3A fragment. The catQ gene contained internal HindII, HaeIII, and DraI restriction sites (...) and was distinct from the catP gene, which was originally cloned (L. J. Abraham, A. J. Wales, and J. I. Rood Plasmid 14:37-46, 1985) from the conjugative C. perfringens R plasmid, pIP401. Hybridization studies were carried out with a 0.35-kb DraI-P fragment of pJIR260 as an internal catQ-specific probe and a 0.38-kb EcoRV-HinfI fragment of pJIR62 as an internal catP-specific gene probe. The results showed that the catP and catQ genes were not similar and that neither probe hybridized with cat genes from other

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1989 Antimicrobial Agents and Chemotherapy

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