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3621. Travail d'équipe: omnipraticien, orthophoniste et audiologiste Full Text available with Trip Pro

Travail d'équipe: omnipraticien, orthophoniste et audiologiste The Speech and Hearing Department of the University of Montréal, in conjunction with 'l'Unité de médecine familiale de Verdun' set up a pilot project grouping family doctors, audiologists and speech pathologists. Information was exchanged on speech and language problems in children, stuttering, voice disorders, aphasia and hearing problems in children and adults. We emphasized the importance of early detection of these problems

1981 Canadian Family Physician

3622. MASA syndrome: new clinical features and linkage analysis using DNA probes. Full Text available with Trip Pro

MASA syndrome: new clinical features and linkage analysis using DNA probes. We describe a two generation family in which two males have the X linked recessive MASA syndrome (mental retardation, aphasia, shuffling gait, and adducted thumbs). A third male in this family died at the age of 15 years from congenital hydrocephalus. In the present family cerebral abnormalities are reported for the first time. Linkage analysis confirms the chromosome localisation at Xq28. A crossover between

1990 Journal of Medical Genetics


Transplantation 0132144 0041-1337 0 Anti-Bacterial Agents 0 Carrier Proteins 0 Immunosuppressive Agents EC 5.1.1.- Amino Acid Isomerases EC Peptidylprolyl Isomerase WM0HAQ4WNM Tacrolimus IM Adult Amino Acid Isomerases antagonists & inhibitors Anti-Bacterial Agents adverse effects Aphasia chemically induced Carrier Proteins antagonists & inhibitors Female Humans Immunosuppressive Agents adverse effects Liver Transplantation Male Peptidylprolyl Isomerase Postoperative Complications Tacrolimus 1990 12 1

1990 Transplantation

3624. The efficacy of treatment for aphasic persons: a meta-analysis. (Abstract)

The efficacy of treatment for aphasic persons: a meta-analysis. A meta-analysis of research findings is reported on the effectiveness of treatments for persons suffering aphasia. Twenty-one studies provided sufficient information for inclusion in the analysis. The magnitudes of three classes of effect sizes were estimated: untreated recovery, treated recovery, and treated versus untreated recoveries. The outcomes witness the clear superiority in performance of persons receiving treatment

1995 Brain and language

3625. Quality of life of stroke survivors: a research synthesis. (Abstract)

functional ability, social support, and healthcare resources. Variables negatively associated with stroke survivors' quality of life were psychological impairment, severity of impairment, severity of aphasia, inappropriate reactions to illness and pessimism, and inability to return to work. Specifically, 22% to 73% of the variance in stroke survivors' quality of life was primarily explained by presence of depression; functional ability, especially the upper extremities; and socialization, in particular

2002 The Journal of neuroscience nursing : journal of the American Association of Neuroscience Nurses

3626. Treatment of residual vestibular schwannoma. (Abstract)

, postoperative complications, hospital stay.All but 2 patients underwent a total removal. Complications recorded were 1 temporary hemiparesis and aphasia, 1 subcutaneous abdominal hematoma, and 2 transitory VIth cranial nerve palsies. No additional treatment was required. The postoperative hospital stays ranged from 3 to 22 days, with a mean of 6.9.Patients with vestibular schwannoma should be treated by an experienced team in a referral center to reduce postoperative morbidity and decrease the need

2002 Otology and Neurotology

3627. Survival in frontotemporal dementia. (Abstract)

classified pathologically as tau-positive and tau-negative.Of the 61 patients with FTD, 26 presented with frontal variant (fvFTD), 9 with semantic dementia, 8 with progressive nonfluent aphasia (PNFA), 9 with associated motor neuron disease (FTD-MND), and 9 with corticobasal degeneration features. There was no difference between the groups in age at symptom onset (overall mean 58.5 +/- 7.8 years), but at diagnosis the PNFA (68.3 +/- 2.7) group was significantly older than the fvFTD (59.9 +/- 7.4) and FTD

2003 Neurology

3628. Plasticity of motor cortex excitability induced by rehabilitation therapy for writing. (Abstract)

Plasticity of motor cortex excitability induced by rehabilitation therapy for writing. The mechanisms of rehabilitation-induced plasticity in the motor system after stroke are not defined. The authors studied seven patients with residual poststroke agraphia, aphasia, and right hemiparesis. After a 40-minute rehabilitation therapy that promoted use of the paretic hand for writing, the authors observed a task-specific increase in recruitment of ipsilateral corticospinal pathways. Rehabilitation

2003 Neurology

3629. Language function and dysfunction in corticobasal degeneration. (Abstract)

Language function and dysfunction in corticobasal degeneration. S: The authors assessed language functioning in corticobasal degeneration (CBD), an area that has received little systematic study. Aphasia has been reported occasionally, and the authors hypothesized that appropriate assessments would reveal at least mild language impairment, particularly affecting phonologic (sound-based) processing, even in cases without frank aphasia.A series of 10 unselected patients with CBD (one (...) of patients had deficits in semantic memory, naming, and reading, but the impairments were usually mild.The authors found phonologic impairment to be a typical feature of CBD. There is substantial overlap between progressive nonfluent aphasia and CBD, and the linguistic impairment can be thought of as a continuum, with mild phonologic impairment at one end and severe aphasia at the other.

2003 Neurology

3630. Slowly progressive spread of the stroke-like lesions in MELAS. (Abstract)

in all four stroke-like episodes, followed by hemianopsia, psychosis, and aphasia. In three episodes, epileptic seizure developed subsequently in close association with a progression of stroke-like lesion. In all four episodes, serial MRI showed a slowly progressive spread of the stroke-like lesion evolving from the temporal cortex to the surrounding parietal or occipital cortex over a few weeks following the onset of initial symptoms. Apparent diffusion coefficient (ADC) maps showed slightly

2003 Neurology

3631. Strokes restricted to the insular cortex. (Abstract)

with partial distribution); 2) gustatory disorder in a patient with left posterior insular infarct; 3) vestibular-like syndrome, with dizziness, gait instability, and tendency to fall, but no nystagmus, in three patients with posterior insular strokes; 4) cardiovascular disturbances, consisting of hypertensive episodes in a patient with a right posterior insular infarct; and 5) neuropsychological disorders, including aphasia (left posterior insula), dysarthria, and transient somatoparaphrenia (right (...) posterior insula).Strokes restricted to the posterior insula may present with pseudothalamic sensory and vestibular-like syndromes as prominent clinical manifestations, but also dysarthria and aphasia (in left lesions), somatoparaphrenia (right lesions) and gustatory dysfunction and blood pressure with hypertensive episodes in right lesions; we did not find acute dysphagia reported in anterior, insular strokes.

2002 Neurology

3632. Progressive affective aprosodia and prosoplegia. (Abstract)

. The authors suggest that this patient has a form of frontotemporal dementia, analogous to primary progressive aphasia but disrupting right frontal lobe-mediated functions.

2003 Neurology

3633. Art and the brain: the influence of frontotemporal dementia on an accomplished artist. (Abstract)

Art and the brain: the influence of frontotemporal dementia on an accomplished artist. A talented artist developed a progressive aphasia syndrome associated with frontotemporal dementia (FTD). As her disease progressed, language and executive skills declined, but her paintings became freer and more original. She demonstrates that artistic development can occur in the setting of language-dominant types of FTD. The study of artistic development in the setting of FTD suggests that language

2003 Neurology

3634. A case of gliosarcoma appearing as ischaemic stroke. Full Text available with Trip Pro

A case of gliosarcoma appearing as ischaemic stroke. Ischaemic stroke attributable to malignant brain tumour is a rarely reported phenomenon and even various imaging techniques including angiography do not necessarily lead to an accurate diagnosis.A 46-year-old, previously healthy man developed apoplectic symptoms with slight right sided hemiparesis and global aphasia. The computed tomography (CT) scan showed lesions of the left temporal lobe and the paraventricular white matter suggestive

2003 Neurosurgery and Psychiatry

3635. Magnetic resonance imaging abnormalities in familial temporal lobe epilepsy with auditory auras. Full Text available with Trip Pro

with auditory auras.We performed detailed clinical and molecular studies as well as MRI evaluation (including volumetry) in all available individuals from one family, segregating FTLE from auditory auras.We evaluated 18 of 23 possibly affected individuals, and 13 patients reported auditory auras. In one patient, auditory auras were associated with déjà vu; in one patient, with ictal aphasia; and in 2 patients, with visual misperception. Most patients were not taking medication at the time, although all

2003 Archives of Neurology

3636. Acute infarction limited to the lenticular nucleus: clinical, etiologic, and topographic features. (Abstract)

to the putamen. Two of them had ataxic motor hemisyndrome and 7 had sensorimotor hemisyndrome (with ataxia in 4, left hemineglect in 1, and deep pain in the arm and leg in 1). Four patients had a lesion of putamen and globus pallidus externus. Three of them had motor hemisyndrome (with nonfluent aphasia in 2 and ataxia in 1) and 1 had ataxic sensorimotor hemisyndrome. All infarcts were in the territory of the medial perforating branches of the medial cerebral artery. Presumed cause of stroke was small-artery (...) disease in 5, artery-to-artery embolism in 4, cardioembolism in 3 and undetermined in 1.Acute lenticular infarction induces mainly hemiparesis but no movement disorder. Associated sensory deficits, aphasia, and hemineglect underline clinically the function of the lenticular nucleus in connection with the prefrontal, temporal, and parietal cortices.

2003 Archives of Neurology

3637. Right orbitofrontal tumor with pedophilia symptom and constructional apraxia sign. (Abstract)

and constructional deficits, including agraphia, resolved following tumor resection.For patients with acquired sociopathy and paraphilia, an orbitofrontal localization requires consideration. This case further illustrates that constructional apraxia can arise from right prefrontal lobe dysfunction. Agraphia may represent a manifestation of constructional apraxia in the absence of aphasia and ideomotor apraxia.

2003 Archives of Neurology

3638. Frontal lobe atrophy due to a mutation in the cholesterol binding protein HE1/NPC2. (Abstract)

, neurolysosomal storage with oligolamellar inclusion and tau-positive neurofibrillary tangles. Of the 119 family members screened, only a married 42-year-old sister displayed symptoms of a dementia. Both women displayed vertical supranuclear ophthalmoplegia; expressive aphasia; concrete, stimulus-bound, perseverative behavior; and impaired conceptualization and planning. Cultured fibroblasts showed decreased cholesterol esterification and positive filipin staining, but no mutation was detected in coding

2002 Annals of Neurology

3639. LGI1 is mutated in familial temporal lobe epilepsy characterized by aphasic seizures. (Abstract)

mutation is associated with autosomal dominant lateral temporal lobe epilepsy in a large Norwegian family showing unusual clinical features like short-lasting sensory aphasia and auditory symptoms.

2002 Annals of Neurology

3640. Myelinoclastic diffuse sclerosis (Schilder's disease): report of a case and review of the literature. (Abstract)

-handed young man presenting with left hemiplegia, aphasia and behavioural changes. Magnetic resonance imaging showed two large lesions, irregular contrast-enhancing, in the subcortical white matter of the right frontal and parietal lobes, and increased intracranial pressure. Definitive diagnosis was made with biopsy. The characteristic pathological features are demyelinization of the white matter, lymphocytic perivascular infiltrates, microglial proliferation and immunohistochemical signs

2002 British Journal of Neurosurgery

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