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221. Double-blind trial of carisoprodol. (PubMed)

Double-blind trial of carisoprodol. 14001807 1998 11 01 2018 12 01 0012-1622 4 1962 Oct Developmental medicine and child neurology Dev Med Child Neurol Double-blind trial of carisoprodol. 499-506 WOODS G E GE eng Journal Article England Dev Med Child Neurol 0006761 0012-1622 21925K482H Carisoprodol OM Athetosis Carisoprodol Cerebral Palsy Double-Blind Method Humans ATHETOSIS CARISOPRODOL CEREBRAL PALSY 1962 10 1 1962 10 1 0 1 1962 10 1 0 0 ppublish 14001807

1998 Developmental Medicine and Child Neurology Controlled trial quality: predicted high

222. Seating orientations and upper extremity function in children with cerebral palsy. (PubMed)

seating position, the subject performed an upper extremity activity on cue. The tests were repeated in reverse sequence. Mean performance times were different at all seating orientations for both types of subjects. Performance time was lowest at the 0-degree orientation during the retest for the subjects with spasticity and highest at 15-degree anterior inclination during the retest for the subjects with athetosis. The results of this study show that orientation of the body in space affects upper

1987 Physical therapy Controlled trial quality: uncertain

223. Transient athetosis induced by tiagabine. (PubMed)

Transient athetosis induced by tiagabine. Movement disorders have been reported with use of different antiepileptic drugs (AEDs). We report a 32-year-old woman, affected by a symptomatic focal drug-resistant epilepsy and a mild hemiparesis, with acute athetoid movements, transiently linked to increasing tiagabine (TGB) therapy. To our knowledge, no other cases of acute athetosis related to TGB have been reported to date. However, we cannot rule out the possibility that involuntary movements (...) were induced by an interaction between TGB and concomitant AEDs, in particular phenobarbital (PB), possibly by increasing GABAergic transmission. We hypothesize that the presence of a static encephalopathy may have influenced the kind of extrapyramidal side effect induced by TGB in our patient, leading to athetosis.

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2006 Epilepsia

224. Progressive choreo-athetosis related to birth anoxia. (PubMed)

Progressive choreo-athetosis related to birth anoxia. 7198140 1982 02 25 2018 11 13 0022-3050 44 10 1981 Oct Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Progressive choreo-athetosis related to birth anoxia. 957 Montagna P P Cirignotta F F Gallassi R R Sacquegna T T eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Athetosis etiology Chorea etiology Female Fetal Hypoxia complications Humans Infant, Newborn Pregnancy 1981

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1981 Journal of neurology, neurosurgery, and psychiatry

225. Athetosis II: the syndrome of mild athetoid cerebral palsy. (PubMed)

Athetosis II: the syndrome of mild athetoid cerebral palsy. We describe 8 patients who presented with continuous, irregular movements occurring independently in individual fingers and, in some cases, toes, in the setting of mild dystonia present since early childhood and not associated with major disability. The finger movements varied from low-amplitude quivering or wriggling to larger amplitude movements in the plane of abduction-adduction as well as flexion-extension; they were asymmetrical (...) but not unilateral. Quivering or working of the facial muscles was seen in 5 patients. Most patients reported worsening of the movements over the years, but there was no other evidence of a progressive neurological disease. We classify the movement disorder as athetosis as described by Hammond and Shaw and the syndrome as mild athetoid cerebral palsy.Copyright 2002 Movement Disorder Society

2002 Movement Disorders

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