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Beau Lines

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101. Intestinal and Multivisceral Transplantation (Treatment)

, line infection, wound infection, pulmonary infection, urinary tract infection, and viral enteritis. Sepsis is not uncommon with acute rejection, and this should always be remembered when evaluating a septic patient. Approximately 40% of small bowel transplant recipients require further surgery during their original inpatient stay; additional surgery is usually the result of infectious complications. [ ] Furthermore, these complications are primarily responsible for the prolonged hospitalizations (...) of these patients; ISB recipients typically are hospitalized for 3 weeks to 3 months, and LSB recipients typically are hospitalized for 3-6 months. Following small bowel transplantation, typical pathogens are enteric organisms, fungal species, or staphylococci (associated with central venous line infections). Bacterial infection appears to be far more common if the colon is included in the allograft; thus, inclusion of the colon is not the recommended protocol in many centers. [ ] Empiric antibiotic selection

2014 eMedicine Surgery

102. Short-Bowel Syndrome (Treatment)

of intestine, the presence or absence of strictures or areas of stasis, bowel dilatation, and the intestinal transit time as described above. Various radiographic techniques, including contrast small-bowel follow-through and computed tomography (CT), are helpful in the decision. Transplant surgery is usually reserved for patients who are dependent on parenteral nutrition, who have run out of venous access, who have had several episodes of central line–related sepsis, or who have begun to manifest (...) . Gastroenterology . 1997 Nov. 113(5):1767-78. . Cavicchi M, Beau P, Crenn P, Degott C, Messing B. Prevalence of liver disease and contributing factors in patients receiving home parenteral nutrition for permanent intestinal failure. Ann Intern Med . 2000 Apr 4. 132 (7):525-32. . Klein S, Nealon WH. Hepatobiliary abnormalities associated with total parenteral nutrition. Semin Liver Dis . 1988 Aug. 8(3):237-46. . Quigley EM, Marsh MN, Shaffer JL, Markin RS. Hepatobiliary complications of total parenteral

2014 eMedicine Surgery

103. Intestinal and Multivisceral Transplantation (Overview)

diminished with the introduction of flexible, silastic, silicone rubber catheters; tunneled, cuffed catheters; and improved line care. [ , , ] Less common indications for intestinal transplantation include locally invasive desmoid tumors, premalignant conditions (Gardener syndrome), and fluid and electrolyte losses unmanageable with TPN. [ ] In summary, intestinal transplantation is a salvage procedure applied to patients who have either anatomic or functional diseases that preclude enteral tolerance (eg (...) of the central veins. Intestinal transplantation is considered when the patient has lost 2 or more common venous access sites, such as the subclavian or internal jugular veins, or when unconventional sites such as the right atrial, transhepatic, or direct inferior vena caval catheters are required. Patient history and previous records should reveal the number and type of organisms responsible for previous central venous line infections. Fungal infections requiring mechanical ventilation or vasopressor

2014 eMedicine Surgery

104. Short-Bowel Syndrome (Follow-up)

of intestine, the presence or absence of strictures or areas of stasis, bowel dilatation, and the intestinal transit time as described above. Various radiographic techniques, including contrast small-bowel follow-through and computed tomography (CT), are helpful in the decision. Transplant surgery is usually reserved for patients who are dependent on parenteral nutrition, who have run out of venous access, who have had several episodes of central line–related sepsis, or who have begun to manifest (...) . Gastroenterology . 1997 Nov. 113(5):1767-78. . Cavicchi M, Beau P, Crenn P, Degott C, Messing B. Prevalence of liver disease and contributing factors in patients receiving home parenteral nutrition for permanent intestinal failure. Ann Intern Med . 2000 Apr 4. 132 (7):525-32. . Klein S, Nealon WH. Hepatobiliary abnormalities associated with total parenteral nutrition. Semin Liver Dis . 1988 Aug. 8(3):237-46. . Quigley EM, Marsh MN, Shaffer JL, Markin RS. Hepatobiliary complications of total parenteral

2014 eMedicine Surgery

105. Non-Hodgkin Lymphoma (Follow-up)

and antimetabolites. Current survival rates for patients with advanced disease are 65-75% for T-cell lymphoblastic lymphomas and 80-90% for those with B-cell lymphomas. Antibiotics If present, fever simply may reflect the underlying malignancy. However, consider beginning empiric, broad-spectrum antibiotic coverage until sepsis or focal infection (eg, due to bowel perforation) is excluded. Central venous access For most patients, a is necessary to manage chemotherapy. If feasible, multiple procedures (eg, line (...) the SFOP described surprising efficacy for monotherapy with vinblastine for relapsing anaplastic LCL, even in patients who previously underwent myeloablative therapy with autologous bone marrow transplantation. [ ] The role of vinblastine in front-line therapy for anaplastic LCL was examined in a Children's Oncology Group protocol (A5941), which compared the standard APO regimen with an experimental therapy that included vinblastine. Myelosuppression was more significant than anticipated and the trial

2014 eMedicine Pediatrics

106. Intestinal Transplantation (Follow-up)

one third of cases, this results in a remission of the PTLD. If improvement is not evident after 2 weeks, all immunosuppression should be discontinued and serious consideration should be given to additional therapeutic measures, including chemotherapy using R-CHOP (rituximab, cyclophosphamide, doxorubicin, vincristine, and prednisone) or adoptive immunotherapy. [ ] Radiotherapy is considered a second-line intervention against PTLD in both pediatric and adult patients. [ ] If necessary (...) , et al. 51Cr-EDTA: a marker of early intestinal rejection in the rat. J Surg Res . 1989 May. 46(5):507-14. . Buchman AL, Iyer K, Fryer J. Parenteral nutrition-associated liver disease and the role for isolated intestine and intestine/liver transplantation. Hepatology . 2006 Jan. 43(1):9-19. . Cavicchi M, Beau P, Crenn P, Degott C, Messing B. Prevalence of liver disease and contributing factors in patients receiving home parenteral nutrition for permanent intestinal failure. Ann Intern Med . 2000

2014 eMedicine Pediatrics

107. Psoriatic Arthritis (Treatment)

with psoriatic arthritis, may include the following: Beau lines Leukonychia Onycholysis Oil spots Subungual hyperkeratosis Splinter hemorrhages Spotted lunulae Transverse ridging Cracking of the free edge of the nail Uniform nail pitting Extra-articular features are observed less frequently in patients with psoriatic arthritis than in those with rheumatoid arthritis (RA) but may include the following: Synovitis affecting flexor tendon sheaths, with sparing of the extensor tendon sheath Subcutaneous nodules (...) of autoimmunity, perhaps mediated by complement activation. The inflammatory nature of the skin and joint lesions in psoriatic arthritis is demonstrated by synovial-lining cell hyperplasia and mononuclear infiltration, resembling the histopathologic changes of RA. However, synovial-lining hyperplasia is less, macrophages are fewer, and vascularity is greater in psoriatic arthritis than in RA synovium. The cytokine profile for psoriatic arthritis reflects a complex interplay between T cells and monocyte

2014 eMedicine.com

108. Psoriatic Arthritis (Treatment)

with psoriatic arthritis, may include the following: Beau lines Leukonychia Onycholysis Oil spots Subungual hyperkeratosis Splinter hemorrhages Spotted lunulae Transverse ridging Cracking of the free edge of the nail Uniform nail pitting Extra-articular features are observed less frequently in patients with psoriatic arthritis than in those with rheumatoid arthritis (RA) but may include the following: Synovitis affecting flexor tendon sheaths, with sparing of the extensor tendon sheath Subcutaneous nodules (...) of autoimmunity, perhaps mediated by complement activation. The inflammatory nature of the skin and joint lesions in psoriatic arthritis is demonstrated by synovial-lining cell hyperplasia and mononuclear infiltration, resembling the histopathologic changes of RA. However, synovial-lining hyperplasia is less, macrophages are fewer, and vascularity is greater in psoriatic arthritis than in RA synovium. The cytokine profile for psoriatic arthritis reflects a complex interplay between T cells and monocyte

2014 eMedicine.com

109. Fingernail psoriasis reconsidered: A case-control study. (PubMed)

examination. The disease severity was measured by the NAPSI.Mean NAPSI score in patients and control subjects was 26.6 and 3.6, respectively. Most items included in the NAPSI were specific for nail psoriasis. Onycholysis and splinter hemorrhages were most frequently observed. Leukonychia was more frequent in control subjects. Longitudinal ridges and Beau lines are not included in the NAPSI but are significantly more frequently seen in patients than in control subjects.Limited sample size (...) was a limitation.The NAPSI was able to discriminate patients with fingernail psoriasis from healthy control subjects. Onycholysis and splinter hemorrhages were the most prevalent fingernail changes in psoriatic patients. Leukonychia was more frequently observed in control subjects, which raises the question of whether leukonychia should remain in the NAPSI. On the other hand, longitudinal ridges and Beau lines occurred more frequently in psoriasis but are not included in the NAPSI.Copyright © 2013 American Academy

2013 Journal of American Academy of Dermatology

110. Unilateral Versus Bilateral Neuromodulation Tests in the Treatment of Refractory Idiopathic Overactive Bladder

for Study: 18 Years and older (Adult, Older Adult) Sexes Eligible for Study: All Accepts Healthy Volunteers: No Criteria Inclusion Criteria: The patient must have given his/her informed and signed consent The patient must be insured or beneficiary of a health insurance plan The patient is available for 1 month of follow-up The patient understands and reads French The patient has symptoms of overactive bladder resistant to first-line treatments (physiotherapy, anticholinergics) The patient suffers from (...) : Laurent Wagner, MD +33.(0)4.66.68.33.00 Contact: Carey M Suehs, Ph D +33.(0)4.66.68.67.88 Locations Layout table for location information France Clinique Beau Soleil Recruiting Montpellier, France, 34070 Sub-Investigator: Antoine Faix, MD CHU de Nantes - Hôtel Dieu Recruiting Nantes Cedex 1, France, 44093 Sub-Investigator: Loïc Le Normand, MD CHU de Nîmes - Hôpital Universitaire Carémeau Recruiting Nîmes Cedex 09, France, 30029 Principal Investigator: Laurent Wagner, MD Sub-Investigator: Stéphane

2012 Clinical Trials

111. Uphold Mesh for the Surgical Treatment of Uterine-predominant Prolapse

Frame: 12 months ] Presence/absence: anatomical success as concerns apical (uterine) support, defined as point C <= stage 1 per POP-Q scoreing (ICS) Anterior vaginal wall anatomical success [ Time Frame: 12 months ] Presence/absence: anatomical success on anterior vaginal wall support, defined as point Ba < = stage 1 per POP-Q scoring (ICS) Change from baseline in PFDI-20 scores [ Time Frame: baseline to 12 lines ] Secondary Outcome Measures : Anterior vaginal wall anatomical success [ Time Frame: 6 (...) Croix Rousse Lyon Cedex 4, France, 69317 APHM - Hôpital de la Conception Marseille Cedex 5, France, 13385 Clinique Beau Soleil Montpellier, France, 34070 CHU de Montpellier - Hôpital Lapeyronie Montpellier, France, 34295 CHU de Nîmes - Hôpital Universitaire Carémeau Nîmes Cedex 09, France, 30029 CH Louis Giorgi Orange, France, 84106 CHU de Poitiers Poitiers, France, 86021 Hôpital Foch Suresnes, France, 92150 Sponsors and Collaborators Centre Hospitalier Universitaire de Nīmes Investigators Layout

2012 Clinical Trials

112. Nail disorders in children: diagnosis and management. (PubMed)

but are usually not the first clue to the diagnosis. Beau lines on several fingernails are very common in children after temperature crest. Tumors are rare in children. Radiographic examination allows confirmation of the diagnosis of subungual exostosis. Other cases should undergo biopsy. Single-digit longitudinal melanonychia in children is mostly due to nevi. Its management should be tailored on a case-by-case basis. Acute trauma should never be underestimated in children and hand surgeons should

2011 American journal of clinical dermatology

114. Nail Deformities and Dystrophies

with lymphedema of limbs and/or chronic respiratory disorders. Yellow Nail Syndrome © Springer Science+Business Media Half-and-half nails (Lindsay nails) occur usually with ; the proximal half of the nail is white, and the distal half is pink or pigmented. Lindsay Nails (Half-and-Half Nails) © Springer Science+Business Media White nails occur with , although the distal third may remain pinker. Terry Nails © Springer Science+Business Media Beau Lines Due to Chemotherapy © Springer Science+Business Media Beau (...) Lines © Springer Science+Business Media Beau lines are horizontal grooves in the nail plate that occur when nail growth temporarily slows, which can occur after infection, trauma, or systemic illness. Onychomadesis similarly results from temporary growth arrest of the nail plate and differs from Beau lines in that the full thickness of the nail is involved, causing a proximal separation of the nail plate from the nail bed. It most frequently occurs several months after but can occur after other

2013 Merck Manual (19th Edition)

115. Acute myeloid leukemia

or , , , Treatment , , Prognosis ~27% (US) Frequency 1 million (2015) Deaths 147,100 (2015) Acute myeloid leukemia ( AML ) is a of the line of , characterized by the rapid growth of abnormal cells that build up in the and and interfere with . Symptoms may include feeling tired, , easy bruising and bleeding, and increased risk of . Occasionally, spread may occur to the brain, skin, or gums. As an , AML progresses rapidly and is typically fatal within weeks or months if left untreated. include , previous (...) and/or the activation of proto-oncogenes. Treatment [ ] First-line treatment of AML consists primarily of , and is divided into two phases: induction and postremission (or consolidation) therapy. The goal of induction therapy is to achieve a complete remission by reducing the number of leukemic cells to an undetectable level; the goal of consolidation therapy is to eliminate any residual undetectable disease and achieve a cure. Hematopoietic stem cell transplantation is usually considered if induction chemotherapy

2012 Wikipedia

116. Acute megakaryoblastic leukemia

) behind these cases of secondary AMKL are unknown although an in at positions q21 and q26, i.e. inv(3)(q21q26), is often seen in these secondary cases of adult-AMKL. Rare cases of adult-AMKL also have . These tumors are malignancies of , i.e. primitive cells that give rise to and cells. In adult-AMKL, mediastinal germ cell tumors that are associated with adult-AMKL are not seminomas (i.e. do not originate from the sperm cell line) and occur before or concomitantly with but not after the diagnosis AMKL (...) , immunochemical evidence that these blast cells bear platelet line-specific markers, and occurrence of the genetic aberrations in these blast cells that have been associated with the disease. Treatment [ ] Adult-AMKL has remained poorly responsive to the treatment regiments used in DS-AMKL and non-DS-AMKL. These treatments have given complete remission rates of 43-50%. Prognosis [ ] The prognosis of adult-AMKL in patients treated for the disease is far below that of other forms of AMKL. Their median overall

2012 Wikipedia

117. Acute lymphoblastic leukemia

, or cranial nerve palsies (CNS involvement) Frequent or unexplained and Weight loss and/or loss of appetite Excessive and unexplained bruising Bone pain, joint pain (caused by the spread of "blast" cells to the surface of the bone or into the joint from the marrow cavity) Breathlessness Enlarged lymph nodes, liver and/or spleen Pitting edema (swelling) in the lower limbs and/or abdomen Petechiae, which are tiny red spots or lines in the skin due to low levels Testicular enlargement Mediastinal mass Cause (...) electromagnetic radiation from power lines has also been associated with a slightly increased risk of ALL. This result is questioned as no causal mechanism linking electromagnetic radiation with cancer is known. High birth weight (greater than 4000g or 8.8lbs) is also associated with a small increased risk. The mechanism connecting high birth weight to ALL is also not known. Evidence suggests that can develop in individuals treated with certain types of chemotherapy, such as and . Delayed infection hypothesis

2012 Wikipedia

118. Kawasaki disease

the original on 2011-11-20 . Retrieved 2010-04-16 . CS1 maint: Archived copy as title ( ) Wang S, Best BM, Burns JC (June 2009). . . 28 (6): 538–9. : . . . Michie C, Kinsler V, Tulloh R, Davidson S (October 2000). . . 83 (4): 353–5. : . . . ^ López Neyra A, Alvarez-Coca González J, Pérez Suárez E, Martínez Pérez J, Rubio Villanueva JL (December 2007). "Líneas de Beau y enfermedad de Kawasaki" [Beau's lines and Kawasaki disease]. Anales de Pediatría (in Spanish). 67 (6): 610–1. : . . González Pascual E

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2012 Wikipedia

119. Mastocytosis

to inactivation of pancreatic enzymes by increased acid) Hepatosplenomegaly Pathophysiology [ ] are located in , including the , the linings of the stomach and intestine, and other sites. They play an important role in the immune defence against bacteria and parasites. By releasing chemical "alarms" such as , mast cells attract other key players of the to areas of the body where they are needed. [ ] Mast cells seem to have other roles as well. Because they gather together around , mast cells may play a part (...) A, Hasserjian R, Thiele J, Borowitz MJ, Le Beau MM, Bloomfield CD, Cazzola M, Vardiman JW (May 2016). "The 2016 revision to the World Health Organization classification of myeloid neoplasms and acute leukemia". Blood . 127 (20): 2391–405. : . . Hermine O, Lortholary O, Leventhal PS, et al. (2008). Soyer HP, ed. . PLOS One . 3 (5): e2266. : . . . Moura DS, Sultan S, Georgin-Lavialle S, Pillet N, Montestruc F, Gineste P, Barete S, Damaj G, Moussy A, Lortholary O, Hermine O (2011). . PLOS One . 6 (10): e26375

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2012 Wikipedia

120. Leukemia

that starts in blood-forming tissue, such as the bone marrow, and causes large numbers of abnormal blood cells ^ World Cancer Report 2014 . World Health Organization. 2014. pp. Chapter 5.13. . Vardiman, JW; Thiele, J; Arber, DA; Brunning, RD; Borowitz, MJ; Porwit, A; Harris, NL; Le Beau, MM; Hellström-Lindberg, E; Tefferi, A; Bloomfield, CD (30 Jul 2009). "The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes (...) , Thiele J, Borowitz MJ, Le Beau MM, Bloomfield CD, Cazzola M, Vardiman JW (2016). "The 2016 revision to the World Health Organization classification of myeloid neoplasms and acute leukemia". Blood . 127 (20): 2391–405. : . . Bhatnagar N, Nizery L, Tunstall O, Vyas P, Roberts I (October 2016). . Current Hematologic Malignancy Reports . 11 (5): 333–41. : . . . Crispino JD, Horwitz MS (April 2017). . Blood . 129 (15): 2103–2110. : . . . Seewald L, Taub JW, Maloney KW, McCabe ER (September 2012). "Acute

2012 Wikipedia

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