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Athetosis

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121. PRRT2 mutation correlated with phenotype of paroxysmal kinesigenic dyskinesia and drug response. (PubMed)

, Fudan University, Shanghai, China. Chen Wan-Jin WJ Ni Wang W Wang Kai-Yan KY Liu Gong-Lu GL Wang Ning N Xiong Zhi-Qi ZQ Xu Jianfeng J Wu Zhi-Ying ZY eng Journal Article Research Support, Non-U.S. Gov't 2013 03 27 United States Neurology 0401060 0028-3878 0 Anticonvulsants 0 Membrane Proteins 0 Nerve Tissue Proteins 0 PRRT2 protein, human 33CM23913M Carbamazepine AIM IM Age of Onset Anticonvulsants therapeutic use Athetosis etiology genetics Carbamazepine therapeutic use Child China Disease

2013 Neurology

122. Movement disorders in cerebrovascular disease. (PubMed)

including chorea, ballism, athetosis, dystonia, tremor, myoclonus, stereotypies, and akathisia. Some of these disorders occur immediately after acute stroke, whereas others can develop later, and yet others represent delayed-onset progressive movement disorders. These movement disorders have been encountered in patients with ischaemic and haemorrhagic strokes, subarachnoid haemorrhage, cerebrovascular malformations, and dural arteriovenous fistula affecting the basal ganglia, their connections

2013 Lancet Neurology

123. The spectrum of movement disorders in children with anti-NMDA receptor encephalitis. (PubMed)

, including chorea (n=4), stereotypic movements (n=4), ataxia (n=3), limb dystonia (n=2), limb myorhythmia (n=2), oromandibular dystonia (n=2), facial myorhythmia, blepharospasm, opisthotonus, athetosis, and tremor (n=1, each). More than a single movement disorder was observed in 6 of these patients. Resolution of the abnormal movements was observed in all patients with immunotherapy; 1 patient improved with tetrabenazine.A wide variety of movement disorders, often in combination, can be observed

2013 Movement Disorders

124. Disorders of bilirubin binding to albumin and bilirubin-induced neurologic dysfunction. (PubMed)

and affinity of albumin, and the presence of displacing substances or infection. Serum unbound bilirubin (UB) concentration may be an ideal marker that reflects changes in bilirubin binding to albumin. Kernicterus, the chronic and with the most severe manifestations beyond BIND, is diagnosed by the presence of motor impairments with athetosis, abnormal magnetic resonance imaging, and/or brainstem auditory-evoked potential findings during infancy and childhood. Preterm infants sometimes have acute bilirubin

2014 Seminars in fetal & neonatal medicine

125. Further evidence of validity of the Modified Melbourne Assessment for neurologically impaired children aged 2 to 4 years. (PubMed)

behaviours for task and time demands on both tools, and (3) scores on the MMA could discriminate between children with mild, moderate, and severe levels of upper limb impairment.  An observational study of 30 children (19 males, 11 females) with neurological impairment aged 2 to 4 years. Twenty-four children had spasticity (20 with a unilateral and four with a bilateral impairment) and two children presented with athetosis, two with ataxia, and two with hypotonia.A high, positive relation was found

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2012 Developmental Medicine and Child Neurology

126. Brainstem cavernous malformations: the role of Gamma Knife surgery. (PubMed)

patients experienced at least 2 instances of repeated bleeding before GKS; these hemorrhages caused neurological deficits including cranial nerve deficits, hemiparesis, hemisensory deficits, spasticity, chorea or athetosis, and consciousness disturbance.The mean size of the CMs at the time of GKS was 3.2 cm(3) (range 0.1-14.6 cm(3)). The mean radiation dose directed to the lesion was 11 Gy with an isodose level at 60.0%. The mean clinical and imaging follow-up time was 40.6 months (range 1.0-150.7

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2012 Journal of Neurosurgery

127. Infantile Cerebellar-Retinal Degeneration Associated with a Mutation in Mitochondrial Aconitase, ACO2. (PubMed)

Infantile Cerebellar-Retinal Degeneration Associated with a Mutation in Mitochondrial Aconitase, ACO2. Degeneration of the cerebrum, cerebellum, and retina in infancy is part of the clinical spectrum of lysosomal storage disorders, mitochondrial respiratory chain defects, carbohydrate glycosylation defects, and infantile neuroaxonal dystrophy. We studied eight individuals from two unrelated families who presented at 2-6 months of age with truncal hypotonia and athetosis, seizure disorder

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2012 American Journal of Human Genetics

128. Exploring Hypertonia in Children With Cerebral Palsy

it is often overlooked in the diagnostic formulation of motor aspects of cerebral palsy, and therefore does not necessarily figure in treatment decision-making. Under-recognised dystonia, when co-existent with spasticity, can produce unpredictable surgical outcomes in the management of gait disorders and associated musculoskeletal deformities. In addition, other abnormal movements such as chorea and athetosis may be observed in these children, adding to the complexity of the movement disorder (...) (dystonia or athetosis) and 3.6% as ataxic.1 However this study also found that when children were assessed face-to-face by a group of expert paediatricians, 19.4% of the population was noted on observation alone to have abnormal movements, which included dyskinesias. This increased with motor severity by Gross Motor Functional Classification System (GMFCS) from 7% (level I) to 45% (level V). We questioned whether in fact recognition of some dyskinesias are "masked" by the presence of spasticity

2012 Clinical Trials

129. Multiple Transplantation of Bone Marrow Derived CD133 Cell in Cerebral Palsy

Exclusion Criteria: Children who have a diagnosis of "mixed" types of CP (i.e. athetosis) or other movement disorders (i.e. ataxia) Children who have had a selective dorsal rhizotomy, are presently are receiving intrathecal Baclofen, or have changed their spasticity medications in the past 6 months. Children who have a metallic or electrical implants Contacts and Locations Go to Information from the National Library of Medicine To learn more about this study, you or your doctor may contact the study

2012 Clinical Trials

130. A Trial Comparing Two Methods of CIMT in the Hemiplegic Child

of bilateral upper limb involvement. Diagnosis/ clinical evidence of athetosis or dystonia. Any medical condition which would cause problems with the cast e.g. chronic eczema. An episode of >2 weeks of prolonged CIMT using a cast /splint in the previous 6 months. Contacts and Locations Go to No Contacts or Locations Provided More Information Go to Layout table for additonal information Responsible Party: Pauline Christmas, Consultant Physiotherapist, University of Birmingham ClinicalTrials.gov Identifier

2012 Clinical Trials

131. Huntington's Chorea

dominant trait and characterized by the onset of progressive CHOREA and DEMENTIA in the fourth or fifth decade of life. Common initial manifestations include paranoia; poor impulse control; DEPRESSION; HALLUCINATIONS; and DELUSIONS. Eventually intellectual impairment; loss of fine motor control; ATHETOSIS; and diffuse chorea involving axial and limb musculature develops, leading to a vegetative state within 10-15 years of disease onset. The juvenile variant has a more fulminant course including

2015 FP Notebook

132. Kernicterus

of the BASAL GANGLIA; BRAIN STEM; and CEREBELLUM and clinically to describe a syndrome associated with HYPERBILIRUBINEMIA. Clinical features include athetosis, MUSCLE SPASTICITY or hypotonia, impaired vertical gaze, and DEAFNESS. Nonconjugated bilirubin enters the brain and acts as a neurotoxin, often in association with conditions that impair the BLOOD-BRAIN BARRIER (e.g., SEPSIS). This condition occurs primarily in neonates (INFANT, NEWBORN), but may rarely occur in adults. (Menkes, Textbook of Child (...) Neurology, 5th ed, p613) Definition (CSP) brain disorder associated with high levels of bilrubin; clinical features include athetosis, muscle spasticity or hypotonia, impaired vertical gaze, and deafness; nonconjugated bilirubin enters the brain and acts as a neurotoxin, often in association with conditions that impair the blood-brain barrier; this condition occurs primarily in neonates with perinatal blood incompatibilities, but may rarely occur in adults. Concepts Disease or Syndrome ( T047 ) MSH

2015 FP Notebook

133. Side Effects of Bone Marrow Derived CD133 Transplantation in Cerebral Palsy

Criteria: Children who have a diagnosis of "mixed" types of CP (i.e. athetosis) or other movement disorders (i.e. ataxia) Children who have had a selective dorsal rhizotomy, are presently are receiving intrathecal Baclofen, or have changed their spasticity medications in the past 6 months. Children who have a metallic or electrical implants Contacts and Locations Go to Information from the National Library of Medicine To learn more about this study, you or your doctor may contact the study research

2011 Clinical Trials

134. Phase III Study of 5LGr to Treat Tic Disorder

for Study: 5 Years to 18 Years (Child, Adult) Sexes Eligible for Study: All Accepts Healthy Volunteers: No Criteria Inclusion Criteria: >1 year history of diagnosed tic disorder; age:5-18 yrs; YGTSS score>=30 at baseline. Exclusion Criteria: Diagnosed with hyperactivity disorder,epilepsy,chorea,autism,obsessive-compulsive disorder,mental retardation,athetosis,Wilson's disease. Any indefinite tic disorder or tic disorders caused by medication. Participation to other studies. Patients with loose stool

2011 Clinical Trials

135. The Effects of Azathioprine (Imuran) on Purine Synthesis in Clinical Disorders of Purine Metabolism (PubMed)

to feedback inhibitors in the patients who responded to the drug.Two children afflicted with the familial disorder of hyperuricemia, choreo-athetosis, and self-mutilation described by Lesch and Nyhan showed no reduction in the activity of the biosynthetic pathway in response to azathioprine. This inability to respond to azathioprine can be directly related to the absence in these patients of the enzyme hypoxanthine-guanine phosphoribosyltransferase which is required for conversion of the drug or its

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1967 Journal of Clinical Investigation

136. Treatment of involuntary movement disorders with tetrabenazine. (PubMed)

had Huntington's chorea, two athetosis, two dystonia musculorum deformans, one hemiballismus, and one spasmodic torticollis. Four of the eight improved patients have continued taking the drug for longer than six months. In a second study seven patients with Huntington's chorea were treated for two weeks each with tetrabenazine (50 mg t.d.s.) and with amantadine (100 mg t.d.s.) and the results assessed by the same method. The choreiform movements of six of these patients were strikingly improved (...) with tetrabenazine therapy, but amantadine had no effect. Tetrabenazine is an effective agent for the suppression of choreiform and ballistic involuntary movements. It is only slightly effective in the treatment of athetosis and spasmodic torticollis. Drowsiness, insomnia, and depression were the most conspicuous unwanted effects, and these may limit the clinical usefulness of the drug.

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1972 Journal of neurology, neurosurgery, and psychiatry

137. Some Clinical Notes and Electroencephalographic Findings in Cerebral Palsy (PubMed)

Some Clinical Notes and Electroencephalographic Findings in Cerebral Palsy 14329256 1996 12 01 2018 12 01 0003-9888 40 1965 Aug Archives of disease in childhood Arch. Dis. Child. SOME CLINICAL NOTES AND ELECTROENCEPHALOGRAPHIC FINDINGS IN CEREBRAL PALSY. 394-401 WOODS G E GE eng Journal Article England Arch Dis Child 0372434 0003-9888 OM Adolescent Ataxia Athetosis Cerebral Palsy Child Electroencephalography Epilepsy Hemiplegia Humans Movement Disorders Muscle Spasticity Paralysis ADOLESCENCE (...) ATAXIA ATHETOSIS CEREBRAL PALSY CHILD ELECTROENCEPHALOGRAPHY EPILEPSY HEMIPLEGIA MOVEMENT DISORDERS PARALYSIS, SPASTIC 1965 8 1 1965 8 1 0 1 1965 8 1 0 0 ppublish 14329256 PMC2019295 J Pediatr. 1950 Oct;37(4):448-54 14779244 Dev Med Child Neurol. 1963 Feb;5:24-31 13957172 Dev Med Child Neurol. 1962 Apr;4:180-3 14472365

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1965 Archives of Disease in Childhood

138. A community program for cerebral palsied children. (PubMed)

A community program for cerebral palsied children. 5954490 1967 03 04 2008 11 20 0008-4409 95 24 1966 Dec 10 Canadian Medical Association journal Can Med Assoc J A community program for cerebral palsied children. 1245-6 Hawke W A WA eng Journal Article Canada Can Med Assoc J 0414110 0008-4409 AIM IM Athetosis therapy Cerebral Palsy rehabilitation Child Child, Preschool Community Health Services Education Hemiplegia therapy Humans Paraplegia therapy 1966 12 10 1966 12 10 0 1 1966 12 10 0 0

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1966 Canadian Medical Association Journal

139. A survey of cerebral palsy in Windsor and Essex County, Ontario. (PubMed)

A survey of cerebral palsy in Windsor and Essex County, Ontario. 5954487 1967 03 04 2008 11 20 0008-4409 95 24 1966 Dec 10 Canadian Medical Association journal Can Med Assoc J A survey of cerebral palsy in Windsor and Essex County, Ontario. 1237-40 McGreal D A DA eng Journal Article Canada Can Med Assoc J 0414110 0008-4409 AIM IM Adolescent Ataxia Athetosis Cerebral Palsy diagnosis epidemiology Child Child, Preschool Education Emotions Female Health Surveys Hemiplegia Humans Male Ontario

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1966 Canadian Medical Association Journal

140. Involuntary movements following stereotactic operations for Parkinsonism with special reference to hemi-chorea (ballismus) (PubMed)

Involuntary movements following stereotactic operations for Parkinsonism with special reference to hemi-chorea (ballismus) 14338118 1996 12 01 2018 12 01 0022-3050 28 1965 Aug Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry INVOLUNTARY MOVEMENTS FOLLOWING STEREOTACTIC OPERATIONS FOR PARKINSONISM WITH SPECIAL REFERENCE TO HEMI-CHOREA (BALLISMUS). 291-303 HUGHES B B eng Journal Article England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 OM Athetosis Chorea (...) Classification Dyskinesias Eyelids Humans Middle Aged Movement Disorders Parkinsonian Disorders Statistics as Topic Stereotaxic Techniques Thalamus Tremor ATHETOSIS CHOREA CLASSIFICATION EYELIDS MIDDLE AGE MOVEMENT DISORDERS PARKINSONISM STATISTICS STEREOTAXIC TECHNICS THALAMUS TREMOR 1965 8 1 1965 8 1 0 1 1965 8 1 0 0 ppublish 14338118 PMC495908 J Neurosurg. 1954 Jul;11(4):409-12 13184348 Brain. 1959 Mar;82(1):104-8 13651497 Neurology. 1958 Oct;8(10):734-40 13590379 J Neurosurg. 1963 Oct;20:860-70 14186081

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1965 Journal of neurology, neurosurgery, and psychiatry

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