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Athetosis

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181. drug-induced movement disorders

drug-induced movement disorders drug-induced movement disorders - General Practice Notebook This site is intended for healthcare professionals General Practice Notebook | Medical search drug-induced movement disorders The following movement disorders may be caused by drugs: tremor parkinsoninism choreo-athetosis acute dystonia tardive dyskinesia ataxia Links: General Practice Notebook General Practice Notebook The information provided herein should not be used for diagnosis or treatment of any

2010 GP Notebook

182. drug induced movement disorders

drug induced movement disorders drug induced movement disorders - General Practice Notebook This site is intended for healthcare professionals General Practice Notebook | Medical search drug induced movement disorders The following movement disorders may be caused by drugs: tremor parkinsoninism choreo-athetosis acute dystonia tardive dyskinesia ataxia Links: General Practice Notebook General Practice Notebook The information provided herein should not be used for diagnosis or treatment of any

2010 GP Notebook

183. drugs causing movement disorders

drugs causing movement disorders drugs causing movement disorders - General Practice Notebook This site is intended for healthcare professionals General Practice Notebook | Medical search drugs causing movement disorders The following movement disorders may be caused by drugs: tremor parkinsoninism choreo-athetosis acute dystonia tardive dyskinesia ataxia Links: General Practice Notebook General Practice Notebook The information provided herein should not be used for diagnosis or treatment

2010 GP Notebook

184. Pelizaeus-Merzbacher Disease

of learning difficulty than is present. These patients may survive to the sixth decade of life or longer. SPG2-like disease - there is spastic paraplegia from childhood, mild , ataxia and athetosis (continual slow movements, especially of the extremities). They usually live to the sixth decade or beyond. Neurological signs progress gradually with periods of relative stability. If they learn to walk they tend to lose it during adolescence (occasionally adulthood). Other general observations may be made

2008 Mentor

185. Neurological Examination of the Lower Limbs

. Neuropathic ulcers and neuropathic joints develop because of a lack of pain perception. Pain is a protective mechanism. When inspecting the motor system, the following points should be assessed: Note the resting posture. Establish whether there is unusual rotation or posture of a joint. Note whether the patient is symmetrical. Look for muscle wasting or hypertrophy: note whether it is focal or diffuse. Look for involuntary movements such as tremor, tics, myoclonic jerks, chorea or athetosis. Look

2008 Mentor

186. Neurological Examination of the Upper Limbs

. Note whether it is focal or diffuse. Look for involuntary movements such as tremor, tics, myoclonic jerks, chorea or athetosis. Look for muscle fasciculation (sign of lower motor neurone disease process). These are subcutaneous twitches over a muscle belly at rest. Tapping the belly may stimulate fasciculation. [ ] Examination of the sensory system Examination of each of the sensory modalities: Light touch Use the light touch of a finger, a piece of cotton wool or a piece of tissue paper

2008 Mentor

187. Abnormal Involuntary Movements

and stereotypies may also be considered to be related but some experts call these 'unvoluntary' because there is an element of voluntary control. Cerebrovascular diseases are a common cause of secondary movement disorders. Post-stroke movement disorders include Parkinsonism and a wide range of hyperkinetic movement disorders, including chorea, ballism, athetosis, dystonia, tremor, myoclonus, stereotypies and akathisia. Classification Athetosis Sinuous, slow, involuntary writhing movements affecting the fingers (...) , thallium, mercury and manganese. Chorea may occur with athetosis and is then called choreoathetosis. Dystonias A dystonia is a sustained muscle contraction, frequently causing repetitive twisting movements or abnormal postures. [ , ] It is a dynamic condition that often changes in severity depending on the posture assumed and on voluntary activity of the area of the body involved. The diagnosis is clinical and there are no specific tests available; therefore, expert opinion should be sought. Dystonias

2008 Mentor

188. Chorea

be considered to be related but some experts call these 'unvoluntary' because there is an element of voluntary control. Cerebrovascular diseases are a common cause of secondary movement disorders. Post-stroke movement disorders include Parkinsonism and a wide range of hyperkinetic movement disorders, including chorea, ballism, athetosis, dystonia, tremor, myoclonus, stereotypies and akathisia. Classification Athetosis Sinuous, slow, involuntary writhing movements affecting the fingers, hands, toes and feet (...) . Chorea may occur with athetosis and is then called choreoathetosis. Dystonias A dystonia is a sustained muscle contraction, frequently causing repetitive twisting movements or abnormal postures. [ , ] It is a dynamic condition that often changes in severity depending on the posture assumed and on voluntary activity of the area of the body involved. The diagnosis is clinical and there are no specific tests available; therefore, expert opinion should be sought. Dystonias may be primary or secondary

2008 Mentor

189. Kernicterus

aged over 1 year, the classical features develop, which include abnormalities in the extrapyramidal, visual and auditory systems. Minor intellectual deficits may be present. Extrapyramidal signs These may occur and the most common and most severe is athetosis, although chorea can also occur. Upper extremities are more severely affected than the lower ones and bulbar nerves may also be involved. Chronic bilirubin encephalopathy causes damage to the basal ganglia. Visual problems These most commonly (...) appear to be very sensitive to bilirubin, even at relatively low levels. This may present as delayed language and so any baby at risk must have hearing assessed. The presenting feature of kernicterus may be childhood deafness. Cognitive defects These do not feature prominently in kernicterus but athetosis or chorea along with hearing defects may give the false impression of learning disability. Dental enamel Shows some hypoplasia and some may show green staining of teeth. More subtle alterations

2008 Mentor

190. athetosis

athetosis athetosis - General Practice Notebook This site is intended for healthcare professionals General Practice Notebook | Medical search athetosis Athetosis describes repetitive involuntary, slow, sinuous, writhing movements, which are especially severe in the hands. There are also elements of postural disturbance. Athetotic movements are around the axis of the limb, as distinct from choreiform movements, which are at right angles to the axis. As with chorea, the lips, jaw and tongue may

2010 GP Notebook

191. Effects of surgical adductor releases for hip subluxation in cerebral palsy: an AACPDM evidence report

eligible for inclusion. The studies included children (mean age 3 to 15 years, where reported) with cerebral palsy, spastic quadriplegia, spastic paraplegia, spastic diplegia, spastic triplegia, paraplegia, hemiplegia, athetosis, athertosis/spastic, dystonic quadriplegia and mixed quadriplegia. Outcomes assessed in the review There were no specific inclusion criteria relating to the outcomes. The included studies used a range of outcome measures relating to the range of movement of the hip, number

2003 DARE.

192. Selective dorsal rhizotomy: meta-analysis of three randomized controlled trials

was not blind. Specific interventions included in the review SDR. The included studies compared SDR plus physiotherapy (PT) with or without occupational therapy versus PT with or without occupational therapy. PT consisted of programmes of stretching, strengthening, and training in functional movements intended to enhance mobility. Participants included in the review Children with spastic cerebral palsy. Children with dystonia, athetosis, ataxia or severe visual impairment were excluded. The mean age

2002 DARE.

193. Guanidinoacetate methyltransferase deficiency

and manifest as chorea, athetosis and ataxia. Etiology GAMT deficiency is due to mutations in the GAMT gene (19p13.3). Different mutations (missense, nonsense, splice site, insertions, deletions) have been identified, located on various exons of the GAMT gene. The most frequently identified mutation, c.327G>A (p.K109K, splice site exon 2), is present in at least one allele in over 50% of families. c.59G>C is most frequently encountered in Portuguese patients. To date no genotype phenotype correlation has

2004 Orphanet

194. Focal hand dystonia after cervical whiplash injury (PubMed)

Focal hand dystonia after cervical whiplash injury 12486287 2004 11 16 2017 11 14 0022-3050 74 1 2003 Jan Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Focal hand dystonia after cervical whiplash injury. 134 Tamburin S S Zanette G G eng Case Reports Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Adult Athetosis diagnosis etiology Dystonia diagnosis etiology physiopathology Electrodiagnosis Evoked Potentials, Somatosensory Hand

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2003 Journal of neurology, neurosurgery, and psychiatry

195. Supported Speed Treadmill Training Exercise Program (SSTTEP) for Marginally Ambulatory Children With Cerebral Palsy

in Baclofen dosing, or casting procedures to the lower extremities; Passive range of motion of lower extremity joints: < 30º contracture of hip in extension as measured by the Thomas Test; passive dorsiflexion range of motion to -15º with the knee extended, and knee extension range (90º/90º test) to 70º and <-20º knee extension. At least 2 years post- dorsal rhizotomy. Exclusion Criteria: Children with "mixed" types of CP (i.e. athetosis) or other movement disorders (e.g. ataxia); Children receiving

2007 Clinical Trials

196. Outcomes of Orthopaedic Surgery Using Gait Laboratory Versus Observational Gait Analysis in Children With Cerebral Palsy

with their physical function. Patients are candidates for orthopaedic surgery including soft tissue and/or bony procedures involving at least 2 levels, in one or both lower extremities (e.g. knee & ankle). Patients must be able to undergo instrumented gait analysis in a motion laboratory. Exclusion Criteria: Presence of dystonia, athetosis, or mixed tone abnormalities. History of orthopaedic lower extremity procedures within the previous 2 years. Patients who have had previous gait laboratory analysis that has

2007 Clinical Trials

197. Energy Costs of Spasticity in Spinal Cord Injury: A Pilot Investigation

amount (65-75% TDEE) and is the most sensitive to changes in spasticity. Dampening spasticity has been reported to increase weight gain and necessitate reduced caloric intake in a child with spastic quadriplegia. Similarly, athetosis in patients with cerebral palsy increased resting metabolic rate (RMR) as compared to control subjects with no athetotic movements. Although several studies have reported energy requirements for persons with neurodevelopmental disabilities, and even SCI, however, none

2007 Clinical Trials

198. Effectiveness of the Innsbruck Sensorimotor Activator and Regulator in improving saliva control in children with cerebral palsy. (PubMed)

7 years 10 months, range 4 to 13 years) were selected. Measures of drooling and feeding skills were taken at baseline, at the completion of a 6-month control phase, and at two more 6-monthly time points after the ISMAR was fitted. Children varied greatly in both the length of time taken to tolerate wearing the ISMAR and duration for which the appliance was worn. Only six children (four females, two males) completed the full study. Their motor disabilities were athetosis (n=3), spastic

2004 Developmental Medicine and Child Neurology

199. Melbourne Assessment of Unilateral Upper Limb Function: construct validity and correlation with the Pediatric Evaluation of Disability Inventory. (PubMed)

with CP (5 to 14 years of age; nine males, nine females) were assessed using the Melbourne Assessment and the Pediatric Evaluation of Disability Inventory (PEDI). Five children had spastic quadriplegia, eight had spastic diplegia, two had spastic hemiplegia and diplegia, two had athetosis, and one had hypotonic quadriplegia with mobile ventilator dependence. Children's performances were statistically correlated using Spearman's rho to establish the relation between these tools. Very high correlation

2003 Developmental Medicine and Child Neurology

200. Clinical (video) findings and cerebrospinal fluid neurotransmitters in 2 children with severe chronic bilirubin encephalopathy, including a former preterm infant without marked hyperbilirubinemia VIDEO. (PubMed)

structures has been described at autopsy after only moderate hyperbilirubinemia, but classic chronic bilirubin encephalopathy without marked hyperbilirubinemia has been reported only rarely. We report a case of a 7-year-old, former 29-weeks' gestation, gravely ill premature infant with a peak bilirubin level of 13.3 mg/dL in the neonatal period. We compare this case with a 12-year-old, former term infant with a peak bilirubin level of 49.4 mg/dL on day 10 of life. Both children have dystonia, athetosis

2005 Pediatrics

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