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Athetosis

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121. PRRT2 mutation correlated with phenotype of paroxysmal kinesigenic dyskinesia and drug response. (Abstract)

, Fudan University, Shanghai, China. Chen Wan-Jin WJ Ni Wang W Wang Kai-Yan KY Liu Gong-Lu GL Wang Ning N Xiong Zhi-Qi ZQ Xu Jianfeng J Wu Zhi-Ying ZY eng Journal Article Research Support, Non-U.S. Gov't 2013 03 27 United States Neurology 0401060 0028-3878 0 Anticonvulsants 0 Membrane Proteins 0 Nerve Tissue Proteins 0 PRRT2 protein, human 33CM23913M Carbamazepine AIM IM Age of Onset Anticonvulsants therapeutic use Athetosis etiology genetics Carbamazepine therapeutic use Child China Disease

2013 Neurology

122. Head drop in Huntington disease: Insights into the pathophysiology. (Abstract)

Head drop in Huntington disease: Insights into the pathophysiology. 23884035 2013 10 24 2013 08 20 1526-632X 81 8 2013 Aug 20 Neurology Neurology Head drop in Huntington disease: insights into the pathophysiology. 769-70 10.1212/WNL.0b013e3182a1aa55 Morgante Francesca F Dipartimento di Neuroscienze, Università di Messina, Italy. fmorgante@gmail.com Girlanda Paolo P Martino Davide D eng Case Reports Journal Article 2013 07 24 United States Neurology 0401060 0028-3878 AIM IM Athetosis diagnosis

2013 Neurology

123. Movement disorders in cerebrovascular disease. (Abstract)

including chorea, ballism, athetosis, dystonia, tremor, myoclonus, stereotypies, and akathisia. Some of these disorders occur immediately after acute stroke, whereas others can develop later, and yet others represent delayed-onset progressive movement disorders. These movement disorders have been encountered in patients with ischaemic and haemorrhagic strokes, subarachnoid haemorrhage, cerebrovascular malformations, and dural arteriovenous fistula affecting the basal ganglia, their connections

2013 Lancet Neurology

124. Kernicterus

of the BASAL GANGLIA; BRAIN STEM; and CEREBELLUM and clinically to describe a syndrome associated with HYPERBILIRUBINEMIA. Clinical features include athetosis, MUSCLE SPASTICITY or hypotonia, impaired vertical gaze, and DEAFNESS. Nonconjugated bilirubin enters the brain and acts as a neurotoxin, often in association with conditions that impair the BLOOD-BRAIN BARRIER (e.g., SEPSIS). This condition occurs primarily in neonates (INFANT, NEWBORN), but may rarely occur in adults. (Menkes, Textbook of Child (...) Neurology, 5th ed, p613) Definition (CSP) brain disorder associated with high levels of bilrubin; clinical features include athetosis, muscle spasticity or hypotonia, impaired vertical gaze, and deafness; nonconjugated bilirubin enters the brain and acts as a neurotoxin, often in association with conditions that impair the blood-brain barrier; this condition occurs primarily in neonates with perinatal blood incompatibilities, but may rarely occur in adults. Concepts Disease or Syndrome ( T047 ) MSH

2015 FP Notebook

125. Huntington's Chorea

dominant trait and characterized by the onset of progressive CHOREA and DEMENTIA in the fourth or fifth decade of life. Common initial manifestations include paranoia; poor impulse control; DEPRESSION; HALLUCINATIONS; and DELUSIONS. Eventually intellectual impairment; loss of fine motor control; ATHETOSIS; and diffuse chorea involving axial and limb musculature develops, leading to a vegetative state within 10-15 years of disease onset. The juvenile variant has a more fulminant course including

2015 FP Notebook

126. Infantile Cerebellar-Retinal Degeneration Associated with a Mutation in Mitochondrial Aconitase, ACO2. Full Text available with Trip Pro

Infantile Cerebellar-Retinal Degeneration Associated with a Mutation in Mitochondrial Aconitase, ACO2. Degeneration of the cerebrum, cerebellum, and retina in infancy is part of the clinical spectrum of lysosomal storage disorders, mitochondrial respiratory chain defects, carbohydrate glycosylation defects, and infantile neuroaxonal dystrophy. We studied eight individuals from two unrelated families who presented at 2-6 months of age with truncal hypotonia and athetosis, seizure disorder

2012 American Journal of Human Genetics

127. Further evidence of validity of the Modified Melbourne Assessment for neurologically impaired children aged 2 to 4 years. (Abstract)

behaviours for task and time demands on both tools, and (3) scores on the MMA could discriminate between children with mild, moderate, and severe levels of upper limb impairment.  An observational study of 30 children (19 males, 11 females) with neurological impairment aged 2 to 4 years. Twenty-four children had spasticity (20 with a unilateral and four with a bilateral impairment) and two children presented with athetosis, two with ataxia, and two with hypotonia.A high, positive relation was found

2012 Developmental Medicine and Child Neurology

128. Brainstem cavernous malformations: the role of Gamma Knife surgery. Full Text available with Trip Pro

patients experienced at least 2 instances of repeated bleeding before GKS; these hemorrhages caused neurological deficits including cranial nerve deficits, hemiparesis, hemisensory deficits, spasticity, chorea or athetosis, and consciousness disturbance.The mean size of the CMs at the time of GKS was 3.2 cm(3) (range 0.1-14.6 cm(3)). The mean radiation dose directed to the lesion was 11 Gy with an isodose level at 60.0%. The mean clinical and imaging follow-up time was 40.6 months (range 1.0-150.7

2012 Journal of Neurosurgery

129. Exploring Hypertonia in Children With Cerebral Palsy

it is often overlooked in the diagnostic formulation of motor aspects of cerebral palsy, and therefore does not necessarily figure in treatment decision-making. Under-recognised dystonia, when co-existent with spasticity, can produce unpredictable surgical outcomes in the management of gait disorders and associated musculoskeletal deformities. In addition, other abnormal movements such as chorea and athetosis may be observed in these children, adding to the complexity of the movement disorder (...) (dystonia or athetosis) and 3.6% as ataxic.1 However this study also found that when children were assessed face-to-face by a group of expert paediatricians, 19.4% of the population was noted on observation alone to have abnormal movements, which included dyskinesias. This increased with motor severity by Gross Motor Functional Classification System (GMFCS) from 7% (level I) to 45% (level V). We questioned whether in fact recognition of some dyskinesias are "masked" by the presence of spasticity

2012 Clinical Trials

130. A Trial Comparing Two Methods of CIMT in the Hemiplegic Child

of bilateral upper limb involvement. Diagnosis/ clinical evidence of athetosis or dystonia. Any medical condition which would cause problems with the cast e.g. chronic eczema. An episode of >2 weeks of prolonged CIMT using a cast /splint in the previous 6 months. Contacts and Locations Go to No Contacts or Locations Provided More Information Go to Layout table for additonal information Responsible Party: Pauline Christmas, Consultant Physiotherapist, University of Birmingham ClinicalTrials.gov Identifier

2012 Clinical Trials

131. Multiple Transplantation of Bone Marrow Derived CD133 Cell in Cerebral Palsy

Exclusion Criteria: Children who have a diagnosis of "mixed" types of CP (i.e. athetosis) or other movement disorders (i.e. ataxia) Children who have had a selective dorsal rhizotomy, are presently are receiving intrathecal Baclofen, or have changed their spasticity medications in the past 6 months. Children who have a metallic or electrical implants Contacts and Locations Go to Information from the National Library of Medicine To learn more about this study, you or your doctor may contact the study

2012 Clinical Trials

132. PROGRESSIVE FAMILIAL CHOREOATHETOSIS WITH CUTANEOUS TELANGIECTASIA Full Text available with Trip Pro

PROGRESSIVE FAMILIAL CHOREOATHETOSIS WITH CUTANEOUS TELANGIECTASIA 13429376 2002 05 01 2018 12 01 0022-3050 20 2 1957 May Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Progressive familial choreoathetosis with cutaneous telangiectasia. 98-104 WELLS C E CE SHY G M GM eng Case Reports Journal Article England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 OM Athetosis Chorea Humans Medical Records Telangiectasis 5732:27556 ATHETOSIS/case reports CHOREA/case

1957 Journal of neurology, neurosurgery, and psychiatry

133. Familial hyperargininaemia. Full Text available with Trip Pro

to parental attitude, a low protein diet (1-5 g/kg) was introduced only late. The infant developed severe mental retardation, athetosis, and spasticity.

1975 Archives of Disease in Childhood

134. Studies of skin fibroblasts from 10 families with HGPRT deficiency, with reference in X-chromosomal inactivation. Full Text available with Trip Pro

Studies of skin fibroblasts from 10 families with HGPRT deficiency, with reference in X-chromosomal inactivation. 5092480 1971 08 23 2018 11 13 0002-9297 23 2 1971 Mar American journal of human genetics Am. J. Hum. Genet. Studies of skin fibroblasts from 10 families with HGPRT deficiency, with reference in X-chromosomal inactivation. 199-210 Migeon B R BR eng Journal Article United States Am J Hum Genet 0370475 0002-9297 0 Culture Media 0 Hypoxanthines 10028-17-8 Tritium AIM IM Athetosis

1971 American Journal of Human Genetics

135. Diseases of the central nervous system. Involuntary movements. Full Text available with Trip Pro

Diseases of the central nervous system. Involuntary movements. 1203674 1976 03 18 2013 11 21 0007-1447 4 5996 1975 Dec 06 British medical journal Br Med J Diseases of the central nervous system. Involuntary movements. 572-4 Mawdsley C C eng Journal Article England Br Med J 0372673 0007-1447 46627O600J Levodopa AIM IM Athetosis etiology therapy Central Nervous System Diseases complications Chorea etiology therapy Face Humans Levodopa therapeutic use Movement Disorders etiology therapy Myoclonus

1975 British medical journal

136. Long-term prognosis of severe accidental head injury. Full Text available with Trip Pro

Long-term prognosis of severe accidental head injury. 817289 1976 07 06 2018 11 13 0035-9157 69 2 1976 Feb Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Long-term prognosis of severe accidental head injury. 137-40 Roberts A H AH eng Journal Article England Proc R Soc Med 7505890 0035-9157 IM Accidents Adolescent Adult Amnesia complications Athetosis diagnosis Brain Injuries diagnosis Brain Stem Coma complications Decerebrate State diagnosis Disability Evaluation Epilepsy, Post

1976 Proceedings of the Royal Society of Medicine

137. Hughlings Jackson Lecture: Hughlings Jackson's american contemporaries: the birth of American Neurology. Full Text available with Trip Pro

Hughlings Jackson Lecture: Hughlings Jackson's american contemporaries: the birth of American Neurology. 785475 1976 10 20 2018 11 13 0035-9157 69 6 1976 Jun Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Hughlings Jackson Lecture: Hughlings Jackson's american contemporaries: the birth of American Neurology. 393-408 Spillane J D JD eng Biography Historical Article Journal Article Review England Proc R Soc Med 7505890 0035-9157 IM Q Athetosis history History, 19th Century Humans

1976 Proceedings of the Royal Society of Medicine

138. Athetoid movements in cervical spondylosis. Full Text available with Trip Pro

Athetoid movements in cervical spondylosis. 5427424 1970 08 26 2008 11 20 0003-4967 29 2 1970 Mar Annals of the rheumatic diseases Ann. Rheum. Dis. Athetoid movements in cervical spondylosis. 195 Bickerstaff E R ER eng Journal Article England Ann Rheum Dis 0372355 0003-4967 IM Arm innervation Athetosis complications Cervical Vertebrae Humans Spinal Diseases complications 1970 3 1 1970 3 1 0 1 1970 3 1 0 0 ppublish 5427424 PMC1010541

1970 Annals of the Rheumatic Diseases

139. Measurement of involuntary arm movement in athetotic patients Full Text available with Trip Pro

spectrum which decreases with increasing frequency, reaching a negligible value between 2-3 Hz. The second component is a rhythmical low frequency movement which is indicated by a predominant peak in the power spectrum at a frequency of 0·3-0·6 Hz. The presence of this peak was predicted in a previous paper because of underdamping demonstrated in the closed loop voluntary control of movement in athetosis. The third component is the athetoid action tremor in which both agonist and antagonist muscle

1974 Journal of neurology, neurosurgery, and psychiatry

140. Letter: Toxic reaction to phenytoin. Full Text available with Trip Pro

Letter: Toxic reaction to phenytoin. 4846140 1974 10 11 2013 11 21 0007-1447 3 5925 1974 Jul 27 British medical journal Br Med J Letter: Toxic reaction to phenytoin. 256-7 Bellman M H MH Haas L L eng Journal Article England Br Med J 0372673 0007-1447 6158TKW0C5 Phenytoin AIM IM Athetosis chemically induced Child Chorea chemically induced Female Humans Infant Phenytoin adverse effects therapeutic use Seizures drug therapy 1974 7 27 1974 7 27 0 1 1974 7 27 0 0 ppublish 4846140 PMC1612009

1974 British medical journal

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