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Acute Vision Loss

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161. Sinusitis, Acute (Treatment)

Sinusitis, Acute (Treatment) Acute Sinusitis Treatment & Management: Approach Considerations, Symptomatic Treatment, Antimicrobial Therapy Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMjMyNjcwLXRyZWF0bWVudA (...) == processing > Acute Sinusitis Treatment & Management Updated: Mar 01, 2018 Author: Itzhak Brook, MD, MSc; Chief Editor: Michael Stuart Bronze, MD Share Email Print Feedback Close Sections Sections Acute Sinusitis Treatment Approach Considerations In June 2013, the American Academy of Pediatrics published updated guidelines on the diagnosis and management of acute bacterial sinusitis in children and adolescents. Changes include the following: Previous diagnostic criteria for acute bacterial sinusitis

2014 eMedicine.com

162. Glaucoma, Angle Closure, Acute (Overview)

Angle-Closure Glaucoma (AACG) Updated: Sep 20, 2018 Author: Albert P Lin, MD; Chief Editor: Inci Irak Dersu, MD, MPH Share Email Print Feedback Close Sections Sections Acute Angle-Closure Glaucoma (AACG) Overview Background Angle closure is defined as the apposition of iris to the trabecular meshwork, which results in increased intraocular pressure (IOP). In acute angle closure (AAC), the process occurs suddenly with a dramatic onset of symptoms, including blurred vision, red eye, pain, headache (...) , and nausea and vomiting. The sudden and severe IOP elevation can quickly damage the optic nerve, resulting in acute angle-closure glaucoma (AACG). AAC is a true ophthalmic emergency, and a delay in treatment can result in blindness. While immediate treatment can sometimes minimize the amount of visual loss, the best treatment is to stop its occurrence in susceptible individuals. [ , , , ] Next: Pathophysiology AAC occurs through a process termed pupillary block. Normally, aqueous humor is produced

2014 eMedicine.com

163. Acute Retinal Necrosis (Overview)

accounts for 5.5% of cases over a 10-year period. [ ] International In Switzerland, acute retinal necrosis accounts for 1.7% of uveitic cases. Mortality/Morbidity Significant visual loss may occur. Retinal detachment is a frequent complication (~50%) [ , , ] and is a cause of legal blindness in some bilateral cases of acute retinal necrosis. Race No clear racial predilection exists. Sex This condition appears to have a predilection for males; however, the extent is not clear. Age Acute retinal necrosis (...) Acute Retinal Necrosis (Overview) Acute Retinal Necrosis: Background, Pathophysiology, Epidemiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTIyMzA0Ny1vdmVydmlldw== processing > Acute Retinal Necrosis

2014 eMedicine.com

164. Acute Retinal Necrosis (Diagnosis)

accounts for 5.5% of cases over a 10-year period. [ ] International In Switzerland, acute retinal necrosis accounts for 1.7% of uveitic cases. Mortality/Morbidity Significant visual loss may occur. Retinal detachment is a frequent complication (~50%) [ , , ] and is a cause of legal blindness in some bilateral cases of acute retinal necrosis. Race No clear racial predilection exists. Sex This condition appears to have a predilection for males; however, the extent is not clear. Age Acute retinal necrosis (...) Acute Retinal Necrosis (Diagnosis) Acute Retinal Necrosis: Background, Pathophysiology, Epidemiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvMTIyMzA0Ny1vdmVydmlldw== processing > Acute Retinal Necrosis

2014 eMedicine.com

165. Glaucoma, Acute Angle-Closure (Follow-up)

medications after LPI. [ ] They also have higher rates of vision loss and subsequent increases in IOP. [ ] It has been hypothesized that the initial attack is often more severe in Asians resulting in greater trabecular damage. Another possibility is the formation of peripheral synechiae (adhesions) causing a creeping angle reclosure. Previous Next: Patient Education For patient education resources, see the , as well as , , , and . Previous References Berkoff DJ, Sanchez LD. An uncommon presentation (...) field loss from primary angle-closure glaucoma: a comparative study of symptomatic and asymptomatic disease. Ophthalmology . 2004 Sep. 111(9):1636-40. . Aung T, Friedman DS, Chew PT, et al. Long-term outcomes in asians after acute primary angle closure. Ophthalmology . 2004 Aug. 111(8):1464-9. . Aung T, Oen FT, Wong HT, et al. Randomised controlled trial comparing the effect of brimonidine and timolol on visual field loss after acute primary angle closure. Br J Ophthalmol . 2004 Jan. 88(1):88-94

2014 eMedicine Emergency Medicine

166. Acute Orbital Compartment Syndrome (Overview)

: Prognosis Acute orbital compartment syndrome with visual acuity loss is associated with a poor prognosis. Permanent blindness occurs if effective therapy is not initiated in a timely manner. Emergent decompressive surgery (ie, lateral canthotomy and inferior cantholysis) may save sight in patients with severe symptoms. However, irreversible visual loss can be expected with retinal ischemia that lasts longer than 120 minutes. Previous References Lima V, Burt B, Leibovitch I, Prabhakaran V, Goldberg RA (...) Acute Orbital Compartment Syndrome (Overview) Acute Orbital Compartment Syndrome: Background, Anatomy, Pathophysiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNzk5NTI4LW92ZXJ2aWV3 processing > Acute Orbital

2014 eMedicine Emergency Medicine

167. Acute Orbital Compartment Syndrome (Follow-up)

=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNzk5NTI4LXRyZWF0bWVudA== processing > Acute Orbital Compartment Syndrome Treatment & Management Updated: Nov 04, 2015 Author: David A Peak, MD; Chief Editor: Robert E O'Connor, MD, MPH Share Email Print Feedback Close Sections Sections Acute Orbital Compartment Syndrome Treatment Approach Considerations The emergency procedure of choice for acute loss of visual acuity associated with acute orbital compartment syndrome is dissection of the lateral canthus and disinsertion of at least the inferior crus of the lateral (...) : Further Inpatient Care Cosmetic repair of lateral canthotomy may be performed in the hospital and usually affords a good outcome. Repair of canthal tendons can be performed at the discretion of the specialist. Repair can be delayed for several days if necessary. Compartment syndrome is one of the accepted indications for hyperbaric oxygen therapy. Case reports of improvement in vision using hyperbaric oxygen as adjunct therapy in acute orbital compartment syndrome exist. Previous Next: Consultations

2014 eMedicine Emergency Medicine

168. Sinusitis, Ethmoid, Acute, Surgical Treatment

orbital abscess, an orbital abscess, superior orbital fissure syndrome, or cavernous sinus thrombosis. This last condition can result in limited ocular motility, proptosis, and loss of vision and can be life threatening. A Taiwanese study, by Chang et al, found that among patients in the report, orbital complications of paranasal sinusitis most commonly arose from ipsilateral maxillary (82%) and ethmoid (76%) sinuses. [ ] A study by Coudert et al reported that in children with acute ethmoiditis (...) Sinusitis, Ethmoid, Acute, Surgical Treatment Surgical Treatment of Acute Ethmoid Sinusitis: Historical Overview, Anatomic Considerations, Sinusitis Complications Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache

2014 eMedicine Surgery

169. Acute Orbital Compartment Syndrome (Treatment)

=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNzk5NTI4LXRyZWF0bWVudA== processing > Acute Orbital Compartment Syndrome Treatment & Management Updated: Nov 04, 2015 Author: David A Peak, MD; Chief Editor: Robert E O'Connor, MD, MPH Share Email Print Feedback Close Sections Sections Acute Orbital Compartment Syndrome Treatment Approach Considerations The emergency procedure of choice for acute loss of visual acuity associated with acute orbital compartment syndrome is dissection of the lateral canthus and disinsertion of at least the inferior crus of the lateral (...) : Further Inpatient Care Cosmetic repair of lateral canthotomy may be performed in the hospital and usually affords a good outcome. Repair of canthal tendons can be performed at the discretion of the specialist. Repair can be delayed for several days if necessary. Compartment syndrome is one of the accepted indications for hyperbaric oxygen therapy. Case reports of improvement in vision using hyperbaric oxygen as adjunct therapy in acute orbital compartment syndrome exist. Previous Next: Consultations

2014 eMedicine Emergency Medicine

170. Acute Orbital Compartment Syndrome (Diagnosis)

: Prognosis Acute orbital compartment syndrome with visual acuity loss is associated with a poor prognosis. Permanent blindness occurs if effective therapy is not initiated in a timely manner. Emergent decompressive surgery (ie, lateral canthotomy and inferior cantholysis) may save sight in patients with severe symptoms. However, irreversible visual loss can be expected with retinal ischemia that lasts longer than 120 minutes. Previous References Lima V, Burt B, Leibovitch I, Prabhakaran V, Goldberg RA (...) Acute Orbital Compartment Syndrome (Diagnosis) Acute Orbital Compartment Syndrome: Background, Anatomy, Pathophysiology Edition: No Results No Results Please confirm that you would like to log out of Medscape. If you log out, you will be required to enter your username and password the next time you visit. https://profreg.medscape.com/px/getpracticeprofile.do?method=getProfessionalProfile&urlCache=aHR0cHM6Ly9lbWVkaWNpbmUubWVkc2NhcGUuY29tL2FydGljbGUvNzk5NTI4LW92ZXJ2aWV3 processing > Acute

2014 eMedicine Emergency Medicine

171. Glaucoma, Acute Angle-Closure (Diagnosis)

, acute angle closure, and acute angle-closure glaucoma were previously used interchangeable. Now, acute angle closure is defined as at least 2 of the following symptoms: ocular pain, nausea/vomiting, and a history of intermittent blurring of vision with halos; and at least 3 of the following signs: IOP greater than 21 mm Hg, conjunctival injection, corneal epithelial edema, mid-dilated nonreactive pupil, and shallower chamber in the presence of occlusion. Primary angle closure is defined (...) of glaucoma in prevalence surveys. Br J Ophthalmol . 2002 Feb. 86(2):238-42. . . Ang LP, Aung T, Chua WH, Yip LW, Chew PT. Visual field loss from primary angle-closure glaucoma: a comparative study of symptomatic and asymptomatic disease. Ophthalmology . 2004 Sep. 111(9):1636-40. . Aung T, Friedman DS, Chew PT, et al. Long-term outcomes in asians after acute primary angle closure. Ophthalmology . 2004 Aug. 111(8):1464-9. . Aung T, Oen FT, Wong HT, et al. Randomised controlled trial comparing the effect

2014 eMedicine Emergency Medicine

172. Glaucoma, Acute Angle-Closure (Overview)

, acute angle closure, and acute angle-closure glaucoma were previously used interchangeable. Now, acute angle closure is defined as at least 2 of the following symptoms: ocular pain, nausea/vomiting, and a history of intermittent blurring of vision with halos; and at least 3 of the following signs: IOP greater than 21 mm Hg, conjunctival injection, corneal epithelial edema, mid-dilated nonreactive pupil, and shallower chamber in the presence of occlusion. Primary angle closure is defined (...) of glaucoma in prevalence surveys. Br J Ophthalmol . 2002 Feb. 86(2):238-42. . . Ang LP, Aung T, Chua WH, Yip LW, Chew PT. Visual field loss from primary angle-closure glaucoma: a comparative study of symptomatic and asymptomatic disease. Ophthalmology . 2004 Sep. 111(9):1636-40. . Aung T, Friedman DS, Chew PT, et al. Long-term outcomes in asians after acute primary angle closure. Ophthalmology . 2004 Aug. 111(8):1464-9. . Aung T, Oen FT, Wong HT, et al. Randomised controlled trial comparing the effect

2014 eMedicine Emergency Medicine

173. Bilateral Visual Loss: More Than Meets The Eye. (PubMed)

Bilateral Visual Loss: More Than Meets The Eye. A 72-year-old woman presented with acute onset bilateral visual loss. She had no other symptoms or signs, but had a complex past medical history including blood transfusions and immunosuppression. T2-weighted magnetic resonance imaging demonstrated bilateral occipital lobe vasogenic edema, consistent with posterior reversible encephalopathy syndrome (PRES). Her vision improved with conservative management.Copyright © 2013 Elsevier Inc. All rights

2012 Survey of Ophthalmology

174. Ganglion Cell Loss in Relation to Visual Disability in Multiple Sclerosis. (PubMed)

Ganglion Cell Loss in Relation to Visual Disability in Multiple Sclerosis. We used high-resolution spectral-domain optical coherence tomography (SD-OCT) with retinal segmentation to determine how ganglion cell loss relates to history of acute optic neuritis (ON), retinal nerve fiber layer (RNFL) thinning, visual function, and vision-related quality of life (QOL) in multiple sclerosis (MS).Cross-sectional study.A convenience sample of patients with MS (n = 122; 239 eyes) and disease-free (...) was significant even accounting for macular RNFL thickness (P = 0.03 for GCL+IPL, P = 0.39 for macular RNFL).We demonstrated that GCL+IPL thinning is most significantly correlated with both visual function and vision-specific QOL in MS, and may serve as a useful structural marker of disease. Our findings parallel those of magnetic resonance imaging studies that show gray matter disease is a marker of neurologic disability in MS.Proprietary or commercial disclosure may be found after the references.Copyright ©

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2012 Ophthalmology

175. Causes of Visual Loss in Retinal Disease

): Simon Taylor, Royal Surrey County Hospital Study Details Study Description Go to Brief Summary: This is a study of visual outcomes in retinal disease that seeks to identify the causes of visual loss. This data will be used to predict which patients are at risk of losing vision and how they can be better treated. Condition or disease Intervention/treatment Uveitis Scleritis Drug: Immunosuppressive Agents Detailed Description: This is a study of visual outcomes in retinal disease (...) Causes of Visual Loss in Retinal Disease Causes of Visual Loss in Retinal Disease - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Causes of Visual Loss in Retinal Disease The safety and scientific validity

2012 Clinical Trials

176. Blurred Vision

Add to Any Platform Loading , MD, Wilmer Eye Institute, Retina Division, Johns Hopkins University School of Medicine Click here for Patient Education NOTE: This is the Professional Version. CONSUMERS: Topic Resources Blurred vision is the most common visual symptom. It usually refers to decreased visual clarity of gradual onset, and corresponds to decreased visual acuity. For sudden, complete loss of vision in one or both eyes (blindness), see . Patients with small visual field defects (eg, caused (...) Suggestive Findings Diagnostic Approach Opacification of eye structures Gradual onset, often risk factors (eg, aging, corticosteroid use), loss of contrast, glare Lens opacification on ophthalmoscopy or slit-lamp examination Clinical evaluation Corneal opacification (eg, posttraumatic or postinfectious scarring) Corneal abnormalities on slit-lamp examination Clinical evaluation Disorders affecting the retina Gradual onset, central vision affected (central scotoma) without loss of peripheral vision

2013 Merck Manual (19th Edition)

177. Early factors associated with axonal loss after optic neuritis. (PubMed)

months later were investigated for their association with later axonal loss.After multivariate analysis, prolonged visual evoked potential latency and impaired color vision, at baseline and after 3 months, were significantly and independently associated with RNFL thinning. Low-contrast acuity measures exhibited significant univariate associations with RNFL thinning.The association of RNFL loss with a prolonged visual evoked potential (VEP) latency suggests that acute and persistent demyelination (...) Early factors associated with axonal loss after optic neuritis. Acute optic neuritis due to an inflammatory demyelinating lesion of the optic nerve is often seen in association with multiple sclerosis. Although functional recovery usually follows the acute episode of visual loss, persistent visual deficits are common and are probably due to axonal loss. The mechanisms of axonal loss and early features that predict it are not well defined. We investigated clinical, electrophysiological

2011 Annals of Neurology

178. Assessment of Homonymous Visual Loss and Its Impact on Visual Exploration, Activities of Daily Living (ADL) and Quality of Life (QoL)

Assessment of Homonymous Visual Loss and Its Impact on Visual Exploration, Activities of Daily Living (ADL) and Quality of Life (QoL) Assessment of Homonymous Visual Loss and Its Impact on Visual Exploration, Activities of Daily Living (ADL) and Quality of Life (QoL) - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved Studies Save this study Warning You (...) have reached the maximum number of saved studies (100). Please remove one or more studies before adding more. Assessment of Homonymous Visual Loss and Its Impact on Visual Exploration, Activities of Daily Living (ADL) and Quality of Life (QoL) (Hemi-Drive) The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our for details. ClinicalTrials.gov Identifier

2011 Clinical Trials

179. Assessment of Advanced Glaucomatous Visual Field Loss and Its Impact on Visual Exploration, Activities of Daily Living (ADL) and Quality of Life (QoL)

, suspicion of glaucoma, ocular hypertension or any other sign of other optic neuropathy, macular degeneration, pathological color vision test results (Ishihara Standard Pseudoisochromatic Plates), eye surgery (except cataract surgery), any type of refractive surgery; history or signs of neuro-ophthalmological diseases, acute infections, diabetic retinopathy, use of miotic drugs. Contacts and Locations Go to Information from the National Library of Medicine To learn more about this study, you or your (...) Assessment of Advanced Glaucomatous Visual Field Loss and Its Impact on Visual Exploration, Activities of Daily Living (ADL) and Quality of Life (QoL) Assessment of Advanced Glaucomatous Visual Field Loss and Its Impact on Visual Exploration, Activities of Daily Living (ADL) and Quality of Life (QoL) - Full Text View - ClinicalTrials.gov Hide glossary Glossary Study record managers: refer to the if submitting registration or results information. Search for terms x × Study Record Detail Saved

2011 Clinical Trials

180. Management of Acute Neurotrauma in Rural and Remote Locations (3rd ed.)

in children and an isolated recording on the chart may be misleading. 2. It is often difficult to decide whether or not there was loss of consciousness at the time of the impact. Concussion may be very brief and not appreciated by observers. 3. Persistent headache and vomiting in a child should be considered as indicating raised ICP until proven otherwise. 4. Blunt trauma to a child’s head may be followed within a short period by the development of acute brain swelling. This may follow what appeared (...) Management of Acute Neurotrauma in Rural and Remote Locations (3rd ed.) The Management of Acute Neurotrauma in Rural and Remote Locations A set of guidelines for the care of head and spinal injuries Neurosurgical Society of Australasia Inc FORWARD These guidelines were first published in 1992 and revised in 2000 and 2009. They are intended to assist medical practitioners responsible for the initial management of patients with neurotrauma in places remote from specialised services. The outcome

2009 ASERNIP-S

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