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Athetosis

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121. Head drop in Huntington disease: Insights into the pathophysiology. (PubMed)

Head drop in Huntington disease: Insights into the pathophysiology. 23884035 2013 10 24 2013 08 20 1526-632X 81 8 2013 Aug 20 Neurology Neurology Head drop in Huntington disease: insights into the pathophysiology. 769-70 10.1212/WNL.0b013e3182a1aa55 Morgante Francesca F Dipartimento di Neuroscienze, Università di Messina, Italy. fmorgante@gmail.com Girlanda Paolo P Martino Davide D eng Case Reports Journal Article 2013 07 24 United States Neurology 0401060 0028-3878 AIM IM Athetosis diagnosis

2013 Neurology

122. PRRT2 mutation correlated with phenotype of paroxysmal kinesigenic dyskinesia and drug response. (PubMed)

, Fudan University, Shanghai, China. Chen Wan-Jin WJ Ni Wang W Wang Kai-Yan KY Liu Gong-Lu GL Wang Ning N Xiong Zhi-Qi ZQ Xu Jianfeng J Wu Zhi-Ying ZY eng Journal Article Research Support, Non-U.S. Gov't 2013 03 27 United States Neurology 0401060 0028-3878 0 Anticonvulsants 0 Membrane Proteins 0 Nerve Tissue Proteins 0 PRRT2 protein, human 33CM23913M Carbamazepine AIM IM Age of Onset Anticonvulsants therapeutic use Athetosis etiology genetics Carbamazepine therapeutic use Child China Disease

2013 Neurology

123. Movement disorders in cerebrovascular disease. (PubMed)

including chorea, ballism, athetosis, dystonia, tremor, myoclonus, stereotypies, and akathisia. Some of these disorders occur immediately after acute stroke, whereas others can develop later, and yet others represent delayed-onset progressive movement disorders. These movement disorders have been encountered in patients with ischaemic and haemorrhagic strokes, subarachnoid haemorrhage, cerebrovascular malformations, and dural arteriovenous fistula affecting the basal ganglia, their connections

2013 Lancet Neurology

124. The spectrum of movement disorders in children with anti-NMDA receptor encephalitis. (PubMed)

, including chorea (n=4), stereotypic movements (n=4), ataxia (n=3), limb dystonia (n=2), limb myorhythmia (n=2), oromandibular dystonia (n=2), facial myorhythmia, blepharospasm, opisthotonus, athetosis, and tremor (n=1, each). More than a single movement disorder was observed in 6 of these patients. Resolution of the abnormal movements was observed in all patients with immunotherapy; 1 patient improved with tetrabenazine.A wide variety of movement disorders, often in combination, can be observed

2013 Movement Disorders

125. Further evidence of validity of the Modified Melbourne Assessment for neurologically impaired children aged 2 to 4 years. (PubMed)

behaviours for task and time demands on both tools, and (3) scores on the MMA could discriminate between children with mild, moderate, and severe levels of upper limb impairment.  An observational study of 30 children (19 males, 11 females) with neurological impairment aged 2 to 4 years. Twenty-four children had spasticity (20 with a unilateral and four with a bilateral impairment) and two children presented with athetosis, two with ataxia, and two with hypotonia.A high, positive relation was found

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2012 Developmental Medicine and Child Neurology

126. Brainstem cavernous malformations: the role of Gamma Knife surgery. (PubMed)

patients experienced at least 2 instances of repeated bleeding before GKS; these hemorrhages caused neurological deficits including cranial nerve deficits, hemiparesis, hemisensory deficits, spasticity, chorea or athetosis, and consciousness disturbance.The mean size of the CMs at the time of GKS was 3.2 cm(3) (range 0.1-14.6 cm(3)). The mean radiation dose directed to the lesion was 11 Gy with an isodose level at 60.0%. The mean clinical and imaging follow-up time was 40.6 months (range 1.0-150.7

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2012 Journal of Neurosurgery

127. Infantile Cerebellar-Retinal Degeneration Associated with a Mutation in Mitochondrial Aconitase, ACO2. (PubMed)

Infantile Cerebellar-Retinal Degeneration Associated with a Mutation in Mitochondrial Aconitase, ACO2. Degeneration of the cerebrum, cerebellum, and retina in infancy is part of the clinical spectrum of lysosomal storage disorders, mitochondrial respiratory chain defects, carbohydrate glycosylation defects, and infantile neuroaxonal dystrophy. We studied eight individuals from two unrelated families who presented at 2-6 months of age with truncal hypotonia and athetosis, seizure disorder

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2012 American Journal of Human Genetics

128. Exploring Hypertonia in Children With Cerebral Palsy

it is often overlooked in the diagnostic formulation of motor aspects of cerebral palsy, and therefore does not necessarily figure in treatment decision-making. Under-recognised dystonia, when co-existent with spasticity, can produce unpredictable surgical outcomes in the management of gait disorders and associated musculoskeletal deformities. In addition, other abnormal movements such as chorea and athetosis may be observed in these children, adding to the complexity of the movement disorder (...) (dystonia or athetosis) and 3.6% as ataxic.1 However this study also found that when children were assessed face-to-face by a group of expert paediatricians, 19.4% of the population was noted on observation alone to have abnormal movements, which included dyskinesias. This increased with motor severity by Gross Motor Functional Classification System (GMFCS) from 7% (level I) to 45% (level V). We questioned whether in fact recognition of some dyskinesias are "masked" by the presence of spasticity

2012 Clinical Trials

129. Multiple Transplantation of Bone Marrow Derived CD133 Cell in Cerebral Palsy

Exclusion Criteria: Children who have a diagnosis of "mixed" types of CP (i.e. athetosis) or other movement disorders (i.e. ataxia) Children who have had a selective dorsal rhizotomy, are presently are receiving intrathecal Baclofen, or have changed their spasticity medications in the past 6 months. Children who have a metallic or electrical implants Contacts and Locations Go to Information from the National Library of Medicine To learn more about this study, you or your doctor may contact the study

2012 Clinical Trials

130. A Trial Comparing Two Methods of CIMT in the Hemiplegic Child

of bilateral upper limb involvement. Diagnosis/ clinical evidence of athetosis or dystonia. Any medical condition which would cause problems with the cast e.g. chronic eczema. An episode of >2 weeks of prolonged CIMT using a cast /splint in the previous 6 months. Contacts and Locations Go to No Contacts or Locations Provided More Information Go to Layout table for additonal information Responsible Party: Pauline Christmas, Consultant Physiotherapist, University of Birmingham ClinicalTrials.gov Identifier

2012 Clinical Trials

131. Kernicterus

of the BASAL GANGLIA; BRAIN STEM; and CEREBELLUM and clinically to describe a syndrome associated with HYPERBILIRUBINEMIA. Clinical features include athetosis, MUSCLE SPASTICITY or hypotonia, impaired vertical gaze, and DEAFNESS. Nonconjugated bilirubin enters the brain and acts as a neurotoxin, often in association with conditions that impair the BLOOD-BRAIN BARRIER (e.g., SEPSIS). This condition occurs primarily in neonates (INFANT, NEWBORN), but may rarely occur in adults. (Menkes, Textbook of Child (...) Neurology, 5th ed, p613) Definition (CSP) brain disorder associated with high levels of bilrubin; clinical features include athetosis, muscle spasticity or hypotonia, impaired vertical gaze, and deafness; nonconjugated bilirubin enters the brain and acts as a neurotoxin, often in association with conditions that impair the blood-brain barrier; this condition occurs primarily in neonates with perinatal blood incompatibilities, but may rarely occur in adults. Concepts Disease or Syndrome ( T047 ) MSH

2015 FP Notebook

132. Huntington's Chorea

dominant trait and characterized by the onset of progressive CHOREA and DEMENTIA in the fourth or fifth decade of life. Common initial manifestations include paranoia; poor impulse control; DEPRESSION; HALLUCINATIONS; and DELUSIONS. Eventually intellectual impairment; loss of fine motor control; ATHETOSIS; and diffuse chorea involving axial and limb musculature develops, leading to a vegetative state within 10-15 years of disease onset. The juvenile variant has a more fulminant course including

2015 FP Notebook

133. Choreoathetosis after subarachnoid hemorrhage related to an aneurysm of the posterior fossa (PubMed)

Gerival G Jr Christo Paulo Pereira PP de Carvalho Gervásio Teles C GT eng Case Reports Journal Article Brazil Clinics (Sao Paulo) 101244734 1807-5932 IM Adolescent Athetosis etiology Chorea etiology Cranial Fossa, Posterior Humans Intracranial Aneurysm complications Male Subarachnoid Hemorrhage complications 2011 12 20 6 0 2011 12 20 6 0 2012 8 22 6 0 ppublish 22179176 S1807-59322011000900026 PMC3164421 J Neurol Neurosurg Psychiatry. 2004 Nov;75(11):1568-74 15489389 No To Shinkei. 1991 Sep;43(9):875

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2011 Clinics

134. Side Effects of Bone Marrow Derived CD133 Transplantation in Cerebral Palsy

Criteria: Children who have a diagnosis of "mixed" types of CP (i.e. athetosis) or other movement disorders (i.e. ataxia) Children who have had a selective dorsal rhizotomy, are presently are receiving intrathecal Baclofen, or have changed their spasticity medications in the past 6 months. Children who have a metallic or electrical implants Contacts and Locations Go to Information from the National Library of Medicine To learn more about this study, you or your doctor may contact the study research

2011 Clinical Trials

135. Phase III Study of 5LGr to Treat Tic Disorder

for Study: 5 Years to 18 Years (Child, Adult) Sexes Eligible for Study: All Accepts Healthy Volunteers: No Criteria Inclusion Criteria: >1 year history of diagnosed tic disorder; age:5-18 yrs; YGTSS score>=30 at baseline. Exclusion Criteria: Diagnosed with hyperactivity disorder,epilepsy,chorea,autism,obsessive-compulsive disorder,mental retardation,athetosis,Wilson's disease. Any indefinite tic disorder or tic disorders caused by medication. Participation to other studies. Patients with loose stool

2011 Clinical Trials

136. Long-term prognosis of severe accidental head injury. (PubMed)

Long-term prognosis of severe accidental head injury. 817289 1976 07 06 2018 11 13 0035-9157 69 2 1976 Feb Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Long-term prognosis of severe accidental head injury. 137-40 Roberts A H AH eng Journal Article England Proc R Soc Med 7505890 0035-9157 IM Accidents Adolescent Adult Amnesia complications Athetosis diagnosis Brain Injuries diagnosis Brain Stem Coma complications Decerebrate State diagnosis Disability Evaluation Epilepsy, Post

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1976 Proceedings of the Royal Society of Medicine

137. Diseases of the central nervous system. Involuntary movements. (PubMed)

Diseases of the central nervous system. Involuntary movements. 1203674 1976 03 18 2013 11 21 0007-1447 4 5996 1975 Dec 06 British medical journal Br Med J Diseases of the central nervous system. Involuntary movements. 572-4 Mawdsley C C eng Journal Article England Br Med J 0372673 0007-1447 46627O600J Levodopa AIM IM Athetosis etiology therapy Central Nervous System Diseases complications Chorea etiology therapy Face Humans Levodopa therapeutic use Movement Disorders etiology therapy Myoclonus

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1975 British medical journal

138. Hughlings Jackson Lecture: Hughlings Jackson's american contemporaries: the birth of American Neurology. (PubMed)

Hughlings Jackson Lecture: Hughlings Jackson's american contemporaries: the birth of American Neurology. 785475 1976 10 20 2018 11 13 0035-9157 69 6 1976 Jun Proceedings of the Royal Society of Medicine Proc. R. Soc. Med. Hughlings Jackson Lecture: Hughlings Jackson's american contemporaries: the birth of American Neurology. 393-408 Spillane J D JD eng Biography Historical Article Journal Article Review England Proc R Soc Med 7505890 0035-9157 IM Q Athetosis history History, 19th Century Humans

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1976 Proceedings of the Royal Society of Medicine

139. The Effects of Azathioprine (Imuran) on Purine Synthesis in Clinical Disorders of Purine Metabolism (PubMed)

to feedback inhibitors in the patients who responded to the drug.Two children afflicted with the familial disorder of hyperuricemia, choreo-athetosis, and self-mutilation described by Lesch and Nyhan showed no reduction in the activity of the biosynthetic pathway in response to azathioprine. This inability to respond to azathioprine can be directly related to the absence in these patients of the enzyme hypoxanthine-guanine phosphoribosyltransferase which is required for conversion of the drug or its

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1967 Journal of Clinical Investigation

140. Difficulty in swallowing, with aspiration pneumonia in infancy. (PubMed)

Difficulty in swallowing, with aspiration pneumonia in infancy. 6025372 1967 07 22 2018 11 13 1468-2044 42 223 1967 Jun Archives of disease in childhood Arch. Dis. Child. Difficulty in swallowing, with aspiration pneumonia in infancy. 308-10 Matsaniotis N N Karpouzas J J Gregoriou M M eng Journal Article England Arch Dis Child 0372434 0003-9888 IM Athetosis complications Cerebral Palsy complications Deglutition Disorders complications etiology Dysautonomia, Familial complications genetics

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1967 Archives of Disease in Childhood

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