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Aphasia

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3821. Veterans Administration cooperative study on aphasia: a comparison of individual and group treatment. (Abstract)

Veterans Administration cooperative study on aphasia: a comparison of individual and group treatment. Five Veterans Administration Medical Centers participated in an investigation designed to compare individual with group treatment for aphasic patients who had suffered a left hemisphere cerebral vascular accident. Patients who met selection criteria were assigned randomly to either traditional, individual, stimulus-response type treatment of specific language deficits or group therapy designed

1981 Journal of speech and hearing research Controlled trial quality: uncertain

3822. Comparison of clinic, home, and deferred language treatment for aphasia. A Veterans Administration Cooperative Study. (Abstract)

Comparison of clinic, home, and deferred language treatment for aphasia. A Veterans Administration Cooperative Study. Aphasic patients who met stringent selection criteria were assigned randomly to three groups: clinic treatment by a speech pathologist for 12 weeks, followed by 12 weeks of no treatment; home treatment by a trained volunteer for 12 weeks, followed by 12 weeks of no treatment; or deferred treatment for 12 weeks, followed by 12 weeks of treatment by a speech pathologist. At 12 (...) weeks after entry, language measures indicated that the clinic-treatment patients made significantly more improvement than did the deferred-treatment patients, and improvement in home-treatment patients did not differ significantly from either clinic- or deferred-treatment patients. At 24 weeks after entry, after deferred-treatment patients had received clinic treatment, there were no significant differences among the groups. These results suggest that clinic treatment for aphasia is efficacious

1986 Archives of neurology Controlled trial quality: uncertain

3823. Comparison of formal language therapy with supportive counseling for aphasia due to acute vascular accident. (Abstract)

Comparison of formal language therapy with supportive counseling for aphasia due to acute vascular accident. A total of 60 right-handed patients with acute aphasia due to left hemispheral stroke were randomly assigned to two modes of therapy for six months, beginning one month after the ictus. Conventional speech therapy provided by professional speech pathologists twice weekly was compared with emotionally supportive counseling therapy, also provided by professional speech pathologists

1987 Archives of neurology Controlled trial quality: uncertain

3824. Aphasia in Clinical Practice Full Text available with Trip Pro

Aphasia in Clinical Practice Aphasia is a central language impairment with word finding and comprehension deficit and paraphasias. The highlights of the essential language tests and the classification based on a scorable assessment are presented. The clinical syndromes of Broca's, global, Wernicke, conduction, anomic and transcortical aphasias are detailed with definition, localization, and prognosis. Modality specific disorders associated with aphasic syndromes are discussed. The management

1983 Canadian Family Physician

3825. Age and type of aphasia in patients with stroke. Full Text available with Trip Pro

Age and type of aphasia in patients with stroke. The age and gender of a series of patients with different types of aphasia were analysed. Regardless of gender, patients with Broca and conduction aphasias were significantly younger than those with Wernicke and global aphasias. Considering the established cerebral localisation of each of those aphasia types, it appears that, with age, stroke in the territory of the middle cerebral artery will tend to either shift posteriorly (producing Wernicke (...) aphasia) or occupy most of the middle cerebral artery territory (producing global aphasia). But in the absence of concurrent verification of the locus of lesion in each of the cases in our sample, a possible alternative hypothesis must be entertained: that there might be age-related changes in the neurophysiological mechanism subserving language, such that some types of aphasia would tend to be more prevalent with age, regardless of lesion location.

1981 Journal of neurology, neurosurgery, and psychiatry

3826. Deep left parietal lobe syndrome: conduction aphasia and other neurobehavioural disorders due to a small subcortical lesion. Full Text available with Trip Pro

Deep left parietal lobe syndrome: conduction aphasia and other neurobehavioural disorders due to a small subcortical lesion. A patient with sudden onset of conduction aphasia in the context of an ischaemic stroke is reported. Other neurological and neuropsychological findings included bilateral ideomotor apraxia, right hemisensory defect and paradoxical left ear extinction on a dichotic listening test. Lesion location, as inferred from magnetic resonance imaging, involved a restricted

1987 Journal of neurology, neurosurgery, and psychiatry

3827. The temporal sequence of aura-sensations in patients with complex focal seizures with particular attention to ictal aphasia. Full Text available with Trip Pro

The temporal sequence of aura-sensations in patients with complex focal seizures with particular attention to ictal aphasia. The sequences of aura sensations in 143 patients with complex partial seizures, were analysed with special emphasis on aphasic symptoms. Anxiety, epigastric sensation and visual hallucination were experienced early in the course of the aura, while illusion of familiarity and aphasia occurred late in the course of the aura. Three groups of interconnections of aura (...) sensations were found which corresponded possibly to the types of seizure constellations proposed by Weiser. Close interconnections between impairment of verbal comprehension during seizures and paroxysmal thought disorder, as well as between paroxysmal paraphasia and illusion of familiarity were noted. Paroxysmal aphasia in patients with complex partial seizures was characterised as a positive symptom in contrast to stable aphasia.

1989 Journal of neurology, neurosurgery, and psychiatry

3828. Progressive aphasia with right-sided extrapyramidal signs: another manifestation of localised cerebral atrophy. Full Text available with Trip Pro

Progressive aphasia with right-sided extrapyramidal signs: another manifestation of localised cerebral atrophy. 2468739 1989 05 26 2018 11 13 0022-3050 52 1 1989 Jan Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Progressive aphasia with right-sided extrapyramidal signs: another manifestation of localised cerebral atrophy. 128-30 Goulding P J PJ Northen B B Snowden J S JS Macdermott N N Neary D D eng Case Reports Letter England J Neurol Neurosurg Psychiatry (...) 2985191R 0022-3050 IM Aphasia pathology Atrophy Basal Ganglia Diseases pathology Cerebral Cortex pathology Dominance, Cerebral physiology Humans Male Middle Aged 1989 1 1 1989 1 1 0 1 1989 1 1 0 0 ppublish 2468739 PMC1032675 J Neurol Neurosurg Psychiatry. 1987 Sep;50(9):1101-9 3499484 Neurology. 1986 Jul;36(7):879-87 3487046 Ann Neurol. 1987 Oct;22(4):527-32 3435071 Arch Neurol. 1988 Apr;45(4):392-6 3258512 J Neurol Neurosurg Psychiatry. 1978 Oct;41(10):903-6 731239 Acta Neurol Scand. 1979 May;59(5

1989 Journal of neurology, neurosurgery, and psychiatry

3829. Recovery from aphasia and neglect after subcortical stroke: neuropsychological and cerebral perfusion study. Full Text available with Trip Pro

Recovery from aphasia and neglect after subcortical stroke: neuropsychological and cerebral perfusion study. Cortical regional cerebral perfusion was assessed by N, N, N1-trimethyl-N1-(2)-hydroxy-3-methyl-5-(I-123) iodobenzyl-1, 3-propanediamine 2 HCl I-123 (HIPDM) and single photon emission computerised tomography (SPECT) in six aphasic and two neglect patients with unilateral subcortical vascular lesions. Assessments were carried out both in the acute phase and after a period ranging from 1 (...) to 6 months after stroke onset. In all patients an almost complete spontaneous recovery occurred and was associated with a significant improvement of cortical perfusion. A relationship between severity of aphasia and degree of cortical hypoperfusion was found, in both the acute and the follow up assessments, in the aphasic subgroup.

1988 Journal of neurology, neurosurgery, and psychiatry

3830. Wernicke aphasia and cardiac embolism. Full Text available with Trip Pro

Wernicke aphasia and cardiac embolism. 3625222 1987 10 22 2017 11 14 0022-3050 50 7 1987 Jul Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Wernicke aphasia and cardiac embolism. 938-9 Harrison M J MJ Marshall J J eng Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Aphasia etiology Aphasia, Wernicke etiology Carotid Artery Thrombosis complications Coronary Disease complications Coronary Thrombosis complications Humans Retrospective Studies

1987 Journal of neurology, neurosurgery, and psychiatry

3831. Spared musical abilities in a conductor with global aphasia and ideomotor apraxia. Full Text available with Trip Pro

Spared musical abilities in a conductor with global aphasia and ideomotor apraxia. A conductor suddenly developed global aphasia and severe ideomotor apraxia as a result of an infarct in the territory of the left middle cerebral artery. Although aphasia and apraxia remained unchanged during the following six years, his musical capacities were largely spared and he was still able to conduct. This case provides some evidence in favour of right hemisphere dominance for music.

1985 Journal of neurology, neurosurgery, and psychiatry

3832. Limb apraxia without aphasia from a left sided lesion in a right handed patient. Full Text available with Trip Pro

Limb apraxia without aphasia from a left sided lesion in a right handed patient. A right handed man had a massive left middle cerebral artery stroke. CT and MRI revealed extensive destruction of both anterior and posterior areas typically associated with language. There was, however, no aphasia, but instead a marked limb apraxia, dyscalculia, dense right visual neglect, and anosognosia. These uncommon dissociations and associations support the hypothesis that cerebral control of motor function

1991 Journal of neurology, neurosurgery, and psychiatry

3833. Depression in acute and chronic aphasia: symptoms, pathoanatomical-clinical correlations and functional implications. Full Text available with Trip Pro

Depression in acute and chronic aphasia: symptoms, pathoanatomical-clinical correlations and functional implications. Depressive alterations were investigated in 21 acute and 21 chronic aphasic patients with single left sided strokes. The assessment of depression was based on a psychometrically evaluated German version of the Cornell Scale for Depression (CDS) and the Research Diagnostic Criteria (RDC). No significant difference was found concerning depression sum-scores between the two aphasic (...) groups. The acute group, however, exhibited significantly higher ratings in items related to physical signs of depression and disturbances of cyclic functions. Patients corresponding to the RDC-syndrome of major depression were only found in the acute group. Neither age, sex nor degree of hemiparesis discriminated the patients on the severity of depressive symptoms. In the acute patient group, nonfluency of aphasia was the only parameter that could be identified which had an effect on the mood

1993 Journal of neurology, neurosurgery, and psychiatry

3834. Familial progressive aphasia: its relationship to other forms of lobar atrophy. Full Text available with Trip Pro

Familial progressive aphasia: its relationship to other forms of lobar atrophy. Two brothers presented with slowly progressive aphasia. One brother, who became behaviourally disturbed only at the end of his illness, was found at necropsy to have predominant left frontotemporal atrophy. The other brother developed severe behavioural disturbances shortly after the onset of language impairment. His brain revealed bilateral frontotemporal atrophy. In both there was non-Alzheimer's disease pathology (...) with the histological features of loss of large cortical nerve cells, spongiform change and mild gliosis. The differential anatomical atrophy supports the view that clinical manifestations of lobar atrophy are dictated by the topographical distribution of a common underlying pathology, linking the syndromes of progressive aphasia to dementia of frontal lobe type (DFT) and DFT with motor neuron disease.

1993 Journal of neurology, neurosurgery, and psychiatry

3835. PET assessment of brain metabolic recovery in aphasia. Full Text available with Trip Pro

PET assessment of brain metabolic recovery in aphasia. 8201362 1994 07 07 2018 11 13 0022-3050 57 5 1994 May Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry PET assessment of brain metabolic recovery in aphasia. 663 Baron J C JC Ford I I eng Letter Comment England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM J Neurol Neurosurg Psychiatry. 1993 Jun;56(6):665-71 8509781 Aphasia diagnostic imaging metabolism Brain metabolism Data Interpretation

1994 Journal of neurology, neurosurgery, and psychiatry

3836. Functional integrity of the structural unaffected left hemisphere in crossed aphasia. Full Text available with Trip Pro

Functional integrity of the structural unaffected left hemisphere in crossed aphasia. 8201363 1994 07 07 2018 11 13 0022-3050 57 5 1994 May Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Functional integrity of the structural unaffected left hemisphere in crossed aphasia. 664 Primavera A A Bandini F F eng Letter Comment England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM J Neurol Neurosurg Psychiatry. 1993 Jun;56(6):665-71 8509781 Aphasia diagnostic

1994 Journal of neurology, neurosurgery, and psychiatry

3837. The Neurobiology of Language Recovery in Aphasia Full Text available with Trip Pro

The Neurobiology of Language Recovery in Aphasia 10716855 2000 04 21 2018 11 13 0093-934X 71 1 2000 Jan Brain and language Brain Lang The neurobiology of language recovery in aphasia. 245-8 Thompson C K CK Department of Communication Sciences and Disorders, Northwestern University, Evanston, IL 60298-3570, USA. ckthom@nwu.edu eng R01 DC001948 DC NIDCD NIH HHS United States R01 DC001948-07 DC NIDCD NIH HHS United States Journal Article Netherlands Brain Lang 7506220 0093-934X IM Aphasia

2000 Brain and language

3838. Mechanisms of recovery from aphasia Full Text available with Trip Pro

Mechanisms of recovery from aphasia 10071090 1999 04 13 2017 11 14 0022-3050 66 2 1999 Feb Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Mechanisms of recovery from aphasia. 136 Baron J C JC eng Comment Editorial England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM J Neurol Neurosurg Psychiatry. 1999 Feb;66(2):155-61 10071093 Aphasia physiopathology Humans 1999 3 10 1999 3 10 0 1 1999 3 10 0 0 ppublish 10071090 PMC1736198

1999 Journal of neurology, neurosurgery, and psychiatry

3839. Unaccusative verb production in agrammatic aphasia: the argument structure complexity hypothesis Full Text available with Trip Pro

Unaccusative verb production in agrammatic aphasia: the argument structure complexity hypothesis This study examined patterns of verb production in narrative samples of eight individuals with agrammatic aphasia and seven education- and age-matched normal subjects. Comprehension and constrained production of two types of intransitive verbs-unaccusatives whose argument structure triggers a complex syntactic derivation and unergatives that are considered syntactically simple- was also tested

2003 Journal of neurolinguistics

3840. Aphasia and Wernicke's arc Full Text available with Trip Pro

Aphasia and Wernicke's arc 11309473 2001 06 21 2017 11 14 0022-3050 70 5 2001 May Journal of neurology, neurosurgery, and psychiatry J. Neurol. Neurosurg. Psychiatry Aphasia and Wernicke's arc. 699 Pearce J M JM eng Historical Article Letter England J Neurol Neurosurg Psychiatry 2985191R 0022-3050 IM Aphasia history History, 17th Century Humans Temporal Lobe 2001 4 20 10 0 2001 6 22 10 1 2001 4 20 10 0 ppublish 11309473 PMC1737333

2001 Journal of neurology, neurosurgery, and psychiatry

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