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Athetosis

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201. Pelizaeus-Merzbacher Disease

of learning difficulty than is present. These patients may survive to the sixth decade of life or longer. SPG2-like disease - there is spastic paraplegia from childhood, mild , ataxia and athetosis (continual slow movements, especially of the extremities). They usually live to the sixth decade or beyond. Neurological signs progress gradually with periods of relative stability. If they learn to walk they tend to lose it during adolescence (occasionally adulthood). Other general observations may be made

2008 Mentor

202. Double-blind trial of carisoprodol. (Abstract)

Double-blind trial of carisoprodol. 14001807 1998 11 01 2018 12 01 0012-1622 4 1962 Oct Developmental medicine and child neurology Dev Med Child Neurol Double-blind trial of carisoprodol. 499-506 WOODS G E GE eng Journal Article England Dev Med Child Neurol 0006761 0012-1622 21925K482H Carisoprodol OM Athetosis Carisoprodol Cerebral Palsy Double-Blind Method Humans ATHETOSIS CARISOPRODOL CEREBRAL PALSY 1962 10 1 1962 10 1 0 1 1962 10 1 0 0 ppublish 14001807

1998 Developmental Medicine and Child Neurology Controlled trial quality: predicted high

203. (+)-PHNO: a new anti-parkinsonian agent which does not induce chorea in MPTP-treated squirrel monkeys. Full Text available with Trip Pro

Dopamine Agents 0 Oxazines 22Z7E0X6OF naxagolide 46627O600J Levodopa 9P21XSP91P 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine IM 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine Administration, Cutaneous Animals Antiparkinson Agents Athetosis chemically induced Chorea chemically induced Dopamine Agents Levodopa toxicity Oxazines toxicity Saimiri 1990 7 1 1990 7 1 0 1 1990 7 1 0 0 ppublish 1975267 PMC488146 Clin Pharmacol Ther. 1971 Mar-Apr;12(2):340-3 5577482 Lancet. 1972 Nov 4;2(7784):981-2 4116875 Arch

1990 Journal of neurology, neurosurgery, and psychiatry

204. Paroxysmal choreoathetosis as presenting symptom of diabetes mellitus. Full Text available with Trip Pro

Paroxysmal choreoathetosis as presenting symptom of diabetes mellitus. 2709027 1989 05 26 2018 11 13 0022-3050 52 1 1989 Jan Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Paroxysmal choreoathetosis as presenting symptom of diabetes mellitus. 133 Haan J J Kremer H P HP Padberg G W GW eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Aged Aged, 80 and over Athetosis etiology Chorea etiology Diabetes Mellitus, Type 2

1989 Journal of neurology, neurosurgery, and psychiatry

205. Carbamazepine-induced choreoathetoid dyskinesias. Full Text available with Trip Pro

Carbamazepine-induced choreoathetoid dyskinesias. 6811702 1982 12 02 2018 11 13 0022-3050 45 6 1982 Jun Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Carbamazepine-induced choreoathetoid dyskinesias. 560 Bimpong-Buta K K Froescher W W eng Case Reports Journal Article England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 33CM23913M Carbamazepine IM Aged Athetosis chemically induced Carbamazepine adverse effects therapeutic use Chorea chemically induced

1982 Journal of neurology, neurosurgery, and psychiatry

206. Self-regulation of spasm and spasticity in cerebral palsy. Full Text available with Trip Pro

Self-regulation of spasm and spasticity in cerebral palsy. Four young adult cerebral palsied subjects with a mixture of spasticity and athetosis attended an experimental reflex training program for three one-hour sessions each week over an 18 month period. During each session on-line measures of contraction level and tonic stretch reflex sensitivity from the biceps brachii muscle were shown to the subject on meter displays. Subjects were asked to attempt to control the displays. They were given

1982 Journal of neurology, neurosurgery, and psychiatry

207. Focal paroxysmal kinesigenic choreoathetosis. Full Text available with Trip Pro

Focal paroxysmal kinesigenic choreoathetosis. 6481380 1984 11 20 2018 11 13 0022-3050 47 9 1984 Sep Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Focal paroxysmal kinesigenic choreoathetosis. 1057-8 Lang A E AE eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Adult Athetosis diagnosis Chorea diagnosis Female Functional Laterality Humans Kinesthesis 1984 9 1 1984 9 1 0 1 1984 9 1 0 0 ppublish 6481380 PMC1028023 Arch Neurol

1984 Journal of neurology, neurosurgery, and psychiatry

208. Painful arm and moving fingers. Full Text available with Trip Pro

Painful arm and moving fingers. 3998744 1985 07 17 2018 11 13 0022-3050 48 4 1985 Apr Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Painful arm and moving fingers. 384-5 Verhagen W I WI Horstink M W MW Notermans S L SL eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Arm innervation Athetosis etiology Brachial Plexus radiation effects Female Fingers innervation Humans Middle Aged Polyneuropathies etiology Radiation

1985 Journal of neurology, neurosurgery, and psychiatry

209. Glue sniffing and movement disorder. Full Text available with Trip Pro

Glue sniffing and movement disorder. 6502186 1985 01 24 2018 11 13 0022-3050 47 11 1984 Nov Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Glue sniffing and movement disorder. 1259 Bartolucci G G Pellettier J R JR eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 3FPU23BG52 Toluene IM Adult Athetosis chemically induced Chorea chemically induced Humans Male Substance-Related Disorders complications Toluene poisoning 1984 11 1

1984 Journal of neurology, neurosurgery, and psychiatry

210. Focal paroxysmal kinesigenic choreoathetosis preceding the development of Steele-Richardson-Olszewski syndrome. Full Text available with Trip Pro

Focal paroxysmal kinesigenic choreoathetosis preceding the development of Steele-Richardson-Olszewski syndrome. 3746331 1986 10 09 2018 11 13 0022-3050 49 8 1986 Aug Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Focal paroxysmal kinesigenic choreoathetosis preceding the development of Steele-Richardson-Olszewski syndrome. 957-9 Adam A M AM Orinda D O DO eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Athetosis

1986 Journal of neurology, neurosurgery, and psychiatry

211. Central pontine myelinolysis causes bilateral loss of deep sensitivity and pseudochoreoathetosis Full Text available with Trip Pro

Central pontine myelinolysis causes bilateral loss of deep sensitivity and pseudochoreoathetosis 9771804 1998 11 10 2017 11 14 0022-3050 65 4 1998 Oct Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Central pontine myelinolysis causes bilateral loss of deep sensitivity and pseudochoreoathetosis. 607-8 Federlein J J Postert T T Przuntek H H Müller T T eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Adult Athetosis etiology

1998 Journal of neurology, neurosurgery, and psychiatry

212. A new neurological entity manifesting as involuntary movements and dysarthria with possible abnormal copper metabolism Full Text available with Trip Pro

, and urinary copper excretion. The onset of neurological symptoms occurred at the age of 15 to 17 years. The common clinical symptoms were involuntary movements and dysarthria. The involuntary movements included dystonia in the neck, myoclonus in the shoulder, athetosis in the neck, and rapid orobuccal movements. The dysarthria consisted of unclear, slow, and stuttering speech. Two of the three patients did not have dementia. A cousin of the female patient had been diagnosed as having Wilson's disease

2001 Journal of neurology, neurosurgery, and psychiatry

213. Use of lamotrigine to treat paroxysmal kinesigenic choreoathetosis Full Text available with Trip Pro

therapeutic use Athetosis drug therapy Chorea drug therapy Humans Kinesthesis drug effects Lamotrigine Male Triazines therapeutic use 2000 7 6 2000 7 6 0 1 2000 7 6 0 0 ppublish 10877627 PMC1736942

2000 Journal of neurology, neurosurgery, and psychiatry

214. Seating orientations and upper extremity function in children with cerebral palsy. (Abstract)

seating position, the subject performed an upper extremity activity on cue. The tests were repeated in reverse sequence. Mean performance times were different at all seating orientations for both types of subjects. Performance time was lowest at the 0-degree orientation during the retest for the subjects with spasticity and highest at 15-degree anterior inclination during the retest for the subjects with athetosis. The results of this study show that orientation of the body in space affects upper

1987 Physical therapy Controlled trial quality: uncertain

215. Phase II Randomized Study of Selective Dorsal Rhizotomy and Physiotherapy Vs Physiotherapy Alone for Spastic Diplegia

to walk or has potential to walk indoors with assistive devices No fixed musculoskeletal deformity greater than 15 degrees Prior orthopedic surgery acceptable if spastic muscle group is in physiologic biomechanical position and retains voluntary power No detectable athetosis, rigidity, ataxia, or other movement disorder No disease-related, treatable condition that may become urgent during study, e.g., hip subluxation requiring adductor release --Patient Characteristics-- Age: 3 to 18 Other

2000 Clinical Trials

216. Focal hand dystonia after cervical whiplash injury Full Text available with Trip Pro

Focal hand dystonia after cervical whiplash injury 12486287 2004 11 16 2017 11 14 0022-3050 74 1 2003 Jan Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Focal hand dystonia after cervical whiplash injury. 134 Tamburin S S Zanette G G eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Adult Athetosis diagnosis etiology Dystonia diagnosis etiology physiopathology Electrodiagnosis Evoked Potentials, Somatosensory Hand

2003 Journal of neurology, neurosurgery, and psychiatry

217. Normal CAG repeat variation at the DRPLA locus in world populations. Full Text available with Trip Pro

States Letter Research Support, U.S. Gov't, P.H.S. United States Am J Hum Genet 0370475 0002-9297 IM Alleles Athetosis genetics Cerebellar Ataxia genetics Chorea genetics Dementia genetics Epilepsies, Myoclonic genetics Genetics, Population Humans Minisatellite Repeats 1995 8 1 1995 8 1 0 1 1995 8 1 0 0 ppublish 7668279 PMC1801546 Nat Genet. 1994 Jan;6(1):9-13 8136840 Nat Genet. 1994 Jan;6(1):14-8 8136826 Hum Mol Genet. 1994 Jan;3(1):45-51 7909252 Nat Genet. 1994 Mar;6(3):225 8012381 Acta Neurol

1995 American Journal of Human Genetics

218. The Xq22 inversion breakpoint interrupted a novel Ras-like GTPase gene in a patient with Duchenne muscular dystrophy and profound mental retardation. Full Text available with Trip Pro

The Xq22 inversion breakpoint interrupted a novel Ras-like GTPase gene in a patient with Duchenne muscular dystrophy and profound mental retardation. A male patient with profound mental retardation, athetosis, nystagmus, and severe congenital hypotonia (Duchenne muscular dystrophy [DMD]) was previously shown to carry a pericentric inversion of the X chromosome, 46,Y,inv(X)(p21.2q22.2). His mother carried this inversion on one X allele. The patient's condition was originally misdiagnosed

2002 American Journal of Human Genetics

219. The MPTP-treated primate as a model of motor complications in PD: primate model of motor complications. (Abstract)

("on-off"). Immediately after each dose of L-dopa, animals show an initial worsening of motor performance ("beginning-of-dose deterioration") and, at the end of action of each dose, they exhibit a decline to levels of disability below those seen at baseline ("rebound worsening"). Dyskinesia (chorea, dystonia, athetosis) appears rapidly in MPTP-treated primates given L-dopa. The greater the degree of nigral denervation, the less exposure to L-dopa that is required to induce dyskinesia. The onset

2003 Neurology

220. Aromatic L-amino acid decarboxylase deficiency: overview of clinical features and outcomes. (Abstract)

, and includes intermittent oculogyric crises and limb dystonia, generalized athetosis, and impaired voluntary movement in all patients. Autonomic dysfunction is characterized by a significant impairment of sympathetic regulation of heart rate and blood pressure, as documented via detailed studies with spectral analysis techniques in two patients. Functional clinical outcomes as a group remain poor, in spite of a variety of attempted treatment interventions, with marked impairment in motor abilities as well

2003 Annals of Neurology

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