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Carcinogens in the Workplace

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1. Carcinogens in the Workplace

Carcinogens in the Workplace Carcinogens in the Workplace Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Carcinogens in the Workplace (...) Carcinogens in the Workplace Aka: Carcinogens in the Workplace , Occupational Exposures Linked to Cancer , Cancer-predisposing Occupational Exposure II. Causes: Carcinogens Aniline and homologs ( s) Beryllium Silica Chromium Cadmium Coal tar pitch volatile substances Diesel exhaust fumes Dinitrotoluenes Benzene (gasoline) Ethylene oxide Ionizing radiation Nickel Silica crystals smoke III. Resources CDC NIOSH Carcinogen List Images: Related links to external sites (from Bing) These images are a random

2018 FP Notebook

2. Toxicological risk at workplace and toxicity as Life Cycle Assessment impact category: Substitution of solvents as an example Full Text available with Trip Pro

naphthas were replaced by n-Butylacetate, 1-Ethoxy-2-propyl acetate and Ethyl-3-ethoxy propionate. In the European Union, the naphthas are classified as mutagens and carcinogens category 1, officially. However, if benzene is below 0.1 %, registrants in the EU proposed to omit this classification, and todays naptha products on the market obviously have benzene contents below 0.1 %. On a first glance, the improvement for workplace safety introduced by the substitution, therefore, is comparatively small (...) Toxicological risk at workplace and toxicity as Life Cycle Assessment impact category: Substitution of solvents as an example Substitution of hazardous substances against less hazardous ones is a central requirement of the European Chemical Regulation REACH (European Regulation 1907/2006/EC). Hazardous substances emitted from products may not only affect the worker; drift off and distribution in the environment may finally result in exposure of the general population. This potential threat

2017 EXCLI journal

3. Adverse health effects of asbestos: solving mysteries regarding asbestos carcinogenicity based on follow-up survey of a Chinese factory Full Text available with Trip Pro

Adverse health effects of asbestos: solving mysteries regarding asbestos carcinogenicity based on follow-up survey of a Chinese factory The present review summarizes the results of several follow-up studies assessing an asbestos product manufacturing plant in Chongqing, China, and discusses three controversial issues related to the carcinogenicity of asbestos. The first issue is the amphibole hypothesis, which asserts that the carcinogenicity of asbestos is limited to amphiboles (...) , such as crocidolite, but not serpentines, such as chrysotile. However, considering the possible multiple component of asbestos carcinogenicity in the presence of tobacco smoke or other carcinogens, chrysotile cannot be regarded as non-carcinogenic. Additionally, in a practical sense, it is not possible to assume "pure" chrysotile due to its ubiquitous contamination with tremolite, which is a type of amphibole. Thus, as the International Agency for Research on Cancer (IARC) assessed, all forms of asbestos

2018 Environmental health and preventive medicine

4. Identifying occupational carcinogens: an update from the IARC Monographs Full Text available with Trip Pro

in the pertinent monograph. The number of known occupational carcinogens has increased over time: 47 agents were identified as known occupational carcinogens in 2017 compared with 28 in 2004. These estimates are conservative and likely underestimate the number of carcinogenic agents present in workplaces. Exposure to these agents causes a wide range of cancers; cancers of the lung and other respiratory sites, followed by skin, account for the largest proportion. The dominant routes of exposure are inhalation (...) and dermal contact. Important progress has been made in identifying occupational carcinogens; nevertheless, there is an ongoing need for research on the causes of work-related cancer. Most workplace exposures have not been evaluated for their carcinogenic potential due to inadequate epidemiologic evidence and a paucity of quantitative exposure data.© Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2018. All rights reserved. No commercial use is permitted unless

2018 Occupational and environmental medicine

5. Prioritising action on occupational carcinogens in Europe: a socioeconomic and health impact assessment. Full Text available with Trip Pro

Prioritising action on occupational carcinogens in Europe: a socioeconomic and health impact assessment. Work-related cancer is an important public health issue with a large financial impact on society. The key European legislative instrument is the Carcinogens and Mutagens Directive (2004/37/EC). In preparation for updating the Directive, the European Commission commissioned a study to provide a socioeconomic, health and environmental impact assessment.The evaluation was undertaken for 25 (...) preselected hazardous substances or mixtures. Estimates were made of the number of cases of cancer attributable to workplace exposure, both currently and in the future, with and without any regulatory interventions, and these data were used to estimate the financial health costs and benefits.It was estimated that if no action is taken there will be >700 000 attributable cancer deaths over the next 60 years for the substances assessed. However, there are only seven substances where the data suggest a clear

2017 British Journal of Cancer

6. Carcinogens in the Workplace

Carcinogens in the Workplace Carcinogens in the Workplace Toggle navigation Brain Head & Neck Chest Endocrine Abdomen Musculoskeletal Skin Infectious Disease Hematology & Oncology Cohorts Diagnostics Emergency Findings Procedures Prevention & Management Pharmacy Resuscitation Trauma Emergency Procedures Ultrasound Cardiovascular Emergencies Lung Emergencies Infectious Disease Pediatrics Neurologic Emergencies Skin Exposure Miscellaneous Abuse Cancer Administration 4 Carcinogens in the Workplace (...) Carcinogens in the Workplace Aka: Carcinogens in the Workplace , Occupational Exposures Linked to Cancer , Cancer-predisposing Occupational Exposure II. Causes: Carcinogens Aniline and homologs ( s) Beryllium Silica Chromium Cadmium Coal tar pitch volatile substances Diesel exhaust fumes Dinitrotoluenes Benzene (gasoline) Ethylene oxide Ionizing radiation Nickel Silica crystals smoke III. Resources CDC NIOSH Carcinogen List Images: Related links to external sites (from Bing) These images are a random

2015 FP Notebook

7. Quantification of N-phenyl-2-naphthylamine by gas chromatography and isotope-dilution mass spectrometry and its percutaneous absorption ex vivo under workplace conditions Full Text available with Trip Pro

Quantification of N-phenyl-2-naphthylamine by gas chromatography and isotope-dilution mass spectrometry and its percutaneous absorption ex vivo under workplace conditions N-Phenyl-2-naphthylamine (P2NA) is an antioxidant used to protect rubbers from flex-cracking. P2NA can be converted in vivo to 2NA, one of the most potent bladder carcinogens. Here, we report the specific and ultra-sensitive quantification of P2NA in the receptor fluid of Franz diffusion cells by gas chromatography and isotope

2017 Archives of toxicology

8. Risk assessment of di(2-ethylhexyl) phthalate in the workplace Full Text available with Trip Pro

was determined according to exposure in workplaces and chemical hazard.In 229 workplaces over the country, 831 tons of DEHP have been used as plasticizers, insecticides, and ink solvent. Calculated 50% lethal dose values ranged from 14.2 to 50 g/kg, as determined via acute toxicity testing in rodents. Chronic carcinogenicity tests revealed cases of lung and liver degeneration, shrinkage of the testes, and liver cancer. The no-observed-adverse-effect level and the lowest-observed-adverse-effect level were (...) Risk assessment of di(2-ethylhexyl) phthalate in the workplace A hazard assessment of di(2-ethylhexyl) phthalate (DEHP), a commonly used workplace chemical, was conducted in order to protect the occupational health of workers. A literature review, consisting of both domestic and international references, examined the chemical management system, working environment, level of exposure, and possible associated risks. This information may be utilized in the future to determine appropriate exposure

2016 Environmental health and toxicology

9. Workplace exposure to diesel and gasoline engine exhausts and the risk of colorectal cancer in Canadian men Full Text available with Trip Pro

Workplace exposure to diesel and gasoline engine exhausts and the risk of colorectal cancer in Canadian men The International Agency for Research on Cancer (IARC) classified diesel exhaust as carcinogenic to humans (Group 1) and gasoline exhaust as a possible carcinogen (Group 2B) based studies of lung cancer, however the evidence for other sites is limited. We addressed this question by investigating exposure to diesel and gasoline emissions with respect to risk of colorectal cancer in men.We

2016 Environmental Health

10. Exposure Monitoring and Risk Assessment of Biphenyl in the Workplace Full Text available with Trip Pro

Exposure Monitoring and Risk Assessment of Biphenyl in the Workplace This study was performed to assess exposure to and the risk caused by biphenyl in the workplace. Biphenyl is widely used as a heat transfer medium and as an emulsifier and polish in industry. Vapor or high levels of dust inhalation and dermal exposure to biphenyl can cause eye inflammation, irritation of respiratory organs, and permanent lesions in the liver and nervous system. In this study, the workplace environment (...) concentrations were assessed as central tendency exposure and reasonable maximum exposure and were shown to be 0.03 and 0.12 mg/m³, respectively. In addition, the carcinogenic risk of biphenyl as determined by risk assessment was 0.14 × 10⁻⁴ (central tendency exposure) and 0.56 × 10⁻⁴ (reasonable maximum exposure), which is below the acceptable risk value of 1.0 × 10⁻⁴. Furthermore, the central tendency exposure and reasonable maximum exposure hazard quotients were 0.01 and 0.06 for oral toxicity, 0.05

2015 International journal of environmental research and public health

11. Carcinogen

carcinogens U.S. National Toxicology Program [ ] The of the is mandated to produce a biennial Report on Carcinogens . As of June 2011, the latest edition was the 12th report (2011). It classifies carcinogens into two groups: Known to be a human carcinogen Reasonably anticipated being a human carcinogen American Conference of Governmental Industrial Hygienists [ ] The (ACGIH) is a private organization best known for its publication of (TLVs) for occupational exposure and monographs on workplace chemical (...) Carcinogen Carcinogen - Wikipedia Carcinogen From Wikipedia, the free encyclopedia A carcinogen is any substance, , or that promotes , the formation of . This may be due to the ability to damage the or to the disruption of cellular processes. Several radioactive substances are considered carcinogens, but their carcinogenic activity is attributed to the radiation, for example and , which they emit. Common examples of non-radioactive carcinogens are inhaled , certain , and smoke. Although

2012 Wikipedia

13. Improving outdoor air quality and health: review of interventions

– some local authorities have successfully used workplace or other levies to fund improvement and use of public transport. Spatial planning can be used to reduce the need for vehicle use by design, and has a wider role in reducing emissions from buildings through energy-efficiency measures and use of renewable energy technologies. Promising local interventions that can help reduce demand for more polluting forms of transport are associated with use of public transport and active travel and include

2019 Public Health England

14. Occupational liver diseases

Occupational liver diseases EASL Clinical Practice Guideline: Occupational liver diseases q European Association for the Study of the Liver ? Summary A variety of chemicals have been linked to occupational liver diseases, including several solvents and mixtures thereof, pesti- cides, and metals. Workplace exposures have been associated withvirtuallytheentirespectrumofacuteandchronicliverdis- eases. However, their prevalence is inadequately quanti?ed and their epidemiology limited. Occupational (...) . 2019EuropeanAssociationfortheStudyoftheLiver.Publishedby Elsevier B.V. All rights reserved. Introduction Occupational exposures can induce liver injury in a similar way to prescription drugs, herbal and dietary supplements, and workplace exposure has been implicated in the full spectrum of liver disease. However, the awareness of hepatologists for thisspeci?caetiologyofliverinjuryislimitedandtheincidence and prevalence of occupational liver diseases (OLDs) remains unknown.Acuteliverinjuryislikelytodecrease, atleastinhigh income countries, given

2019 European Association for the Study of the Liver

15. Return to School Considerations

), if indicated, and proper ventilation. The use of EPA approved disinfectants against COVID-19 is recommended ( ). When possible, only products labeled as (eg, Safer or Designed for the Environment), containing active ingredients such as hydrogen peroxide, ethanol, citric acid, should be selected from this list, because they are less toxic, are not strong respiratory irritants or asthma triggers, and have no known carcinogenic, reproductive, or developmental effects. When EPA-approved disinfectants (...) for entrance to or exclusion from school. regarding antibody testing for COVID-19 is that serologic test results should not be used to make decisions about grouping people residing in or being admitted to congregate settings, such as schools, dormitories, or correctional facilities. Additionally, serologic test results should not be used to make decisions about returning people to the workplace. The CDC states that serologic testing should not be used to determine immune status in individuals until

2020 American Academy of Pediatrics

16. Management of Alcohol-Related Liver Disease

that heavyalcoholintakeassociateswithincreasedriskofcardiomy- opathy, hypertension, atrial arrhythmias and haemorrhagic stroke, light–moderate drinkers seem to have a lower risk of coronaryarterydisease. 13 Thispositiveeffectofalcoholmayoff- set some of the large array of negative health consequences of even moderate alcohol consumption. However, alcohol is a recognised carcinogen, and no threshold level of consumption existsfortheriskofcancer.Alcoholconsumptionhasbeenasso- ciated with an increased risk of several cancers, and in at least four of them (...) or liver cirrhosis, i.e. patients presenting with bilateral parotid gland hypertrophy, muscle wasting, malnutrition, Dupuytren’s contracture, gynecomastia or extensive spider naevi. Screening for ALD should be performed in high-risk popula- tions, such as those in alcohol rehabilitations clinics, or harmful drinkers identi?ed by their GP. Screening in the workplace would be extremely helpful, although dif?cult to implement. 26 The best way to do such screening is still debatable. The Southampton traf?c

2018 European Association for the Study of the Liver

17. Closed-system transfer-devices for limiting exposure to cytotoxic anti-cancer drugs in healthcare professionals, patients and visitors

Organisational issues/context 15 Identified research gaps 16 Conclusion 16 Equality and diversity 17 Acknowledgements 17 References 18 SHTG Evidence Synthesis | 4 Definitions Antineoplastic: Blocking the formation of neoplasms (growths that may become cancer) 1 . Cytotoxic drug: A substance that kills cells, including cancer cells. These agents may stop cancer cells from dividing and growing and may cause tumors to shrink in size 1 . Carcinogen: Any substance that causes cancer 1 . Genotoxic: Denoting (...) , distribution, administration and disposal are exposed to low doses of cytotoxic drugs over many years. 3 Hazardous drugs, which include cytotoxic drugs, are defined as drugs which are carcinogenic, genotoxic, teratogenic, and those that can result in reproductive toxicity, and organ toxicity at low doses. 4 Cytotoxic drugs, or antineoplastic agents, have anti-tumour and immunosuppressive functions and are used to treat cancer and disorders such as rheumatoid arthritis and multiple sclerosis. 3 Cytotoxic

2019 SHTG Advice Statements

18. Tobacco Cessation Treatment

sustained attention. Keeping it on the problem list also alerts the patient’s other providers to reiterate smoking cessation advice at their visits and facilitates coordination of tobacco treatment efforts across providers. Exposure to Tobacco Smoke. Because SHS exposure increases nonsmokers’ CVD risk (23), all patients should also be asked about SHS exposure routinely in clinical practice. Smoke-free policies in the United States have made most public places and many workplaces, restau- rants, and bars

2019 American College of Cardiology

19. Muscle-invasive and Metastatic Bladder Cancer

cancer. Int J Radiat Oncol Biol Phys, 2012. 83: 953. 28. Zamora-Ros, R., et al. Flavonoid and lignan intake in relation to bladder cancer risk in the European Prospective Investigation into Cancer and Nutrition (EPIC) study. Br J Cancer, 2014. 29. Schistosomes, liver flukes and Helicobacter pylori. IARC Working Group on the Evaluation of Carcinogenic Risks to Humans. Lyon, 7-14 June 1994. IARC Monogr Eval Carcinog Risks Hum, 1994. 61: 1. 30. Gouda, I., et al. Bilharziasis and bladder cancer: a time

2019 European Association of Urology

20. Risk factors for breast cancer: A review of the evidence 2018

178 Summary 180 5 Appendix A Acknowledgements 192 Breast cancer risk factors: A review of the evidence v Appendix B IARC and WCRF/AICR classifications 194 Appendix C IARC and WCRF/AICR categories of evidence and criteria for grading carcinogenicity 196 Appendix D Data tables 200 Glossaries 512 Abbreviations 519 References 529 Breast cancer risk factors: A review of the evidence vi Figures Figure 4.1 Age–specific incidence of breast cancer in Australia, by age group, 2017 16 Figure 4.2 Age (...) Table 5.1 Evidence classifications 180 Table 5.2 Summary of risk estimates for factors where the body of evidence has been classified as either ‘Convincing’ or ‘Probable’ 183 Table C.1 International Agency for Research on Cancer (2015): Categories of evidence of carcinogenicity 196 Table C.2 World Cancer Research Fund/American Institute for Cancer Research (2018): Criteria for grading evidence for cancer prevention 198 Table D.1 Birthweight and risk of breast cancer 200 Table D.2 Height and risk

2018 Cancer Australia

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